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1 donor for patient 1, which subsequently grew M. hominis.
2 d to 24 (29.6%) of the 81 women negative for M. hominis.
3 a suggest donor-to-recipient transmission of M. hominis.
4 mutator element in the vaa gene that governs M. hominis adherence and highlight the importance of mut
5 T. vaginalis in symbiosis with M. hominis or M. hominis alone triggers a noncanonical type I interfer
6        Increasing levels of G. vaginalis and M. hominis and decreasing levels of lactobacilli were si
7 ergence of Vaa could affect the adherence of M. hominis and evasion of antibody-mediated immunity, th
8 he results of these studies demonstrate that M. hominis and M. hominis antigen are potent stimulators
9 acteria will not be reliable for recovery of M. hominis and that specialized media and incubation con
10 ase demonstrates the pathogenic potential of M. hominis and the need for rapid recognition of the org
11 hree log(10) bacterial counts (G. vaginalis, M. hominis, and lactobacilli) in our model improved the
12 hese studies demonstrate that M. hominis and M. hominis antigen are potent stimulators of type II epi
13 erial vaginosis was strongly associated with M. hominis (aOR = 8.01, 95%CI:5.99-10.71), but was not a
14 tative bacterial counts for lactobacilli and M. hominis are better correlates of CVL HIV RNA than are
15 m the joint synovial fluid of a patient with M. hominis-associated arthritis, which indicated that Va
16 terial counts (P=.006; inverse association), M. hominis bacterial counts (P=.0001; positive associati
17 ate analyses, we found that G. vaginalis and M. hominis bacterial counts, Candida vaginitis, and herp
18              Clinicians should be aware that M. hominis can cause surgical site infections, and may n
19  had log(10) G. vaginalis counts and log(10) M. hominis counts greater than 6.81 and 4.82, respective
20             Here we describe the recovery of M. hominis from a brain abscess associated with a postpa
21       PCR quantification of G. vaginalis and M. hominis from CVL is significantly more sensitive than
22 e to challenge with LPS, U. urealyticum, and M. hominis in a concentration-dependent fashion.
23             No BacT/ALERT bottles containing M. hominis in simulated blood cultures were flagged posi
24 e data strongly suggest that the presence of M. hominis in the lungs of premature infants may initiat
25                Surfactant suppressed LPS and M. hominis induced TNF-alpha production in a dose-depend
26 ng chronic, active arthritis associated with M. hominis infection and is highly immunogenic in the hu
27 cated that Vaa phase variation occurs during M. hominis infection in the natural host.
28 ransplant recipients presented with invasive M. hominis infections at multiple sites characterized by
29             In a single center, a cluster of M. hominis infections were identified in lung transplant
30 eloped surgical site infections, including 2 M. hominis infections.
31 ICU were found to have genetically unrelated M. hominis isolates, excluding patient-to-patient transm
32                                          The M. hominis-like mycoplasma neither inhibits nor enhances
33                                 Evidence for M. hominis neutrophil extracellular trap escape is also
34               Only 7 women were positive for M. hominis; none were allele 2 homozygotes as opposed to
35 nfection with T. vaginalis in symbiosis with M. hominis or M. hominis alone triggers a noncanonical t
36                                Mechanisms of M. hominis pathogenicity are still largely obscure, and
37                 We investigated a cluster of M. hominis surgical site infections in patients who unde
38 ich correlated precisely with the ability of M. hominis to adhere to cultured human cells.
39  findings do not support routine testing for M. hominis, U. urealyticum, and U. parvum in nonpregnant
40                           We investigated if M. hominis, U. urealyticum, and U. parvum were associate
41 lvovaginal candidiasis (VVC), and tested for M. hominis, U. urealyticum, and U. parvum, and 4 nonvira
42 immunity and provide additional knowledge on M. hominis virulence and survival in the host.
43 n requirements and relatively slow growth of M. hominis warrant that dependence on automated systems
44            After adjusting for STIs and VVC, M. hominis was associated with abnormal vaginal discharg
45                      In stratified analyses, M. hominis was associated with self-reported vaginal mal
46                                         Only M. hominis was associated with symptoms/signs, and these
47 s, Ureaplasma spp. was detected in 23 (82%), M. hominis was detected in 3 (11%), and both were detect
48            Viability of clinical isolates of M. hominis was maintained for 7 days in BacT/ALERT media
49                                 Importantly, M. hominis was not associated with symptoms/signs in wom