ライフサイエンスコーパス: MALT lymphoma

1 ciated with salivary gland MALT lymphoma (SG-MALT-lymphoma).

2 phoepithelial lesions, a hallmark of gastric MALT lymphoma.

3 may eventually develop into low-grade B-cell MALT lymphoma.

4 olution of H. pylori-associated gastritis to MALT lymphoma.

5 is associated with HP dependence of gastric MALT lymphoma.

6 atients with an increased risk of developing MALT lymphoma.

7 tion signature to that of the salivary gland MALT lymphoma.

8 varying size in four patients with low-grade MALT lymphoma.

9 patients initially diagnosed elsewhere with MALT lymphoma.

10 ic marginal zone in a single case of gastric MALT lymphoma.

11 recurrent t(11;18)(q21;q21) translocation in MALT lymphoma.

12 pe 4 (CXCR4) has been previously observed in MALT lymphoma.

13 bacter pylori-positive gastritis and gastric MALT lymphoma.

14 of-function mechanism in the pathogenesis of MALT lymphoma.

15 iological, and molecular genetic features of MALT lymphoma.

16 ular mechanisms of primary SS and primary SS/MALT lymphoma.

17 , both proteins are translocation targets in MALT lymphoma.

18 lize and optimize treatment of patients with MALT lymphoma.

19 clinical management of patients with gastric MALT lymphoma.

20 have been implicated in the pathogenesis of MALT lymphoma.

21 on potentially bridges LELs to genesis of SG-MALT-lymphoma.

22 gastritis and other low-grade gastric B-cell MALT lymphomas.

23 cosal B-cell traffic are also operational in MALT lymphomas.

24 come in the management of gastric, low-grade MALT lymphomas.

25 clones of concurrent gastric and intestinal MALT lymphomas.

26 nt role in the clonal expansion of low-grade MALT lymphomas.

27 the time being in patients with other non-GI MALT lymphomas.

28 otic therapy in nongastrointestinal (non-GI) MALT lymphomas.

29 alternative to radiotherapy for conjunctival MALT lymphomas.

30 not as favorable as the treatment results of MALT lymphomas.

31 a state-of-the-art summary of ocular adnexal MALT lymphomas.

32 c signaling and implicate its dysfunction in MALT lymphomas.

33 on to the IGH locus can promote formation of MALT lymphomas.

34 ll these low-grade lesions are classified as MALT lymphomas.

35 pathogenetic pathways in the development of MALT lymphomas.

36 ased on the distinction between MALT and non-MALT lymphomas.

37 cation of mucosa-associated lymphoid tissue (MALT) lymphoma.

38 2;q32) of mucosa-associated lymphoid tissue (MALT) lymphoma.

39 cation in mucosa-associated lymphoid tissue (MALT) lymphoma.

40 cation in mucosa-associated lymphoid tissue (MALT) lymphoma.

41 lmarks of mucosa-associated lymphoid tissue (MALT) lymphoma.

42 gastric mucosae-associated lymphoid tissue (MALT) lymphoma.

43 s in some mucosa-associated lymphoid tissue (MALT) lymphomas.

44 ations in mucosa-associated lymphoid tissue (MALT) lymphomas.

45 Studies on the use of doxycycline for MALT lymphomas (137 patients) reported complete response

46 igin) and mucosa-associated lymphoid tissue (MALT) lymphoma (19 of 45 = 42%) (postgerminal center), b

47 ar lymphoma (3 cases) and mucosa-associated (MALT) lymphoma (3 cases) strong (2+) pRB staining was li

48 20 (6.7%) mucosa-associated lymphoid tissue (MALT) lymphomas, 4 of 42 (9.5%) follicular lymphomas, an

49 ll clone, whereas 15 of 25 t(11;18)-negative MALT lymphomas (60%) showed trisomy of chromosomes 18 (n

50 can occur, and in studies reporting only on MALT lymphomas (884 patients), the 5-year and 10-year di

51 11 of 12 polymerase chain reaction-positive MALT lymphomas (92%).

52 tiple histologic subtypes of lymphoma or non-MALT lymphomas (988 patients) reported local control rat

53 have high-grade MALT lymphoma with low-grade MALT lymphoma abutting the tumor.

54 sing test for assessment of residual gastric MALT lymphomas after H pylori eradication.

55 Thirty-seven MALT lymphomas (all previously evaluated for t(11;18) by

56 is work establishes a molecular link between MALT lymphoma and ABC-DLBCL, and provides mouse models t

57 niques have aided in the distinction between MALT lymphoma and other lymphoproliferative disorders an

58 howed the same sized dominant product in the MALT lymphoma and the follicular lymphoma.

59 (q21;q21) translocation occurs frequently in MALT lymphomas and creates a chimeric NF-kappaB-activati

60 t(11;18)(q21,q21) is found in 30% of gastric MALT lymphomas and is associated with a failure to respo

61 ximab for mucosa-associated lymphoid tissue (MALT) lymphoma and steroids for prolonged cough.

62 why GPR34 genetic changes associate with SG-MALT-lymphoma and how these mutations contribute to the

63 ge cell lymphoma (LCL) arising subsequent to MALT lymphoma, and 16 controls were tested by FISH using

64 ts with primary SS, patients with primary SS/MALT lymphoma, and subjects without primary SS (non-prim

65 cosal venules are similar in normal MALT and MALT lymphomas, and factors controlling normal mucosal B

66 mphomas have a more indolent course than non-MALT lymphomas, and in the conjunctiva a more conservati

67 ly in aggressive and antibiotic unresponsive MALT lymphomas, and may further implicate the biologic i

68 on to systemic disease seems high, even with MALT lymphomas, and treatment with radiation is still re

69 disease, mucosa-associated lymphoid tissue (MALT) lymphoma, and gastric adenocarcinoma.

70 Apart from gastric MALT lymphoma, antibiotic therapies have been adequately

71 MALT lymphomas are characterized genetically by the t(11

72 show that concurrent gastric and intestinal MALT lymphomas are derived from the same clone and sugge

73 depth of infiltration of the wall by gastric MALT lymphoma as measured by endoscopic ultrasound has b

74 disease, mucosa-associated lymphoid tissue (MALT) lymphoma, as well as hyperplastic polyps, hyperpla

75 rin-mediated cell adhesion, while primary SS/MALT lymphoma-associated modules were enriched with gene

76 ixafor-PET for detection of residual gastric MALT lymphoma at follow-up were 97.0%, 95.0%, 100.0%, 10

77 Homing receptor expression in MALT lymphoma B cells was heterogeneous, however, in lin

78 ithin chromosome 18 DNA in three examples of MALT lymphoma bearing this translocation.

79 he LCL and its clonally identical antecedent MALT lymphoma both showed t(11;18).

80 x cases of concurrent gastric and intestinal MALT lymphomas by polymerase chain reaction (PCR) amplif

81 ) and without ("pure" DLBCL) the features of MALT lymphomas, can achieve long-term complete remission

82 cell development, these findings suggest the MALT lymphoma cell of origin may be a germinal center B

83 he LCL had trisomy 12, and a small subset of MALT lymphoma cells had trisomy 3 and/or 12.

84 d from colonization of lymphoid follicles by MALT lymphoma cells, following which the tumor cells wer

85 zone B cells are the normal counterparts of MALT lymphoma cells.

86 r gastric mucosa-associated lymphoid tissue (MALT) lymphoma cells preferentially to localize around r

87 For low-grade MALT lymphomas confined to the gastric wall and without

88 As many as 50% of low-grade MALT lymphomas contain an (11;18)(q21; q21) chromosomal

89 f gastric mucosa-associated lymphoid tissue (MALT) lymphoma currently relies on esophagogastroduodeno

90 nvolvement of the marginal zone when gastric MALT lymphomas disseminate to the spleen, which is in ke

91 rminal caspase recruitment domain (CARD), in MALT lymphomas due to the recurrent t(1;14)(p22;q32).

92 e management of patients with ocular adnexal MALT lymphoma, especially monoclonal antibody therapy an

93 nd 8 hub genes for distinguishing primary SS/MALT lymphoma from primary SS.

94 f gastric mucosa-associated lymphoid tissue (MALT) lymphoma (GML).

95 In the MALT lymphoma group, time-matched gastric biopsies were

96 chronic gastritis tissue, those with MALT or MALT lymphoma had an increase in PCNA labeling index of

97 mitantly, the BCL10 protein is stabilized in MALT lymphomas harboring this fusion.

98 Importantly, primary MALT lymphomas harbouring the API2-MALT1 fusion uniquely

99 The pathogenesis of gastric MALT lymphoma has been elucidated to a large degree in r

100 gy of the mucosa-associated lymphoid tissue (MALT) lymphomas, has been implicated in inflammatory pro

101 MALT lymphomas have a more indolent course than non-MALT

102 In gastric MALT lymphoma Helicobacter pylori is the most common sti

103 . pylori-associated follicular gastritis and MALT lymphomas high endothelial venules coexpressed muco

104 ents with mucosa-associated lymphoid tissue (MALT) lymphomas (HR = 0.26, 95%CI: 0.11-0.59, p = 0.001)

105 study pathogenesis and treatment of gastric MALT lymphoma in humans.

106 a higher than expected incidence of gastric MALT lymphoma in immunosuppressed transplant recipients

107 iption of mucosa-associated lymphoid tissue (MALT) lymphoma in 1983 rapid advances have been made in

108 low-grade mucosa-associated lymphoid tissue (MALT) lymphoma in the parotid gland.

109 have been associated with the development of MALT lymphoma including t(11;18) and alterations in Bcl-

110 A subset of H. pylori-positive gastric MALT lymphomas, including infiltrative tumors, may respo

111 1;q21) in mucosa-associated lymphoid tissue (MALT) lymphoma induces proteolytic cleavage of NF-kappaB

112 The MALT-lymphoma International Prognostic Index (MALT-IPI)

113 Gastric MALT lymphoma is a model malignancy for examination of h

114 When low-grade MALT lymphoma is suspected on the basis of barium study

115 be involved in the growth of salivary gland MALT lymphomas is further suggested by the noted restric

116 f gastric mucosa-associated lymphoid tissue (MALT) lymphoma is dependent on Helicobacter pylori infec

117 Gastric mucosa-associated lymphoid tissue (MALT) lymphoma is indolent and often associated with Hel

118 Mucosa-associated lymphoid tissue (MALT) lymphoma is the most common extranodal lymphoid ce

119 Mucosa-associated lymphoid tissue (MALT) lymphoma is the most common extranodal lymphoid ne

120 enesis of mucosa-associated lymphoid tissue (MALT) lymphomas is associated with independent chromosom

121 g gastric mucosa-associated lymphoid tissue (MALT) lymphoma linked with Helicobacter pylori infection

122 For MALT lymphomas, local control, disease-free survival, an

123 API2-MALT1-positive MALT lymphomas manifest antibiotic resistance and aggres

124 Mucosa-associated lymphoid tissue (MALT) lymphomas most frequently involve the gastrointest

125 h gastric mucosa-associated lymphoid tissue (MALT) lymphoma, much less is known about the value of an

126 Among low-grade gastric MALT lymphoma, mutations were found in 3 of 11 tumors th

127 chronic gastritis (n = 7), MALT (n = 12), or MALT lymphoma (n = 12) were undertaken.

128 pulmonary mucosa-associated lymphoid tissue (MALT) lymphoma (n = 33) and compared the results to GEP

129 The majority of data were available for MALT lymphoma of the ocular adnexa (OAML) including a to

130 Mucosa-associated lymphoid tissue (MALT) lymphoma of the stomach (MLS) has recently been de

131 lymphoma (FL), marginal zone lymphoma (MZL), MALT lymphoma or B-small lymphocytic lymphoma (B-SLL) ce

132 s in early MESA-associated lesions represent MALT lymphomas or more benign types of expansions has be

133 have furthered the understanding of gastric MALT lymphoma pathogenesis, clinical behavior, and treat

134 , accessible, and effective tool to identify MALT lymphoma patients at risk of poor outcomes.

135 nce imaging (MRI) examinations of 26 gastric MALT lymphoma patients, and 20 [68Ga]Pentixafor-PET/MRI

136 obtained by merging 3 independent cohorts of MALT lymphoma patients.

137 r adnexal mucosa-associated lymphoid tissue (MALT) lymphoma (POAML) is the most common orbital tumor,

138 with antibiotics can be followed by gastric MALT lymphoma regression in most cases.

139 oma occurred 6 months after excision and the MALT lymphoma remained indolent during the course of her

140 ier work in establishing that salivary gland MALT lymphomas represent a highly selected B-cell popula

141 s for the treatment of patients with gastric MALT lymphoma requiring further treatment beyond H pylor

142 ts with primary SS and those with primary SS/MALT lymphoma revealed pathways and molecular targets as

143 specifically associated with salivary gland MALT lymphoma (SG-MALT-lymphoma).

144 anaplastic large cell (Ki-1+) lymphoma, 0/2 MALT lymphoma) showed hypermethylation of the promoter.

145 High-grade MALT lymphoma tended to show a slightly higher mutation

146 Early-stage, low-grade gastric MALT lymphoma that was associated with Helicobacter pylo

147 Frequent use of V1-69 appears to differ from MALT lymphomas that develop at other sites.

148 the transformation of H. pylori gastritis to MALT lymphoma, the extent of cell proliferation, cell vi

149 g the translocation t(1;18)(q21;q21) forms a MALT lymphoma, the growth of which is independent of H.

150 s efficacy on high-grade transformed gastric MALT lymphomas, the DLBCL(MALT).

151 AI was 3.4- and 1.4-fold higher in MALT and MALT lymphoma tissue, respectively, in the same comparis

152 he AI and Cdc2/Cdk1 or cyclin B1 in MALT and MALT lymphoma tissues.

153 ed tumor onset and induced transformation of MALT lymphoma to activated B-cell diffuse large-cell lym

154 his truly represented preferential homing of MALT lymphoma to the splenic marginal zone, we have now

155 Another MALT lymphoma translocation, t(11;18)(q21;q21), fuses th

156 h gastric mucosa-associated lymphoid tissue (MALT) lymphoma, treatment-related toxicity should be min

157 The results of treatment of non-MALT lymphomas using radiotherapy also were good, but th

158 On the other hand, the AI index of MALT lymphoma was 2.5-fold lower than that for MALT tiss

159 ntibacterial therapy in patients with non-GI MALT lymphomas was performed.

160 orambucil Plus Rituximab Versus Rituximab in MALT Lymphoma) was launched to compare chlorambucil alon

161 medical records of six patients with gastric MALT lymphoma were retrospectively reviewed.

162 our liver transplant recipients with gastric MALT lymphoma were reviewed.

163 modules related to primary SS and primary SS/MALT lymphoma were significantly enriched with genes kno

164 (VH and VL) expressed by five salivary gland MALT lymphomas were cloned and sequenced.

165 een adequately tested only in ocular adnexal MALT lymphomas where upfront doxycycline may be a reason

166 ecutive patients with stage I to IIE gastric MALT lymphoma who obtained a pathologic remission after

167 t endoscopy demonstrated early-stage gastric MALT lymphoma with associated Helicobacter pylori gastri

168 latter patient was found to have high-grade MALT lymphoma with low-grade MALT lymphoma abutting the

169 Only one other MALT lymphoma with t(11;18) showed aneuploidy (trisomy 3

170 The local control rate of MALT lymphomas with treatments involving radiotherapy av

171 ry showed mucosa-associated lymphoid tissue (MALT) lymphoma with immunoglobulin kappa monotype.

172 elapsed Helicobacter pylori-negative gastric MALT lymphoma, with stage I-IV disease.