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1                                              MAPC cells also promoted Treg activation by inducing CD4
2                                              MAPC suppression required licensing and proceeded via ID
3                                              MAPC-SMCs entrapped in fibrin vascular molds became circ
4                                              MAPC-treated kidneys demonstrated improved urine output
5                                              MAPC-treated kidneys demonstrated improved urine output
6                                              MAPCs also induce a shift in macrophages from an M1, or
7                                              MAPCs are believed to be derived from the bone marrow st
8                                              MAPCs can be distinguished from MSCs on the basis of cel
9                                              MAPCs limited donor T-cell proliferation and GVHD-induce
10                                              MAPCs may therefore be an ideal cell for in vivo therapi
11                                              MAPCs significantly decrease MMP-9 (matrix metalloprotei
12                                              MAPCs suppressed T cell proliferation and Th1 and Th17 c
13                                              MAPCs therefore may constitute a source of cells for tre
14                                              MAPCs, which can be expanded in vitro and maintained in
15                                     C57BL /6 MAPCs were infused intravenously into C57BL /6, Rag-2(-/
16  have implications for the use of allogeneic MAPCs and possibly other immunomodulatory nonhematopoiet
17                                        Among MAPC-derived GFP+CD45.2+ cells in the bone marrow of eng
18 oduced in mice by iliac artery ligation, and MAPCs were administered intramuscularly on day 1.
19 iated recipients given allogeneic marrow and MAPCs; such MAPCs expressed MHC class I antigens in tiss
20                                     MSCs and MAPCs can be successfully labeled with (18)F-FDG for mol
21 e the successful deployment of both MSCs and MAPCs for the amelioration of human autoimmunity and all
22 ditionally, mice were injected with MSCs and MAPCs prelabeled with (18)F-FDG, and stem cell biodistri
23       Therefore, direct labeling of MSCs and MAPCs with (18)F-FDG is a suitable technique to noninvas
24                                           As MAPCs proliferate extensively without obvious senescence
25                                      Because MAPCs can be expanded in culture without obvious senesce
26 IDO) resulted in decreased Treg induction by MAPC cells demonstrating IDO involvement.
27 es play a critical role in Treg induction by MAPC cells.
28 ential factor for Treg induction secreted by MAPC cells.
29       Although hematopoietic contribution by MAPCs was comparable to control KTLS HSCs, approximately
30 ely to account for the generation of HSCs by MAPCs.
31 (Flk1(+)) multipotent adult progenitor cell (MAPC) that copurifies with mesenchymal stem cells from p
32          Multipotent adult progenitor cells (MAPC cells), an adult adherent bone-marrow derived strom
33          Multipotent Adult Progenitor Cells (MAPC((R)) ) possess potent immunomodulatory properties t
34          Multipotent adult progenitor cells (MAPCs) are bone marrow-derived stem cells that have exte
35          Multipotent adult progenitor cells (MAPCs) are marrow-derived pluripotent stem cells with a
36          Multipotent adult progenitor cells (MAPCs) are nonhematopoietic stem cells capable of giving
37 und that multipotent adult progenitor cells (MAPCs) can affect both macrophages and dystrophic neuron
38 at human multipotent adult progenitor cells (MAPCs) have the ability to modulate macrophage activatio
39 SCs) and multipotent adult progenitor cells (MAPCs) in vitro with (18)F-FDG and to investigate the po
40 ort that multipotent adult progenitor cells (MAPCs) isolated from rat, murine, porcine, and human bon
41  with multipotential adult progenitor cells (MAPCs) promotes recovery of blood flow through the recru
42 imitive, multipotent adult progenitor cells (MAPCs) that can differentiate into most mesodermal cells
43 lations, multipotent adult progenitor cells (MAPCs), and bone marrow and adipose tissue-derived mesen
44 e termed multipotent adult progenitor cells (MAPCs), can be expanded for >120 population doublings.
45 ate that multipotent adult progenitor cells (MAPCs), isolated from green fluorescent protein (GFP)-tr
46 SCs) and multipotent adult progenitor cells (MAPCs), represent attractive immunomodulatory cell thera
47 t MAPCs would colocalize with donor T cells, MAPCs were injected directly into the spleen at bone mar
48                               We demonstrate MAPCs, as well as MSCs, are able to egress from the micr
49                          Our study describes MAPC cell dependent Treg phenotypic changes and provides
50 responses and illustrate the requirement for MAPC colocalization to sites of initial donor T-cell act
51 ruitment when stimulated with perfusate from MAPC-treated kidneys (P < .001).
52               Furthermore, SMCs derived from MAPCs (MAPC-SMCs) demonstrated functional L-type calcium
53                                 Furthermore, MAPCs favored the proliferation of regulatory T cells du
54 ietic progenitor subsets and functional GFP+ MAPC-derived lymphocytes that were functional.
55 mmune regulatory potential of clinical-grade MAPCs are limited.
56 modulatory characteristics of clinical-grade MAPCs shown in this study suggest that they can be regar
57 tion to assess the ability of clinical-grade MAPCs to control T cell responses that drive immunopatho
58  transplantation into a non-irradiated host, MAPCs engraft and differentiate to the haematopoietic li
59 itive monocytes were significantly higher in MAPC-treated mice than in the control groups at days 3 a
60 10-fold-greater infiltration of monocytes in MAPC-treated than control-treated ischemic limbs at all
61 stribution of flow to microvascular units in MAPC-treated mice.
62 r expansion of microvascular blood volume in MAPC-treated mice than in controls.
63 rily through prostaglandin E(2) synthesis in MAPCs, which resulted in decreased proinflammatory cytok
64                                  Intravenous MAPCs altered the immune response in the spinal cord and
65                            In limb ischemia, MAPCs stimulate the recruitment of proangiogenic monocyt
66 ion and persistence of reporter gene-labeled MAPCs are maximized after intra-arterial delivery or hos
67 To study in vivo biodistribution, we labeled MAPCs with luciferase for sequential quantification of b
68                                     Mouse (m)MAPCs differentiate into mesenchymal lineage cells as we
69 In addition to these effects on macrophages, MAPCs promote sensory neurite outgrowth, induce sproutin
70        Furthermore, SMCs derived from MAPCs (MAPC-SMCs) demonstrated functional L-type calcium channe
71                            In C57BL /6 mice, MAPCs were transiently detected only in the chest compar
72 plants into lethally irradiated C57BL6 mice, MAPCs are of donor origin.
73 rol KTLS HSCs, approximately 10(3)-fold more MAPCs were required for efficient engraftment.
74                                    Moreover, MAPC cell-induced Tregs (miTregs) have a more suppressiv
75                                    Moreover, MAPC-SMCs may lend themselves to tissue engineering appl
76                                    Moreover, MAPCs altered the balance away from positive and toward
77 ver biodistribution studies revealed that no MAPCs were found in the cord and instead preferentially
78  We investigated the potential capability of MAPC cells in kidney NMP.
79  immunosuppressive capacity and mechanism of MAPC-induced suppression of T-cell alloresponses and ill
80 f SCI, we found that intravenous delivery of MAPCs one day, but not immediately, after SCI significan
81                                The origin of MAPCs within bone marrow is unknown.
82 d here multipotent adult progenitor cells or MAPCs--differentiate, at the single cell level, not only
83       Immunofluorescence revealed prelabeled MAPC cells in the perivascular space of kidneys during N
84 all-animal PET experiments with radiolabeled MAPCs and MSCs injected intravenously in mice showed a p
85                           Mouse MSCs and rat MAPCs were used for (18)F-FDG uptake kinetics and tracer
86                        Human, mouse, and rat MAPCs, cultured on Matrigel with FGF-4 and HGF, differen
87   Kidneys were randomly allocated to receive MAPC treatment or control.
88                         NK depletion reduced MAPC elimination.
89 we demonstrate that, in an in vitro setting, MAPC cells increase Treg frequencies by promoting Treg p
90 en injected into an early blastocyst, single MAPCs contribute to most, if not all, somatic cell types
91 ents given allogeneic marrow and MAPCs; such MAPCs expressed MHC class I antigens in tissues.
92                                     As such, MAPCs hold promise for tissue injury repair after transp
93                     When given systemically, MAPCs did not home to sites of allopriming and did not s
94          These results provide evidence that MAPC-SMCs are phenotypically and functionally similar to
95                 Our results demonstrate that MAPCs exert their primary effects in the periphery and p
96                 Our results demonstrate that MAPCs have therapeutic benefits after spinal cord injury
97 a-treated cremaster muscle demonstrated that MAPCs migrate to perimicrovascular locations and potenti
98                               To ensure that MAPCs would colocalize with donor T cells, MAPCs were in
99                           Here, we show that MAPCs can also differentiate into hepatocyte-like cells
100     These responses are sustained beyond the MAPC lifespan, suggesting that paracrine effects promote
101 macrophage activation, and prior exposure to MAPC secreted factors can reduce macrophage-mediated axo
102 monocytes was 10-fold greater in response to MAPC-conditioned than basal media.
103    Optical imaging of luciferase-transfected MAPCs indicated that cells survived for 1 week.
104 y factors that affect adoptively transferred MAPCs.
105 milar to neonatal SMCs and that the in vitro MAPC-SMC differentiation system may be an ideal model fo
106                                    In vitro, MAPCs cultured with VEGF differentiate into CD34(+), VE-
107                                    In vitro, MAPCs expressed low levels of major histocompatibility c
108                                    In vitro, MAPCs potently suppressed allogeneic T-cell activation a
109  B cells, and NK cells contribute to in vivo MAPC rejection.
110                                     In vivo, MAPCs can differentiate in response to local cues into e
111 the gastrointestinal tract is increased when MAPCs are transplanted in a minimally irradiated host.
112      One of the proposed mechanisms by which MAPC cells modulate immune responses is via the inductio
113 es evidence of potential mechanisms by which MAPC cells promote Treg differentiation.
114                                   Widespread MAPC biodistribution and long-term persistence were seen
115                         Kidneys treated with MAPC cells demonstrate improvement in clinically relevan

 
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