ライフサイエンスコーパス: MCD

1 MCD apparently evolved toward preventing the nonspecific

2 MCD behavior is tunable through net-charge: increasing n

3 MCD catalyzes the unprecedented oxidation of an alpha-me

4 MCD feeding triggered steatosis, hepatic lipid storage,

5 MCD induction further potentiated the defects in insulin

6 MCD saturation magnetization data for valence-delocalize

7 MCD should be subdivided into HHV-8-associated MCD and H

8 MCD was assumed when IMR>/=29.1 (75(th) percentile).

9 MCD-fed ATGL-KO mice, although partially protected from

10 MCD/C5s manifest prominent extranodal dissemination and

11 MCDs can synergize with a speckle-associated RNA recogni

12 MCDs place a substantial burden on affected individuals,

13 Following 2 (MCD groups) or 3 (HFD groups) weeks of treatment with G4

14 (n = 9) samples from 32 patients with HHV-8 MCD and compared them with patients with KS (n = 24) and

15 re markedly decreased in patients with HHV-8 MCD and were undetectable in 6 of them.

16 oreover, iNKT cells from patients with HHV-8 MCD displayed a proliferative defect after stimulation w

17 Compared with other ACDs, MCD contains an approximately 170-residue-long N-termina

18 The mainstay of treatment for adult MCD, oral glucocorticoids, is based on two randomized co

19 nificant across subgroups (children, adults, MCD/MesGN, and FSGS; P<0.01).

20 Consequently, KLHL14 inactivation allows MCD cells to maintain NF-kappaB signaling in the presenc

21 s infusions of FGF21 for 4 weeks while on an MCD diet had reduced steatosis and peroxidative damage,

22 Castleman's disease (MCD), in addition to an MCD-related disorder involving systemic elevation of pro

23 e recapitulated in aged mice treated with an MCD inhibitor (CBM-3001106), and these mice also demonst

24 methods, including UV-vis-NIR absorption and MCD spectroscopies, single-crystal X-ray structure deter

25 solute bioavailabilities obtained by DCH and MCD nasal administration were 6% and 15%, respectively.

26 trin microparticles loaded with DFO (DCH and MCD, respectively) were obtained by spray drying.

27 THF)5][Fe8Me12], which combined with EPR and MCD studies is shown to be consistent with Kochi's S = 1

28 between eruptive cherry hemangiomatosis and MCD.

29 ith concomitant HHV8-associated lymphoma and MCD at relapse.

30 We fed mice custom MCS and MCD formulas containing 4 different carbohydrate-fat com

31 on of diffuse atherosclerotic narrowings and MCD.

32 ges in disposition were caused by ob/ob- and MCD diet-specific decreases in the kidney mRNA expressio

33 that recapitulates features seen in PEL and MCD by gene expression and cell phenotype analysis, allo

34 EPR, resonance Raman, and MCD spectroscopies have been used to determine the redox

35 e reference plane for MRW and MRA, BMO area, MCD, mean ALCSD, PLTT, RNFLT and visual field parameters

36 D should be subdivided into HHV-8-associated MCD and HHV-8-negative MCD or iMCD.

37 HHV-8-associated MCD may be considered as a single clinicopathological en

38 MCD), human herpes virus-8 (HHV8)-associated MCD (HHV8-MCD), and polyneuropathy, organomegaly, endocr

39 ll disorder, skin changes (POEMS)-associated MCD (POEMS-MCD).

40 in synthetic and natural speckle-associated MCDs abolishes these activities, identifying a key role

41 or laboratory findings that were present at MCD diagnosis predicted subsequent relapse, and the medi

42 guration were used to calculate: (i) average MCD in each cortical lobe, (ii) number of overlapping co

43 restricted disruption of lipid rafts by beta-MCD jettisoned the D(1) R from the brush border, decreas

44 rate attenuated hepatic inflammation in both MCD-fed and LPS-treated ATGL-KO mice.

45 In contrast, disruption of caveolae by MCD treatment or Cav-3 knockdown abolished the protectio

46 ent 2Cu2 in solution was now investigated by MCD and EPR spectroscopy.

47 dress this deficit, we combined magnetic CD (MCD) spectroscopy and protein film electrochemistry (PFE

48 Circular dichroism (CD) and magnetic CD (MCD) studies demonstrate that chloride binding triggers

49 Using circular dichroism (CD), magnetic CD (MCD), and variable-temperature, variable-field (VTVH) MC

50 n (Abs)/circular dichroism (CD)/magnetic CD (MCD)/variable temperature, variable field (VTVH) MCD spe

51 des unicentric CD (UCD) and multicentric CD (MCD), the latter of which is divided into idiopathic MCD

52 From CD, MCD, and VTVH MCD, p-anisidine addition is found to mini

53 Abs/CD/MCD/VTVH MCD data exhibit 12 transitions that are assign

54 een characterized using UV-vis absorption/CD/MCD, EPR, Mossbauer, and resonance Raman spectroscopies.

55 A comprehensive MCD spectral analysis coupled with a molecular field mod

56 of a subsequent lymph node biopsy confirmed MCD.

57 Mice fed the custom MCD formulas developed varying degrees of hepatic steato

58 veolae disruptors (methyl-beta-cyclodextrin (MCD) and Nystatin) and Ang II stimulation variably incre

59 lesterol with 5 mm methyl-beta-cyclodextrin (MCD) caused a substantial increase in the rate of agonis

60 In comparison with DCH, MCD enhanced the in vitro DFO permeation across lipophil

61 ice deficient for malonyl CoA decarboxylase (MCD(-/-)), a mouse model of reduced fat oxidation, were

62 Malonyl-CoA decarboxylase (MCD) has been a target of investigation because it reduc

63 ates and inhibits malonyl CoA decarboxylase (MCD), an enzyme that produces acetyl CoA from malonyl Co

64 e of MRW (+2.64 degrees , p<.001), decreased MCD (-11.6mum, p=.007), and decreased mean ALCSD (-18.91

65 aluated in the methionine-choline deficient (MCD) and streptozotocin-western diet nonalcoholic fatty

66 e by feeding a methionine-choline deficient (MCD) diet up to 8 weeks.

67 re we used the methionine-choline deficient (MCD) model of NASH to characterize the possible involvem

68 rticularly the methionine-choline deficient (MCD) model, profound changes are seen in redox enzymes a

69 llenged with a methionine-choline-deficient (MCD) diet as a nutritional model of NASH or lipopolysacc

70 chow or a methionine and choline-deficient (MCD) diet for 1 week were divided into 4 groups: control

71 ) or fed a methionine and choline-deficient (MCD) diet to induce experimental NASH and underwent MR i

72 ced by CCl4 or methionine-choline-deficient (MCD) diet, liver injury and fibrosis were attenuated in

73 feeding mice a methionine-choline-deficient (MCD) diet, the degree of liver damage is related to diet

74 using the methionine and choline-deficient (MCD) diet, which depletes methyl groups and results in a

75 ed by the methionine- and choline-deficient (MCD) diet, which was reasoned to be due to increased hep

76 (ALD) and methionine and choline-deficient (MCD) diet-induced liver injury.

77 diet or a methionine- and choline-deficient (MCD) diet.

78 ere fed with a methionine-choline-deficient (MCD) diet.

79 e and choline [methionine-choline-deficient (MCD) diet] is a well-established model of nonalcoholic s

80 placed on methionine- and choline-deficient (MCD), high-fat, or control diets for 8-16 weeks.

81 We also measured mast cell degranulation (MCD)72 h post-injury.

82 (2S)-methylsuccinyl-CoA dehydrogenase (MCD) belongs to the family of FAD-dependent acyl-CoA deh

83 values of the magnitude of current density (MCD) obtained from the configuration were used to calcul

84 Myocardial microvascular density (MCD) in aFGF-NP+UTMD group was up to 35n/hpf, much highe

85 membrane opening (BMO) area, mean cup depth (MCD), anterior lamina cribrosa surface depth (ALCSD), pr

86 with malformations in cortical development (MCD) or spinal muscular atrophy with lower extremity pre

87 se of malformations of cortical development (MCD), typically lissencephaly, pachygyria and polymicrog

88 Malformations of cortical development (MCDs) are neurodevelopmental disorders that result from

89 Malformations of cortical development (MCDs) compose a diverse range of disorders that are comm

90 ) are malformations of cortical development (MCDs) that are highly associated with medication-resista

91 In the magnetic circular dichroism (MCD) measurements, we observed giant Zeeman splitting wi

92 We show that magnetic circular dichroism (MCD) of sun's ultraviolet C light by oxygen in Archaean

93 absorption and magnetic circular dichroism (MCD) spectra show weak ligand-field transitions between

94 e Mossbauer and magnetic circular dichroism (MCD) spectroscopies of well-defined and in situ formed m

95 absorption, and magnetic circular dichroism (MCD) spectroscopies show that CuZ degrees is a 1-hole (i

96 absorption and magnetic circular dichroism (MCD) spectroscopies.

97 Magnetic circular dichroism (MCD) spectroscopy and computational studies provide insi

98 crystals using magnetic circular dichroism (MCD) spectroscopy and SQUID magnetometry.

99 ble-temperature magnetic circular dichroism (MCD) spectroscopy to experimentally evaluate this excite

100 s on the use of magnetic circular dichroism (MCD) spectroscopy to validate the results of TD-DFT calc

101 absorption, and magnetic circular dichroism (MCD) spectroscopy, coupled with DFT and highly correlate

102 estigated using Magnetic Circular Dichroism (MCD) spectroscopy.

103 nance (EPR) and magnetic circular dichroism (MCD) yields two intermediates corresponding to neutral t

104 dichroism (CD), magnetic circular dichroism (MCD), and variable temperature variable field (VTVH) MCD

105 dichroism (CD), magnetic circular dichroism (MCD), and variable-temperature, variable-field (VTVH) MC

106 s ((119)Sn-NMR, magnetic circular dichroism (MCD), electron paramagnetic resonance (EPR), SQUID, UV-v

107 divided into 4 groups: control (chow diet), MCD diet, chow diet plus G49, and M+G49 (MCD diet plus G

108 fed male Sprague-Dawley rats a control diet, MCD diet, or betaine-supplemented MCD (MCD+B) diet for 8

109 ding sign of multicentric Castleman disease (MCD) and other lymphoproliferative diseases.

110 ociated with multicentric Castleman disease (MCD) and primary effusion lymphoma (PEL).

111 )-associated multicentric Castleman disease (MCD) is a lymphoproliferative inflammatory disorder comm

112 )-associated multicentric Castleman disease (MCD) is a polyclonal B-cell lymphoproliferative disorder

113 oma (KS) and multicentric Castleman disease (MCD), a life-threatening, virally induced B-cell lymphop

114 ma (PEL) and multicentric Castleman disease (MCD).

115 a (PEL), and multicentric Castleman disease (MCD).

116 a subset of multicentric Castleman disease (MCD).

117 esis of KSHV multicentric Castleman disease (MCD).

118 Minimal change disease (MCD) is the etiology of 10%-25% of cases of nephrotic sy

119 e the frequencies of minimal change disease (MCD), focal segmental glomerulosclerosis (FSGS), LN and

120 ephrotic syndrome of minimal change disease (MCD), mesangial proliferative GN (MesGN), or FSGS may be

121 uria are features of minimal-change disease (MCD).

122 cells from multicentric Castleman's disease (MCD) and Kaposi's sarcoma (KS) lesions, suggesting that

123 Multicentric Castleman's disease (MCD) describes a heterogeneous group of disorders involv

124 coma (KS), multicentric Castleman's disease (MCD), and primary effusion lymphoma (PEL).

125 oma (PEL), multicentric Castleman's disease (MCD), and the inflammation-driven neoplasm Kaposi's sarc

126 (PEL), and multicentric Castleman's disease (MCD), in addition to an MCD-related disorder involving s

127 a, PEL and multicentric Castleman's disease (MCD), in addition to proinflammatory activities observed

128 (PEL), and multicentric Castleman's disease (MCD).

129 (PEL), and multicentric Castleman's disease (MCD).

130 (PEL) and multicentric Castleman's disease (MCD).

131 phoma, and multicentric Castleman's disease (MCD).

132 the treatment of motor conversion disorder (MCD).

133 etella lacking the mature C-terminal domain (MCD), suggesting the direct interaction between AC domai

134 trinsically disordered mixed-charge domains (MCDs) in nuclear speckle condensation.

135 arrowings, and microcirculatory dysfunction (MCD) contribute to limit myocardial flow.

136 ations and thereby assignments of low-energy MCD bands associated with the Fe-Fe interaction.

137 ignals, we unambiguously assign a low-energy MCD feature of [Mn(IV)(O)(N4py)](2+) as the (4)E excited

138 Importantly, mice fed MCD starch-palmitate accumulated as much hepatic palmita

139 ulated as much hepatic palmitate as mice fed MCD sucrose-oleate, yet their degree of liver injury was

140 By contrast, mice fed MCD sucrose-palmitate developed severe liver injury, wor

141 tty acid oxidation were suppressed following MCD induction.

142 pose herein a generalized stepwise model for MCD/C5 and PENLs pathogenesis, whereby acquisition of fo

143 e expressing a muscle specific transgene for MCD (Tg-fMCD(Skel)) stabilized posttranslationally by th

144 The diagnostic pathway for MCDs is complex owing to wide variations in presentation

145 solated endothelial cells and monocytes from MCD-fed L1-10-treated mice showed reduced expression of

146 t), MCD diet, chow diet plus G49, and M+G49 (MCD diet plus G49).

147 roviral therapy and use of rituximab in HHV8-MCD have improved outcomes in HHV8-MCD.

148 b in HHV8-MCD have improved outcomes in HHV8-MCD.

149 n herpes virus-8 (HHV8)-associated MCD (HHV8-MCD), and polyneuropathy, organomegaly, endocrinopathy,

150 rdinates, and (iii) cortical areas with high MCD.

151 sociated multicentric Castleman disease (HIV+MCD) with rituximab-based approaches has dramatically im

152 cohort of 84 patients with biopsy-proven HIV+MCD were treated with risk-stratified rituximab-based th

153 Four patients (5%) died of refractory HIV+MCD and 80 achieved clinical remission.

154 isome has been investigated by docking human MCD onto the peroxisomal import protein peroxin 5, which

155 on based on mutational landscapes identified MCD/C5 tumors as specific ABC-DLBCLs with unfavorable cl

156 which we propose referring to as idiopathic MCD (iMCD).

157 e latter of which is divided into idiopathic MCD (iMCD), human herpes virus-8 (HHV8)-associated MCD (

158 The increased severity of NASH in immunized MCD-fed mice involved liver recruitment and the T helper

159 al features of the most common and important MCDs.

160 In MCD mice, we observed a significant decrease in HP DHA t

161 In MCD, infected B cells, although polyclonal, express a mo

162 In MCD-fed WT mice, hepatic AnxA1 increased in parallel wit

163 ating the features seen in infected cells in MCD.

164 ic expression of proinflammatory cytokine in MCD-fed mice.

165 e degree of hepatic fibrosis was enhanced in MCD-fed AnxA1 KO mice, an effect associated with augment

166 educed hepatocyte ballooning and fibrosis in MCD diet-fed mice and was associated with reduced hepati

167 ro functional results in cells maintained in MCD medium.

168 es on TBL1XR1, a gene recurrently mutated in MCD/C5s and PENLs, suggest that aberrant memory B cells

169 on to proinflammatory activities observed in MCD-like "Kaposi's sarcoma-associated herpesvirus-induce

170 Decreased fat oxidation in MCD(-/-) mice resulted in the accumulation of lipid inte

171 treatment improves the hepatic phenotype in MCD- or LPS-challenged ATGL-knockout (KO) mice.

172 between hepatic gene expression profiles in MCD diet-fed wild-type and untreated Lrh-1(-/-) mice sug

173 CRF was significantly reduced in MCD and did not return to normal values even after PK.

174 of My-T-BCR-dependent NF-kappaB signaling in MCD DLBCL and suggest that the genetic status of KLHL14

175 Inducing MCD activity >5-fold in skeletal muscle over two weeks d

176 In this article, the international MCD network Neuro-MIG provides consensus recommendations

177 thus, contribute to the pathogenesis of KSHV MCD.

178 KSHV-MCD activity is associated with (18)F-FDG PET abnormalit

179 KSHV-MCD activity was associated with hypermetabolic symmetri

180 KSHV-MCD is characterized by systemic inflammation caused, in

181 None developed KSHV-MCD; 6 died with median survival from KICS diagnosis 13.

182 ociated multicentric Castleman disease (KSHV-MCD) is a lymphoproliferative disorder, most commonly se

183 ociated multicentric Castleman disease (KSHV-MCD) is characterized by severe inflammatory symptoms ca

184 (PEL), and a form of Castleman disease (KSHV-MCD).

185 ) and hIL-6, and other cytokines during KSHV-MCD flare and remission in 21 patients with 34 flares an

186 cytokine syndrome (KICS) distinct from KSHV-MCD was reported.

187 ough rituximab has reported activity in KSHV-MCD, its use is often associated with KS progression.

188 immunofluorescence staining of involved KSHV-MCD lymph nodes reveals that most plasmablasts expressin

189 Using NCI KSHV-MCD response criteria, major clinical and biochemical re

190 plications for the development of novel KSHV-MCD therapies targeting IL-6 and its downstream signalin

191 is is a key step in the pathogenesis of KSHV-MCD and other KSHV-induced diseases.

192 n important step in the pathogenesis of KSHV-MCD and PEL.

193 ons for the diagnosis and monitoring of KSHV-MCD and shed light on its pathobiologic mechanism.

194 thus, contribute to the pathogenesis of KSHV-MCD and the activity of zidovudine and valganciclovir in

195 We show that a number of KSHV-MCD lymph node plasmablasts express vIL-6 but do not hav

196 ducing plasmablasts from lymph nodes of KSHV-MCD patients coexpress XBP-1s.

197 o toxic moieties, enabling treatment of KSHV-MCD with these drugs.

198 Within a natural history study of KSHV-MCD, we prospectively evaluated rituximab 375 mg/m(2) co

199 ed plasmablasts is a central feature of KSHV-MCD.

200 and valganciclovir in the treatment of KSHV-MCD.

201 R-Dox is effective in symptomatic KSHV-MCD and may be useful in patients with concurrent KS.

202 lasts from lymph nodes of patients with KSHV-MCD express vIL-6 but not ORF45, a KSHV lytic gene.

203 ion in the lymph nodes of patients with KSHV-MCD is predominantly found in cells with XBP-1.

204 aphy (PET) findings in 27 patients with KSHV-MCD.

205 temperature magnetic circular dichroism (LT MCD) spectroscopy in combination with quantum-chemical c

206 ology and diagnostic approaches for the main MCD subtypes and collected data on current practices and

207 diet, MCD diet, or betaine-supplemented MCD (MCD+B) diet for 8 wk and collected blood and tissue.

208 0.68 mug/mL (DCH) to 14.37 +/- 1.69 mug/mL (MCD) 30 min after insufflation of microparticles.

209 Moreover, MCD were able to promote the DFO permeation across monol

210 .77 +/- 0.06 mum (DCH) to 3.47 +/- 0.05 mum (MCD); the aerodynamic diameters were about 1.1 mum and t

211 The in vivo role of muscle MCD expression in the development of insulin resistance

212 specially in the MYD88(L265P), CD79B mutant (MCD) genetic subtype of diffuse large B cell lymphoma (D

213 into HHV-8-associated MCD and HHV-8-negative MCD or iMCD.

214 ma concentrations were 4.8-fold higher in ob/MCD mice compared with WT/control.

215 ct1 mRNA expression was only decreased in ob/MCD mice.

216 respectively, through direct calculations of MCD spectra and independent determination of the MCD C-t

217 sequencing tubulin genes in large cohorts of MCD patients has detected tubulin mutations in only 1-13

218 changes demonstrated by CM in the course of MCD.

219 coronary narrowings with variable degrees of MCD.

220 esults identify key sequence determinants of MCD-promoted speckle condensation and link the dynamic m

221 n Unit (years 2007-2011) with a diagnosis of MCD were included.

222 K in patients with preoperative diagnosis of MCD, and 28 normal eyes.

223 showed the classic histological features of MCD, and LANA-1 immunostaining identified HHV-8-infected

224 Induction of MCD in skeletal muscle resulted in a suppression of mito

225 Acute induction of MCD in the skeletal muscle of obese and glucose intolera

226 ensitivity were determined upon induction of MCD.

227 ividual with MCD and a comprehensive list of MCD-related genes with their associated phenotypes.

228 e dismutase 2 in muscle but not the liver of MCD(-/-) mice.

229 A and FK506 on proteinuria in a rat model of MCD induced by puromycin aminonucleoside (PAN) and in vi

230 evaluate the characteristics and outcomes of MCD patients admitted to a specialist neuropsychiatric i

231 y to the BCR in the endoplasmic reticulum of MCD cell line models and promotes the turnover of immatu

232 To determine the role of MCD in skeletal muscle of diet induced obese and insulin

233 herry hemangiomata is the presenting sign of MCD.

234 were able to shift substrate specificity of MCD toward succinyl-CoA through active-site mutagenesis.

235 Here we report the crystal structure of MCD at a resolution of 1.37 A.

236 pert clinicians in the diagnostic work-up of MCDs with the aim of improving patient management worldw

237 Assignments are based on MCD/Abs intensity ratios, transition energies, polarizat

238 SL had a phenotype similar to the WT mice on MCD and LPS challenge.

239 inactivation of CES1 aggravated alcohol- or MCD diet-induced liver inflammation and liver injury, li

240 ucial role in protection against alcohol- or MCD diet-induced liver injury.

241 hat in addition to decreasing fat oxidation, MCD inhibition also has novel effects on protein acetyla

242 , skin changes (POEMS)-associated MCD (POEMS-MCD).

243 As expected, betaine prevented MCD diet-induced NASH.

244 calvarial porosity and significantly reduced MCD, compared with vehicle-treated controls.

245 Only 1 patient died of relapsed MCD (at fifth relapse 9.4 years after initial diagnosis)

246 -8 and immune cells that cause HHV-8-related MCD.

247 hose described in HIV-positive HHV-8-related MCD.

248 The diabetes group showed similar results (MCD, CVF and cardiac myocyte apoptosis index) to other a

249 Here, we review MCD in adults with particular focus on the evidence for

250 d pathobiological characteristics of several MCDs have been recently elucidated.

251 Patients with severe, long-standing MCD can achieve significant improvements in functioning

252 trol diet, MCD diet, or betaine-supplemented MCD (MCD+B) diet for 8 wk and collected blood and tissue

253 The low-temperature MCD spectra of complex 1 exhibit three pseudo A-term sig

254 rs and demonstrate that variable-temperature MCD spectroscopy provides a means of detecting and inves

255 -NIR absorption, CD and variable-temperature MCD, and protein-film electrochemistry.

256 ysis of the signs of the experimental C-term MCD signals, we unambiguously assign a low-energy MCD fe

257 ed metabolic dysfunction, demonstrating that MCD inhibitors may have utility in the battle against ch

258 We found that MCD diet-fed, liver-specific LRH-1 knockout mice (Lrh-1(

259 ional and phenotypic profiling suggests that MCD/C5s, as a class, represent bona fide MB tumors.

260 The MCD diet increased hepatic levels of FGF21 messenger RNA

261 The MCD diet significantly increased plasma half-life and me

262 The MCD spectrum of biferrous MIOX shows two ligand field (L

263 ate that the diabetic ob/ob genotype and the MCD disease model alter kidney transporter expression an

264 direct interaction between AC domain and the MCD is required for the inhibitory effect.

265 IOP values did not differ between the MCD (11.25 +/- 1.69 mm Hg) and PK (12.0 +/- 2.67 mm Hg)

266 CRF did not differ between the MCD (8.34 +/- 2.12 mm Hg) and the PK (8.66 +/- 1.66 mm H

267 lizing brain membranes were decreased by the MCD diet, and these improved modestly with betaine.

268 ting the pathological changes induced by the MCD diet.

269 e serum albumin (MDA-BSA) before feeding the MCD diet.

270 CCT was significantly lower in the MCD group (423 +/- 47 mmu) than in the PK group (541 +/-

271 ity as carbohydrate-derived palmitate in the MCD model of fatty liver disease.

272 In the MCD model of steatohepatitis, carbohydrate-derived palmi

273 oxic when combined with dietary sugar in the MCD model, presumably by enhancing hepatic de novo lipog

274 ear to do so by stimulating methylation; the MCD+B diet worsened hyperhomocysteinemia and depressed l

275 spectra and independent determination of the MCD C-term signs from the corresponding electron donatin

276 responses observed in wild-type mice on the MCD diet were also observed in Lrh-1(-/-) mice on a norm

277 nd hepatic regeneration ratio in mice on the MCD diet.

278 Based on the MCD structure, we were able to shift substrate specifici

279 Casp8 expression in hepatocytes reduced the MCD-dependent increase in apoptosis and decreased expres

280 In mice fed up to 8 weeks with the MCD diet the extension of liver injury and lobular infla

281 thylation potential 8-fold compared with the MCD diet.

282 18 cases and 12 HIV-negative HHV-8-unrelated MCD cases showed marked discrepancies.

283 ectively, compared with cells from untreated MCD diet-fed mice.

284 Herein, we use UV-vis, CD, XAS, EPR, VT/VH-MCD, and resonance Raman spectroscopies, augmented with

285 al approach using X-ray diffraction; UV/vis, MCD, IR, EPR, and NMR spectroscopy; and quantum chemistr

286 The VT MCD spectra of the enzymatic S = 2 Fe(IV) horizontal lin

287 From CD, MCD, and VTVH MCD, p-anisidine addition is found to minimally perturb

288 Abs/CD/MCD/VTVH MCD data exhibit 12 transitions that are assigned as d-d

289 variable-temperature, variable-field (VTVH) MCD spectroscopies are used to determine the geometric a

290 /variable temperature, variable field (VTVH) MCD spectroscopies to obtain detailed insight into its g

291 variable-temperature, variable-field (VTVH) MCD spectroscopies, 4-AP is found to bind directly to th

292 d variable temperature variable field (VTVH) MCD spectroscopies.

293 variable-temperature, variable-field (VTVH) MCD studies of non-alpha-KG-containing forms and the con

294 tivity; NRVS focuses on the Fe(III), whereas MCD reflects the spin-allowed transitions mostly on the

295 recurrence in 10 children and 20 adults with MCD/MesGN (n=22) or FSGS who had suffered >/=2 recurrenc

296 dynein motility in vitro are associated with MCD.

297 This study enrolled 24 eyes with MCD, 25 eyes that underwent PK in patients with preopera

298 w that can be applied to any individual with MCD and a comprehensive list of MCD-related genes with t

299 sult in underestimating IOP in patients with MCD and in those undergoing PK for this stromal dystroph

300 entrations were slightly increased in the WT/MCD and ob/control groups, whereas plasma concentrations