ライフサイエンスコーパス: MCT

1 MCT achieved better results with significantly lower reo

2 MCT feeding stimulated jejunal-epithelial thymic stromal

3 MCT in the ES stroma were more degranulated than in thos

4 MCT of A. funestus switched from 2 AM in Lokohoue and 3

5 MCT rats developed pulmonary arterial hypertension, RV f

6 MCT suppressed antigen absorption into blood but stimula

7 MCT-4 was in turn regulated by EMMPRIN (CD147) as determ

8 MCT-injected rats developed severe PH by day 21 and prog

9 MCT-RVfib (but not left ventricular fibroblasts) display

10 MCT-RVfib manifest a DNMT1-HIF-1alpha-PDK-mediated, cham

11 MCT-sensitized mice experienced IgG-dependent anaphylaxi

12 d the malignant T cell-amplified sequence 1 (MCT-1/MCTS1) oncoprotein support noncanonical translatio

13 , we identify monocarboxylate transporter-1 (MCT-1) as a receptor used by HERV-T for attachment and i

14 il phase (sunflower oil-LCT or NEOBEE(R)1053-MCT) and emulsifiers (WPI, Tween 80 - T80 or WPI/T80 mix

15 OS (80% olive oil and 20% SO), or MOSF (30% MCTs, 25% olive oil, 30% SO, and 15% FO).

16 tion or at follow-up at 52 weeks (HIIT, 39%; MCT, 25%; RRE, 34%; P=0.16).

17 ent levels of monocarboxylate transporter-4 (MCT-4) specialized in secreting lactate from glycolytic

18 colysis (monocarboxylate transporter type 4 [MCT-4]), and angiogenesis (vascular endothelial growth f

19 n triglycerides (MCTs) and 50% SO], MSF (50% MCTs, 40% SO, and 10% fish oil (FO)], OS (80% olive oil

20 the 106 eyes (77 patients) included were 54 MCT and 52 CAS eyes.

21 Several airway diseases exhibit abnormal MCT, including asthma, chronic bronchitis, and cystic fi

22 importance in the CNS, little is known about MCT expression and lactate function in the PNS.

23 An acute MCT with a comparable baseline was available in 71 of 82

24 The magnitude of increase in acute MCT above the threshold predicted by consensus equation

25 Analyses confirmed that a rise in acute MCT greater than that defined by the equation had a sens

26 2 mg/L) has been proposed to interpret acute MCT in mast cell activation syndrome (MCAS).

27 in a subgroup of participants (n = 16) after MCT supplementation.

28 ly and precipitously during 23-32 days after MCT.

29 and increased IL-4 and IL-13 secretion after MCT/EW challenge.

30 human mast cells were suppressed by LA in an MCT-dependent manner.

31 are always, sometimes, or never a part of an MCT.

32 with tumor volume (R = 0.903, P = 0.005) and MCT 1 (R = 0.85, P = 0.032) and HIF1alpha expression (R

33 27), HK-3 (P = 0.013), VEGF (P = 0.049), and MCT-4 (P = 0.020).

34 ession were observed for GLUT-1, GLUT-3, and MCT-4.

35 among all participants regardless of age and MCT (61.1+/-1.8% and 0.64+/-0.05, respectively).

36 her agent alone, indicating that GABA(B) and MCT inhibitors, alone and in combination, represent pote

37 The operator's impact on CCT and MCT measurements is insignificant in all devices.

38 CCT and MCT measurements show moderate agreement between instrum

39 ence of oil phase composition (vitamin D and MCT), surfactant-to-oil ratio (SOR), surfactant type (Tw

40 DENR and MCT-1 form a heterodimer, which binds to the ribosome.

41 atomic details of the mechanism of DENR and MCT-1 interaction.

42 cal load influences electrical dynamics) and MCT (how mechanical load alters cell signalling and Ca(2

43 12 weeks was not different between HIIT and MCT (P=0.45); left ventricular end-diastolic diameter ch

44 here was also no difference between HIIT and MCT in peak oxygen uptake (P=0.70), but both were superi

45 rmed by siRNA-mediated knockdown of LDHA and MCT-4, which decreased lactate secretion and macrophage

46 nt regions outside the GA were measured, and MCT and CVI from the entire scan area were measured.

47 validated against manual segmentations, and MCT measurements were shown to be in good agreement (P <

48 Control diet (sham and MCT group) and isocaloric nutritional intervention (MCT

49 boratory containing varying ratios of FO and MCTs, and subsequently FO- and 50:50 FO:MCT-ILE plus 500

50 gistic relation between alpha-tocopherol and MCTs.

51 ine decreased in the order LCT approximately MCT>>orange oil; whereas beta-carotene bioaccessibility

52 sodium lactate revealed that LA effects are MCT-1- and pH-dependent.

53 To assess MCT, we tracked movement of radiodense microdisks in air

54 ol ELPCs (expressing nuRFP alone) attenuated MCT-induced right ventricular systolic pressure increase

55 tein into the oocyte cytosol did not augment MCT transport function.

56 Correlations between MCT and choroidal vessel metrics of CVV, CSV, CVI, and C

57 However, there was no difference between MCT and the corn oil control supplement in the intestina

58 The number of observed taxa between MCT and healthy skin surfaces was detected, showing a de

59 Both MCT and MCTC in tumour islets were higher in ES (20.0 an

60 Since both MCT and MCTC infiltrating tumour islets in ES NSCLC pati

61 s that was inhibited by niflumic acid and by MCT siRNA knockdown, and significantly reduced in the pr

62 Such exchange is catalyzed by MCT transporters, which cotransport lactate and protons

63 Overall, the murine model of PH elicited by MCT mimics loss of body weight and diaphragm muscle weak

64 However, it is unclear if PH induced by MCT in mice reproduces the loss of body weight and diaph

65 rmalities in a murine model of PH induced by MCT.

66 studies, and these studies are supported by MCT homology modeling and computational inhibitor-dockin

67 lic interactions are most likely mediated by MCTs.

68 microenvironment, H(+)-coupled transport by MCTs tends to drive lactate from the interstitium into t

69 lock ancHTenv mediated infection, by causing MCT-1 depletion from cell surfaces.

70 onstration of chemical sensing using on-chip MCT waveguides, monolithically fabricated IR sensing sys

71 eta-carotene entrapped within protein-coated MCT droplets was more stable than within T80-MCT systems

72 study was to test whether FO-ILEs containing MCTs and/or additional alpha-tocopherol decrease the inf

73 Limitations of current MCT assays and of current animal models of human disease

74 bicarbonate transport, explaining defective MCT that occurs even in the presence of adequate PCL hyd

75 airway surface liquid contributes to delayed MCT beyond that caused by airway dehydration alone and i

76 further the mechanism of regulation of DENR-MCT-1 activities in unconventional translation initiatio

77 Dietary MCTs promote allergic sensitization and anaphylaxis by a

78 We sought to test how dietary MCTs affect food allergy.

79 s and which unites our findings with earlier MCT studies.

80 mayonnaise remained intact containing either MCT oil or LSO.

81 However, a significant difference favouring MCT was found on the BDI-II at post treatment (-5.49 [95

82 Significant differences favouring MCT, also maintained over time, were observed for most s

83 Mixed FO/MCT and the addition of alpha-tocopherol to FO improved

84 ared with UPD-fed controls, whereas 50:50 FO:MCT, 30:70 FO:MCT, FO + AT, and 50:50 + AT groups had si

85 and MCTs, and subsequently FO- and 50:50 FO:MCT-ILE plus 500 mg/L alpha-tocopherol (FO + AT and 50:5

86 Mice that received 30:70 FO:MCT developed mild hepatosteatosis.

87 fed controls, whereas 50:50 FO:MCT, 30:70 FO:MCT, FO + AT, and 50:50 + AT groups had significantly lo

88 Following MCT 74% of patients compared with 52% in CBT met formal

89 ter surgery was -0.03 mm (-0.34 to 0.40) for MCT and +1.35 mm (-0.64 to 1.62) for CAS (P < .001).

90 Normal airways compensate for MCT-driven H(+) secretion by secreting HCO3(-), a proces

91 and extent of lipid digestion was higher for MCT- than LCT-emulsions, which was attributed to differe

92 impact of 4 wk of supplementation with 20 g MCT oil/d or 20 g corn oil/d on the kinetics of apolipop

93 As a result, we found that these glycolytic/MCT-deficient cells resumed growth by redirecting their

94 Group MCT targeting cognitive biases vs neuropsychological tra

95 mately fish oil > orange oil > mineral oil > MCT.

96 bioaccessibility decreased in the order LCT>>MCT>orange oil.

97 2 weeks of supervised interventions of HIIT, MCT, or a recommendation of regular exercise (RRE).

98 structure of the heterodimer formed by human MCT-1 and the N-terminal domain of DENR at 2.0- angstrom

99 ased level of p63 transcript in hypertrophic MCT cells (an established cell model of chondrocyte matu

100 Impaired MCT was not due to periciliary liquid depletion; rather,

101 Thus, impaired MCT is a primary defect in CF, suggesting that submucosa

102 In MCT cells, a mouse renal proximal tubule cell line, ATP

103 In MCT cells, inhibition of gamma-secretase similarly atten

104 In MCT rats, dichloroacetate, at therapeutic levels in the

105 The percentage of IOP reduction was 52.1% in MCT and 45.5% in CAS (P = .1616).

106 ber atrophy was induced (P < 0.05) by 22% in MCT compared to sham mice, but prevented in MCT + NI gro

107 glaucoma surgery was required in 5.5% (3) in MCT and 63.4% (33) in CAS.

108 HIIT and -1.2 mm (-3.6 to 1.2 mm; P=0.34) in MCT.

109 HIF-1alpha activation in MCT-RVfib reflected increased DNMT (DNA methyltransferas

110 hallenges of sensitized mice with antigen in MCT significantly aggravated anaphylaxis compared with c

111 (all p < 0.05) and these were attenuated in MCT + NI mice.

112 Complete success was 69% (37 cases) in MCT and 23% (12 cases) in CAS.

113 The ability to quantify in vivo changes in MCT may have utility in pre-clinical research studies de

114 he aim of this study was to image changes in MCT produced by a rehydrating treatment based on hyperto

115 Age-related changes in MCT, CVV, CSV, and CVI were studied in the entire scanni

116 gery, qualified success was 85% (46 eyes) in MCT and 37% (19 eyes) in CAS.

117 utes phylum and Corynebacteriaceae family in MCT skin surface when compared to the healthy contralate

118 in with MCT, as well as prolonged feeding in MCT-based diets, caused spontaneous allergic sensitizati

119 ES protocol, but there was no improvement in MCT performance.

120 dose of AE produced the most improvement in MCT with dose-dependent changes in Klotho in the blood.

121 s suggest that the CAIV-mediated increase in MCT transport activity requires direct binding between C

122 d MCT4 resulted in a significant increase in MCT transport activity, even in the nominal absence of C

123 were statistically significant increases in MCT in the isotonic and HS-P308 groups.

124 MCT compared to sham mice, but prevented in MCT + NI group.

125 H/HeJ mice were fed peanut butter protein in MCT, LCT (peanut oil), or LCT plus an inhibitor of chylo

126 a-tocopherol during storage being similar in MCT and LSO mayonnaises, but being stable in mixed oil m

127 d the role of the sirtuin deacylase Sirt5 in MCT metabolism by feeding Sirt5 knockout mice (Sirt5KO)

128 ring supervised HIIT and 80% above target in MCT.

129 duced pulmonary preferential vasodilation in MCT induced PAH rats.

130 tment with the 3-mAb cocktail during initial MCT/EW immunization induced EW tolerance.

131 up) and isocaloric nutritional intervention (MCT + NI) were administered.

132 e viscoelasticity of interfacial layers (LCT/MCT-1% WPI).

133 omoted an increase of viscoelasticity of LCT/MCT-T80 (0.5%WPI/0.5%T80 and 1%T80 w/w) interfaces, but

134 e reduced the interfacial tension of the LCT/MCT-water systems.

135 MMP-9/TIMP-1 ratio, and significantly lower MCT-1 and CD98 levels, factors associated with EMMPRIN f

136 In general, many MCTs exist for a given network; here we characterize a b

137 enomenology can be reproduced by microscopic MCT, thus challenging the conventional belief that exist

138 pharmacological efficacy in a monocrotaline (MCT) induced rat model of PAH.

139 mice by weekly injections of monocrotaline (MCT) for 8 weeks.

140 a single subcutaneous dose of monocrotaline (MCT, 60 mg/kg) to induce PH-associated RVF (PH, n=24) or

141 Sprague-Dawley rats received monocrotaline (MCT; 60 mg/kg) or saline.

142 RFP) were tested in rats with monocrotaline (MCT)-induced PAH.

143 ts with pressure-induced RVF (monocrotaline [MCT] injection, n = 25; controls with saline injection,

144 model of PH induced by drug (monocrotaline, MCT) has been extensively used in mice to examine the et

145 nalogue of Northern methanocarbathymidine (N-MCT), 2 and 3, are reported.

146 We show that 2'-F incorporation on the N-MCT scaffold has a strong stabilizing effect on duplex t

147 ich was pathologically activated in normoxic MCT-RVfib.

148 At 1 year, 94.3% of MCT cases achieved qualified success compared to 34.6% o

149 In ES 44% of MCT and 37% of MCTC expressed TNFalpha in the tumour isl

150 Therefore, we developed an in vivo assay of MCT, and here we describe its use in newborn wild-type p

151 CAIV-mediated augmentation of MCT activity was independent of the CAIV catalytic funct

152 d not suppress CAIV-mediated augmentation of MCT transport activity.

153 rkers to support the clinical development of MCT inhibitors now in clinical trials.

154 hows, for the first time, that disruption of MCT binding to their chaperon, Basigin, may be an effect

155 Regional distributions of MCT and CVI were analyzed using a grid centered on the f

156 tential mechanisms underlying the effects of MCT on triglyceride-rich lipoprotein (TRL) metabolism ha

157 We assessed the clinical efficacy of MCT compared to current best psychotherapy practice, CBT

158 was corroborated by the strong expression of MCT-4, EMMPRIN and LDHA in perivascular macrophages in M

159 er, our results suggest that facilitation of MCT transport activity by CAII requires direct binding b

160 Our results show that facilitation of MCT transport activity requires direct binding of CAIV t

161 The increased granularity of MCT data provided by this assay may provide an opportuni

162 Understanding the impact of MCT blockade on tumor cell metabolism may help develop c

163 investigate the ultrastructural mechanics of MCT by measuring fibril strain at different chemically i

164 However, the mechanisms of MCT have not been characterized at the nanoscale.

165 Understanding the mechanisms of MCT quantitatively may have applications in development

166 ped to explain the biophysical mechanisms of MCT.

167 he right ventricle revealed that a number of MCT-altered genes and neurotransmitter pathways (dopamin

168 the chief investigator is the originator of MCT and group differences in time under therapy.

169 Preliminary data on bacterial population of MCT dermis, obtained only on three dogs, demonstrated an

170 In the presence of MCT, the overall uptake of BMV was increased and provide

171 hanced from both polymers in the presence of MCT.

172 ta composition is related to the presence of MCT.

173 The functional relevance of MCT-4/EMMPRIN interaction was affirmed by lower macropha

174 terogeneity of taxa over the skin surface of MCT, at both inter- and intra-individual level.

175 Importantly, the effects of MCTs could be mimicked by adding Pluronic L81 to LCTs, a

176 metabolic dysfunction resulting from loss of MCTs.

177 substrate binding and transport mechanism of MCTs, elucidate the mode of action of three anti-cancer

178 Compared with corn oil, MCT supplements had no significant effect on plasma lipo

179 otein) and extracellular CAIV (expressed) on MCT transport activity, were additive.

180 indicating the removing of oxidized films on MCT wafers, which is difficult to achieve using single H

181 observed in electrochemical measurements on MCT wafers.

182 5, and 4.74 nm are achieved, respectively on MCT wafers after CMP.

183 in terms of protease content (tryptase-only [MCT], tryptase + chymase [MCTC]) and tumour necrosis fac

184 mice after 6-8 weeks of saline (control) or MCT (600 mg/kg) injections.

185 ped for mercury cadmium telluride (HgCdTe or MCT) semiconductors.

186 mRNA are released by eIF1/eIF1A, Ligatin, or MCT-1/DENR.

187 ing a so-called minimal control topology, or MCT).

188 Inhibition of HDACs or MCTs decreases acetate export and lowers pH(i), particul

189 Our MCT analysis allows us to rationally identify the micros

190 Outward MCT activity is, however, thermodynamically inhibited by

191 he advantage of junctional transmission over MCT in vivo.

192 The consensus equation (peak MCT should be>1.2x baseline tryptase+2 mg/L) has been pr

193 rtile range (IQR) time from reaction to peak MCT was 1.34 (0.82-2.51) hours.

194 s, including the host-derived formyl peptide MCT-ND4, we found that the PSMalpha peptides lacked the

195 Finally, the polished MCT wafers are cleaned and dried by deionized water and

196 itor dichloroacetate was started 7 days post-MCT.

197 hondrocyte maturation) than in proliferative MCT cells.

198 liquid lipid nanoparticles (LLNs) with pure MCT.

199 to HIIT at 90% to 95% of maximal heart rate, MCT at 60% to 70% of maximal heart rate, or RRE.

200 stacyclin synthase-expressing ELPCs reversed MCT-induced PAH.

201 which preserves the structure of the DENR's MCT-1-binding interface that is essential for the dimeri

202 Finally, we develop a simplified schematic MCT model which corroborates our microscopically founded

203 Timed serial MCT measurements were mapped against the consensus equat

204 Compared to sham, MCT mice increased heart weight by 7%, RV thickness by 1

205 on for the storage effect to all of standard MCT's spatial and temporal coexistence mechanisms, and a

206 vel MIR sensing approach utilizes structured MCT chips fabricated via molecular beam epitaxy (MBE) as

207 MCT droplets was more stable than within T80-MCT systems.

208 or structure-guided drug discovery targeting MCTs.

209 d nitrogen cooled mercury cadmium telluride (MCT) detector and compare their performance to a commerc

210 eguides made from mercury-cadmium-telluride (MCT)-a material to date exclusively used for mid-infrare

211 as a treadmill-based maximum capacity test (MCT).

212 The methacholine challenge test (MCT) is commonly used to assess airway hyperresponsivene

213 n was also considerably higher for LCT- than MCT-emulsions, which may impact the subsequent absorptio

214 n E after digestion was higher for LCT- than MCT-emulsions, which was attributed to the greater solub

215 We argue that MCT's mathematical complexity and subtlety have obscured

216 We show that MCT is NP-hard, and present an exact algorithm based on

217 ng with Raman micro-spectroscopy showed that MCT was primarily confined to the inclusions within the

218 less evidence from this study suggests that MCT had considerable beneficial effects in treating depr

219 t analyses demonstrated that patients in the MCT group had significantly greater reductions in the co

220 lyses also demonstrated that patients in the MCT group had significantly greater reductions in the PA

221 ydrophobic films, it was more soluble in the MCT inclusions in hydrophilic films, suggesting its incr

222 ixed-abrasive lapping is used to machine the MCT wafers, and the lapping solution is deionized water.

223 Administration of the MCT inhibitor l-lactate increased GHB renal and total cl

224 We investigated the ability of the MCT to differentiate participants with a physician's dia

225 The utility of the MCT to rule out a diagnosis of asthma depends on racial

226 We sought to determine if disruption of the MCT-Basigin interaction may be achieved with a small mol

227 luate the sensitivity and specificity of the MCT.

228 se findings highlight that inhibition of the MCT/BSG complexes alone or in combination with phenformi

229 Secondly, the MCT wafers are polished using the developed CMP slurry.

230 psychological training group relative to the MCT group.

231 crophage transmigration in culture using the MCT-4 inhibitor, alpha-cyano-4-hydroxy-cinnamic acid (CH

232 directly interfacing the waveguide with the MCT detector element may be envisaged.

233 Modern coexistence theory (MCT), formalised by Chesson, holds out the promise of do

234 s and first-principles mode-coupling theory (MCT) of star-polymer vitrimers.

235 t a recent treatment, metacognitive therapy (MCT), might be more effective, by targeting mental contr

236 Phenformin addition to these MCT-disrupted cells in normoxic and hypoxic conditions i

237 ow deficits (FDs), mean choroidal thickness (MCT), and choroidal vascularity index (CVI) were investi

238 estigated included mean choroidal thickness (MCT), choroidal vessel volume (CVV), choroidal stroma vo

239 of magnitude upon implementation of thinner MCT waveguides.

240 door biting (POB) and median catching times (MCT) were compared.

241 The mutable collagenous tissue (MCT) of echinoderms (e.g., sea cucumbers and starfish) i

242 85 were randomly allocated to MCT and 89 to CBT.

243 The transition from CAS to MCT can be easily achieved, even in difficult cases or t

244 Sirt5 in regulating the hepatic response to MCT and may shed light into the pathogenesis of periport

245 HIIT was not superior to MCT in changing left ventricular remodeling or aerobic c

246 is superior to moderate continuous training (MCT) in reversing cardiac remodeling and increasing aero

247 Metacognitive training (MCT) is targeted at cognitive biases involved in the pat

248 upling, MEC) and mechano-chemo-transduction (MCT) mechanisms at cell and molecular levels which coupl

249 have developed a novel mucociliary transit (MCT) measurement that uses synchrotron phase contrast X-

250 Mucociliary transport (MCT) is an innate defense mechanism that removes particu

251 d (PCL) hydration and mucociliary transport (MCT) rates, a relationship frequently invoked but never

252 rized by a deficit in mucociliary transport (MCT), a process that traps and propels bacteria out of t

253 d to the parent monocarboxylate transporter (MCT) inhibitor cyano-hydroxycinnamic acid (CHC).

254 s including the monocarboxylate transporter (MCT), the sodium hydrogen exchanger (NHE), and V-Type AT

255 Monocarboxylate transporters (MCT) modulate tumor cell metabolism and offer promising

256 inhibitors for monocarboxylate transporters (MCT) or replacing LA with sodium lactate revealed that L

257 onocarboxylate lactate-H(+) co-transporters (MCTs) with AR-C155858.

258 Proton-coupled monocarboxylate transporters (MCTs) are carriers of high-energy metabolites such as la

259 Proton-coupled monocarboxylate transporters (MCTs) mediate the exchange of high energy metabolites li

260 Monocarboxylate transporters (MCTs) mediate the proton-coupled exchange of high-energy

261 of the cell by monocarboxylate transporters (MCTs), preventing further reductions in pH(i).

262 inhibition of monocarboxylate transporters (MCTs), resulting in a negative feedback on glycolytic ra

263 e exported via monocarboxylate transporters (MCTs), supporting the formation of an acidic microenviro

264 e-transporting monocarboxylate transporters (MCTs), we used a combination of primary cell culture stu

265 proton-coupled monocarboxylate transporters (MCTs), which are encoded by members of the Slc16a family

266 nd protons via monocarboxylate transporters (MCTs), which exacerbates extracellular acidification and

267 tate efflux on monocarboxylate transporters (MCTs).

268 ocytes through monocarboxylate transporters (MCTs).

269 We introduce the Multiple Consensus Tree (MCT) problem to simultaneously cluster T and infer a con

270 rols (LCT) or medium chain triacylglycerols (MCT) as carrier oils.

271 ating the lipidic medium-chain triglyceride (MCT) into polymeric film-forming systems (FFS) for topic

272 component of the medium-chain triglyceride (MCT) ketogenic diet.

273 either saturated medium chain triglyceride (MCT) oil or unsaturated purified linseed oil (LSO), were

274 Orange oil, medium-chain triglyceride (MCT) oil, and WPI were used to make stable nanoemulsions

275 , being blends of medium chain triglyceride (MCT) oil, glyceryl stearate (GS) or hydrogenated palm oi

276 Medium-chain triglyceride (MCT) supplements are used by clinicians to treat patient

277 oil composition (medium-chain triglyceride (MCT) to long-chain triglyceride (LCT) ratio) and total c

278 following order: medium chain triglycerides (MCT) > corn oil approximately fish oil > orange oil > mi

279 lycerides (LCT), medium chain triglycerides (MCT) or orange oil as carrier oils.

280 Medium-chain triglycerides (MCT), containing C(8)-C(12) fatty acids, are used to tre

281 l (SO)], MS [50% medium-chain triglycerides (MCTs) and 50% SO], MSF (50% MCTs, 40% SO, and 10% fish o

282 e suggested that medium-chain triglycerides (MCTs) and alpha-tocopherol have anti-inflammatory proper

283 oral gavage with medium-chain triglycerides (MCTs) plus egg white (EW) and was characterized by incre

284 ted that dietary medium-chain triglycerides (MCTs), which bypass mesenteric lymph and directly enter

285 or assessing serum total mast cell tryptase (MCT) in anaphylaxis.

286 ogs affected by spontaneous mast cell tumor (MCT), using skin contralateral sites as intra-animal hea

287 direct interaction between CAIV and the two MCT chaperones basigin (CD147) and embigin (GP70).

288 ed adjuvant, with microcrystalline tyrosine (MCT), monophosphoryl lipid A (MPLA) and calcium phosphat

289 ic distribution (VOL(AAP), VOL(PAP), and VOL(MCT)), and percentage of relative volume increase (VOL(A

290 Suppression was MCT-1 dependent and reproducible with sodium lactate or

291 Whereas MCT, CVV, and CSV decrease with age, the CVI and CSVR re

292 However, it remains uncertain whether MCT abnormalities contribute to the genesis of disease o

293 ng a signaling molecule that correlated with MCT performance in the AE conditions, but also highly co

294 and CSV showed significant correlations with MCT (all P < 0.0001).

295 copy imaging and nanoindentation of FFS with MCT revealed two-phase structured films with softer incl

296 or TSLP mAbs before initial oral gavage with MCT/EW to suppress FA development; treatment with the sa

297 A single gavage of peanut protein with MCT, as well as prolonged feeding in MCT-based diets, ca

298 The LV of rats with MCT-induced RVF exhibited electrophysiologic remodeling:

299 ndicate that short-term supplementation with MCT has a neutral effect on TRL apo B-48 and VLDL apo B-

300 ic hydroxypropyl cellulose, with and without MCT.