ライフサイエンスコーパス: MDC

1 MDC biogenesis requires the ER-mitochondria encounter st

2 MDC frequencies tended to be reduced in HIV infection (1

3 MDC mRNA and protein expression were markedly induced in

4 MDC-associated IL-12 production was markedly reduced in

5 MDC-enriched cells were either CD123(+) or CD123(-), but

6 MDC-expressing DCs specifically attracted T lymphocytes

7 MDC-L (ADAM 28), a member of the ADAM (a disintegrin and

8 MDC-staining and the GFP-AtATG8e fusion protein can now

9 MDCs activated with lactobacilli clearly skewed CD4(+) a

10 MDCs and PDCs exposed to either rAd5 or rAd35 encoding f

11 MDCs derived in vitro suppressed angiogenesis in vivo th

12 MDCs were more susceptible to both rAd35 and rAd5 than w

13 < .001), IL-10 (P = .01), IP-10 (P = .0001), MDC (P < .001), MIP-1alpha, (P < .001), MIP-1beta (P = .

14 of CC (I-309, Exodus-1, TARC, RANTES, MCP-1, MDC, and MIP-1 alpha and -1 beta), CXC (GRO-alpha, -beta

15 quence in the disintegrin domain of ADAM-15 (MDC-15; metargidin) highlighted ADAM-15 as a protein par

16 ADAM 28 (MDC-L) is expressed by human lymphocytes and contains a

17 IL-12p70, IL-13, IL-16, IP-10, MCP-1, MCP-4, MDC, MIP-1a, TARC, TNFB) was associated with diminished

18 participants (21/112) did not meet accepted MDC for Moebius syndrome because they had abduction defi

19 st a potential role for the lymphocyte ADAM, MDC-L, in the interaction of lymphocytes with alpha(4)be

20 Consistent with this finding, virtually all MDCs isolated from the tissues or draining lymph nodes o

21 30 cross-sectional subjects revealed that an MDC:PDC ratio more than or equal to 1.78 was associated

22 Two configurations of anammox MDCs (anaerobic-anammox cathode MDC (AnA(mox)MDC) and ni

23 Treatment of platelets with SDF-1 and MDC rapidly exposed P-selectin (CD62P) on the cell surfa

24 In contrast, TARC, MCP-1, and MDC were not induced, suggesting the existence of distin

25 Whereas ADAM8, ADAM15, and MDC-L (ADAM28) are expressed in specific cell types and

26 We found that ADAM8, ADAM15, and MDC-L, but not ADAM17, catalyzed ectodomain shedding of

27 es remotely the expression of TARC/CCL17 and MDC/CCL22 in the lungs.

28 ttern of CCR4 and its ligands TARC/CCL17 and MDC/CCL22 in the peripheral blood and skin of patients w

29 ssion of the two CCR4 ligands TARC/CCL17 and MDC/CCL22.

30 egulated chemokine; also known as CCL17) and MDC (macrophage-derived chemokine; CCL22).

31 pes (interleukin-8, interleukin-12, GRO, and MDC).

32 determined in vitro, but only methylone and MDC (1, 3 mg/kg, iv) produced significant elevations in

33 or the PK study, we found that methylone and MDC peaked early (Tmax=15-45 min) and were short lived (

34 rea-under-the-curve values for methylone and MDC were greater than dose-proportional, suggesting non-

35 atic biopsies which correlated with TARC and MDC expression and airway obstruction.

36 (LPS)-induced production of MCP-1, TARC, and MDC by DCs was clearly enhanced.

37 covery of both recipient MDSCs (P < .01) and MDCs (P < .01) is significantly increased when the alkyl

38 Intermediate monocytes and MDCs were highly frequent in the airways compared with p

39 As a result, both PDCs and MDCs preferentially transmitted HIV-1 to the responding

40 Soon after HIV-1 exposure, both PDCs and MDCs were able to transfer the virus to T cells in the a

41 nocytes and B cells or unstimulated PDCs and MDCs.

42 rin domain of MDC-L was inhibited by an anti-MDC-L monoclonal antibody (mAb), Dis1-1.

43 ined the role of Stat1 in obesity-induced AT MDC polarization and inflammation and insulin resistance

44 ivation, early and persistently in AT and AT MDCs of wild-type mice fed a high-fat diet (HFD).

45 A positive association was observed between MDC-associated IL-12 production and HCV-specific T cell

46 T cells conditioned with the B7-H1-blocked MDCs had a more potent ability to inhibit autologous hum

47 g IAV infection, we stimulated primary blood MDCs and inflammatory monocyte-derived DCs (MDDCs) with

48 TLR ligand was observed to enhance both MDC and PDC activation of naive CD4 T cells.

49 C express COX-1, but, unlike monocytes, both MDC populations constitutively express COX-2.

50 Both MDCs and PDCs were found to express the primary receptor

51 lone is extensively metabolized in rats, but MDC is the only centrally active metabolite that could c

52 4(+) T cells in a dose-dependent manner, but MDCs stimulated the highest frequencies of pp65-specific

53 resection is inhibited by gamma-H2AX and by MDC-1 (mediator of DNA damage checkpoint 1), which binds

54 action between RhoA and ROCK-2 is blocked by MDC and TGaseM, indicating a role for transglutaminase a

55 also up-regulates DC-SIGN gene expression by MDC.

56 IL-12 production and CD80/CD86 expression by MDC/PDC mixtures was further enhanced by CD40L and ISS-O

57 Further, second-wave aggregation induced by MDC in platelet-rich plasma was inhibited by aspirin, AD

58 JAK2, and this interaction was inhibited by MDC.

59 The release of TR6 by MDC was dependent on the activation of p42/p44 mitogen-a

60 n of GM3 and attenuation of virus capture by MDCs.

61 oprotein-pseudotyped lentivirus particles by MDCs were severely attenuated upon depletion of GSLs fro

62 esence or absence of mono-dansyl cadaverine (MDC), a TG inhibitor.

63 (mox)MDC)) were compared with an air cathode MDC (CMDC), operated in fed-batch mode.

64 s of anammox MDCs (anaerobic-anammox cathode MDC (AnA(mox)MDC) and nitration-anammox cathode MDC (NiA

65 (AnA(mox)MDC) and nitration-anammox cathode MDC (NiA(mox)MDC)) were compared with an air cathode MDC

66 Our data implicate TSLP, TARC/CCL17, MDC/CCL22, and IP-10/CXCL10 in asthma pathogenesis.

67 s expressing mRNA encoding TSLP, TARC/CCL17, MDC/CCL22, and IP-10/CXCL10, but not I-TAC/CXCL11 and I-

68 mucosa expressing mRNA for TSLP, TARC/CCL17, MDC/CCL22, and IP-10/CXCL10, but not I-TAC/CXCL11 and I-

69 venance of TSLP, Th2-attracting (TARC/CCL17, MDC/CCL22, I-309/CCL1), and Th1-attracting (IP-10/CXCL10

70 erum concentration of CXCL10 (IP-10), CCL22 (MDC), CCL17 (TARC), CCL-2 (MCP-1) and CCL-13 (MCP-4) in

71 in the local chemokine production of CCL22 (MDC) and CCL17 (TARC), two chemokines previously shown t

72 Nasal eotaxin-2, CCL22/MDC, and monocyte chemoattactant protein-1 (MCP-1) level

73 uniformly suppressed the production of CCL22/MDC, a chemokine associated with infiltration of T regul

74 the functional role of CCR3/eotaxin and CCR4/MDC during the in vivo recruitment of Th2 cells, we have

75 After TLI/ATS + BMT, Gr-1(low)CD11c(+) MDCs and Gr-1(high)CD11c(neg) myeloid-derived suppressor

76 ude non-DCs, Lin(-)HLA-DR(+)CD11c(+)CD123(-) MDCs and Lin(-)HLA-DR(+)CD11c(-)CD123(+) PDCs were ident

77 prevented in mice with myeloid-derived cell (MDC)-specific deletion of TNFR1.

78 itation-anammox microbial desalination cell (MDC) for resource-efficient wastewater treatment and des

79 Freshly purified myeloid dendritic cells (MDC) and plasmacytoid DC (PDC) obtained from subjects wi

80 Myeloid, CD1a-sorted dendritic cells (MDC) productively replicated human immunodeficiency viru

81 nocytes and myeloid-derived dendritic cells (MDC) released the protein exclusively following stimulat

82 unction of monocyte-derived dendritic cells (MDC).

83 n of human CD11c(+) myeloid dendritic cells (MDCs) and CD123(+) plasmacytoid dendritic cells (PDCs),

84 CD11c(+) macrophages/dendritic cells (MDCs) are increased and display the classically activate

85 od monocyte-derived myeloid dendritic cells (MDCs) express B7-H1, a member of the B7 family, on the c

86 capture of HIV-1 by mature dendritic cells (MDCs) is mediated by an interaction between the glycosph

87 ansion of recipient myeloid dendritic cells (MDCs) that induce contact-dependent expansion of donor T

88 By contrast, myeloid dendritic cells (MDCs) were absent from malignant ascites.

89 Monocyte-derived cells (MDCs), including professional antigen-presenting cells s

90 Microbial desalination cells (MDCs) are an emerging concept for simultaneous wastewate

91 Microbial desalination cells (MDCs) are an emerging concept for simultaneous water/was

92 ocalcifications, masses, and density change (MDC, cm(2)/year) was estimated using 1,940 sister pairs.

93 Macrophage-derived chemokine (MDC) is a potent chemoattractant for antigen-specific T

94 tralization of macrophage-derived chemokine (MDC) with Ab caused a significant reduction of CCR4-posi

95 ine (TARC) and macrophage-derived chemokine (MDC)), CD (10 proteins), and PS (kynureninase (KYNU)).

96 tional role of macrophage-derived chemokine (MDC), a potent mononuclear cell chemoattractant, during

97 r (TNF)-alpha, macrophage-derived chemokine (MDC), and C10, known to enhance bacterial clearance.

98 ctor (GM-CSF), macrophage-derived chemokine (MDC), and macrophage inflammatory protein 1alpha (MIP-1a

99 kines, RANTES, macrophage-derived chemokine (MDC), macrophage inflammatory protein-1beta (MIP-1beta),

100 orted that the macrophage-derived chemokine (MDC), thymus activation-regulated chemokine (TARC), and

101 ta chemokines [macrophage-derived chemokine (MDC), thymus and activation-regulated chemokine (TARC),

102 ; in addition, macrophage-derived chemokine (MDC), thymus and activation-regulated chemokine (TARC),

103 t (MCP-1, MCP-5, monocyte-derived chemokine (MDC), thymus and activation-related chemokine (TARC), C1

104 ine (TARC) and macrophage-derived chemokine (MDC).

105 ands eotaxin and monocyte-derived chemokine (MDC).

106 10)/CXCL10 and macrophage-derived chemokine (MDC)/CCL22 production were evaluated at the mRNA or at t

107 (TARC)/CCL17, macrophage-derived chemokine (MDC)/CCL22, I-309/CCL1) and Th1-attracting (IFN-gamma-in

108 rine (EPI) and macrophage-derived chemokine (MDC, CCL22).

109 MCP-3/CCL7) or macrophage-derived chemokine (MDC/CCL22), elicited anti-gp120 antibodies with high tit

110 ARC/CCL17) and macrophage-derived chemokine (MDC/CCL22), which preferentially attract type 2 T helper

111 or CCL17), and macrophage-derived chemokine (MDC; or CCL22).

112 le protein-10, macrophage-derived chemokine [MDC], and platelet factor-4 [PF-4]) to be expressed by C

113 and CCL22 (or macrophage-derived chemokine; MDC), in Th2-type cytokine-dominated responses in the lu

114 o induces the secretion of the CC-chemokines MDC and TARC.

115 OX40L, the production of the Th2-chemokines MDC (macrophage-derived chemokine/CCL22) and TARC (thymu

116 These findings suggest that the chemokines MDC, TARC, and SDF-1, which may be produced during infla

117 ferent from that of secretion of chemokines (MDC, I-309, MIP-1alpha, MIP-1beta, and RANTES), cytokine

118 alled the mitochondrial-derived compartment (MDC) that is generated from mitochondria in response to

119 curacy and minimum detectable concentration (MDC).

120 ecular Probes]), autophagic vacuole content (MDC), SA-beta-galactosidase (FDG), and cathepsin activit

121 In contrast, MDCs, which express TLR3, TLR4, and TLR7, responded to p

122 that networks within multiple-demand cortex (MDC) become active when diverse skills and behaviors are

123 ing, networks within multiple-demand cortex (MDC), which control higher cognitive functions, such as

124 oebius syndrome minimum diagnostic criteria (MDC) (congenital, nonprogressive facial palsy, and abduc

125 ted that degrakines based on SDF-1 (CXCL12), MDC (CCL22) and RANTES (CCL5) specifically inactivate th

126 assays showed that SDF-1, which binds CXCR4; MDC, which binds CCR4; and ELC, which binds CCR7, mediat

127 Myeloid CD11c DC (MDC), which may have inflammatory functions, and plasmac

128 Myeloid CD11c+ DC (MDC), which may have inflammatory functions, and plasmac

129 ells (DCs), we evaluated myeloid-derived DC (MDC) and plasmacytoid-derived DC (PDC) frequencies and f

130 based on phenotypic markers: the myeloid DC (MDC) and the plasmacytoid DC (PDC).

131 macytoid DCs (PDCs) 40-fold and myeloid DCs (MDCs) 20-fold less frequent in HIV(+) nodes than in cont

132 c cells (DCs); CD11c(+)CD123(-) myeloid DCs (MDCs) and CD11c(-)CD123(+) plasmacytoid DCs (PDCs).

133 ecies of Lactobacillus on human myeloid DCs (MDCs) and found that they modulated the phenotype and fu

134 how different subsets of human myeloid DCs (MDCs) involved in tissue inflammation are affected by in

135 Myeloid DCs (MDCs) were identified as CD1c(+)CD11c(+)CD14(-)HLA-DR(+)

136 acytoid DCs (PDCs) and CD11c(+) myeloid DCs (MDCs) were susceptible to both a CCR5- and a CXCR4-using

137 rformed to identify mature DCs, myeloid DCs (MDCs), and plasmacytoid DCs (PDCs) by using antibodies a

138 ed subpopulations of monocytes, myeloid DCs (MDCs), and plasmacytoid DCs in the lung mucosa.

139 asmacytoid DCs (PDCs), CD11c(+) myeloid DCs (MDCs), monocytes, and B cells.

140 a biotin-independent malonate decarboxylase (MDC), which allows them to use malonate as the sole carb

141 The key to developing MDCs is to understand fundamental problems, such as the

142 We conclude that an elevated MDC:PDC ratio associates with early small bowel allograf

143 We conclude that an elevated MDC:PDC ratio associates with liver graft rejection, whi

144 Blockade of B7-H1 enhanced MDC-mediated T-cell activation and was accompanied by do

145 PDCs to augment angiogenesis while excluding MDCs to prevent angiogenesis inhibition, demonstrating a

146 Lactobacillus-exposed MDCs up-regulated HLA-DR, CD83, CD40, CD80, and CD86 and

147 conversion of MDSCs to PD ligand-expressing MDCs, and increased donor naturally occurring Treg recov

148 n pediatric control muscle, there were fewer MDCs than PDCs, and the distributions of MDCs and PDCs w

149 e critical producers of cytokines (IL-12 for MDC and type I/II IFNs for PDC) and are functionally dif

150 In addition, for MDC, having a family history of breast cancer was associ

151 Stat1 is a key transcription factor for MDC polarization into the M1-like phenotype.

152 The estimated heritability for MDC was essentially null (2%; 95% CI, -8% to 12%).

153 l 3-kinase, signaling pathways important for MDC maturation and survival.

154 press CCR4, the common specific receptor for MDC and TARC.

155 on or the number of PDC, suggests a role for MDC in viral control.

156 e infiltrating lymphocytes, and staining for MDC was present on scattered individual cells throughout

157 How cells form MDCs is unclear.

158 Furthermore, MDC-mediated capture and transmission of MA mutant virus

159 .004) was associated with numerically higher MDC counts and significantly lower PDC frequencies (P=0.

160 Among rejectors, a significantly higher MDC:PDC ratio (P=0.004) was associated with numerically

161 sures analysis showed a significantly higher MDC:PDC ratio (P=0.043, F-test) among rejectors, compare

162 Our data show that different human MDC subsets may play distinct roles during IAV infection

163 dulated the phenotype and functions of human MDCs.

164 Immature MDC (I-MDC) and mature MDC express COX-1, but, unlike monocytes

165 By immunohistochemistry, we identified MDCs throughout different anatomical locations of the lu

166 Immature MDC (I-MDC) and mature MDC express COX-1, but, unlike mo

167 COX-2-mediated PG production impacts MDC function as maturing these cells in the presence of

168 COX-2 is functional in MDC as a specific inhibitor, NS-398, significantly reduc

169 Thus, the function of ERMES and Gem1 in MDC biogenesis may extend beyond their conventional role

170 dds ratio, 0.79; adjusted P=0.03) but not in MDC.

171 mice fed an HFD (16 weeks) had reductions in MDC (mainly CD11c(+) macrophage) M1-like polarization an

172 arameters to achieve optimal desalination in MDCs.

173 Cells enriched in MDCs expressed CD86, moderate CD80, and little CD40, but

174 hese results suggest that TNFR1 signaling in MDCs is a critical mediator of both the detrimental and

175 sing mice with specific knockout of Stat1 in MDCs (cKO).

176 ether, our results demonstrate that Stat1 in MDCs plays an important role in obesity-induced MDC M1-l

177 IL-12 was sustained in MDCs exposed to all three Lactobacillus species in the p

178 ignificantly, however, mice lacking TNFR1 in MDCs succumbed to listeria infection, although they disp

179 conversion of LC3-I to LC3-II and increased MDC-labeled autophagic vacuoles.

180 s plays an important role in obesity-induced MDC M1-like polarization and AT inflammation and contrib

181 upernatant collected from long-term infected MDC, which had been exposed to an X4R5 virus 45 days ear

182 Interestingly, MDC-mediated capture of Nipah and Hendra virus (recently

183 Draining lymph node LDC:MDC ratios induced by the protective immunogen were sign

184 In conclusion, in the human lung MDCs were twice as numerous and expressed higher levels

185 We propose that this causal role makes MDC candidate target for experimental therapeutics; for

186 Immature MDC (I-MDC) and mature MDC express COX-1, but, unlike monocytes, both MDC popul

187 our unrelated participants, also not meeting MDC, had large-angle exotropia, vertical gaze deficiency

188 Three percent of participants (3/112) met MDC but also had restricted vertical gaze.

189 The remaining 88 participants (79%) met MDC and had full vertical gaze.

190 I metabolites: 3,4-methylenedioxycathinone (MDC), 3,4-dihydroxy-N-methylcathinone (HHMC), and 4-hydr

191 Methylone, MDC, and HHMC were substrate-type releasers at monoamine

192 which was correlated with plasma methylone, MDC, and HHMC concentrations.

193 Treatment with monodansylcadarevine (MDC), a selective TGase inhibitor or down-regulation of

194 D Western blotting and monodansylcadaverine (MDC) staining to detect autophagic vacuoles.

195 which is inhibited by monodansylcadaverine (MDC; an inhibitor of transglutaminase activity) and TGas

196 s, the fluorescent dye monodansylcadaverine (MDC) and a green fluorescent protein (GFP)-AtATG8e fusio

197 h the TGase inhibitor, monodansylcadaverine (MDC), converted RA from a differentiation factor to an a

198 Cs in 0.3 M sucrose or monodansylcadaverine (MDC), which both inhibit clathrin-coated pit formation,

199 d p62, GFP-LC3 puncta, monodansylcadaverine (MDC) staining, and transmission electron microscopy.

200 effects, we found that monodansylcadaverine (MDC), which binds to the enzyme (transamidase) active si

201 to block tTG function (monodansylcadaverine; MDC) or c-Src kinase activity (PP2) disrupted the format

202 007 mW/m(3)) was higher than that of AnA(mox)MDC (444 mW/m(3)) and CMDC (952 mW/m(3)).

203 A(mox)MDC, significantly higher than AnA(mox)MDC (84%) and CMDC (77%).

204 x)MDC performed better than CMDC and AnA(mox)MDC in terms of power density, COD removal and salt remo

205 MDCs (anaerobic-anammox cathode MDC (AnA(mox)MDC) and nitration-anammox cathode MDC (NiA(mox)MDC)) we

206 0.022 kWh/m(3) >CMDC-0.019 kWh/m(3) >AnA(mox)MDC-0.021 kWh/m(3)) among the three configurations.

207 he maximum power density produced by NiA(mox)MDC (1,007 mW/m(3)) was higher than that of AnA(mox)MDC

208 The NiA(mox)MDC performed better than CMDC and AnA(mox)MDC in terms

209 Further, net energy balance of the NiA(mox)MDC was the highest (NiA(mox)MDC-0.022 kWh/m(3) >CMDC-0.

210 ) and nitration-anammox cathode MDC (NiA(mox)MDC)) were compared with an air cathode MDC (CMDC), oper

211 (NH(4)(+)-N) removal was achieved in NiA(mox)MDC, significantly higher than AnA(mox)MDC (84%) and CMD

212 of the NiA(mox)MDC was the highest (NiA(mox)MDC-0.022 kWh/m(3) >CMDC-0.019 kWh/m(3) >AnA(mox)MDC-0.0

213 CD11c(+)CD14(-) lineage-negative (MDC-enriched) LAF cells were isolated and shown to be mu

214 Interestingly, MDDCs but not MDCs were protected against IAV infection after LPS (TLR

215 DC from HCV-infected persons, the ability of MDC to activate naive CD4 T cells in the presence or abs

216 Data were obtained from 163 cases of MDC and 6214 cases of non-Meckelian ileal cancer, betwee

217 e report here that the disintegrin domain of MDC-L is recognized by the leukocyte integrin alpha(4)be

218 roteins possessing the disintegrin domain of MDC-L supported adhesion of the T-lymphoma cell line, Ju

219 of Jurkat cells to the disintegrin domain of MDC-L was inhibited by an anti-MDC-L monoclonal antibody

220 N in the WKY rat, and whether the effects of MDC were dependent on its receptor CCR4.

221 mice resulting from transgenic expression of MDC resulted in acceleration of clinical disease.

222 the absolute number and median frequency of MDC in HIV-infected individuals were similar to those ob

223 Blocking the function of MDC did not affect the developing of the disease from da

224 Furthermore, in vitro HIV infection of MDC from normal subjects cultured with cocaine and/or HI

225 The interaction of MDC-L with alpha(4)beta(1) may potentially regulate meta

226 lucrin levels accumulated in the presence of MDC.

227 ia, thereby identifying the opposing role of MDC TNFR1 in autoimmunity and bacterial infection.

228 secreted cytokines and chemokines typical of MDC upon stimulation with a TLR-4 agonist and both subse

229 atical model to simulate dynamic behavior of MDCs for the first time through evaluating multiple fact

230 R-mitochondria contacts in the biogenesis of MDCs.

231 data highlight the functional capacities of MDCs and PDCs in naive as well as healthy, infected maca

232 wer MDCs than PDCs, and the distributions of MDCs and PDCs were similar in pediatric control skin sam

233 Formation of MDCs requires the import receptors Tom70/71, and failure

234 ty and thus has highlighted the potential of MDCs as an energy-efficient technology to address water-

235 re was increased CD83 and CD86 expression on MDC from HCV-infected persons, the ability of MDC to act

236 GM3 on virus particles and CD169/Siglec-1 on MDCs.

237 Therefore, upregulation of B7-H1 on MDCs in the tumor microenvironment downregulates T-cell

238 rontal midline node of the cingulo-opercular MDC affected learning rates specifically during the init

239 into a mitochondrial-derived compartment, or MDC, followed by release through mitochondrial fission a

240 n with recombinant RABV expressing GM-CSF or MDC protected significantly more mice against intracereb

241 ibody blocked aggregation induced by TARC or MDC.

242 Optimally TLR ligand-stimulated PDCs or MDCs exposed to CMV or HIV-1 Ags enhanced autologous CMV

243 nsduced dendritic cells (DCs) overexpressing MDC would enhance the T cell-mediated humoral immune res

244 r malignancies and in 13% of these patients, MDC was the first malignancy.

245 Combined decline rate of KLK-7, PEDF, MDC and ANGPTL4 by Week 16 represented biomarkers for th

246 ma production induced by AT-2 SIV-presenting MDC/PDC-enriched mixtures.

247 st, stimulation with TLR7/8 ligand protected MDCs but not MDDCs from IAV infection.

248 esent the crystal structure of a Pseudomonas MDC and give insights into its catalytic mechanism and f

249 ther investigated the binding of recombinant MDC-L disintegrin domain (rDis-Fc) in solution.

250 mutants exhibit distinct abilities to rescue MDC formation in ERMES-depleted strains and are incapabl

251 pleted strains and are incapable of rescuing MDC formation in cells lacking Gem1.

252 ons were observed in the replication sample (MDC).

253 al data from both a bench- and a large-scale MDC system.

254 on the nature of hepatitis; the data specify MDCs as the essential cell type of TNFR1 function in T c

255 iduals from the Malmo Diet and Cancer study (MDC) into baseline cystatin C quintiles (n=4757).

256 n=1015) and the Malmo Diet and Cancer Study (MDC; n=746).

257 TARC, MDC, and SDF-1 increased intracellular calcium concentra

258 d 559 serum samples were evaluated for TARC, MDC, IL-10, and sCD163 by immunoassay.

259 After therapy TARC, MDC, and IL-10 correlated with PFS and overall survival

260 latory T cells (Tregs) that express the TARC/MDC-specific chemokine receptor CCR4, thus generating an

261 microstructure-designed direct-current TENG (MDC-TENG) with rationally patterned electrode structure

262 a TLR-7 agonist by secreting IFN-alpha than MDC-enriched cells.

263 an allogeneic mixed lymphocyte response than MDC matured without this inhibitor.

264 10-fold more proinflammatory cytokines than MDCs upon ex vivo stimulation.

265 and male human participants to confirm that MDC was most active in the initial stages of learning a

266 This observation demonstrated that MDC activity is important for learning.

267 In this study, we also found that MDC activity on M omicron/M phi in this GN was at least

268 rating characteristic analysis revealed that MDC:PDC ratio more than or equal to 1.52 was associated

269 These results suggest that MDC is critically involved in the development of anti-GB

270 al of malignant T cells and demonstrate that MDCs are abundant within the tumor microenvironment of T

271 Here, we show that MDCs are dynamic structures that form and stably persist

272 The MDC and TARC receptor CCR4 was expressed on platelets, a

273 The MDC generated higher current with higher salt concentrat

274 The MDC model will be helpful with determining operational p

275 The MDC-TENG realizes both the miniaturized device and high

276 y was achieved for phantom inserts above the MDC of 1 MBq/mL.

277 dding from the plasma membrane to access the MDC-dependent trans infection pathway.

278 l bias based on measurements found below the MDC were recorded.

279 ongitudinal and cross-sectional cohorts, the MDC:PDC ratio was higher and was associated with decreas

280 ermore, optimal operating conditions for the MDC used in this study were estimated to have an acetate

281 When fed with 4 M NaCl, the MDC achieve a current density of 300 A m(-3) (anode volu

282 e inverse association with viral load of the MDC number, but not of IFN-alpha secretion or the number

283 It was able to predict the response of the MDC with time under various conditions, and also provide

284 Additionally, recognition of the MDC-L disintegrin domain required "activation" of lympho

285 pression of the glomerular expression of the MDC/CCL22 chemokine and down-regulation of MIP-1alpha/CC

286 tochondrial dysfunction, suggesting that the MDC pathway provides protection to mitochondria in times

287 Thus, the MDC-TENG achieves a record high charge density of ~5.4 m

288 opose adding "full vertical motility" to the MDC for Moebius syndrome.

289 Adhesion of Jurkat cells to the MDC-L disintegrin domain was specifically inhibited by a

290 HIV-1 exposure of the MDCs and PDCs did not inhibit their ability to present c

291 In addition, an antagonist to MDC may represent a prime drug target for therapeutic ap

292 rthermore, adhesion of various cell lines to MDC-L correlated with expression of the integrin alpha(4

293 ide evidence that noninvasive stimulation to MDC nodes can enhance learning rates, thereby demonstrat

294 Herein, a tubular MDC was operated under a wide range of salt concentratio

295 pression of TLR4, TRIF, and MyD88 in the two MDC subsets regulated the ability of the cells to enter

296 ncoding gp120 either alone or as fusion with MDC.

297 Treatment with MDC or TGase-1 siRNA decreased Stat-3 but not Akt phosph

298 rmines the ability of HIV-1 to interact with MDCs.

299 resent study demonstrated that a node within MDC, located in midline frontal cortex, becomes active d

300 these cells in the presence of NS-398 yields MDC that stimulate significantly more IFN-gamma in an al