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1 MDF during trophic transfer of MeHg leading to enrichmen
2 MDF encodes a putative RS domain protein with a predicte
3 MDF expression is modulated by osmotic and cold stress,
4 MDF expression is not defective in the bodenlos, pin1 or
5 MDF identifies patients at risk of relapse and poor outc
6 MDF is required for the correct splicing and expression
7 all ions that fall outside of the GSH adduct MDF template windows, the processed full scan MS chromat
10 Moreover, the sensitivities with FIGS and MDF were equal for all As species, allowing for the poss
12 249 patients were prospectively evaluated by MDF for RD, and presence of RD was correlated with disea
14 er abiotic reduction pathways, and combining MDF with the observed MIF allows the distinction from ph
17 -1 or a second spindle checkpoint component, MDF-2, failed to arrest the cell cycle, exhibited chromo
19 but the mass dependent isotope compositions (MDF; delta(202)Hg) were not (r(2) = 0.26, p = 0.16), ref
20 ase (RD) by multidimensional flow cytometry (MDF) in children treated on Children's Oncology Group AM
21 measured by multidimensional flow cytometry (MDF) is a key early prognostic indicator and is strongly
23 tions in the Arabidopsis MERISTEM-DEFECTIVE (MDF) gene lead to a loss of stem cell and meristematic a
24 Measurements of Hg isotopic mass-dependent (MDF) and mass-independent fractionation (MIF) in food we
25 indicating lower degrees of mass-dependent (MDF) and mass-independent Hg fractionation (MIF) (respec
26 the wear rate of sample after the different MDF pass numbers using the corresponding hardness magnit
27 appearance of myogenic determination factor (MDF) transcripts in developing chick limbs and other emb
29 of a single member of the MyoD gene family (MDF) is necessary and sufficient to establish the positi
30 raphene behave like massless Dirac fermions (MDFs) with linear energy-momentum dispersion (1, 2) , pr
31 as designed to apply the mass defect filter (MDF) approach to the screening and identification of rea
33 four functions: Marker Dependency Filtering (MDF) to correct for known dependency between omics marke
35 Development of microbiota-directed foods (MDFs) that selectively increase the abundance of benefic
39 y-derived metric (the Max-min Driving Force, MDF), which enables objective ranking of pathways by the
40 by application of multi-directional forging (MDF) as a well-known severe plastic deformation method.
43 ish Hg isotope mass-dependent fractionation (MDF) during biotic methylation (-1.20 to +0.58 per thous
44 leads to both mass-dependent fractionation (MDF) of Hg isotopes and mass-independent fractionation (
45 this is due to mass-dependent fractionation (MDF) of up to -0.9 per thousand between IHg and MMHg.
46 ve equilibrium mass-dependent fractionation (MDF) with enrichment of heavier isotopes in the oxidized
47 sampling, the mass dependent fractionation (MDF, delta(202)Hg) of GEM taken up by the PAS was lower
48 atively narrow mass-dependent fractionation (MDF, delta(202)Hg; +/- 0.08 per thousand, 2SD) ranges (-
49 ependent and mass-independent fractionation (MDF and MIF) of methylmercury (MeHg) during trophic tran
50 with calculation of mean dominant frequency (MDF) and relative power of delta, theta, alpha and beta
51 e [MELD] >20, Maddrey discriminant function [MDF] >32) were randomized to receive methylprednisolone
54 und that Hg mass dependent fractionation (Hg-MDF) values in sediments mostly reflect a mixing between
58 cipitation, have determined which individual MDFs reside at promoters of several receptor subunit gen
59 PGC arrest by two mechanisms, one involving MDF-2 and another that is independent of other SAC compo
60 West-Haven criteria) and various MMSE items, MDF showed no correlation with any of MMSE items as well
61 Fe(II) in open systems results in a kinetic MDF with a larger epsilon compared to other abiotic redu
62 te checkpoint mechanism in which a core Mad1(MDF-1)/Mad2(MDF-2) signal generated at kinetochores is i
63 s orchestrate the integration of a core Mad1(MDF-1)/Mad2(MDF-2)-based signal, with a largely independ
64 tically and physically with SAC protein MAD1/MDF-1, whose nuclear envelope accumulation requires NPP-
65 t mechanism in which a core Mad1(MDF-1)/Mad2(MDF-2) signal generated at kinetochores is integrated wi
66 e the integration of a core Mad1(MDF-1)/Mad2(MDF-2)-based signal, with a largely independent Mad3(SAN
67 mes from the fact that subtly elevating Mad2(MDF-2) levels bypasses the requirement for BUB-3 and Mad
70 Expression of a non-phosphorylatable mutant MDF-1 partially suppressed the defect in the starvation-
75 lt2 double mutants have unaffected levels of MDF RNA, indicating that MDF acts upstream of PIN and PL
76 y defined as containing background levels of MDF transcripts which are thought to be nonfunctional.
82 nowledge of both the bioactive components of MDFs and the mechanisms underlying microbe-microbe inter
86 n of stable oxidative metabolites with other MDF templates, and determination of metabolite molecular
87 This result is in contrast to net positive MDF (+0.4 to +0.8 per thousand) previously observed in l
89 mitotic delay and localizes the SAC protein MDF-1/MAD1 to the kinetochore facing away from the spind
90 s showed very similar Hg isotope signatures (MDF delta(202)Hg: -0.2 per thousand to -0.5 per thousand
91 ergence of a Dirac band structure supporting MDFs has been observed in AG using molecular (5) , atomi
93 aused by mdf-1 hemizygosity, suggesting that MDF-1 causes the PGC arrest by two mechanisms, one invol
94 in L1 larvae lacking DAF-18, suggesting that MDF-1 regulates germ cell proliferation as a downstream
99 uggests a unique pathway responsible for the MDF of Hg isotopes during methylation by this strain reg
100 se results demonstrate a requirement for the MDF-dependent pathway in regulating PIN/PLT- and WUS/CLV
101 scan by a triple quadrupole instrument, the MDF approach was more sensitive and selective in screeni
102 The GSH adduct screening capability of the MDF approach, together with the utility of accurate mass