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1                                              MDF during trophic transfer of MeHg leading to enrichmen
2                                              MDF encodes a putative RS domain protein with a predicte
3                                              MDF expression is modulated by osmotic and cold stress,
4                                              MDF expression is not defective in the bodenlos, pin1 or
5                                              MDF identifies patients at risk of relapse and poor outc
6                                              MDF is required for the correct splicing and expression
7 all ions that fall outside of the GSH adduct MDF template windows, the processed full scan MS chromat
8 ither uniquely by one MDF or randomly by all MDFs.
9                                  We analyzed MDF of end-induction bone marrow samples from a historic
10    Moreover, the sensitivities with FIGS and MDF were equal for all As species, allowing for the poss
11                              In general, any MDF protein that is expressed also is present on transcr
12 249 patients were prospectively evaluated by MDF for RD, and presence of RD was correlated with disea
13 of target genes correlates with occupancy by MDF, in particular, herculin.
14 er abiotic reduction pathways, and combining MDF with the observed MIF allows the distinction from ph
15 y, the epsilon subunit gene becomes a common MDF target and begins to be expressed.
16  spindle assembly checkpoint (SAC) component MDF-1/MAD1 is required for the PGC arrest.
17 -1 or a second spindle checkpoint component, MDF-2, failed to arrest the cell cycle, exhibited chromo
18 ffects its binding to another SAC component, MDF-2/MAD2.
19 but the mass dependent isotope compositions (MDF; delta(202)Hg) were not (r(2) = 0.26, p = 0.16), ref
20 ase (RD) by multidimensional flow cytometry (MDF) in children treated on Children's Oncology Group AM
21 measured by multidimensional flow cytometry (MDF) is a key early prognostic indicator and is strongly
22                      The MERISTEM-DEFECTIVE (MDF) Arabidopsis gene encodes an SR-related family prote
23 tions in the Arabidopsis MERISTEM-DEFECTIVE (MDF) gene lead to a loss of stem cell and meristematic a
24  Measurements of Hg isotopic mass-dependent (MDF) and mass-independent fractionation (MIF) in food we
25  indicating lower degrees of mass-dependent (MDF) and mass-independent Hg fractionation (MIF) (respec
26  the wear rate of sample after the different MDF pass numbers using the corresponding hardness magnit
27 appearance of myogenic determination factor (MDF) transcripts in developing chick limbs and other emb
28          The myogenic determination factors (MDFs) are transcriptional activators that target E boxes
29  of a single member of the MyoD gene family (MDF) is necessary and sufficient to establish the positi
30 raphene behave like massless Dirac fermions (MDFs) with linear energy-momentum dispersion (1, 2) , pr
31 as designed to apply the mass defect filter (MDF) approach to the screening and identification of rea
32 ates were filtered by mass defect filtering (MDF) and multiple-group comparison.
33 four functions: Marker Dependency Filtering (MDF) to correct for known dependency between omics marke
34 red to a standard miniature diffusion flame (MDF) atomizer.
35    Development of microbiota-directed foods (MDFs) that selectively increase the abundance of benefic
36          The measured enrichment factors for MDF and MIF (epsilon(202)Hg and E(199)Hg) ranged from 1.
37 haracterization of GEM (+/-0.3 per mille for MDF, +/-0.05 per mille for MIF, 2SD).
38 patterning, as splicing targets required for MDF function in the meristem.
39 y-derived metric (the Max-min Driving Force, MDF), which enables objective ranking of pathways by the
40 by application of multi-directional forging (MDF) as a well-known severe plastic deformation method.
41                Mass-dependent fractionation (MDF) and mass-independent fractionation (MIF) may cause
42                Mass-dependent fractionation (MDF) and mass-independent fractionation (MIF) of Hg isot
43 ish Hg isotope mass-dependent fractionation (MDF) during biotic methylation (-1.20 to +0.58 per thous
44  leads to both mass-dependent fractionation (MDF) of Hg isotopes and mass-independent fractionation (
45 this is due to mass-dependent fractionation (MDF) of up to -0.9 per thousand between IHg and MMHg.
46 ve equilibrium mass-dependent fractionation (MDF) with enrichment of heavier isotopes in the oxidized
47  sampling, the mass dependent fractionation (MDF, delta(202)Hg) of GEM taken up by the PAS was lower
48 atively narrow mass-dependent fractionation (MDF, delta(202)Hg; +/- 0.08 per thousand, 2SD) ranges (-
49 ependent and mass-independent fractionation (MDF and MIF) of methylmercury (MeHg) during trophic tran
50 with calculation of mean dominant frequency (MDF) and relative power of delta, theta, alpha and beta
51 e [MELD] >20, Maddrey discriminant function [MDF] >32) were randomized to receive methylprednisolone
52 gic complete remission at EOI1, 46 (24%) had MDF-detectable disease.
53              The difference in delta(202)Hg (MDF) and Delta(199)Hg (MIF) between fish tissues and foo
54 und that Hg mass dependent fractionation (Hg-MDF) values in sediments mostly reflect a mixing between
55              Seston presents intermediate Hg-MDF and odd Hg-MIF values falling between sediments and
56 dness and wear rate are improved by imposing MDF process.
57                          Large variations in MDF and MIF are observed in fish and provide new insight
58 cipitation, have determined which individual MDFs reside at promoters of several receptor subunit gen
59  PGC arrest by two mechanisms, one involving MDF-2 and another that is independent of other SAC compo
60 West-Haven criteria) and various MMSE items, MDF showed no correlation with any of MMSE items as well
61  Fe(II) in open systems results in a kinetic MDF with a larger epsilon compared to other abiotic redu
62 te checkpoint mechanism in which a core Mad1(MDF-1)/Mad2(MDF-2) signal generated at kinetochores is i
63 s orchestrate the integration of a core Mad1(MDF-1)/Mad2(MDF-2)-based signal, with a largely independ
64 tically and physically with SAC protein MAD1/MDF-1, whose nuclear envelope accumulation requires NPP-
65 t mechanism in which a core Mad1(MDF-1)/Mad2(MDF-2) signal generated at kinetochores is integrated wi
66 e the integration of a core Mad1(MDF-1)/Mad2(MDF-2)-based signal, with a largely independent Mad3(SAN
67 mes from the fact that subtly elevating Mad2(MDF-2) levels bypasses the requirement for BUB-3 and Mad
68 etochore localization is independent of Mad2(MDF-2).
69 uence on the Hg isotope composition of MMHg (MDF) in sediments and aquatic organisms.
70  Expression of a non-phosphorylatable mutant MDF-1 partially suppressed the defect in the starvation-
71         The poplar leaves exhibited negative MDF (-3.18 to -1.22 per thousand) and small positive MIF
72                                          Non-MDF sample's surface shows spalling and delamination, wh
73     It is also shown that the application of MDF process changed the mechanism of wear.
74               We have analyzed expression of MDF and acetylcholine receptor subunits in cultured mous
75 lt2 double mutants have unaffected levels of MDF RNA, indicating that MDF acts upstream of PIN and PL
76 y defined as containing background levels of MDF transcripts which are thought to be nonfunctional.
77                                  The loss of MDF-2 or another SAC component also caused inappropriate
78                            Overexpression of MDF leads to the activation of markers of embryonic iden
79 PDL-1 functions in a kinetochore receptor of MDF-1/MAD1 to induce SAC function.
80 n Bacteroides and control the selectivity of MDF components.
81                The phosphorylation status of MDF-1 affects its binding to another SAC component, MDF-
82 nowledge of both the bioactive components of MDFs and the mechanisms underlying microbe-microbe inter
83 ty of states consistent with the presence of MDFs.
84 tified 2 Akt kinase phosphorylation sites on MDF-1.
85 ive gene is occupied, either uniquely by one MDF or randomly by all MDFs.
86 n of stable oxidative metabolites with other MDF templates, and determination of metabolite molecular
87   This result is in contrast to net positive MDF (+0.4 to +0.8 per thousand) previously observed in l
88 lecular events and interactions that precede MDF expression in myogenic precursor cells.
89  mitotic delay and localizes the SAC protein MDF-1/MAD1 to the kinetochore facing away from the spind
90 s showed very similar Hg isotope signatures (MDF delta(202)Hg: -0.2 per thousand to -0.5 per thousand
91 ergence of a Dirac band structure supporting MDFs has been observed in AG using molecular (5) , atomi
92 naffected levels of MDF RNA, indicating that MDF acts upstream of PIN and PLT gene expression.
93 aused by mdf-1 hemizygosity, suggesting that MDF-1 causes the PGC arrest by two mechanisms, one invol
94 in L1 larvae lacking DAF-18, suggesting that MDF-1 regulates germ cell proliferation as a downstream
95 on-of-function allele of mdf-1 suggests that MDF-1 has a dual role during development.
96                                          The MDF offset occurred more during the sorption of GEM rath
97                                  Because the MDF offset was consistent across field studies and labor
98 types, that is not directly addressed by the MDF model.
99 uggests a unique pathway responsible for the MDF of Hg isotopes during methylation by this strain reg
100 se results demonstrate a requirement for the MDF-dependent pathway in regulating PIN/PLT- and WUS/CLV
101  scan by a triple quadrupole instrument, the MDF approach was more sensitive and selective in screeni
102   The GSH adduct screening capability of the MDF approach, together with the utility of accurate mass
103 tion energy, suggesting the emergence of the MDF linear dispersion in the AG.
104  while expressing the Pax3 gene and prior to MDF gene activation.
105 llowed a mass-dependent fractionation trend (MDF; Delta(33)S <= +/-0.2 mUr).
106                  We propose a model in which MDF controls splicing in the root meristem to promote st
107                             Therefore, while MDF family members act positively during myogenic differ
108 n addition, SPDL-1 coimmunoprecipitates with MDF-1/MAD1 in vivo.
109                             Negative or zero MDF and MIF signatures are typical of geological Hg sour

 
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