ライフサイエンスコーパス: MFG

1 MFG from mammary gland epithelial cells (MEC) or raw mil

2 MFG was administered for 12 days starting 24 hours after

3 MFG were separated into six size groups (1-3 mum) from s

4 MFG-E8 (also termed lactadherin) and developmental endot

5 MFG-E8 also decreased in erbB2(+) human cancers and erbB

6 MFG-E8 and its homologue Del1 may represent relevant tar

7 MFG-E8 augmented melanoma cell resistance to apoptosis,

8 MFG-E8 bound to phosphatidylserine and triggered reorien

9 MFG-E8 can be developed as novel treatment for renal isc

10 MFG-E8 deficiency accelerated the onset of disease in a

11 MFG-E8 deficiency in mice led to the accumulation of une

12 MFG-E8 expression correlated significantly with fractalk

13 MFG-E8 gene expression was significantly decreased in WB

14 MFG-E8 mRNA and protein were increased in angiogenic isl

15 MFG-E8 was shown to attenuate the progression of inflamm

16 MFG-E8(-/-) mice displayed impaired efferocytosis associ

17 MFG-E8-deficient human melanoma cells also showed increa

18 MFG-E8-deficient RPE in primary culture retained normal

19 MFG-E8-mRNA was significantly overexpressed in CP and is

20 MFG-E8-producing myofibroblasts mainly originated from r

21 Intervention Patients participated in 1 of 2 MFGs (MFG-adherence or MFG-standard) or treatment as usu

22 or the expression of milk fat globule EGF 8 (MFG-E8) in antigen-presenting cells, and that MFG-E8-med

23 Here, we show that milk fat globule EGF-8 (MFG-E8), a secreted protein expressed at high levels in

24 regulation of milk fat globule-EGF factor 8 (MFG-E8) as a contributor to breast cancer progression us

25 Milk fat globule EGF factor 8 (MFG-E8) binds to apoptotic cells and facilitates their r

26 Milk fat globule- EGF factor 8 (MFG-E8) is a bridge protein that facilitates efferocytos

27 at globule epidermal growth factor-factor 8 (MFG-E8) is a peripheral glycoprotein that acts as a brid

28 at globule-epidermal growth factor factor 8 (MFG-E8) is expressed in several tissues and mediates div

29 Milk fat globule-EGF factor 8 (MFG-E8) maintains the intestinal homeostasis by enhancin

30 at globule epidermal growth factor-factor 8 (MFG-E8) was originally identified for phagocytosis of ap

31 t globule protein epidermal growth factor 8 (MFG-E8), increases 2.3-fold in abundance in old aorta.

32 geting factor milk fat globule-EGF factor 8 (MFG-E8), stimulated collagen uptake and degradation by a

33 rTK) and Milk fat globule EGF-like factor 8 (MFG-E8), were transiently up-regulated by macrophages/mi

34 milk fat globule-epidermal growth factor 8 (MFG-E8), which promotes apoptotic engulfment, and determ

35 llular ligand milk fat globule-EGF factor 8 (MFG-E8).

36 Milk fat globule-EGF factor 8 (MFG-E8)/lactadherin participates in several cell surface

37 nd its ligand milk fat globule EGF factor-8 (MFG-E8) but not the receptor tyrosine kinase MerTK.

38 Milk-fat globule EGF factor-8 (MFG-E8, SED1, lactadherin) is known to mediate the phago

39 Consistently, exogenously added MFG-E8 inhibited receptor activator of NF-kappaB ligand-

40 Administration of antibody against MFG-E8 significantly attenuated the increase in M2 polar

41 ntration pattern throughout lactation in all MFG size groups.

42 Mfge8 knock-out mice or by coinjection of an MFG-E8 receptor (VR) inhibitor into the rat striatum.

43 ed that p63 regulates MFG-E8 expression, and MFG-E8 knockdowns sensitized triple-negative breast canc

44 to-parietal attentional network, the IPS and MFG/IFG appear to be most heavily involved in attentive

45 functional connectivity between the PCC and MFG/vACC during a working memory task and at rest by exa

46 Our results indicate that the SC and MFG transcriptome are representative of MGT and LCMEC an

47 C strength between the dACC and both SFG and MFG were positively correlated (P = 0.012, and P = 0.007

48 he superior and middle frontal gyri (SFG and MFG) and insula, but higher zALFF in the occipital-tempo

49 ing MFG-E8 in mice by administration of anti-MFG-E8 antibody or targeted deletion of the MFG-E8 gene

50 Accordingly, local microinjection of anti-MFG-E8 mAb exacerbated periodontal bone loss in wild-typ

51 ranslation analysis demonstrated that aortic MFG-E8 mRNA and protein levels increase with aging in se

52 The data indicate that arterial MFG-E8 significantly increases with aging and is a pivot

53 SED1, also known as MFG-E8, is a secreted protein composed of two EGF repeat

54 tors such as c-mer and glycoproteins such as MFG-E8 were found to participate in the clearance of apo

55 Using a wound-healing assay, MFG-E8 was shown to promote the migration of intestinal

56 uman CD34 cells used the retroviral backbone MFG.

57 as a hemispheric asymmetry in the MDMR-based MFG findings, with literacy associated with the left MFG

58 um that was affected by Cx43 was found to be MFG-E8 (milk fat globule epidermal growth factor 8), whi

59 ce suggests an immunomodulatory link between MFG-E8 and the pro-inflammatory chemokine fractalkine, w

60 -1beta may moderate the relationship between MFG-related connectivity and depressive symptoms.

61 s uniquely activated for updating [bilateral MFG (BA 8) and left supramarginal gyrus (BA 39)], inhibi

62 Our data show that blocking MFG-E8-dependent phagocytosis preserves live neurons, im

63 ic bacteria like E. coli weren't affected by MFG size from both sources.

64 sufficient to fully restore phagocytosis by MFG-E8-deficient RPE.

65 hat osteoclasts express and are regulated by MFG-E8.

66 nversely dependent manner when stimulated by MFG-E8 and efferocytosis.

67 In human melanoma cells, MFG-E8 knockdown attenuated Akt and Twist signaling and

68 In conclusion, MFG-E8 is a novel homeostatic regulator of osteoclasts t

69 cting these cells with retroviral construct (MFG) expressing TGF-beta RII.

70 ressed the activity of a reporter containing MFG-E8 3'UTR.

71 By contrast, MFG displayed movement-related information during the se

72 In contrast, MFG-E8 expression was present at high levels in triple-n

73 In contrast, MFG-E8(-/-) mice that received wild-type bone marrow sho

74 gyrus and ventral anterior cingulate cortex (MFG/vACC).

75 Finally, reduced local efficiency of dACC/MFG during the task was significantly associated with an

76 in flexibility of local efficiency of R dACC/MFG significantly predicted inhibition performance, cons

77 ingulate cortex/medial frontal gyrus (R dACC/MFG).

78 Depleting MFG-E8 in mice by administration of anti-MFG-E8 antibody

79 Wound macrophage-derived MFG-E8 was recognized as a critical driver of wound angi

80 les supported these findings for MEC-derived MFG.

81 After detecting MFG-E8 mRNA in murine tumor cell lines, we sought to det

82 ncer cells increased efferocytosis, elevated MFG-E8 protein expression levels, and induced macrophage

83 nged cells with SNA-NC(anti-miR99b) enhanced MFG-E8 expression in the cells.

84 fronto-parietal regions, including IPS, FEF, MFG and IFG, in addition to regions in visual cortex.

85 Following MFG-E8-mediated engulfment of apoptotic cells, myofibrob

86 e isolated from CP tissues and evaluated for MFG-E8 mRNA expression after fractalkine stimulation.

87 that serum response factor is important for MFG-E8 production in myofibroblasts.

88 (n = 9) and 95% and 100%, respectively, for MFG (n = 19).

89 ese findings delineate pleiotropic roles for MFG-E8 in the tumor microenvironment and raise the possi

90 Less fractionated MFG regimens of every 48 and 72 hours are safe and as ef

91 ne marrow-derived osteoclast precursors from MFG-E8-deficient (Mfge8(-/-)) mice underwent increased r

92 tudied mice lacking expression of functional MFG-E8 to test the contribution of this integrin ligand

93 Furthermore, MFG-E8 supplementation strongly increased POS binding by

94 Mean field games (MFG) and mean field control (MFC) are critical classes o

95 potential/current, a microfluidic generator (MFG) is demonstrated using patterned micropillar arrays

96 mes to DCs is mediated via milk fat globule (MFG)-E8/lactadherin, CD11a, CD54, phosphatidylserine, an

97 sm underlying the shift in milk-fat-globule (MFG) mean diameter upon changing the concentrate-to-fora

98 epithelial cells (LCMEC), milk fat globules (MFG) and antibody-captured milk mammary epithelial cells

99 pid composition of bovine milk fat globules (MFG) and their membranes (MFGM) was investigated.

100 rmation about the role of milk fat globules (MFGs) in high-fat dairy systems, such as cheese, and con

101 Bulk RNA sequencing of milk fat globules (MFGs), milk cells, and breast tissue revealed that MFG-d

102 portal function is the bridging glycoprotein MFG-E8.

103 The glycoprotein MFG-E8 mediates phagocytic clearance of apoptotic cells

104 ttributed to increased secretion of F1-group MFG, while fat content and composition in the other MFG

105 cessus group (MCAG), the M. fortuitum group (MFG), and M. mucogenicum.

106 x, primarily within the middle frontal gyri (MFG).

107 e middle frontal and inferior temporal gyri (MFG and ITG) and resistant (cerebellum) to classical AD

108 sociation between left Middle Frontal Gyrus (MFG) activity during the vSAT task and the PANSS score d

109 eased rsFC in the left middle frontal gyrus (MFG) and bilateral inferior parietal lobe (IPL) of the D

110 cordings from the left middle frontal gyrus (MFG) and precentral gyrus (PCG) of a person with tetrapl

111 cycle within the left middle frontal gyrus (MFG) and the right prefrontal cortex.

112 nced expression in the middle frontal gyrus (MFG) and the subcallosal cingulate gyrus (SCG) showed en

113 iFC), highlighting the middle frontal gyrus (MFG) for both competencies.

114 rence and in the right middle frontal gyrus (MFG) for the double interference condition.

115 nt and the surrounding middle frontal gyrus (MFG) shows that both gyral and sulcal components of the

116 ncentration within the middle frontal gyrus (MFG) successfully classified whether an adolescent studi

117 f the anterior insula, middle frontal gyrus (MFG), and anterior cingulate cortex (ACC).

118 ontal eye field (FEF), middle frontal gyrus (MFG), and inferior frontal gyrus (IFG).

119 rontal junction (IFJ), middle frontal gyrus (MFG), inferior frontal gyrus, and intraparietal sulcus c

120 r frontal gyrus (SFG), middle frontal gyrus (MFG), LIP, anterior intraparietal sulcus (IPSa)] that ma

121 recentral gyrus (PcG), middle frontal gyrus (MFG), orbital frontal cortex, and two regions of inferio

122 of damage to the left middle frontal gyrus (MFG).

123 The right middle/inferior frontal gyrus (MFG/IFG), which is included in the FPCN, showed greater

124 mporal gyrus [ITG] and middle frontal gyrus [MFG]), we tested associations between brain tissue conce

125 The higher MFG mean diameter in whole raw LCHF milk might therefore

126 expressed histidine-tagged recombinant human MFG-E8 (rhMFG-E8) is protective in various disease condi

127 verified by SDS-PAGE with the standard human MFG-E8 loaded as control and, mass spectrometry followed

128 a retroviral vector expressing human apoA-I (MFG-HAI) had 95% lower atherosclerotic lesion area than

129 We first identified MFG-E8 downregulation in invasive lesions in transgenic

130 These results identify MFG-E8 as the first extracellular ligand in the retina t

131 tumor cell lines, we sought to determine if MFG-E8 influenced tumorigenesis in Rip1-Tag2 transgenic

132 nt stimuli in the frontal regions (ACG, IFG, MFG) than the control group.

133 A 60-hr survival study was conducted in MFG-E8 and recombinant murine MFG-E8-treated wild-type m

134 c islets and tumor burdens were decreased in MFG-E8-deficient Rip1-Tag2 mice compared with those in c

135 Mice deficient in MFG-E8 develop lupus-like autoimmunity associated with a

136 nic islets and tumors, were also detected in MFG-E8-deficient mice.

137 fractalkine led to a significant increase in MFG-E8 expression.

138 less likely to be hospitalized than those in MFG-standard (chi(2) = 8.2; P = .04) and treatment as us

139 carcinomas were modestly underrepresented in MFG-E8-deficient mice, but tumor frequencies and surviva

140 o advanced glycated end products inactivated MFG-E8, recognizing a key mechanism that complicates dia

141 on of extracellular growth factors including MFG-E8.

142 tidylserine-recognizing molecules, including MFG-E8, TIM-1, -3, and -4, CD300a, BAI1, and stabilin-1

143 n of MFG-E8 in CP, with noticeably increased MFG-E8 immunoreactivity in tubular complexes.

144 g aorta to angiotensin II markedly increases MFG-E8 and enhances invasive capacity to levels observed

145 east cancer progression and small inhibitory MFG-E8 RNAs accelerated ER(+) breast cancer cell prolife

146 After renal ischemia-reperfusion injury, MFG-E8 mRNA and protein expressions were significantly d

147 esults suggest that a decrease in intestinal MFG-E8 impairs intestinal mucosal repair in sepsis.

148 Moreover, in septic mice, intestinal MFG-E8 expression was downregulated, which correlated wi

149 ssociated with down-regulation of intestinal MFG-E8 and impairment of enterocyte migration.

150 on of SNA-NC(anti-miR99b) rescued intestinal MFG-E8 expression in LPS-induced septic mice and attenua

151 an cancers and erbB2 transgenic mice lacking MFG-E8 showed accelerated tumor formation.

152 mine concentration was constant in the large MFG, but dropped twofold in the small MFG.

153 abolome of the bacteria incubated with large MFG had reduced concentrations of metabolites important

154 ing left intraparietal sulcus (IPS) and left MFG/IFG.

155 task-set) to the right IFG/POp and the left MFG respectively.

156 In addition, the rsFC of the left MFG was inversely correlated with the Trail Making Test-

157 ings, with literacy associated with the left MFG, whereas numeracy associated with the right MFG (R.M

158 rrant activation differences within the left-MFG region may describe a dysregulation of attentional n

159 sMerTK release, whereas the integrin ligand MFG-E8 markedly increases both phagocytosis and sMerTK l

160 alphavbeta5 integrin receptor and its ligand MFG-E8, thus generating a phagocytic peak.

161 In a murine model of melanoma, MFG-E8 enhanced tumorigenicity and metastatic capacity t

162 ention Patients participated in 1 of 2 MFGs (MFG-adherence or MFG-standard) or treatment as usual.

163 of alternate dosing regimens of micafungin (MFG) for the treatment of experimental subacute dissemin

164 In wild-type mice, MFG-E8 attenuates the vaccination activity of GM-CSF-sec

165 In septic WT mice, MFG-E8 mRNA and protein levels were suppressed by 49 and

166 Moreover, MFG-E8 administration into infarcted hearts restored car

167 Moreover, MFG-E8 knockdown tumor cells cultured with wild-type or

168 tidylserine binding domain and an RGD motif, MFG-E8 helps target HIV-1 VLPs to alphav integrin bearin

169 Using ectopically expressed mouse MFG-E8, a secreted phosphatidylserine-binding protein, w

170 a microRNA that is predicted to target mouse MFG-E8 3'UTR.

171 A recombinant murine MFG-E8 (0.4 mug/20 g) was given intraperitoneally at the

172 Treatment with recombinant murine MFG-E8 recovered renal dysfunction, significantly suppre

173 Administration of recombinant murine MFG-E8 reduces neutrophil migration through upregulation

174 oration and, in contrast, recombinant murine MFG-E8-treated wild-type mice showed a significant impro

175 s conducted in MFG-E8 and recombinant murine MFG-E8-treated wild-type mice.

176 ng PS blocking antibodies, annexin V, mutant MFG-E8 unable to bind VR, or VR antagonist).

177 Mutated MFG-E8, which binds viral envelope phosphatidylserine wi

178 Treg induction, whereas a dominant-negative MFG-E8 mutant potentiates GM-CSF-stimulated tumor destru

179 tion was affected by lactation stage but not MFG size.

180 Absence of MFG-E8 also led to decreases in tumor vascular permeabil

181 chemistry analysis confirmed accumulation of MFG-E8 in CP, with noticeably increased MFG-E8 immunorea

182 y was to determine whether administration of MFG-E8 attenuates renal ischemia-reperfusion injury.

183 previously shown that the administration of MFG-E8-rich exosomes from immature dendritic cells promo

184 In situ analyses of MFG-E8 expression in estrogen receptor (ER) positive cas

185 the door to much-anticipated applications of MFG and MFC models that are beyond reach with existing n

186 Study groups consisted of MFG at 1 mg/kg every 24 hours (MFG1), 2 mg/kg every 48 h

187 r strengthened differential contributions of MFG connections to each competency.

188 ed enterocyte migration, whereas deletion of MFG-E8 impeded mucosal healing in mice with sepsis.

189 Mean diameter of MFG, determined in raw whole milk, was 0.4 mum larger in

190 ile retaining the single promoter feature of MFG responsible for high virus titer and enhanced protei

191 e studies clearly indicate the importance of MFG-E8 in ameliorating neutrophil infiltration and sugge

192 uggests the role of miR-99b in inhibition of MFG-E8 gene expression.

193 Lack of MFG-E8 only minimally reduced retinal adhesion.

194 Lack of MFG-E8 or integrin blockade severely limits HIV-1 VLP sp

195 In contrast, lack of MFG-E8, like lack of alphavbeta5 receptor, eliminated al

196 e cancer patients presented higher levels of MFG-E8 compared with controls, a novel finding in human

197 The expression and localization of MFG-E8 was investigated in CP (n=62), and normal pancrea

198 Conversely, microinjection of MFG-E8 inhibited bone loss in experimental mouse periodo

199 PK of MFG were determined on day 7 by high-performance liquid

200 PK of MFG were linear and the parameter means +/- SD were Vc =

201 ation of closure, upholding the potential of MFG-E8-directed therapeutics in diabetic wound care.

202 sis, we hypothesized that down-regulation of MFG-E8 is mediated via the LPS-CD14 pathway, eventually

203 dependent manner and the down-regulation of MFG-E8 mRNA expression in CLP-induced sepsis was attenua

204 esent in the gut, the physiological roles of MFG-E8 in the intestinal mucosa have not been explored.

205 Thus, targeting of MFG-E8 within this signaling axis pathway is a potential

206 Transplantation of MFG-E8(-/-) bone marrow to MFG-E8(+/+) mice resulted in

207 of 293GPG cells using a modified version of MFG.SnlsLacZ, in which the cytomegalovirus IE promoter w

208 ant change in the size and zeta-potential of MFGs.

209 articipated in 1 of 2 MFGs (MFG-adherence or MFG-standard) or treatment as usual.

210 oglia were genetically deficient in MerTK or MFG-E8, both of which mediate phosphatidylserine-recogni

211 e isolation of total RNA directly from SC or MFG released into milk during lactation.

212 kdown tumor cells cultured with wild-type or MFG-E8-deficient macrophages resulted in increased SOCS3

213 ile fat content and composition in the other MFG size groups remains unchanged.

214 hPSCs overexpress MFG-E8 upon fractalkine stimulation in vitro, which unde

215 work for the numerical solution of potential MFG and MFC models.

216 Using the proposed MFG to power a single nanowire-based pH sensor, a self-p

217 of medin amyloid (a fragment of the protein MFG-E8, also known as lactadherin) are found in the vasc

218 soluble phosphatidylserine-binding protein, MFG-E8, enhances transduction.

219 c deficiency of the Medin precursor protein, MFG-E8, eliminates not only vascular aggregates but also

220 athways, and is inhibited by blocking the PS/MFG-E8/VR pathway (by adding PS blocking antibodies, ann

221 n iFC profiles to both competencies (e.g., R.MFG with cerebellum).

222 as numeracy associated with the right MFG (R.MFG).

223 as that numeracy was negatively related to R.MFG connections with the default network, which has been

224 cultures, but restored by adding recombinant MFG-E8, without affecting inflammation.

225 Soluble or POS-bound recombinant MFG-E8 was sufficient to fully restore phagocytosis by M

226 mucosal healing and suggest that recombinant MFG-E8 may be beneficial for the treatment of bowel inju

227 Topical recombinant MFG-E8 induced resolution of wound inflammation, improve

228 Treatment with recombinant MFG-E8 restored enterocyte migration, whereas deletion o

229 Endotoxemia reduced MFG-E8 mRNA expression in a dose dependent manner and th

230 Silencing MFG-E8 RNA substantially reduces MFG-E8 expression and VSMCs invasion capacity.

231 eporter assays all showed that p63 regulates MFG-E8 expression, and MFG-E8 knockdowns sensitized trip

232 ively, our study suggests that LPS represses MFG-E8 expression and disrupts enterocyte migration via

233 nd F-actin recruitment via Rac1 both require MFG-E8-ligated alphavbeta5 integrin.

234 l nanoparticle-conjugate capable of rescuing MFG-E8 gene expression and maintaining intestinal epithe

235 all and large (2.3 and 7.0 um, respectively) MFG were isolated from cow milk and used as a substrate

236 present study, we have used the retrovirus, MFG-IRAP, to transfer the human IL-1 receptor antagonist

237 We therefore aimed to reveal MFG-E8 roles in regulating lung neutrophil infiltration

238 y using offline TMS over right IPS and right MFG.

239 f the DAN, as well as the left IPL and right MFG/IFG of the VAN.

240 Higher right MFG-mid-anterior cingulate cortex (mACC) connectivity wa

241 , whereas numeracy associated with the right MFG (R.MFG).

242 In contrast, perturbation of the right MFG had no effect on any of the conflict types.

243 right hemisphere frontal regions (FEF, SFG, MFG).

244 Silencing MFG-E8 RNA substantially reduces MFG-E8 expression and V

245 compositional regulation for large and small MFG at different lactation stages.

246 large MFG, but dropped twofold in the small MFG.

247 Bacteria incubated with small MFG had increased concentration of metabolites related t

248 between treatments were found in a specific MFG subgroup with the diameter of 3.3 mum (F1), with hig

249 Splenic MFG-E8 expression, phagocytic activity, and apoptosis we

250 In the survival study, MFG-E8 mice showed a significant deterioration and, in c

251 iorating neutrophil infiltration and suggest MFG-E8 as a novel therapeutic potential for ALI.

252 ment and raise the possibility that systemic MFG-E8 blockade might prove therapeutic for melanoma pat

253 ciated with higher medication adherence than MFG-standard or treatment as usual only (F = 6.41; P = .

254 FG-E8) in antigen-presenting cells, and that MFG-E8-mediated uptake of apoptotic cells is a key deter

255 However, we show here that MFG-E8 can mediate phagocytosis of viable neurons during

256 In this work, we test the hypothesis that MFG-E8 helps resolve inflammation, supports angiogenesis

257 This study investigated the hypothesis that MFG-E8-mediated efferocytosis promotes M2 polarization.

258 These experiments indicate that MFG activation is reliably evoked by exemplars from arbi

259 Together, our data indicate that MFG-E8 plays an important role in the maintenance of int

260 milk cells, and breast tissue revealed that MFG-derived RNA closely mirrors luminal milk cells.

261 Taken together, our results show that MFG-E8 is expressed in triple-negative breast cancers as

262 Here we show that MFG-E8 was expressed in intestinal lamina propria macrop

263 Furthermore, we showed that MFG GABA not only classifies whether an adolescent is st

264 Here, we have shown that MFG-E8 controls phagocytic ingestion of cell fragments a

265 We have recently shown that MFG-E8 treatment attenuates organ injury, inflammatory r

266 Dual immunolabeling shows that MFG-E8 colocalizes with both angiotensin II and monocyte

267 together, these novel findings suggest that MFG-E8 blockade may be a promising tool for future immun

268 These observations suggest that MFG-E8 deficiency promotes immune responses to self anti

269 udy, we demonstrated for the first time that MFG-E8 is significantly up-regulated in CP patients and

270 lying variational primal-dual structure that MFGs exhibit and phrase it as a convex-concave saddle-po

271 The MFG-adherence participants had a longer time to first ho

272 ted with AD when comparing the groups in the MFG and ITG, respectively: linoleic acid (p < 0.0001, p

273 e former novels now evoked activation in the MFG, but the former target circles did not.

274 that both gyral and sulcal components of the MFG have greater myelin content in deeper compared with

275 caudal/medial SFG and a small region of the MFG in a majority of the subjects analyzed.

276 -MFG-E8 antibody or targeted deletion of the MFG-E8 gene resulted in a slowing of enterocyte migratio

277 lored neural network parameterization of the MFG/MFC solution helps us avoid any spatial discretizati

278 sing the problem in this manner, solving the MFG can be interpreted as a special case of training a g

279 n of the 293GPG packaging cell line with the MFG.SnlsLacZ retroviral vector construct, it was possibl

280 This MFG activation did not differ as a function of the requi

281 The continuous electrical output makes this MFG particularly suitable as a power source in self-powe

282 Thus, MFG-E8 production is down-regulated in sepsis by LPS-CD1

283 Thus, MFG-E8 stimulates rhythmic POS phagocytosis by ligating

284 ransplantation of MFG-E8(-/-) bone marrow to MFG-E8(+/+) mice resulted in impaired wound closure and

285 At the end of the 1-year treatment, MFG-adherence was associated with higher medication adhe

286 Using MFG-based retrovirus vectors containing oxidase genes, w

287 The replication-defective retroviral vector MFG was used for GM-CSF gene transfer.

288 The retroviral vector MFG was used to transfer the G156A MGMT (deltaMGMT) cDNA

289 C were transduced with the retroviral vector MFG-enhanced green fluorescence protein as a marker gene

290 urine leukemia virus-based retroviral vector MFG-S encoding the human form of p47phox, we performed e

291 el, we have used a murine retroviral vector, MFG, that expresses the green fluorescent protein (GFP)

292 s uncover a unique role of efferocytosis via MFG-E8 as a mechanism for macrophage polarization into t

293 globule epidermal growth factor-factor VIII (MFG-E8) functions as a bridging molecule to promote the

294 e epidermal growth factor (EGF)-factor VIII (MFG-E8) mediates the clearance of apoptotic cells.

295 globule-epidermal growth factor-factor VIII (MFG-E8), a membrane-associated secretory glycoprotein, i

296 rrow cells infected with the parental virus (MFG).

297 , and green tea extract were added to washed MFGs to examine possible interactions.

298 However, whether MFG-E8 can regulate neutrophil function to alleviate inf

299 elated WM performance, FFA connectivity with MFG predicted LTM improvements.

300 Treatment of VSMCs with MFG-E8 increases MCP-1 expression and VSMCs invasion tha