ライフサイエンスコーパス: MRF

1 MRF can also be used to identify the presence of a speci

2 MRF could be used to measure both pre- and post-contrast

3 MRF has shown promise in improving the diagnosis of clin

4 MRF is a nuclear protein containing an evolutionarily co

5 MRF pulse sequences have thousands of tunable parameters

6 MRF T(1) and T(2) mapping was performed successfully in

7 MRF thus provides an alternative way to quantitatively d

8 MRFs included diabetes mellitus (DM), hypertension, hype

9 cific transcription factors, including HLH-1/MRF and UNC-120/SRF, which together orchestrate specific

10 In total, 171 MRF neurons that projected to the C5-C6 ventral horn wer

11 Among lower metabolic burden (<=2 MRFs), statin use was associated with a 15% lower risk o

12 mong those with higher metabolic burden (>=3 MRFs).

13 A total of 94 MRF neurons responsive to bilateral electrical stimulati

14 s, they require E protein partners to form a MRF-E protein heterodimer, which represents the function

15 Semi-automatic segmentation incorporating a MRF model was performed in all patients below the C4 ver

16 Additional MRF reconstructions using the first nine segments of the

17 n appropriate pattern-recognition algorithm, MRF inherently suppresses measurement errors and can thu

18 Although MRFs are essential for myogenic commitment and different

19 e structural motifs that are conserved among MRFs: an alanine-threonine (Ala-Thr) dipeptide of the ba

20 pharyngeal muscle fate both by promoting an MRF-associated myogenic program in myoblasts and by main

21 val tail muscle development and thus that an MRF-dependent myogenic regulatory network probably exist

22 In contrast, myf 5 and MRF 4 mRNAs are detected at significant levels in embryo

23 Myf 5 and MRF 4 transcripts are detected in stage 15 forelimbs, wh

24 A survey of MyoD, myf 5, and MRF 4 transcripts in other embryonic tissues reveals tha

25 roduct required for normal development), and MRF-2 (which represses expression from the cytomegalovir

26 technique (FF(Dixon)), water T2 mapping, and MRF T1-FF, from which the FF measured with MRF T1-FF (FF

27 lix family (bHLH) (myoD, myf-5, myogenin and MRF-4) to induce myogenic differentiation.

28 with a high percentage in RRN, PoO, PoC, and MRF.

29 ed by Hes/Hey downstream of Notch as well as MRF activities are integrated at the level of the p57(ki

30 troponin I reporter gene activity as well as MRF-directed transcription from a multimerized skeletal

31 and aldolase, but also myoD and myogenin as MRFs.

32 est-retest variability and agreement between MRF and HFA-SFr were analyzed using Bland-Altman plots a

33 Test-retest variability between MRF tests found a bias of -0.18 dB and 95% limits of agr

34 ion using a Uni-ZAPTM XR vector and XL1-Blue MRF' host.

35 In addition to the anonymization afforded by MRF, this format also facilitates the decoupling of the

36 The methods used by MRF-MGL efficiently characterised IMD isolates and infor

37 y element of p57(kip2) directly activated by MRFs in myoblasts but repressed by the Notch targets Hes

38 opagate and amplify the signals initiated by MRFs.

39 Deep-learning-aided CEST-MRF may also be amenable to the characterization of host

40 sfer magnetic resonance fingerprinting (CEST-MRF) aided by deep learning.

41 ulated magnetic resonance fingerprints, CEST-MRF can generate quantitative maps of intratumoural pH a

42 also show that in a healthy volunteer, CEST-MRF yielded molecular parameters that are in good agreem

43 Ci-MRF is the sole myogenic regulatory factor (MRF) of the

44 Conversely, inhibiting Ci-MRF activity with antisense morpholinos down-regulated t

45 invertebrate chordate Ciona intestinalis (Ci-MRF).

46 mple in vivo assay based on misexpressing Ci-MRF in the notochord of Ciona embryos.

47 Ala-Thr dipeptide is necessary for normal Ci-MRF function, and that while eliminating the C/H domain

48 ity and revealed the myogenic activity of Ci-MRF by inducing the expression of four muscle marker gen

49 Injection of Ci-MRF mRNA into eggs resulted in increased embryonic muscl

50 ndividually has no demonstrable effect on Ci-MRF, simultaneous loss of both motifs significantly redu

51 We conclude that Ci-MRF is required for larval tail muscle development and t

52 Within the CNS, MRF is specifically expressed by postmitotic oligodendro

53 Consequently, MRF-MRF comparison for remote homology detection shall b

54 e the function of each of the four conserved MRF genes in zebrafish, an organism that has retained a

55 model very short-range residue correlation, MRFs can model long-range residue interaction pattern an

56 st - Magnetic Resonance Fingerprinting (DC - MRF) methodology is described that can detect and indepe

57 nts from both single agent and dual agent DC-MRF studies demonstrated significant correlations with e

58 rast - Magnetic Resonance Fingerprinting (DC-MRF) methodology was extended for in vivo application an

59 vivo study demonstrates the potential for DC-MRF to provide a useful dual-agent MRI platform.

60 Ca, including its dynamic analysis as in DCE-MRF.

61 Deep MRF provides solutions to these challenges by providing

62 ol for quantitative molecular MRI using deep MRF.

63 Different MRF neuronal groups were found to respond to locomotion,

64 of muscle determination and differentiation (MRFs) together with two candidate markers of satellite c

65 genes on the networks using a local discrete MRF model.

66 n to a skeletal muscle lineage by disrupting MRF function via a mechanism that is independent of the

67 strate differential requirements of distinct MRFs for the induction of microRNA gene expression durin

68 ty that the regulative behavior of distinct, MRF-expressing populations explains the functional compe

69 gest that the differential use of duplicated MRF paralogues in this novel two-component myogenic syst

70 atlas segmentation methods like SEGMA and EM-MRF with multiple atlases, our method improved accuracy

71 ctation maximization-Markov random field (EM-MRF) method.

72 Hh is known to enhance MRF expression.

73 oes not alter the ability of CKII to enhance MRF transcriptional activity, suggesting that the effect

74 These findings establish MRF as a critical transcriptional regulator essential fo

75 of a mixed waste material recovery facility (MRF), anaerobic digestion, and waste-to-energy combustio

76 ting muscle cells, muscle regulatory factor (MRF) 4 is normally the last of the four MRFs to be expre

77 The activity of myogenic regulatory factor (MRF) genes is essential for vertebrate muscle developmen

78 the first of the myogenic regulatory factor (MRF) genes to be activated in preexisting somites in a r

79 Myogenic regulatory factor (MRF) genes, MYOD1, MYOG, MYF6 and MYF5, are critical for

80 iption factor myelin gene regulatory factor (MRF) is required to maintain the integrity of myelin in

81 The myogenic regulatory factor (MRF) MYF5 is the earliest to be expressed during myogene

82 -MRF is the sole myogenic regulatory factor (MRF) of the ascidian Ciona intestinalis, an invertebrate

83 we have named myelin gene regulatory factor (MRF), as a transcriptional regulator required for CNS my

84 grams that require muscle regulatory factor (MRF)-family genes.

85 tified host factors [Mu replication factors (MRF alpha 2)], which displace the transpososome in an AT

86 The myogenic regulatory factors (MRFs) are a subclass of a much larger group of basic hel

87 lated family of myogenic regulatory factors (MRFs) are expressed during somitogenesis although cells

88 Myogenic regulatory factors (MRFs) are required for mammalian skeletal myogenesis.

89 Myogenic regulatory factors (MRFs) are vital transcription factors that act at multip

90 ssion levels of myogenic regulatory factors (MRFs) between fast- versus slow-growing yellow perch Per

91 e expression of myogenic regulatory factors (MRFs) myf5 and myod in specific muscle precursor cell po

92 g the activity of muscle regulatory factors (MRFs) of the Myod family above a threshold.

93 tch pathway and myogenic regulatory factors (MRFs) orchestrate the proliferation, specification and d

94 in vertebrates myogenic regulatory factors (MRFs) such as MyoD1 alone are required and sufficient fo

95 Myogenic regulatory factors (MRFs), including Myf5, MyoD (Myod1) and Myog, are muscle

96 Myogenic regulatory factors (MRFs), muscle-specific transcription factors, are implic

97 rganized by the myogenic regulatory factors (MRFs), Myf5, MyoD, Myf6, and myogenin, where myogenin pl

98 helix-loop-helix muscle regulatory factors (MRFs), such as MyoD, to convert nonmuscle cells to a myo

99 in are dominant myogenic regulatory factors (MRFs), which are involved in control of muscle-specific

100 gets of the key myogenic regulatory factors (MRFs)--MyoD and myogenin--and Myocyte Enhancer Factor 2

101 s a regulation by muscle regulatory factors (MRFs).

102 he MyoD family of muscle regulatory factors (MRFs).

103 etween CTCF and myogenic regulatory factors (MRFs).

104 rs known as the myogenic regulatory factors (MRFs): Myf5, Mrf4, myogenin and MyoD.

105 n, and the four myogenic regulatory factors (MRFs.) Most mutant satellite cells entered the cell cycl

106 B proteins, the myogenic regulatory factors (MRFs: MyoD, myogenin, Myf-5 and MRF4/Myf-6), and the hem

107 op-helix (bHLH) myogenic regulatory factors (MRFs; MyoD, Myf5, myogenin and MRF4) and Pax3, a paired-

108 f myogenic regulatory transcription factors (MRFs) are well known to govern lineage commitment and di

109 rs of the myogenic regulatory factor family (MRFs) and on extrinsic cellular cues, including Wnt sign

110 genic regulatory factors of the Myod family (MRFs) are transcription factors essential for mammalian

111 genic regulatory factors of the myod family (MRFs) are transcription factors essential for mammalian

112 A linear correlation was observed between FF(MRF) and FF(Dixon) (R(2) = 0.97, P < .001).

113 rom which the FF measured with MRF T1-FF (FF(MRF)) and water T1 were derived.

114 s embryo, we show that CeMyoD is a bona fide MRF that can convert almost all cells to a muscle-like f

115 icle, we propose a methylation random field (MRF) method to detect mQTLs by testing the association b

116 eq, which is based on a Markov random field (MRF) model to appropriately accommodate gene network inf

117 ted MRI (WBDWI) using a Markov random field (MRF) model to derive tumor total diffusion volume (tDV)

118 nt algorithm based on a Markov random field (MRF) model, called MRFSeq, that uses additional gene coe

119 datasets by defining a Markov random field (MRF) over super-voxels in the foreground and applying mo

120 We use a Markov random field (MRF) prior to map the network connections among genes.

121 s article, we develop a Markov random field (MRF)-based method for identifying genes and subnetworks

122 d visual field test (Melbourne Rapid Field, (MRF)) conducted at the bedside aided swift and appropria

123 e key components: 1) a Markov Random Fields (MRF) representation of a protein family; 2) a scoring fu

124 The tablet-based Melbourne Rapid Fields (MRF) visual field (VF) test and the IMOvifa Smart Visual

125 n be fully captured by Markov random fields (MRFs).

126 Background MR fingerprinting (MRF) provides rapid and simultaneous quantification of m

127 Magnetic resonance fingerprinting (MRF) is a method to extract quantitative tissue properti

128 Magnetic resonance fingerprinting (MRF) is a rapidly developing fast quantitative mapping t

129 transfer magnetic resonance fingerprinting (MRF) is an emerging approach for noninvasive in vivo ima

130 we term 'magnetic resonance fingerprinting' (MRF)--that permits the simultaneous non-invasive quantif

131 During mouse development Myf5 is the first MRF to be expressed and it acts by integrating multiple

132 f MRF, indicating an ongoing requirement for MRF in the expression of these genes.

133 Test-retest for MRF also showed strong correlation for MD (ICC = 0.983)

134 s, we have developed the Mapped Read Format (MRF), a compact data summary format for both short and l

135 ivered to the medullary reticular formation (MRF) by diffusion from a cannula inserted through a guid

136 The mesencephalic reticular formation (MRF) is formed by the pedunculopontine and cuneiform nuc

137 ile inputs to medullary reticular formation (MRF) neurons after acute and chronic dorsal column (DC)

138 s onto single medullary reticular formation (MRF) neurons.

139 of the medial medullary reticular formation (MRF) participate in generating vestibulo-respiratory res

140 in the medial medullary reticular formation (MRF) provide inputs to phrenic and abdominal motoneurons

141 e lateral mesencephalic reticular formation (MRF) that in turn project to the nucleus reticularis pon

142 ts of the mesencephalic reticular formation (MRF), namely the pedunculopontine and cuneiform nuclei.

143 silateral mesencephalic reticular formation (MRF), periaqueductal gray, Kolliker-Fuse nucleus, and po

144 r part of the medullary reticular formation (MRF).

145 and many cells ultimately expressed all four MRFs simultaneously.

146 whereas Id3 interacted weakly with all four MRFs.

147 tor (MRF) 4 is normally the last of the four MRFs to be expressed.

148 s investigating the added value of post-GBCA MRF in PCa, including its dynamic analysis as in DCE-MRF

149 m density-weighted from simultaneous 3D (1)H MRF/(23)Na MRI in the brain at 7 T showed high repeatabi

150 ification measured with simultaneous 3D (1)H MRF/(23)Na MRI in the brain at 7 T, from ten healthy vol

151 ematic errors dominate over random errors in MRF scans under clinically relevant conditions of high u

152 tudy, we sought to evaluate the variation in MRF T(1) measurements post gadolinium-based contrast age

153 ptide of the basic domain of an invertebrate MRF behaves as a myogenic code.

154 Purpose To evaluate a rapid kidney MRF technique at 3.0 T in phantoms, healthy volunteers,

155 Repeat kidney MRF scans for the three patients with ARPKD on successiv

156 Materials and Methods A 15-second kidney MRF acquisition was designed with 12 acquisition segment

157 the hit kinases could be connected to known MRFs, directly or through one interaction node.

158 In mice lacking MRF within the oligodendrocyte lineage, premyelinating o

159 sed into the dorsal/lateral PAG, the lateral MRF, or the superficial layers of the SC did not affect

160 rch Foundation Meningococcus Genome Library (MRF-MGL) exploits whole-genome sequencing (WGS) for this

161 elegans embryos lacking activity of the lone MRF ortholog HLH-1, indicating that additional myogenic

162 ing with the Melbourne Rapid Fields-macular (MRF-m) iPad application.

163 We conclude that CTCF modulates MRF functional interactions in the orchestration of myog

164 ortening effect, compromising the ability of MRF T(1) to identify transition zone lesions.

165 Genetic ablation of MRF in mature oligodendrocytes within the adult CNS resu

166 were rapidly downregulated after ablation of MRF, indicating an ongoing requirement for MRF in the ex

167 eks and peaking at 8 weeks after ablation of MRF.

168 the modulator reduce the binding activity of MRF, as well as the repressing activity on the enhancer.

169 Ciona requires the combinatorial activity of MRF, MyoD and Early B-cell Factor (Ebf).

170 e-specific gene and that multiple classes of MRF-regulated genes exist in Ciona.

171 consistent with substantial conservation of MRF-directed myogenesis in chordates and demonstrate for

172 Here, we perform de novo automated design of MRF pulse sequences by applying physics-inspired optimiz

173 on algorithm has discovered the existence of MRF pulse sequences with intrinsic robustness against sh

174 The expression of MRF is differentiation specific; the DNA binding activit

175 sults demonstrate that ongoing expression of MRF within the adult CNS is critical to maintain mature

176 The feasibility of MRF at 1.5 T and 3 T was demonstrated in the pancreas.

177 This work investigates the feasibility of MRF parameter mapping for pancreatic imaging in the pres

178 udy tested the hypothesis that the firing of MRF neurons whose axons could be antidromically activate

179 muscle development is largely independent of MRF function.

180 g pathway(s) that mediates the inhibition of MRF-induced myogenesis by oncogenic Ras, we tested two t

181 Knockdown of MRF in oligodendrocytes by RNA interference prevents exp

182 Loss of MRF function leads to loss of myogenesis by specific pop

183 myelin genes; conversely, overexpression of MRF within cultured oligodendrocyte progenitors or the c

184 s two possibilities: that two populations of MRF neurons provide independent inputs to inspiratory an

185 multigene neighborhoods in the regulation of MRF genes.

186 The role of MRF has also been advocated in modulation of state of ar

187 nally incorporated entropy and uniformity of MRF metrics, as well as morphometric features from the m

188 FGFs, which interferes with the activity of MRFs, suppressed the expression of miR-1, miR-206 and mi

189 gh up-regulation of distinct combinations of MRFs.

190 We found that directed expression of MRFs in the neural tube of chicken embryos induced ectop

191 in the quiescent state before expression of MRFs or desmin and distinguish them from fibroblasts.

192 ere, we define Ascl2 as a novel inhibitor of MRFs.

193 sequester their transcriptional activity on MRF genes.

194 Demonstrating the effect of GBCA on MRF T(1) relaxometry in patients also paves the way for

195 1, encoding HLH-1 (CeMyoD) which is the only MRF-related factor in the nematode.

196 ic regulatory networks were MRF-dependent or MRF-independent.

197 nstream of myelin regulatory factor (MYRF or MRF), a transcriptional regulator that specifically acti

198 al interactions modeled using a second-order MRF guide the LS contour evolution towards the target ki

199 ith DM having the highest impact among other MRFs.

200 lting in ~7,000,000 simulated (19)F-paraGEST MRF patterns of different Ln(3+) concentrations.

201 tic resonance fingerprinting ((19)F-paraGEST MRF), a rapid signal acquisition, encoding, and analysis

202 We, therefore, recommend performing MRF T(1) prior to DCE imaging to maintain its benefit fo

203 ative Ebf-binding site adjacent to predicted MRF binding sites in the Ciona Mymk promoter.

204 Unlike previous MRF-based methods, SMURFLite is computationally feasible

205 improved precision in comparison with prior MRF implementations.

206 The proposed MRF has two major advantages over existing approaches.

207 In conclusion, the proposed MRF is a useful tool to identify novel mQTLs, especially

208 The proposed MRF model is implemented in the R package MRFscRNAseq av

209 Conclusions: The proposed MRF-based model efficiently utilizes the known pathway s

210 We detected a novel nuclear protein, MRF, that binds to multiple sites on the modulator which

211 water and fat fraction (FF) quantification (MRF T1-FF) for providing markers of fatty replacement an

212 abling fast and free-breathing quantitation, MRF has the potential to add value during the clinical c

213 ns with MyoD, indicating that CTCF regulates MRF-mediated muscle differentiation.

214 investigate the role of myogenic regulators (MRFs), Myf5, MyoD, Myogenin and MRF4 in the regulation o

215 ence (i.e. 62%) in a subset of PN-responsive MRF neurons is significantly greater than for the subset

216 greater than for the subset of PN-responsive MRF neurons that did not respond to urinary bladder dist

217 Free Grammars and Markov Random Fields (SCFG/MRF).

218 The SCFG/MRF models are constructed using atomic-resolution RNA 3

219 A mutation in myog, encoding a second MRF, has little obvious phenotype at early stages, but e

220 Lite, a method that combines both simplified MRFs and simulated evolution to substantially improve re

221 na exhibits a similar reliance on its single MRF-family gene, and diverse mechanisms activate Ci-Mrf

222 ry and expiratory motoneurons or that single MRF neurons have collateralized projections to both grou

223 e role in embryonic myogenesis of the single MRF gene of the invertebrate chordate Ciona intestinalis

224 hibits myogenic differentiation by targeting MRFs and facilitates the generation of postnatal satelli

225 Subjects underwent whole-brain 3 Tesla MRF acquisition, the reconstruction of which generated T

226 Competitive binding assays demonstrate that MRF requires the presence of multiple A+T stretches for

227 simplest explanation for these data is that MRF neurons that receive input from the vestibular nucle

228 We propose that MRF binds over the entire modulator and exerts repressor

229 Agreement analysis revealed that MRF tended to generate significantly higher MD (bias, 3.

230 Agreement analysis revealed that MRF tended to generate significantly higher MD (bias, 3.

231 These results suggest that MRF may act as a repressor of enhancer function.

232 We discovered that MRFs and MEF2 regulate a remarkably extensive array of t

233 We found that MRFs play an unexpectedly wide-ranging role in directing

234 The MRF alpha 2 transition factors, assembled into a prerepl

235 The MRF underestimated MD and overestimated PSD values compa

236 The MRF-MGL represents an effective, broadly applicable mode

237 The MRF-ML framework presented in this study demonstrated st

238 Although the MRF alpha 2 components are apparently not encoded by cur

239 help registration in textureless areas, the MRF over super-voxels efficiently propagates motion info

240 In particular, we compare the MRF prior to a situation where independent Bernoulli pri

241 r simulation studies show that employing the MRF prior improves on selection accuracy.

242 glaucoma, the sensitivity was 60.9% for the MRF and 78.3% for the HFA (P = 0.10); respective specifi

243 However, the MRF showed potential for screening in a low-resource set

244 ct evidence to demonstrate that cells in the MRF relay vestibular signals monosynaptically to respira

245 ion of the clique potential functions in the MRF so its maximum a posteriori estimation can be reduce

246 lutionarily ancient, but that changes in the MRF targets for particular signals contribute to myogeni

247 nists (if endogenous) acting at sites in the MRF would be effective muscle relaxants during pregnancy

248 le group of brainstem neurons located in the MRF.

249 Lidocaine or muscimol injections into the MRF produced a large increase in diaphragm and abdominal

250 both clinical MRI and MAP were negative, the MRF-ML models correctly distinguished FCD patients from

251 determine whether functional lesions of the MRF affect inspiratory and expiratory muscle responses t

252 ectron microscopy that MyoD, a member of the MRF family, also plays a role in fetal synapse formation

253 respect, MyoD and MRF4, both members of the MRF family, exist in vivo as phosphoproteins and contain

254 e bladder were found for 51% (n = 48) of the MRF neurons tested.

255 uctions using the first nine segments of the MRF profiles (11-second acquisition time) were in good a

256 xtensive electrophysiological mapping of the MRF using extracellular recordings at rest and during lo

257 tly located in the magnocellular part of the MRF, but were absent from both the dorsal and ventral re

258 study staff also trained participants on the MRF tablet with instructions to take weekly tests at hom

259 gative and mean PSD was more positive on the MRF than the HFA in both groups (all P < 0.001).

260 after dextrose deposits, further reduced the MRF-evoked EMG responses over the course of 1 h.

261 hrough simulations, we demonstrated that the MRF had improved power over other existing methods in de

262 These data support the hypothesis that the MRF participates in generating vestibulo-respiratory res

263 Thus, the MRF-SRF and YAP-TEAD pathways interact indirectly throug

264 ltiple spinal pathways from the penis to the MRF may correspond to different functions, including tho

265 automated perimetry tests at home using the MRF online system on their own personal computers.

266 locomotor neuronal system present within the MRF in behaving NHPs under normal conditions, in accorda

267 es of a locomotor neuronal system within the MRF in behaving NHPs.

268 Although the MRFs are unique in their ability to confer a myogenic ph

269 express either MyoD or myf5 first among the MRFs; most cells then expressed both myf-5 and MyoD simu

270 However, the MRFs can be computationally prohibitive when beta strand

271 ng that the effect of CKII expression on the MRFs is indirect.

272 Given that the MRFs require dimerization with E protein partners to act

273 Most (135/171 or 79%) of these MRF neurons lacked spontaneous firing.

274 of a locomotor neuronal circuit within these MRF in behaving primates.

275 functional compensatory activities of these MRFs.

276 including all sequences deleted in the three MRF knockout alleles, with a basal promoter and a lacZ r

277 ormation from the male external genitalia to MRF, and (2) ascending bilateral projections in the vent

278 in myogenic cells and in the embryo prior to MRF expression but absent in nonmyogenic fibroblasts.

279 gans (bladder, descending colon, urethra) to MRF.

280 ethod of Multipliers) algorithm aligning two MRFs.

281 scoring function measuring similarity of two MRFs; and 3) an efficient ADMM (Alternating Direction Me

282 glaucoma and control participants underwent MRF and Humphrey Field Analyzer (HFA) testing.

283 qualitatively different from previously used MRF pulse sequences and achieve fourfold shorter scan ti

284 ts with glaucoma with prior experience using MRF platform were recruited.

285 participants were prospectively imaged using MRF framework.

286 2D classification with ensemble models using MRF T1 and T2 mean and standard deviation from gray matt

287 In vivo MRF kidney T1 and T2 assessments in healthy adult volunt

288 In vivo MRF-based kidney T1 and T2 values with and without B(1)

289 Utilization of the mixed waste MRF was sensitive to the efficiency of material separati

290 )e; the lower values were when a mixed waste MRF was used, and the higher values when anaerobic diges

291 e earliest myogenic regulatory networks were MRF-dependent or MRF-independent.

292 ues model nested pairs and insertions, while MRF ideas handle crossing interactions and base triples.

293 ar-distal enhancer chromatin associated with MRF genes in Mb and Mt than previously reported from stu

294 d MRF T1-FF, from which the FF measured with MRF T1-FF (FF(MRF)) and water T1 were derived.

295 Visual field testing with MRF performed at home showed strong correlation for mean

296 ifferentiation state, might collaborate with MRFs.

297 Ascl2 competes with MRFs for binding to E-boxes in the promoters of muscle g

298 between patients without MRFs and those with MRFs but no DM (P > .2 for all liver outcomes).

299 6 pairs of matched patients with and without MRFs for analysis.

300 cept for comparison between patients without MRFs and those with MRFs but no DM (P > .2 for all liver