ライフサイエンスコーパス: MRS

1 MRS also showed an effect of fingolimod on glutamate lev

2 MRS data were acquired from occipital cortex.

3 MRS may provide supplementary biochemical information an

4 MRS metabolite peak-area ratios (n=160) of NAA-creatine

5 MRS plays a complementary role to MRI, by increasing its

6 MRS revealed the number of neurons in the left hypothala

7 MRS, plays significant complementary role and should be

8 to both real-time noninvasive imaging by 13C MRS as well as therapeutic response.

9 ined combination algorithms, designed for 1D MRS, for 2D MRS with both internal and external referenc

10 and myocardial function were assessed by 1H MRS imaging and MRI at 3 T.

11 ate and glutamine (Glx), were measured by 1H MRS in the left dorsolateral prefrontal cortex (l-DLPFC)

12 g proton magnetic resonance spectroscopy (1H MRS), this study assessed whether dysregulation of the h

13 1H-MRS showed adequate discriminant validity, but limited r

14 gastrocnemius by 1H-MRS and HPLC to compare signal quality and convergent va

15 However, carnosine quantification by 1H-MRS has some potential limitations that warrant a thorou

16 , proton magnetic resonance spectroscopy (1H-MRS) has been used as a non-invasive alternative to quan

17 g proton magnetic resonance spectroscopy (1H-MRS) in healthy subjects.

18 Cross-sectional design using 3-T 1H-MRS in participants recruited from university-based psyc

19 HFC was measured using 1H-MRS prior to test meal administration (before time 0) an

20 We used in vitro 1H-MRS to verify signal linearity and possible noise source

21 Inclusion criteria were single voxel 1H-MRS studies reporting glutamate, glutamine or Glx values

22 uring acetylcarnitine concentrations with 1H-MRS is feasible on clinical MR scanners and support the

23 tion algorithms, designed for 1D MRS, for 2D MRS with both internal and external references.

24 n, can be non-invasively quantified using 2D MRS method of double quantum filtered correlation spectr

25 Criteria for 2HG MRS were established to make a presumptive molecular dia

26 e data provide a basis for incorporating 2HG MRS into clinical management of IDH-mutated gliomas.

27 Patients and Methods 2HG MRS was performed in 136 patients using point-resolved s

28 Results Quantitative 2HG MRS was technically and biologically reproducible.

29 scan for tissue segmentation followed by 31P MRS, chemical shift imaging scan with 84 voxels of data

30 orus-31 magnetic resonance spectroscopy (31P MRS) allows for the quantification of bioenergetic molec

31 31P-MRS represents a promising technique to assess the patho

32 31P-MRS was acquired from cerebral motor regions and from ti

33 ntagonists of protease-activated receptor 4, MRS 2179, and clopidogrel) were also protected from seve

34 -exposed normal controls (TENC) underwent 4T MRS of the left and right hippocampus.

35 ticipants with acutely treated HIV, except a MRS alteration in basal ganglia choline.

36 Adding MRS and MRI to the protocol allows us to define the natu

37 left striatum during tDCS and an additional MRS measurement in the left DLPFC immediately after the

38 ssed only in mouse islets and the A3 agonist MRS 5698 inhibited glucose-induced insulin secretion fro

39 stablished that the IDH1 mutation induces an MRS-detectable reprogramming of pyruvate metabolism, whi

40 ance was superior to MELD, MELD-Na, CTP, and MRS at all time points (e.g., 30-day postoperative morta

41 ive fibers and mtDNA multiple deletions, and MRS displayed defective oxidative metabolism in muscle a

42 DTI and MRS seem to be more useful imaging methods to assess the

43 Our combined fMRI and MRS investigation showed that the stronger ATL BOLD resp

44 agnosis after deployment (combined MRS-I and MRS-II cohort meta-analysis odds ratio, 1.47; 95% CI, 1.

45 articles are highly irregular and jagged and MRS powder particles are rough, but primarily rounded.

46 The resulting LRS and MRS 3D-printed materials exhibit similar, but distinct i

47 inally, we discuss the potential for LRS and MRS ink components to be reclaimed and recycled, as well

48 Both LRS and MRS inks exhibit similar rheological and 3D-printing cha

49 The LRS and MRS powders are characterized by distinct, highly inhomo

50 Lunar and Martian regolith simulant (LRS and MRS, respectively) architectures using ambient condition

51 ruited 223 infants who all underwent MRI and MRS at a median age of 7 days (IQR 5-10), with 190 (85%)

52 MRI and MRS examinations were performed on variable dates (10-29

53 reased scanning time, combination of MRI and MRS is currently not recommended as a first line investi

54 In vivo MRI and MRS showed that at 17-23 months of age there was a signi

55 Combination of MRI and MRS showed the highest diagnostic accuracy among the ima

56 imulation (EFS) in the presence of L-NNA and MRS 2500 enhanced ICC-IM Ca(2+) transients.

57 ponding centers of mass in (18)F-FET PET and MRS imaging of Cho/NAA, determined by simultaneously acq

58 cles that are abundant in the facies bearing MRS, but not in the host sandstone and black shale.

59 o better understand the relationship between MRS-visible neurochemicals, the BOLD signal change, and

60 algorithms developed for conventional brain MRS.

61 or 48 h; aEEG was acquired throughout, brain MRS acquired at 24 and 48 h and cell death (TUNEL) evalu

62 ne whether quantitative assessment of 2HG by MRS could serve as a noninvasive clinical imaging biomar

63 onses to nerve stimulation were abolished by MRS-2500 and not observed in muscles with genetic deacti

64 nts and IJPs elicited by EFS were blocked by MRS-2500, a P2Y1 antagonist, and absent in P2ry1((-/-))

65 purines and these responses were blocked by MRS-2500.

66 Additionally, NAAG levels measured by MRS and cortical efficiency during working memory measur

67 Conclusion 2HG concentration as measured by MRS was reproducible and reliably reflected the disease

68 ry/inhibitory network activity as proxied by MRS measurements of GABA and glutamate.

69 a novel agent for clinically relevant (13)C MRS studies of energy metabolism and further provides op

70 Hyperpolarized (13)C MRS was performed on the myocardium of 8 sham-operated c

71 vation of glutamate and hyperpolarized (13)C MRS-detectable glutamate production from either pyruvate

72 uating glycolysis using hyperpolarized (13)C MRS.

73 d time resolution using hyperpolarized (13)C MRS.

74 (13)C-MRS also revealed a reduction in glucose flux to glutama

75 (13)C-MRS dynamic acquisition demonstrated in an axial slab in

76 H probe, 3-aminopropylphosphonate, and (13)C-MRS measurements of the H(13)CO3(-)/(13)CO2 peak intensi

77 (13)C-magnetic resonance spectroscopy ((13)C-MRS) magnetization transfer measurements.

78 (13)C-magnetic resonance spectroscopy ((13)C-MRS), which revealed a reduction in metabolism of hyperp

79 fied in post-ATI basal ganglia total choline MRS, suggesting an alteration in neuronal membranes.

80 we also calculated ADC values and Cho Cr/Cit MRS ratios for all patients.

81 We collected MRS measurements in the left DLPFC and left striatum dur

82 of PTSD diagnosis after deployment (combined MRS-I and MRS-II cohort meta-analysis odds ratio, 1.47;

83 se to UDCA and elevated IgG-levels confirmed MRS (p = 0.03), DS (p = 0.04), biochemical response (p =

84 However, conventional MRS requires large voxels compared with fMRI, because of

85 eurochemical and BOLD signals (combined fMRI-MRS) to different image contrasts in human V1 at 7 tesla

86 es of interest (for example, metabolites for MRS or MRI) to fill the pore channels (incipient wetness

87 -day postoperative mortality C-statistic for MRS = 0.766; 95% CI, 0.676-0.855) in terms of discrimina

88 lite developed exclusively in the fossilized MRS thus provides a new biosignature for metasediments d

89 he interplay between fMRI connectivity, (1)H MRS and clinical data was explored by applying moderatio

90 The reviewed (1)H MRS and PET/SPECT studies support the theory of hypofunc

91 Using a multi-voxel (1)H MRS approach at 3 Tesla with high spatial resolution and

92 These results demonstrate potential of (1)H MRS at ultrahigh fields.

93 Using (1)H MRS in combination with chemometrics identified several

94 s confirmed in vivo, where the observed (1)H MRS increase in glutamate/glutamine occurred prior to tu

95 emic variability for 5 days followed by (1)H MRS scanning in the occipital lobe to measure the change

96 The present cross-sectional 3T (1)H MRS study is the first to use a multivoxel approach to s

97 proton magnetic resonance spectroscopy ((1)H MRS) in the pregenual anterior cingulate (pgACC) and occ

98 Proton MR spectroscopy ((1)H MRS) was used to divide MDD subjects into two subgroups:

99 proton magnetic resonance spectroscopy ((1)H MRS) were performed immediately before and after the adm

100 proton magnetic resonance spectroscopy ((1)H MRS), especially in bipolar I disorder (BD-I).

101 Collectively, our findings identify (1)H MRS-detectable elevation of glutamate and hyperpolarized

102 HFC was assessed by (1)H MRS.

103 uld broaden the applications of routine (1)H MRS.

104 0001) and liver triglyceride content by (1)H-MRS (P < 0.0001) were worse in NASH with elevated ALT.

105 roton magnetic resonance spectroscopy ((1) H-MRS) was established based on the 95th percentile in a g

106 roton magnetic resonance spectroscopy ((1) H-MRS), abdominal fat using magnetic resonance imaging (MR

107 ized with the following studies: liver (1) H-MRS; euglycemic insulin clamp with measurement of glucos

108 (1)H-MRS and clinical ratings at baseline were assessed for a

109 ageing in these brain regions using 7T (1)H-MRS and findings indicate that glia-related metabolites

110 ipid content as assessed by noninvasive (1)H-MRS and lipid profiling independent of changes in hepati

111 Conventional MR imaging and (1)H-MRS are important complementary tools in the diagnostics

112 vely enrolled and acquired high quality (1)H-MRS at median 33.0 (IQR 31.6-35.2) weeks PMA from a voxe

113 (1)H-MRS at ultra-high field strength can potentially improve

114 concentrations were determined by using (1)H-MRS before and 3 and 5 h after a high-fat with added pro

115 (1)H-MRS data were processed using LCModel software to calcul

116 (1)H-MRS data were processed using LCModel software to calcul

117 Consistent with the (1)H-MRS data, steatosis on liver biopsy was also significant

118 s associated with a significant in vivo (1)H-MRS detectable reduction in 2HG but not with significant

119 (1)H-MRS improves the diagnostic accuracy in the prediction o

120 We prospectively collected high quality (1)H-MRS in 59 premature infants born </=32 weeks and 61 heal

121 ivity at ultra-high field, we performed (1)H-MRS in 60 healthy human volunteers to asses age-related

122 However, (1)H-MRS measurements revealed significantly higher GABA/Wate

123 Measurements of (1)H-MRS metabolites were obtained in the posterior cingulate

124 emical exchange saturation transfer and (1)H-MRS methods, lower levels of glial glutamate transporter

125 They underwent 7T (1)H-MRS of the ACC, DLPFC, hippocampus, and thalamus and per

126 (1)H-MRS of two cell lines genetically modified to express ID

127 x (dACC) was acquired as a secondary 3T (1)H-MRS outcome using a MEGA-PRESS sequence.

128 We correlated the (1)H-MRS PDFF findings with SPCs (r = 0.92; P < .001).

129 tive study to determine the accuracy of (1)H-MRS PDFF in the measurement of steatosis using histopath

130 (1)H-MRS PDFF results correlated with histopathology results

131 We collected clinical, serologic, (1)H-MRS PDFF, and liver biopsy data from 94 adult patients w

132 hed controls underwent ultra-high field (1)H-MRS scans of the upper limb motor cortex and pons, ALS F

133 ation done by using the dynamic MRI and (1)H-MRS showed 91.5% diagnostic accuracy with 75.0% sensitiv

134 rgeted metabolomics analysis of in vivo (1)H-MRS spectra discriminated between IDH-mutant tumors and

135 rations were measured continuously with (1)H-MRS using a specific lactate detection method.

136 (1)H-MRS was a better predictor of study withdrawal due to AL

137 Three dimensional (1)H-MRS was acquired in young schizophrenia subjects (N = 36

138 (1)H-MRS was used to measure hippocampal glutamate concentrat

139 ng magnetic resonance imaging (MRI) and (1)H-MRS with choline (Cho) signal-to-noise ratio (SNR) measu

140 proton magnetic resonance spectroscopy ((1)H-MRS) acquisition scheme that uses an ultrahigh magnetic

141 We determined liver fat ((1)H-MRS) and clinical characteristics including features of

142 proton magnetic resonance spectroscopy ((1)H-MRS) and fibrosis estimated as stiffness using transient

143 proton magnetic resonance spectroscopy ((1)H-MRS) and whether the addition of protein can modulate th

144 d (1)H magnetic resonance spectroscopy ((1)H-MRS) at ultra-high-field in the nucleus accumbens and in

145 Proton MR spectroscopy ((1)H-MRS) has been used to assess regional neurochemical brai

146 proton magnetic resonance spectroscopy ((1)H-MRS) in 28 adults with ASD and 29 age-matched typically

147 asured by proton magnetic spectroscopy ((1)H-MRS) in preterm infants with advancing post-menstrual ag

148 proton magnetic resonance spectroscopy ((1)H-MRS) in the rACC to examine the function and neurochemis

149 proton magnetic resonance spectroscopy ((1)H-MRS) investigating glutamate in DLPFC.

150 Proton magnetic resonance spectroscopy ((1)H-MRS) studies have examined glutamatergic abnormalities i

151 Proton MR spectroscopy ((1)H-MRS) studies of in vivo neurochemical changes across the

152 proton magnetic resonance spectroscopy ((1)H-MRS), instead of collecting and analyzing liver biopsy s

153 proton magnetic resonance spectroscopy ((1)H-MRS).

154 proton magnetic resonance spectroscopy ((1)H-MRS).

155 magnetic resonance spectroscopy ((31)P/(1)H-MRS).

156 g (1)H-magnetic resonance spectroscopy ((1)H-MRS).

157 proton magnetic resonance spectroscopy ((1)H-MRS); (2) severity of liver disease by biopsy (n = 293);

158 continuously with J-difference-editing (1)H-MRS, and time curves were analyzed using nonlinear mixed

159 abolites were examined with whole-brain (1)H-MRS, in early schizophrenia.

160 rformance of a fractal n-back task, and (1)H-MRS, respectively.

161 Using 7T (1)H-MRS, we assessed levels of mIns, tCr, and tCho (glia-rel

162 er, which was detected by using in vivo (1)H-MRS.

163 evels of N-acetyl-aspartate measured by (1)H-MRS; and hypomyelination in PFC as evidenced by relevant

164 olarized magnetic resonance spectroscopy (HP MRS) using dynamic nuclear polarization (DNP) is a techn

165 However, the HP MRS polarization decays in the liquid state according to

166 e dehydrogenase deficiency, in particular if MRS shows elevated succinate.

167 Cases showed significant improvements in MRS scores (p<0.001), mobility (p<0.001) and ADL (p=0.00

168 patient the follow-up examinations included MRS with the assessment of metabolite ratios (NAA/Cr, Ch

169 This is the largest MRS study of GABA in schizophrenia and the first to exam

170 ise protocol and muscle studied by localized MRS.

171 (LBG), modified corn and rice starches (MCS, MRS)) to an infant formula on both in vitro mineral avai

172 MRI MRS has more diagnostic accuracy than MRI alone for dete

173 obese and 11 healthy subjects using a 3T MRI/MRS scanner, and IR in the obese subjects was documented

174 Using a combined functional MRI/MRS approach, our results demonstrate that susceptible-

175 controls and a brain phantom using a 3 T MRI/MRS scanner.

176 t 2HG could serve as a potential noninvasive MRS-detectable metabolic biomarker of IDHmut glioma resp

177 We tested the association of MRS with 61 behavioural phenotypes and found that whilst

178 However, publications of MRS, PET and SPECT are limited only providing anecdotal

179 to overcome this low spatial specificity of MRS by predicting local glutamate and GABA based on func

180 ols (13.2+/-2.2 years) provided at least one MRS scan.

181 ch terms (magnetic resonance spectroscopy OR MRS) AND (glutamate OR glut* OR GLX) AND (schizophrenia

182 The AUC of our MRS was 0.724 and higher than risk scores created using

183 ial capacity, and, in addition, static (31)P-MRS also revealed differences in the Pi-to-ATP exchange

184 iduals through dynamic, static, and ST (31)P-MRS at 7T.

185 in spectral and temporal resolution of (31)P-MRS at ultra high fields (i.e., 7T) uncovers new potenti

186 hyperinsulinemic-euglycemic clamp and (31)P-MRS before, during, and after near-maximal isometric cal

187 lity of using high spectral resolution (31)P-MRS data, acquired at rest, as a marker of oxidative met

188 (31)P-MRS measurements of the chemical shift of the pH probe,

189 In addition, as the dynamic (31)P-MRS requires a complex setup and patient exercise, our a

190 horus magnetic resonance spectroscopy ((31)P-MRS), in particularly dynamic (31)P-MRS, provides a powe

191 y ((31)P-MRS), in particularly dynamic (31)P-MRS, provides a powerful tool for the non-invasive inves

192 F with magnetic resonance spectroscopy-PDFF (MRS-PDFF), biochemical triglyceride extraction, and hist

193 on echo-planar spectroscopic imaging (PEPSI) MRS scans of pregenual anterior cingulate cortex (pACC)

194 h psychosocial and mental health phenotypes, MRS were more strongly associated with lifestyle and soc

195 chosis by means of echo time-averaged proton MRS at 4T.

196 jects by using the echo time-averaged proton MRS technique at 4T (i.e., modified point resolved spect

197 arametric liver MR, including hepatic proton MRS, T1- and T2*-mapping yielding "iron-corrected T1" [c

198 d diffusion-weighted MRI and thalamic proton MRS 4-14 days after birth.

199 Thalamic proton MRS measures acquired soon after birth in neonatal encep

200 Using proton MRS, we measured glutamate concentrations in ACC and occ

201 ve SNR or accelerate the acquisition of PUFA MRS from breast in a clinical setting.

202 )F-FDG PET and hyperpolarized (13)C-pyruvate MRS imaging simultaneously for tumor tissue characteriza

203 at combined (18)F-FDG PET and (13)C-pyruvate MRS imaging was possible in a single session of approxim

204 With this spectral quality, MRS was used to interrogate a number of metabolic proper

205 included ventricular enlargement and reduced MRS-derived creatine levels.

206 y acquired, 3-dimensional spatially resolved MRS imaging data, were compared.

207 We also created a methylation risk score (MRS) to predict MDD status 6 years later.

208 of dominant stricture (DS), Mayo Risk Score (MRS), immunosuppression, biochemical response to UDCA an

209 ration of VOCAL-Penn to the Mayo Risk Score (MRS), Model for End-Stage Liver Disease (MELD), Model fo

210 ylation, we tested whether DNAm risk scores (MRS), trained on 1223 MDD cases and 1824 controls, could

211 etic divergence from multiregion sequencing (MRS) data, we demonstrate that it is feasible to disting

212 Consequently, a single MRS voxel may cover areas with distinct cytoarchitecture

213 nalysis (EPMA) and micro-Raman spectrometry (MRS).

214 troduction of combined PET/MR spectroscopic (MRS) imaging, it is now possible to directly and indirec

215 MR spectroscopy (MRS) changes of increased Glutamate/glutamine, reduced m

216 ng the results of series of MR spectroscopy (MRS) examinations in the course of BBE.

217 sine ((18)F-FET) and proton MR spectroscopy (MRS) imaging of cell turnover measured by the ratio of c

218 bution of cerebral CT, MRI, MR spectroscopy (MRS), positron emission tomography (PET) and single-phot

219 resonance imaging (MRI) and MR spectroscopy (MRS).

220 proton magnetic resonance (MR) spectroscopy (MRS) biomarkers as early predictors of the neurodevelopm

221 lism, (13)C magnetic resonance spectroscopy (MRS) allows following the fate of (13)C-enriched substra

222 Magnetic resonance spectroscopy (MRS) allows for noninvasive measurement of metabolites i

223 ls, such as magnetic resonance spectroscopy (MRS) and magnetic resonance imaging (MRI), contributes t

224 we combined magnetic resonance spectroscopy (MRS) and resting state fMRI and demonstrated an inverse

225 ies, proton magnetic resonance spectroscopy (MRS) and tissue biochemistry, sampling individuals with

226 ging (MRI), magnetic resonance spectroscopy (MRS) and transrectal ultrasound (TRUS) in detecting pros

227 ) and novel magnetic resonance spectroscopy (MRS) approaches.

228 assessed by magnetic resonance spectroscopy (MRS) are used as markers of neuroinflammation in neurode

229 , employing magnetic resonance spectroscopy (MRS) at 7 T.

230 rence (MQC) magnetic resonance spectroscopy (MRS) at the expense of losing half of the signal.

231 I) and (1)H magnetic resonance spectroscopy (MRS) data were obtained from participants with OCD (n=49

232 (HP) (13)C magnetic resonance spectroscopy (MRS) has the unique ability to detect real-time metaboli

233 e GABA with magnetic resonance spectroscopy (MRS) have been inconsistent.

234 ng tDCS and magnetic resonance spectroscopy (MRS) in 17 healthy participants.

235 ole of (1)H magnetic resonance spectroscopy (MRS) in demonstrating these metabolic changes and to rev

236 rized (13)C magnetic resonance spectroscopy (MRS) is a developing imaging technique that enables non-

237 (31)P magnetic resonance spectroscopy (MRS) is widely used for non-invasive investigation of mu

238 ues such as magnetic resonance spectroscopy (MRS) may cover anatomically and functionally distinct ar

239 E STATEMENT Magnetic resonance spectroscopy (MRS) measures local glutamate and GABA noninvasively.

240 Magnetic resonance spectroscopy (MRS) measures of glutamate in the striatum and hippocamp

241 ined by 13C magnetic resonance spectroscopy (MRS) of hyperpolarized pyruvate, we confirmed that FX11

242 pose Proton magnetic resonance spectroscopy (MRS) of the brain can detect 2-hydroxyglutarate (2HG), t

243 rized (13)C magnetic resonance spectroscopy (MRS) provides unprecedented opportunities to obtain clin

244 s in proton magnetic resonance spectroscopy (MRS) resulting from the replacement of (1)H with (2)H.

245 novel (17)O magnetic resonance spectroscopy (MRS) technique on R6/2 mice to directly determine rates

246 ield proton magnetic resonance spectroscopy (MRS) to determine whether PTSD is associated with lower

247 SD, we used magnetic resonance spectroscopy (MRS) to measure glutamate (Glx = glutamate + glutamine)

248 sting-state magnetic resonance spectroscopy (MRS) to measure task-related blood-oxygen level-dependen

249 study used magnetic resonance spectroscopy (MRS) to quantify the concentration of GABA in human moto

250 asured with magnetic resonance spectroscopy (MRS) was associated with OCD and/or CBT response.

251 Proton magnetic resonance spectroscopy (MRS) was used to test the hypothesis that older particip

252 s using 7-T magnetic resonance spectroscopy (MRS) were measured.

253 using (31)P magnetic resonance spectroscopy (MRS), and glycolysis and fatty acid oxidation were measu

254 e and brain magnetic resonance spectroscopy (MRS), and muscle biopsy and fibroblasts were analyzed.

255 S using 31P-magnetic resonance spectroscopy (MRS), the modality of choice to assess energy metabolism

256 using (31)P magnetic resonance spectroscopy (MRS), we demonstrated that pHLIP Var3 was retained in tu

257 he value of magnetic resonance spectroscopy (MRS)-based metabolic changes for detection of response t

258 to identify magnetic resonance spectroscopy (MRS)-detectable metabolic biomarkers associated with IDH

259 bjects with magnetic resonance spectroscopy (MRS).

260 lution (1)H magnetic resonance spectroscopy (MRS).

261 ration with magnetic resonance spectroscopy (MRS).

262 sting-state magnetic resonance spectroscopy (MRS).

263 le spinning magnetic resonance spectroscopy (MRS).

264 measured by magnetic resonance spectroscopy (MRS).

265 uccinate on magnetic resonance spectroscopy (MRS).

266 I) and (1)H magnetic resonance spectroscopy (MRS).

267 ith [(19)F] magnetic resonance spectroscopy (MRS).

268 g (DTI) and magnetic resonance spectroscopy (MRS).

269 g 7T proton magnetic resonance spectroscopy (MRS).

270 C] pyruvate magnetic resonance spectroscopy (MRS).

271 chniques, such as single voxel spectroscopy (MRS) and diffusion tensor imaging (DTI), in children wit

272 s been shown to host mat-related structures (MRS) and, in this regard, these rocks offer a unique opp

273 first phase of the Marine Resiliency Study (MRS-I) included 1415 male Marines, 59 of whom developed

274 second phase of the Marine Resiliency Study (MRS-II) included 745 male Marines, 25 of whom developed

275 Mitchell-Riley syndrome (MRS) is caused by recessive mutations in the regulatory

276 Using single-voxel 3-T MRS, healthy term neonates (n = 31, mean postconception

277 ns with re-experiencing symptoms affirm that MRS indices of hippocampus neuron integrity and glutamat

278 The MRS explained 1.75% of the variance in MDD (beta = 0.338

279 The MRS was also associated with incident cases of MDD who w

280 de PRS (beta = 0.384, p = 4.69 x 10(-9)) the MRS remained associated with MDD (beta = 0.327, p = 5.66

281 tic effects explained 22% of variance in the MRS, with a further 19% explained by pedigree-associated

282 We report evidence for a reduction in the MRS-measured concentration of GABA specific to learning.

283 Neither the postmortem nor the MRS studies directly address the physiological propertie

284 removing smokers from the training set, the MRS strongly associated with BMI (beta = 0.053, p = 0.02

285 No relationships to MRS glutamate and amphetamine-induced subclinical positi

286 Quantitative exchanged-label turnover MRS should broaden the applications of routine (1)H MRS.

287 termed quantitative exchanged-label turnover MRS, only requires deuterium-labelled glucose and standa

288 Using MRS, we monitored the brain metabolic response to lipopo

289 ium particles was further investigated using MRS which enabled us to verify the reliability of the re

290 and tumor glutamine pool size measured using MRS (r(2) = 0.71).

291 Taurine levels, measured using MRS, showed a very strong inverse correlation with GFAP

292 ee, depressed outpatients using single-voxel MRS, to measure absolute glutamate concentrations in bas

293 ll nine Couinaud segments using single-voxel MRS-PDFF (n=117) and tissue wedges for biochemical trigl

294 ing status was also strongly associated with MRS (beta = 0.440, p <= 2 x 10(-16)).

295 he first report that PTSD is associated with MRS markers of hippocampus Glu excess, together with ind

296 RI-PDFF showed an excellent correlation with MRS-PDFF (r=0.984, confidence interval 0.978-0.989) and

297 ompared between groups and correlations with MRS glutamate, subclinical psychopathological and neuroc

298 ndoderm, and its absence in individuals with MRS specifically impairs formation of endocrine cells of

299 To determine why individuals with MRS specifically lack pancreatic endocrine cells, we mic

300 was assessed through linear regression with MRS-PDFF, triglyceride extraction, and histology.