ライフサイエンスコーパス: MSD

1 MSD-HSCT (n = 32), compared with standard care (n = 35)

2 es flanked by basic amino acids in the HIV-1 MSD that function to anchor the glycoprotein in the lipi

3 polar head groups, is proposed for the HIV-1 MSD.

4 We aimed to enroll 220 MSD cases per year from selected health centers serving

5 atal in the community without treatment; (3) MSD seen at sentinel centers is a proxy for fatal diarrh

6 es were compared between 1,052 URD and 3,514 MSD BMT recipients with CML in CP1.

7 y ligase-PROTAC-target protein complex and a MSD assay to measure cellular degradation of the target

8 Furthermore, indications for a MSD were found by UPLC/MS/MS measurements.

9 In children lacking a MSD, increasingly MUD HSCT is being considered as first-

10 = 0.93), Abeta42-EI (r = 0.93), and Abeta42-MSD (r = 0.95) assays compared with the classic Abeta42-

11 ctors (including knowledge and beliefs about MSDs) between occupational groups should allow the study

12 In addition, MSD-IAA demonstrated better sensitivity than our mIAA fr

13 qPCR detected 155 additional MSD cases with high copy numbers of ipaH, a 90% increase

14 r rate of T-cell recovery was observed after MSD/MFD HCT.

15 re encouraging, with similar EFS rates after MSD, 6/6 matched UCB, 5/6 UCB, and 10/10 matched UD.

16 ds and were significantly protective against MSD in Mozambique and India.

17 In comparison, ion trap noise from Agilent MSD-Trap-SL is larger than the Q-TOF noise and is propor

18 allows direct quantitative comparisons among MSD curves and between velocity autocorrelation function

19 An MSD case was defined as a child with a diarrheal illness

20 died 36 patients with obstructive HCM and an MSD and compared them with 15 patients with HCM and no o

21 These data favor an MSD over a MUD in patients age >/=50 years.

22 m inflammatory profile was assessed using an MSD 30-plex array.

23 ding to diabetes determine high affinity and MSD-IAA reactivity.

24 se the major attributable LSD-associated and MSD-associated pathogens are the same, implementing vacc

25 n back-propagation neural network (BPNN) and MSD analysis using a sliding window.

26 estimate the annual number of MSD cases and MSD incidence rate attributable to a pathogen or group o

27 tical copolymer including MMD, FTD, CCD, and MSD.

28 immunoassays (modified INNOTEST, FL, EI, and MSD) may correlate better with the antibody-independent

29 between sanitation and hygiene exposures and MSD.

30 GSK and MSD varicella-containing vaccines (VCVs) were considered

31 significant difference between Haplo-HCT and MSD-HCT in terms of nonrelapse mortality (P = .06), prog

32 Therefore, LRRD and MSD are analog and digital force transducers, respective

33 ere will be important for diagnosing MLD and MSD patients and for monitoring the efficacy of therapeu

34 re not significantly different after MUD and MSD transplants.

35 ll domain-swapping contacts between NBDs and MSD cytoplasmic loops in opposite halves of the protein

36 on at the last follow-up between the URD and MSD recipients; however, MSD recipients were more likely

37 Fatigue and MSDs impact psychomotor performance; therefore, these re

38 With comparable effectiveness as MSD VCV at lower costs, GSK VCV may offer higher value f

39 hospitalised were eligible for inclusion as MSD.

40 tion by using the designed network, and ASD, MSD, VOE and RVD improved from 27.8 to 21, 147 to 124, 0

41 parallel case-control studies, one assessing MSD and the other less-severe diarrhoea (LSD).

42 However, traditional time-averaged MSD analysis of individual trajectories remains linear a

43 achieve greater accuracy than time-averaged MSD analysis on simulated trajectories while only requir

44 of MSDs after 4 years among women (for back MSDs, risk ratio (RR) = 1.58, 95% confidence interval (C

45 1.59, 95% CI: 1.21, 2.07) and men (for back MSDs, RR = 1.50, 95% CI: 1.05, 2.15; for degenerative MS

46 both types of MSDs after 11 years (for back MSDs, RR = 1.72, 95% CI: 1.21, 2.43; for degenerative MS

47 Because MSD refolding may turn off GPIb-IX's mechanosensory sign

48 Participants with both MSD and LSD had significantly more linear growth falteri

49 hringer Ingelheim (Canada), Pfizer (Canada), MSD, Chest, Heart and Stroke Scotland, and The Stroke As

50 echnology from Meso Scale Discovery Company (MSD-IAA) detects as positive 61% (61 of 100) of new-onse

51 Among a subset (n = 2,874) comprising MSD cases and their age-, gender-, and location-matched

52 ynamics analysis, which extends conventional MSD analysis to measure diffusive motion in confined tra

53 ts obtained from mean square displacement (D(MSD)) data were 20-100% larger for the two uncharged PDI

54 phy (MLD) and multiple sulfatase deficiency (MSD) displayed a clear deficit in the enzymatic activity

55 generation in multiple sulfatase deficiency (MSD), a severe lysosomal storage disorder caused by muta

56 interval (CI): 1.15, 2.18; for degenerative MSDs, RR = 1.59, 95% CI: 1.21, 2.07) and men (for back M

57 = 1.50, 95% CI: 1.05, 2.15; for degenerative MSDs, RR = 1.61, 95% CI: 1.16, 2.22) and both types of M

58 = 1.72, 95% CI: 1.21, 2.43; for degenerative MSDs, RR = 1.68, 95% CI: 1.25, 2.46) among men only, but

59 nfounders, job strain predicted degenerative MSDs among women after 4 and 11 years of follow-up.

60 Multi-specificity design (MSD), on the other hand, involves considering the stabil

61 ch we call Multivariate Shapelets Detection (MSD), that allows for early and patient-specific classif

62 hod called Multivariate Shapelets Detection (MSD), which extracts patterns from all dimensions of the

63 A more sustainable development (MSD) scenario introduces an impact fee that developers m

64 The mean standard deviation (MSD) was calculated to be 3.65 when alpha = 0.01.

65 m-maximum (MIN-MAX), map standard deviation (MSD), and pattern standard deviation (PSD), were calcula

66 uences of acute moderate-to-severe diarrhea (MSD) among a censused population of children aged 0-59 m

67 associated with moderate-to-severe diarrhea (MSD) among children <5 years of age, and thereby the att

68 d incidence) of moderate-to-severe diarrhea (MSD) in children <60 months of age at 7 sites in sub-Sah

69 isk factors for moderate-to-severe diarrhea (MSD) in children less than 5 y of age.

70 with or without moderate-to-severe diarrhea (MSD).

71 cally attended moderate-to-severe diarrhoea (MSD) among children aged 0-59 months residing in censuse

72 onsequences of moderate-to-severe diarrhoea (MSD) in children aged 0-59 months.

73 ut also in terms of mean squared-difference (MSD), Lloyd's K-means Clustering algorithm is more effic

74 ons (SAM) or the method of serial dilutions (MSD).

75 interpret using conventional two-dimensional MSD analysis.

76 Using Meso Scale Discovery (MSD) assay, the mHTT signal detected with MW1 Ab correla

77 ), EUROIMMUN (EI), and Meso Scale Discovery (MSD) assays.

78 Here we developed a Meso Scale Discovery (MSD)-based screening platform to quantify endogenous NMN

79 k and degenerative musculoskeletal diseases (MSDs).

80 tudy was to review musculoskeletal disorder (MSD) prevalence among surgeons performing minimally inva

81 hesis that common musculoskeletal disorders (MSDs) and associated disability are importantly influenc

82 n of work-related musculoskeletal disorders (MSDs) in surgeons is high and no overarching strategy fo

83 ral scaling of the mean-square displacement (MSD proportional, variant tau (alpha), with alpha = 2/3)

84 Conventional mean-square displacement (MSD) analysis of single-particle trajectories often assu

85 A local mean-square displacement (MSD) analysis separates ballistic motion phases, which w

86 en evaluated using mean square displacement (MSD) analysis.

87 is examined by the mean-square displacement (MSD) and the fractional diffusion and fractional kinetic

88 ditionally use the mean square displacement (MSD) as an order parameter characterizing dynamics.

89 ensemble-averaged mean square displacement (MSD) exhibits superdiffusive behaviour.

90 namics governs the mean square displacement (MSD) of water molecules, with a clear cage stage charact

91 tracked particle's mean-square displacement (MSD).

92 e been limited to mean-squared displacement (MSD) analysis.

93 he combination of mean squared displacement (MSD) and cumulative distribution function (CDF) analysis

94 aracterization of mean-squared displacement (MSD) curves for migrating cells, describes a motion in w

95 ure the resulting mean squared displacement (MSD).

96 Mean-squared displacements (MSD) and protein resilience on the picosecond-to-nanosec

97 on the measured mean squared displacements (MSD).

98 antly affects DWS mean-square displacements (MSDs) in dense colloidal emulsions.

99 obtained from the mean-square displacements (MSDs).

100 es as well as monomer sequence distribution (MSD), gradient separation only by CCD was not possible.

101 ) and juxtamembrane mechanosensitive domain (MSD).

102 ces unfolding of this mechanosensory domain (MSD) on the platelet surface.

103 vered a juxtamembrane mechanosensory domain (MSD) within the GPIbalpha subunit of GPIb-IX.

104 ately 1-395) and a membrane-spanning domain (MSD) (residues approximately 396-655); however, its exac

105 eucine) within the membrane-spanning domain (MSD) abolished protein functionality in infectivity assa

106 he residues in the membrane-spanning domain (MSD) and also immediately flanking it (T140 to L163).

107 ing domain and one membrane-spanning domain (MSD) likely work as dimers, whereas full-length transpor

108 The membrane-spanning domain (MSD) of the envelope (Env) glycoprotein from human (HIV)

109 The 27-residue membrane-spanning domain (MSD) of the HIV-1 glycoprotein gp41 bears conserved sequ

110 The membrane-spanning domain (MSD) of the human immunodeficiency virus type 1 (HIV-1)

111 y of the F-MLV Env membrane-spanning domain (MSD) to its incorporation into HIV-1 particles.

112 D) followed by one membrane-spanning domain (MSD).

113 loops (CL) of the membrane-spanning domains (MSD).

114 ssembled from two membrane-spanning domains (MSDs) and two nucleotide-binding domains (NBDs).

115 region (MPER) and membrane-spanning domains (MSDs) of viral glycoproteins have been shown to be criti

116 d consists of two membrane spanning domains (MSDs), two cytosolic nucleotide binding domains (NBDs),

117 CFTR contains two membrane-spanning domains (MSDs), two nucleotide-binding domains (NBDs), and a regu

118 ts interface with membrane-spanning domains (MSDs).

119 EFS after HLA-matched sibling donor (MSD) and 6/6 matched unrelated cord blood (CB) donor wer

120 ukocyte antigen (HLA)-matched sibling donor (MSD) HSCT remains the gold standard for SAA patients you

121 do not have an HLA-identical sibling donor (MSD).

122 patients lacking HLA-matched sibling donors (MSD).

123 ved transplants from matched sibling donors (MSD; n = 21) and unrelated donors (UD; n = 29), using fl

124 HCT from matched sibling and family donors (MSDs, MFDs) had significantly better overall survival (8

125 ncy disease who have matched sibling donors (MSDs) can proceed to hematopoietic cell transplantation

126 oviruses with defective major splice donors (MSDs) can activate novel splice sites to produce HIV-1 t

127 HLA-matched sibling (matched sibling donors [MSDs]) BMT in 66 patients.

128 estigated the cause of the midsystolic drop (MSD) in left ventricular (LV) ejection velocities that a

129 We use this to correct DWS MSDs by up to a factor of 4 and then calculate [Formula:

130 ric MSD than in children with non-dysenteric MSD (HR 0.20, 95% CI 0.05-0.87, p=0.032), and lower in c

131 n with LSD than in those with non-dysenteric MSD (HR 0.29, 0.14-0.59, p=0.0006).

132 ildren aged 12-59 months with non-dysenteric MSD, 31 occurred among 942 children qPCR-positive for Sh

133 death was lower in children with dysenteric MSD than in children with non-dysenteric MSD (HR 0.20, 9

134 iffusive" regime is only a transient effect, MSD proportional, varianttau(alpha),alpha<1.

135 rved after receipt of transplant from either MSD or URD.

136 on experimental data, both net and ensemble MSD analysis converge to similar values; however, the ne

137 number of trajectories required for ensemble MSD to achieve the same confidence interval.

138 ighly conserved core residues of the HIV Env MSD, in addition to serving as a membrane anchor, play a

139 592 case children aged <5 y old experiencing MSD and for 12,390 asymptomatic age, gender, and neighbo

140 Risk of death is increased following MSD and, to a lesser extent, LSD.

141 tive incidence of graft failure was 9.5% for MSD and 14.5% for UD HSCT.

142 erall survival at 2 years was 95% +/- 5% for MSD and 83% for UD HSCT (p 0.34).

143 Primary outcomes were to characterise, for MSD and LSD, the pathogen-specific attributable risk and

144 ticing open defecation was a risk factor for MSD in children <5 y old in Kenya.

145 a statistically significant risk factor for MSD in Kenya, Mali, Mozambique, and Pakistan.

146 pathogen was isolated and the odds ratio for MSD and the pathogen from conditional logistic regressio

147 Upon exposure to pulling forces, MSD unfolding accelerates exponentially over a force sca

148 worse than those after receipt of BMTs from MSDs (5-year survival, 55% v 63%; RR, 1.35; 95% CI, 1.17

149 ansplants from URDs compared with those from MSDs remains undefined.

150 l cord blood, are comparable with those from MSDs.

151 than those after receipt of transplant from MSDs.

152 romatograph with Mass-Selective Detector (GC-MSD) employing solid-phase microextraction (SPME).

153 The use of SPME-GC-MSD is an effective method to detect volatiles in beers

154 les gas chromatography/mass spectrometry (GC/MSD) with a novel scheme for cryogen-free low-temperatur

155 conformational variability of the HIV-1 gp41 MSD.

156 ), more negative MIN-MAX (P<0.0001), greater MSD (P<0.0001), and larger PSD (P<0.0001).

157 A mean deviation (MD) > MSD indicates a significant difference.

158 It is of interest that in holo-Mb, higher MSD values are observed for the residues outside the hem

159 However, MSD measurements are highly susceptible to static error

160 between the URD and MSD recipients; however, MSD recipients were more likely to achieve B-cell recons

161 95% confidence interval [95% CI], 6%-21%) in MSD groups (P = .15), with respective thalassemia-free s

162 nd control households, are evaluated both in MSD cases and in the population.

163 of the time after 30 years of development in MSD or business as usual (BAU) scenarios, respectively.

164 integral components of the neuropathology in MSD and that modulation of astrocyte function may impact

165 r T-cell engraftment similar to that seen in MSD recipients, and reconstitution rates are adequate.

166 materials, coincident with the cage stage in MSD, the non-Gaussian parameter indicates a significant

167 essential statistical features of the local MSD algorithm.

168 Specifically, the values of the mean MSD exponent and effective diffusion coefficients can be

169 etiologic agents may be responsible for most MSD; (2) a definition of MSD can be crafted that encompa

170 The native MSD unfolds at a resting rate of 9 x 10(-3) s(-1).

171 binding small molecules with or without NBD1/MSD interface mutation.

172 ether, our findings indicate that the NMNAT2-MSD platform provides a sensitive phenotypic screen to d

173 ivity and large dynamic range of this NMNAT2-MSD platform allowed us to screen the Sigma LOPAC librar

174 The IAA detectable by radioassay, but not MSD-IAA, were usually of lower affinity compared with th

175 a subset of IAA with current radioassay (not MSD-IAA) represents biologic false positives in terms of

176 We have developed a novel MSD algorithm, which we refer to as REstrained CONvergen

177 The entire RD group and 88% of MSD group had sustained engraftment.

178 roach that greatly increases the accuracy of MSD measurements is presented herein by combining experi

179 nitation facilities may reduce the burden of MSD in children.

180 For each included case of MSD or LSD, we enrolled one to three community control c

181 ted at six of the seven sites; only cases of MSD and controls were enrolled at the seventh site.

182 tions showed that most attributable cases of MSD and LSD were due to rotavirus, Cryptosporidium spp,

183 esponsible for most MSD; (2) a definition of MSD can be crafted that encompasses cases that might oth

184 search is warranted to assess the effects of MSD-HSCT on clinical outcomes and over longer follow-up.

185 ic strategies will calculate the fraction of MSD attributable to each pathogen and the incidence, fin

186 would substantially increase the fraction of MSD cases that are attributable to Shigella.

187 d location-matched controls, the fraction of MSD cases that were attributable to Shigella infection i

188 hogen-specific population-based incidence of MSD, to guide investments in research and public health

189 k a subset of neurological manifestations of MSD to astrocyte dysfunction.

190 on are used to estimate the annual number of MSD cases and MSD incidence rate attributable to a patho

191 of the incidence, etiology, and outcomes of MSD among infants and young children in sub-Saharan Afri

192 hogens, is estimated using the proportion of MSD cases from whom the pathogen was isolated and the od

193 djusted attributable fraction, proportion of MSD cases taken to a sentinel health center (SHC), numbe

194 The unstressed refolding rate of MSD is ~17 s(-1) and slows exponentially over a force sc

195 nalyzed comparing outcomes for recipients of MSD >/=50 (n = 1415) versus MUD <50 years (n = 757).

196 The repeatability of MSD was better than 0.4 mum.

197 wo other households can increase the risk of MSD in young children, compared to using a private facil

198 but there may be other pathways of onset of MSDs in old age.

199 To summarize the prevalence of MSDs among surgeons performing laparoscopic surgery, we

200 chanical exposure for possible prevention of MSDs in working life and around the time of retirement,

201 1.61, 95% CI: 1.16, 2.22) and both types of MSDs after 11 years (for back MSDs, RR = 1.72, 95% CI: 1

202 Joint exposure predicted both types of MSDs after 4 years among women (for back MSDs, risk rati

203 ears, both exposures predicted both types of MSDs among men.

204 Similar polyQ length-dependent effects on MSD signals were confirmed using endogenous full length

205 nts undergoing either Haplo-HCT (n = 180) or MSD-HCT (n = 807) following reduced-intensity conditioni

206 deletions and insertions within the MPER or MSD result in functionally inactive proteins.

207 concentration, quantified via either SAM or MSD or simply a calibration curve, is of great importanc

208 conditioned HCT from either URDs (n = 37) or MSDs (n = 66).

209 xplanations for the roles of these and other MSD features remain unclear.

210 In particular, the method outperforms MSD analysis in the data-poor limit, thereby giving acce

211 e is validated, and direct improvements over MSD analysis are shown.

212 Differences in thermodynamic parameters, MSD, and resilience were observed for both proteins.

213 separate and joint exposures did not predict MSDs.

214 servation rates are better captured by RECON MSD over multiple conformations and thus multiple local

215 rtantly, the sequence space sampled by RECON MSD resembled the evolutionary sequence space of flexibl

216 ation-specific burden of common work-related MSDs and described a framework for prevention via occupa

217 l ergonomics as a determinant, and reporting MSD prevalence.

218 D) rather than an older HLA-matched sibling (MSD).

219 lecular dynamics simulations yielded similar MSD and resilience values for the two proteins.

220 Although simple, MSD analysis has several pitfalls, including difficulty

221 Our simulations' MSD curves reproduce the three experimentally observed t

222 Since MSD and LSD have similar aetiologies, interventions targ

223 el method for quantifying FAE using a single MSD immunoassay is also reported and confirms previous f

224 d the heme group in holo-Mb showed a smaller MSD and higher resilience compared to the same residue g

225 ltilevel Suicide Data for the United States (MSD-US) replicate several individual and contextual find

226 oximately three times more cases of LSD than MSD in the population, more deaths are expected among ch

227 The MSD group received calcineurin inhibitor-based GVHD prop

228 The MSD in velocity and flow occurs in patients with gradien

229 The MSD scenario generates more tax revenue and water saving

230 By analyzing the MSD, we find that upon densification, our colloidal syst

231 ortant role in the communication between the MSD and NBD as it is predicted to be located at the inte

232 mation about stochastic processes beyond the MSD.

233 In our experiments, the MSD method outperformed the baseline methods, achieving

234 In the MSD approach instead of the sequence of standard additio

235 the RD group and 8% (95% CI, 3%-16%) in the MSD group (P = .83).

236 l glass, marked by logarithmic growth in the MSD toward that previous plateau value.

237 r deleted one, two, or three leucines in the MSD were constructed.

238 marked by the appearance of a plateau in the MSD whose magnitude shrinks with increasing density.

239 n HIV-1 Env upon addition of leucines in the MSD, with +1 and +2 leucine mutations greatly reducing E

240 ide chain snorkels to the inner leaflet, the MSD peptide assumes a metastable conformation consistent

241 the outer leaflet of the viral membrane, the MSD assumes a metastable conformation where the highly-c

242 With medical abolition of obstruction the MSD disappears and the duration and length of contractio

243 144) and a proline near the beginning of the MSD (P148).

244 (T139 and T140) and at the N terminus of the MSD (S143 and T144) and a proline near the beginning of

245 Mechanical unfolding of the MSD activates GPIb-IX signaling into platelets, leading

246 he C terminus (positions 159 and 160) of the MSD as being the boundaries of the two interfaces.

247 However, the amino acid composition of the MSD core does influence the ability of Env to mediate ce

248 We show here that conservation of the MSD core sequence is not required for normal expression,

249 ematic biomechanical characterization of the MSD in its native, full-length receptor complex and a re

250 P with the amino and carboxyl termini of the MSD located intracellularly.

251 Env constructs were deleted, all six of the MSD mutants were able to produce infectious particles.

252 in its predicted hydrophilic regions of the MSD.

253 fects from the local microenvironment on the MSD and potentially generate misleading conclusions abou

254 SRSF2, we analyzed splicing responses on the MSD-L cell line and found that the missense mutation of

255 These data indicate that the MSD is caused by premature termination of LV segmental s

256 Our measurements confirm that the MSD is relatively unstable, with a folding free energy o

257 We showed that the MSD method can classify the time series early by using a

258 We find that the MSD peptide assumes a stable tilted alpha-helical confor

259 etime is prolonged by LRRD unfolding) to the MSD to enhance its unfolding, resulting in unfolding coo

260 n the haploidentical group compared with the MSD group (63% v 91%; P = .001).

261 l is shorter in obstructed patients with the MSD than in nonobstructed HCM patients: septal contracti

262 the role of specific amino acids within the MSD core, we initially replaced the core region with 12

263 diverse transmembrane pathways formed by the MSDs.

264 By contrast, in CFTR the MSDs form a pathway for passive anion flow that is gated

265 nucleotide-binding domains and two or three MSDs function as monomers.

266 ad similar 5-year overall survival (100%) to MSD recipients.

267 Here, we provide an alternative to MSD analysis using the jump distance distribution (JDD),

268 -Cy provides survival outcomes comparable to MSD-HCT, with a significantly lower risk of chronic GVHD

269 Compared to MSD algorithms that enforce (constrain) an identical seq

270 nor hematopoietic stem cell transplantation (MSD-HSCT) can reduce velocities in patients with SCA is

271 Importantly, the unfolded MSD can refold with or without applied forces.

272 The unfolded MSD, particularly the juxtamembrane 'Trigger' sequence t

273 or complex and a recombinant, unglycosylated MSD in isolation.

274 The members of the wwPDB-RCSB PDB (USA), MSD-EBI (Europe), PDBj (Japan) and BMRB (USA)-have remed

275 We use MSD-based analyses to identify contractile steps and fin

276 Using MSD as the reference standard, we determined the optimal

277 of inflammatory cytokines was measured using MSD.Measurements and Main Results: Adding IFN-beta to MD

278 For Haplo-HCT v MSD-HCT, 3-year rates of nonrelapse mortality (15% v 13%

279 use of persistently elevated TCD velocities, MSD-HSCT was significantly associated with lower TCD vel

280 incidence per 100 child-years for LSD versus MSD, by age stratum, for rotavirus was 22.3 versus 5.5 (

281 unfolding intensifies Ca(2+) signal whereas MSD unfolding affects the type of Ca(2+) signal.

282 dren without diarrhoea matched to cases with MSD (n=3597) or LSD (n=4236).

283 223 (2.0%) of 11 108 children with MSD and 43 (0.3%) of 16 369 matched controls died betwee

284 ys later (range 50-90 days) in children with MSD and LSD and in their respective controls.

285 e first eight or nine eligible children with MSD and LSD at each site during each fortnight in three

286 tched controls, in addition to children with MSD and matched controls, were recruited at six of the s

287 ov 14, 2012, we recruited 2368 children with MSD, 3174 with LSD, and one to three randomly selected c

288 RD (50% and 39%, respectively) compared with MSD (22% and 5%, respectively) recipients (P < .01 for b

289 ts (71% and 60%, respectively) compared with MSD recipients (92% and 89%, respectively; P < .01 for b

290 < .0001) were higher after MUD compared with MSD transplants.

291 ed outcomes after Haplo-HCT using PT-Cy with MSD-HCT in patients with lymphoma, using the Center for

292 iagnostic capacity in countries dealing with MSD.

293 Children aged 0-59 months with MSD seeking care at sentinel health centres were recruit

294 In children younger than 24 months with MSD, infection with typical enteropathogenic Escherichia

295 transplant outcomes similar to patients with MSD grafts.

296 r progression to diabetes were positive with MSD-IAA assay.

297 her islet autoantibodies) were positive with MSD-IAA.

298 V, and only cost-effective at long-term with MSD VCV at pound 20,000 per QALY gained threshold.

299 d among children with LSD than in those with MSD.

300 engraftment compared with 97% for those with MSDs; however, estimated 5-year overall and event-free s