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1 MSV enablement increased nAb-PTX efficacy and survival i
2 MSV-nAb-PTX also augmented the accumulation of paclitaxe
3 MSVs provided both a high degree of sensitivity and rapi
4 MSVs, conventional culture, and direct immunofluorescenc
5 g/ul of purified genomic DNA obtained from a MSV-infected maize plant, a sensitivity 10(5) times high
7 e identity between Zambian and other African MSV isolates indicate that this LAMP assay can be used f
9 augmented the accumulation of paclitaxel and MSV in the liver, specifically in macrophages, whereas p
10 ogists individually evaluated DBT images and MSVs of 67 masses (30 malignant, 37 benign) in 67 women
11 transformed Madin-Darby canine kidney cells (MSV-MDCK) have highly reduced levels of E-cadherin and b
12 oloney sarcoma virus-transformed MDCK cells (MSV-MDCK) express low levels of Na,K-ATPase beta(1)-subu
13 the quantified symptom intensities of cloned MSV isolates in differentially resistant maize genotypes
14 thesized two of the most extreme conceivable MSV chimeras, each effectively carrying 182 recombinatio
15 specific and reproducible and able to detect MSV in as little as 10 fg/ul of purified genomic DNA obt
19 Likewise, miR-146a/-181b packaged in ESTA-MSV efficiently suppressed the chemokines, CCL2, CCL5, C
20 Targeted delivery of STAT3 siRNA in ESTA-MSV resulted in knockdown of STAT3 expression in 48.7% o
24 Our data supported our hypothesis that ESTA-MSV microparticle-mediated delivery of miR-146a/-181b am
25 ioaptamer-conjugated multistage vector (ESTA-MSV) drug carrier to bone marrow for the treatment of br
26 E-selectin-targeting multistage vector (ESTA-MSV) to inflamed endothelium covering atherosclerotic pl
28 somal fraction in beta(1)-subunit expressing MSV-MDCK cells compared with MSV-MDCK cells, indicating
29 pha(1)-subunit in beta(1)-subunit expressing MSV-MDCK cells was six- to sevenfold higher compared wit
31 here has been an increase in how extensively MSV colonizes the cells upon which transmission vectors
35 yields upto 440 GCUPS for SSV, 277 GCUPS for MSV and 14.3 GCUPS for P7Viterbi all with 100 % accuracy
39 alpha(1)-subunit protein were comparable in MSV-MDCK and beta(1)-subunit expressing MSV-MDCK cells.
41 e lamellipodia and suppress cell motility in MSV-MDCK cells, independent of Na,K-ATPase ion transport
44 murine sarcoma and leukemia virus complex (M-MSV/M-MuLV), and the induced immune response was monitor
46 in (EGFP) gene expression compared with MLV, MSV LTR, phosphoglycerate kinase, and CMV promoters in T
47 to a nanoporous solid multistage nanovector (MSV) to enable the passage of the drug through the tumor
48 tilizes heuristic-pipeline which consists of MSV/SSV (Multiple/Single ungapped Segment Viterbi) stage
52 mbined PTVs and pathogenic/likely pathogenic MSVs in BRCA1, BRCA2, and TP53 and PTVs in CHEK2 and PAL
53 as for PTVs and pathogenic/likely pathogenic MSVs in BRCA2 (ER-positive BC) and TP53 and PTVs in CHEK
54 evidence of associations of PTVs and/or rare MSVs with CBC risk or BCSS for the 25 suspected BC genes
55 DNA binding motifs into the Rep78-resistant MSV long terminal repeat results in a promoter that has
56 was up to 5-fold enrichment of the targeted MSV in the bone marrow of mice bearing early or late sta
60 a biologically active form of c-ski and the MSV LTR are required for the development of the muscular
61 Our results indicate that MyoD-E12 binds the MSV enhancer with higher affinity and higher cooperativi
62 application to actual FA titration data, the MSV function is simulated, and its predictive ability is
63 ndition of a static quenching mechanism, the MSV postulates an underlying 1:1 fulvic acid (FA)/copper
67 of MyoD, as a heterodimer with E12, with the MSV enhancer, which has six E-box targets for MyoD famil
70 variants (PTVs) and rare missense variants (MSVs) in nine known (ATM, BARD1, BRCA1, BRCA2, CHEK2, PA
73 ink lung and A549 cell lines in shell vials (MSVs) for the detection of respiratory viruses in 159 sp
75 under the control of a murine sarcoma virus (MSV) long terminal repeat (LTR) express the transgene in
76 -ski oncogene from the murine sarcoma virus (MSV) promoter-enhancer display preferential hypertrophy
77 repeat (LTR) promoters-murine sarcoma virus (MSV), MoMLV (MLV), and the LTR (termed Rh-MLV) that is d
81 isease (MSVD), caused by Maize streak virus (MSV; genus Mastrevirus), is one of the most severe and w
82 e intensive tasks within the pipeline (viz., MSV/SSV and P7Viterbi stages) still stand to benefit fro
83 work, we modify the multisite Stern-Volmer (MSV) equation for fitting fluorescence titration curves.
84 unit expressing MSV-MDCK cells compared with MSV-MDCK cells, indicating that in mammalian cells the N
86 y better with DBT (range, 3.2-4.4) than with MSV (range, 3.8-4.8) for all four readers, with one read
87 masses as BI-RADS 4 or 5 with DBT than with MSV, at a cost of five false-positive biopsy recommendat
88 s demonstrated that macrophages treated with MSV-nAb-PTX remained viable and were able to internalize