ライフサイエンスコーパス: MTT

1 MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

2 MTT activity was only significantly reduced by FG4592 or

3 MTT and LDH assays confirmed cytotoxicity of perfluorooc

4 MTT assay of the HPLC fractions identified an active fra

5 MTT assay showed that cell proliferation on denuded AM w

6 MTT assay showed that PEG5K-Fmoc-VE2/DOX exerted signifi

7 MTT assay was performed to quantify mitochondrial stress

8 MTT assays revealed that, of all 30 compounds tested, co

9 MTT assays showed only minor effects of the thioethers a

10 MTT cytotoxicity assay and confocal microscopy images we

11 MTT produced sustained improvement in OR team function,

12 MTT was defined as antibiotics, probiotics, synbiotics,

13 MTT was generally inversely related to BF.

14 MTT, qPCR and immunoblotting assays tested the effects o

15 MTTs in group 2 on days 0 and 2 were significantly longe

16 A total of 4863 MTT debriefings were analyzed.

17 Caspase-3/7, MTT and TUNEL assays elucidated that pEpo-AFPL-TK transf

18 the most active compounds was confirmed in a MTT efficacy assay.

19 MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each compartment

20 l MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(

21 We administered an MTT to 12 obese T2D patients and 15 obese nondiabetic (N

22 d in a cellular HDAC inhibition assay and an MTT assay for cytotoxicity.

23 using a modified Boyden chamber assay and an MTT assay, respectively.

24 Cell proliferation was measured by an MTT assay.

25 of the delivery system was studied using an MTT assay, and by studying the histology of skin samples

26 and to growth factors was evaluated using an MTT assay.

27 (KYSE30) cancer cells were studied using an MTT assay.

28 EPO protein bioactivity was confirmed via an MTT proliferation bioassay.

29 Cellular toxicity was assessed with an MTT dye reduction assay.

30 a significant difference in baseline BF and MTT values between responders and nonresponders (P < or

31 ary permeability or between vessel count and MTT.

32 ll viability was assessed by cell counts and MTT assay, and apoptosis was measured by nucleosomal deg

33 ll viability were measured using H(2)DCF and MTT assays, respectively.

34 nin was carried out using both live/dead and MTT assays.

35 7) than control rats, and lower MTR, DV, and MTT (P = .014, .001, and .010, respectively; alpha = .01

36 There was an increase in F(a), ART, DV, and MTT and a decrease in PV in patients with advanced fibro

37 o compare F(a), F(p), F(t), ART, PV, DV, and MTT between groups.

38 Trypan blue exclusion and MTT assays evaluated TSA cytotoxicity to the cornea.

39 Using trypan blue exclusion and MTT assays, there was no evidence of cell toxicity with

40 n primarily by thioflavin T fluorescence and MTT.

41 here were significant differences in GFR and MTT(K) between the acute dysfunction group (36.4 mL/min

42 and PS values were significantly higher and MTT values were significantly lower (P<.01) with the cur

43 s within the as-synthesized zeolites ITW and MTT that, in conjunction with synchrotron X-ray diffract

44 or as-synthesized siliceous zeolites ITW and MTT.

45 LDH and MTT assays demonstrate that peptide 1a is toxic toward S

46 Cell death was assayed by LDH and MTT methods.

47 with histology, cell volume measurement, and MTT assay.

48 Electron microscopy and MTT assay of the mutants revealed abnormal mitochondria

49 ondral area with low or no detectable PF and MTT adjacent to the joint surface, which was surrounded

50 PF and MTT were correlated with CCl(4) treatment duration from

51 ts (P < .001 and P = .03, respectively), and MTT was shorter on day 2 than on day 0 in group 2 (P = .

52 Nuclear staining and MTT assays following dsRNA stimulation revealed two subp

53 tic resonance (NMR), chemical synthesis, and MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

54 cal, hypochlorous acid), cytotoxicity assay (MTT) and quantification of TNF-alpha production in RAW 2

55 -diphenyltetrazolium bromide formazan assay (MTT assay) as a reporter of Abeta-mediated neuronal cell

56 From these preliminary cytotoxicity assays (MTT) we found that C8-propyl-catechin gallate was more a

57 tional effects analyzed using kinase assays, MTT assays were used to assess cell viability as a marke

58 Two cell viability assays, MTT and lactate dehydrogenase (LDH), and an assay measur

59 ity observed for C(alc) (147.0% +/- 3.1%) at MTT assay in MDCK1 cell culture.

60 A cell-based MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazoliu

61 e found to be active in the human cell-based MTT metabolic activity assay.

62 MDCT-based MTT and PBF measurements demonstrate globally increased

63 Both MTT and PrestoBlue assays showed higher cell viability a

64 hiazol 2-yl)-2,5-diphenyltetrazolium bromide MTT assays.

65 l)-2,5-diphenyl diphenyltetrazolium bromide (MTT) assay and a lactate dehydrogenase (LDH) release ass

66 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assay, flow cytometry with propidium iodide, gene e

67 hiazolyl-2)-2,5-diphenyltetrazolium bromide (MTT) assay.

68 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) assay.

69 iazol-2-yl]-2,5-diphenyltetrazolium bromide (MTT) assay.

70 azol-2-cyl)-2,5-Diphenyltetrazolium Bromide (MTT) assay.

71 azole-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction assay, superoxide scavenging activity, re

72 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction, lactate dehydrogenase release, and [(3)H

73 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT), a substrate commonly used to measure cell viabilit

74 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT), and AlamarBlue cell viability assays.

75 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT), apoptosis and colony formation), and chelation of

76 iazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT).

77 azol-2-yl)-2,5-diphenyl tetrasodium bromide (MTT) and Alamar Blue cell viability assays.

78 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assay and cell counting, the expression of alpha-SM

79 azol-2-yl]-2,5 diphenyl tetrazolium bromide (MTT) assay with photoabsorbance testing was used to asse

80 azolyl)-2,5-diphenyl-2H-tetrazolium bromide (MTT) assay, >28.90 to 13,157.89].

81 methylthiazolyldiphenyl-tetrazolium bromide (MTT) assay, Hoechst staining and caspase-3 activation, a

82 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assay, lactate dehydrogenase (LDH) release assay, H

83 methylthiazolyldiphenyl-tetrazolium bromide (MTT) assays and significant tumorigenicity in in vivo al

84 azol-2-yl)-2,5-diphenyl tetrazolium bromide (MTT) assays.

85 azol-2-yl)-2,5-diphenyl-tetrazolium bromide (MTT), and apoptosis assays.

86 azol-2-yl)-2,5-diphenyltetrazolium bromide, (MTT) whereas apoptosis was determined by DNA fragmentati

87 azol-2-yl)-2,5-diphenyltetrazolium bromide] (MTT), glucose-6-phosphate dehydrogenase (G6DP), and calc

88 images, with within-patient CVs for BF, BV, MTT, and PS of 11.2%, 14.4%, 5.5% and 12.1%, respectivel

89 For tumors, BF, BV, MTT, and PS values and reproducibility varied by analyti

90 lammatory, antimicrobial and antioxidant, by MTT, nitric oxide inhibitory assay, agar disc diffusion

91 mbrane matrix and fibronectin was assayed by MTT.

92 The affect of Usp and Imu1-3 was assessed by MTT and Comet assays, infection assays, caspase 3/7 acti

93 ty on PC12 cell lines (viability assessed by MTT assay and intracellular ROS production by DCFH-DA as

94 The cell proliferation rate was assessed by MTT assay and with the electric cell-substrate impedance

95 ell viability in wild-type mice, assessed by MTT assay, was approximately half of that in contralater

96 Viability of RGC-5 cells was assessed by MTT assays.

97 ellular reducing equivalents was assessed by MTT dye reduction and NAD(P)H assays, and cell survival

98 to the aggregation reactions, as assessed by MTT metabolic activity measurements.

99 ity and metabolic activity was calculated by MTT assay.

100 l of human PDLMSCs and GMSCs was compared by MTT assay.

101 rmal keratinocytes (NHEKs) were conducted by MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

102 viability was assessed after 1 to 3 days by MTT assay.

103 umor cell growth in vitro as demonstrated by MTT and clonogenic assays.

104 Cell viability was detected by MTT assay.

105 neuroblastoma SH-SY5Y cells as determined by MTT and LDH release assays.

106 l viability and apoptosis were determined by MTT assay and caspase-3 (DEVDase) activity.

107 ed by cell count, toxicity was determined by MTT assay, and neoplastic transformation was assessed by

108 12 pheochromocytoma cells were determined by MTT assay, while the cell differentiation was evaluated

109 ur human cancer cell lines was determined by MTT assay, yielding dose- and cancer cell line-dependent

110 Cell viability was determined by MTT assay.

111 mercial HEK cell-produced hOPN determined by MTT assay.

112 l lines (A549, CH1, SW480) was determined by MTT assays, yielding IC(50) values of 6-60 muM for three

113 Cell viability was determined by MTT reduction or LDH release assays.

114 and fibrin-based scaffolds was evaluated by MTT and live/dead assay.

115 in HBVEC treated with Heme was evaluated by MTT and TUNEL assay.

116 Cytotoxicity was evaluated by MTT assay and flow cytometry analysis, while genotoxicit

117 y in a cancerous cell lines was evaluated by MTT assay in the presence and absence of the corona.

118 GNP-HCIm cytotoxicity was evaluated by MTT assay, apoptosis/necrosis and phosphorylated-cAbl (c

119 alcein-AM, and cytotoxicity was evaluated by MTT assay.

120 of MCF-7 and SK-BR-3 cells was evaluated by MTT assays.

121 orioallantoic membrane (CAM) was examined by MTT assay, BrdU labeling, cell proliferation assay, cell

122 ced T cell/FLS proliferation was examined by MTT assay.

123 iability and cell death were investigated by MTT, terminal deoxynucleotidyl transferase-mediated dUTP

124 rative activity on human tumor cell lines by MTT assay, for antioxidant potential by DPPH, ABTS and F

125 tions in tumor cell survival, as measured by MTT and crystal violet assays, regardless of IGF1 pre-tr

126 Cell viability/apoptosis was measured by MTT assay and Annexin V/PI staining , activation related

127 s, and ~45% cancer cell death as measured by MTT assay, when illuminated with 980 nm NIR light.

128 Cell proliferation was measured by MTT assay.

129 Cell growth was measured by MTT assays.

130 peripheral blood mononuclear cells (PBMC) by MTT assay.

131 Tramp-C1 cells affects cell proliferation by MTT assays.

132 Cytoprotection was quantified by MTT assay.

133 Cell growth test by MTT assay showed that induction of Cx50P59A decreased ce

134 herapeutic effect was determined in vitro by MTT assay, [(18)F]fluorodeoxyglucose (FDG)- and [(18)F]f

135 MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

136 tective/cytotoxic effects upon Caco-2 cells (MTT, cell cycle and reactive oxygen species (ROS)) were

137 f fragmented beta2m fibrils rescued cellular MTT reduction.

138 meters were also optimized for colorimetric (MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

139 (C)) and fractional MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(

140 (T)), and the collecting system compartment (MTT(C)) were calculated.

141 mpartment (MTT(A)), the tubular compartment (MTT(T)), and the collecting system compartment (MTT(C))

142 of the tracer for the vascular compartment (MTT(A)), the tubular compartment (MTT(T)), and the colle

143 Conversely, MTT was longer with the distributed parameter model than

144 py, ELISA), toxicity assays in cell culture (MTT and lactate dehydrogenase in human SH-SHY5Y cells, m

145 vival, which was detected by flow cytometry, MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

146 Cytotoxicity (MTT test) and genotoxicity (micronuclei assay) were not

147 The cytotoxicity (MTT assay) on HaCaT cell line demonstrated high cytocomp

148 alpha neutralisation and rTNFalpha decreased MTT and NFI further.

149 -dimethylthiazol-2-yl)-3,5-diphenylformazan (MTT) to quantitatively determine tissue viability.

150 methylthiazol-2-yl)-2,5-diphenyltetrazolium (MTT) and clonogenic assays]; indolequinones displayed po

151 retrospective and prospective thought (i.e., MTT), here we explored the possibility that the spatiali

152 f spatiotemporal data, we suggest estimating MTTs at the disequilibrium state via long-term data assi

153 The Pearson coefficient for blood flow, MTT, K(trans), and PS at the two ROIs demonstrated good

154 One year following MTT, case delays decreased (23% to 10%, P < 0.0001), mea

155 nt LoA were -35% for BF,-43% for BV, 33% for MTT, and -79% for PS.

156 , good (>0.75) for PS, and lower (>0.38) for MTT, irrespective of software version.

157 readers for BF were -33%; for BV, -39%; for MTT, 55%; and for PS, -93%.

158 s were isolated and seeded (0.5 x 10(4)) for MTT assay.

159 for BV, or an increase of more than 55% for MTT, could be considered beyond the analysis variability

160 At their EC50 concentrations for MTT reduction, all four APs disrupted cellular ultrastru

161 etween TBMES and osteonecrosis was found for MTT (P = .09) and PF (P = .75) in the surrounding area.

162 dies of structure-directing agents (SDA) for MTT-type zeolites, a large number of amines or quaternar

163 e successfully predicted to be selective for MTT phases.

164 ) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T

165 According to the obtained data from MTT assay, these structures have concentration-dependent

166 C sink globally; (b) the SSA-induced bias in MTT and NEP correlates negatively with forest age, which

167 ed climate change, spatiotemporal changes in MTT are likely to be underestimated, thereby resulting i

168 significant reduction in BF and increase in MTT (P < or =.05).

169 L-15 and rTNFalpha, limited the reduction in MTT and nuclear fusion index (NFI) associated with rTNFa

170 Biological evaluation includes MTT and cellular HDAC assays on sensitive and chemoresis

171 ed to evaluate cytotoxicity by incorporating MTT dye, and genotoxicity was evaluated with the micronu

172 nificantly reduced LDH release and increased MTT activity compared to vehicle (1% DMSO) pretreatment.

173 conceptualization of time that may influence MTT as well as other temporally relevant cognitive pheno

174 es in neuroscience and biologically inspired MTT methods in computer vision and discuss the ways in w

175 OR personal completed a 1-day intensive MTT training.

176 ircumscribed rim of high PF and intermediate MTT, which was only found in joints with osteonecrosis,

177 rounded by a rim of high PF and intermediate MTT.

178 actional MTT of each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(

179 MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

180 rived was MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each com

181 rtment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

182 ) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

183 each compartment (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

184 Also derived was MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of

185 rest were drawn in areas of high PF and long MTT on each parametric map.

186 w MTT that was surrounded by an area of long MTT and low PF.

187 ubchondral elongated area of high PF and low MTT that was surrounded by an area of long MTT and low P

188 y (EC50 = 1.1 x 10(-9) M vs 1.8 x 10(-11) M, MTT test) in agreement with a reduced binding affinity (

189 18.9 mL/100 mL per minute +/- 11.0 and mean MTT was 213.3 seconds +/- 56.8.

190 ude the NIHSS and the perfusion measurements MTT and Tmax.

191 y were analysed in methylthiazoltetrazolium (MTT) and trypan blue assays.

192 A multidisciplinary MTT committee reviewed and rectified any systems problem

193 rIL-15 increased the thickness of myotubes (MTT) from both age groups to a similar extent.

194 rvival of serum-starved C2C12, HSM, and NCM (MTT, trypan blue) and prevented taxol-induced apoptosis

195 This is the largest case analysis of MTT and one of the few to document an impact of MTT on o

196 conditions necessary for crystallization of MTT phases in borosilicate preparations with some of the

197 hat are selective for the crystallization of MTT-type zeolite phases.

198 nstrate the long-term safety and efficacy of MTT as a potential therapy to treat children with ASD wh

199 and one of the few to document an impact of MTT on objective measures of operating room function and

200 Results of MTT- and anchorage-independent growth assays and cell cy

201 -aged ecosystems; and (c) the sensitivity of MTT to temperature and precipitation was 22% and 42% low

202 ion and the following assays were performed: MTT, CellTox(TM) Green Cytotoxicity, CellTiter-Glo(R), N

203 gh-level debriefing/problem-solving process, MTT can be a foundation for improving OR performance.

204 tions these molecules usually do not produce MTT phases.

205 f these materials were evaluated by protein, MTT, and LDH assays, which demonstrate that all the clic

206 hat enrolled persons with NAFLD who received MTT rather than placebo or usual care.

207 APs impaired the ability of HBMECs to reduce MTT which was followed by decreased Trypan blue exclusio

208 and mHAMs have similar potencies in reducing MTT activity, mHAMs induced greater Nrf2-mediated oxidat

209 Cell viability-related (MTT assay) studies clearly showed that nitrones 1-3 and

210 In this paper, we review MTT studies in neuroscience and biologically inspired MT

211 hree patients with high initial BF and short MTT showed poor response.

212 Rectal cancer showed higher BF and shorter MTT compared with those of normal rectum (P < or =.05).

213 treatment (EC(50) = 34.69 nM to 7.52 muM; SI-MTT = 5.24 to > 1,441.33) settings.

214 treatment (EC(50) = 0.14 muM to 0.55 muM; SI-MTT = 70.12 to >357.14) or cotreatment (EC(50) = 34.69 n

215 In contrast, the very similar MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

216 cell viability at 24 and 72 hours post-SMA (MTT assay).

217 ements, microscopic examination and standard MTT viability assays.

218 the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT(C)) and fractional MTT of each compartment (MTT(A/K)

219 (MTT(A/K) = MTT(A)/MTT(K), MTT(T/K) = MTT(T)/MTT(K), MTT(C/K) = MTT(C)/MTT(K)).

220 ults using a conventional cytotoxicity test (MTT).

221 logical stimulus, such as a mixed meal test (MTT), has not been determined.

222 on electron microscopy (TEM), scratch tests, MTT reduction cytotoxicity, HOS cell bioactivity (human

223 using a more traditional methyl tetrazolium (MTT) cytotoxicity assay at selected time points followin

224 The MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

225 The MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazodiu

226 The MTT assay confirmed these results for PRO 2000 (4% and 0

227 The MTT assay was confounded by the reduction of the MTT rea

228 The MTT assay was used to test the extract on the effect of

229 The MTT(A/K) was significantly higher in the acute rejection

230 The MTT(T/K) was significantly higher in the ATN group (mean

231 analyzed by immunostaining for Ki67 and the MTT assay.

232 cell proliferation-enhancing activity by the MTT assay and anchorage-independent growth in soft agar.

233 th cell lines was normal, as measured by the MTT assay and markers of cytotoxicity, cell cycle, apopt

234 A/CWO/PTX") formulation was confirmed by the MTT assay in cancer cell cultures.

235 Cell viability was assessed by the MTT assay.

236 ls in 2D, whose toxicity was measured by the MTT assay.

237 prostate cancer cell proliferation from the MTT assay and flow cytometry investigation.

238 ytotoxic refill fluids were effective in the MTT assay, and ethyl maltol, which was in over 50% of th

239 In the MTT assay, ExMh showed the highest cytotoxicity, especia

240 % of the refill fluids were cytotoxic in the MTT assay.

241 l lines Y79 and WERI-Rb1 with the use of the MTT assay, BrdU incorporation assay, flow cytometry, imm

242 lines (CH1, SW480, and A549) by means of the MTT assay, featuring IC(50) values to the low micromolar

243 lines (CH1, SW480 and A549) by means of the MTT colorimetric assay.

244 assay was confounded by the reduction of the MTT reagent by honey's reducing sugars and phenolic comp

245 had lesions of the MD without lesions of the MTT.

246 e to the toxic effects of glutamate than the MTT assay.

247 latin at micromolar concentrations using the MTT and cell invasion assay.

248 1, T24 and 5637 cells was assessed using the MTT assay.

249 gree of cell death was established using the MTT assay.

250 the flavonoids after 24 hours, by using the MTT assay.

251 e cytotoxic activity was evaluated using the MTT assay.

252 riple-negative breast cancer cells using the MTT assays.

253 micals were tested for cytotoxicity with the MTT assay using human lung fibroblasts and epithelial ce

254 tion in cell survival as determined with the MTT assay.

255 e level of cell death was monitored with the MTT assay.

256 48-h treatment with FeSO(4), tested with the MTT viability assay.

257 n the one-dimensional pore zeolites with the MTT, TON, and MTW frameworks.

258 ic effects of microbiome-targeted therapies (MTTs) in persons with NAFLD.

259 -label trial of Microbiota Transfer Therapy (MTT) that combined antibiotics, a bowel cleanse, a stoma

260 wer (>0.30 and >0.39) for mean transit time (MTT) and permeability surface area product (PS), respect

261 Parameter maps for mean transit time (MTT) and plasma flow (PF) were evaluated qualitatively a

262 ribution volume (DV), and mean transit time (MTT) of gadopentetate dimeglumine.

263 ration rate (GFR) and the mean transit time (MTT) of the tracer for the vascular compartment (MTT(A))

264 (BF), blood volume (BV), mean transit time (MTT), and capillary permeability-surface area product we

265 (BF), blood volume (BV), mean transit time (MTT), and permeability-surface area product (PS) for tum

266 (BF), blood volume (BV), mean transit time (MTT), and permeability-surface area product were measure

267 Distribution volume (DV), mean transit time (MTT), and portal fraction (PF) of blood inflow were calc

268 flow (BF), blood volume, mean transit time (MTT), and vascular permeability-surface area product.

269 Blood flow, blood volume, mean transit time (MTT), permeability-surface area product, extraction frac

270 BV), blood flow (BF), and mean transit time (MTT), were calculated at the primary site.

271 ary blood flow (PBF), and mean transit time (MTT).

272 (BF), Blood Volume (BV), Mean Transit Time (MTT)] and permeability parameters [including endothelial

273 ficantly underestimated mean turnover times (MTTs) by 29%, thereby leading to a 4.83-fold underestima

274 cantly shorter than the distant liver tissue MTT at 2.5 mug/mL only (9.7 vs 15.3 sec, P = .0006).

275 antly shorter than the adjacent liver tissue MTT at angiotensin II doses of 2.5 mug/mL (9.7 vs 15.8 s

276 ligence, the human multiple-target tracking (MTT) ability is rarely imitated in computer-vision algor

277 postulated that the mammillo-thalamic tract (MTT)/anterior thalamic nucleus (AN) complex would be cri

278 Medical team training (MTT) has been touted as a way to improve teamwork and pa

279 nd now, this faculty for mental time travel (MTT) is dependent upon an underlying cognitive represent

280 Tumor MTT was significantly shorter than the adjacent liver ti

281 Unlike MTT, WST-1 reagent is reduced extracellularly through tr

282 l lines of NIH/3T3, CCD18-Co and B98-5 using MTT assay.

283 The lutein emulsions were analysed using MTT assay on the gut enterocyte cell line Caco-2 and the

284 ntial function of miR-497 in HUVECs by using MTT and TUNEL assays.

285 proliferation/survival in these cells using MTT, (3)H-thymidine uptake and Annexin-V apoptosis assay

286 d MDCK cells against ADR (demonstrated using MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

287 sing human tumor cells was established using MTT assays.

288 ible hepatic toxicity was investigated using MTT assay on HepG2 cells.

289 A (normal epithelial breast cell line) using MTT assay, where they showed highest inhibitory activity

290 re and after treatment with nifurtimox using MTT assays.

291 FEPO's cytotoxicity was tested using MTT (2-(4,5-dimethyl-2-thiazolyl)-3,5-diphenyl-2H-tetraz

292 he pancreatic cancer cell lines tested using MTT and colony formation assays.

293 ox and BSO and evaluated for viability using MTT assays.

294 ponders existed in K(trans), Kep, Ve, Ve_SD, MTT, BV_SD and MTT_SD (P < 0.05).

295 PA treatment by analyzing cell viability via MTT assay, neurosphere formation, and endoplasmic reticu

296 ere confirmed by Apoptag and cell viability (MTT) assays supporting the ability of PDT-BIAS to induce

297 Also derived was MTT for the whole kidney (MTT(K) = MTT(A) + MTT(T) + MTT

298 Cell proliferation was measured with MTT [3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazoliu

299 ls up to 0.25 mg/ml extract as measured with MTT and LDH-leakage assays.

300 ve oxygen species (ROS) were quantified with MTT, ApoTox-Glo, and ROS-Glo H(2)O(2) assays, respective

301 By comparison, for as-synthesized zeolite MTT, F(-) anions reside within the 10-ring channels and