ライフサイエンスコーパス: MVV

1 MVV infection induces axonal damage with some areas of n

2 +/- 0.24 versus 0.95 +/- 0.31 L, p < 0.001), MVV (28 +/- 11 versus 41 +/- 13 L, p < 0.001), resting P

3 ctional receptors for the two North American MVV strains tested, unlike the Icelandic MVV and similar

4 EV CO, and lytic and nonlytic North American MVV strains to cells of different species.

5 some common motifs between the HIV-1 Vif and MVV Vif are involved in recruiting Cul5, different deter

6 tepwise multiple regression model, %DCO and %MVV combined were the best predictors of dyspnea severit

7 icate a differential receptor recognition by MVV strains which is unrelated to cytopathic phenotype.

8 ral constructs and plasmids expressing CAEV, MVV, or vesicular stomatitis virus envelope glycoprotein

9 in), Watts (45 +/- 48 versus 71 +/- 59), V E/MVV (88 +/- 33 versus 79 +/- 14), and HRmax (117 +/- 17

10 h mechanical ventilatory limitation (V Emax/ MVV 0.81 versus 0.58; p = 0.02).

11 and catalytic core domains are critical for MVV IN strand transfer activity in vitro and for virus i

12 Icelandic MVV strains, which are lytic in tissue culture, have a w

13 d the wide species distribution of Icelandic MVV receptors and the narrow host range of CAEV.

14 can MVV strains tested, unlike the Icelandic MVV and similar to CAEV, were limited to cells of rumina

15 ve improvement in dyspnea were the change in MVV, change in resting arterial PaO2, and change in FEV1

16 en demonstrated not only to carry infectious MVV but also to be hosts of the virus themselves.

17 conserved and unique structural elements of MVV Vif and its common and distinct interaction modes wi

18 eudotyped with the envelope glycoproteins of MVV K1514, CAEV CO, and lytic and nonlytic North America

19 dvance our understanding of the mechanism of MVV Vif recruitment of cellular factors and the evolutio

20 functional receptors is a common property of MVV strains or related to cytopathic phenotype, we teste

21 LEDGF/p75 results in gross redistribution of MVV integration sites in human and ovine cells.

22 rt the cryo-electron microscopy structure of MVV Vif in complex with CypA and E3 ligase components.

23 tion assays to study motifs required for the MVV Vif to bind zinc ion, Cul5, and the cofactor CypA.

24 cruiting Cul5, different determinants in the MVV Vif are required for cofactor binding and stabilizat

25 tudies suggest that the early effects of the MVV tat peptide may involve glutamate neurotoxicity via

26 peptide derived from the basic region of the MVV transactivating protein Tat causes considerable neur

27 4 L/min), which represented 60 +/- 2% of the MVV.

28 unction with photobleaching reveals that the MVV intasome can bind a variable number, up to sixteen m

29 ception of the change in Dyspneic Index [(VE/MVV)100] at maximum exercise.

30 s 12.1 [10.4-14.8] vs 9.4 [9.1-12.4]), or VE/MVV (80.4% [72.6-88.3] vs 57.8 [52.1-92.6] vs 63.9 [34.5

31 ilation/maximum ventilatory volume ratio [VE/MVV]) were measured continuously in patients with COPD d

32 VEmax/MVV was 67.2 +/- 4.0% in SLT recipients and 48.5 +/- 3.6

33 of their 12-s maximum voluntary ventilation (MVV) until task failure.

34 and increased maximum voluntary ventilation [MVV]) following the surgically induced reduction in resi

35 minant lentiviruses ovine maedi-visna virus (MVV) and caprine arthritis-encephalitis virus (CAEV) cau

36 nodeficiency virus (BIV), maedi-visna virus (MVV) and equine infectious anemia virus (EIAV) readily i

37 Lentiviruses such as Maedi Visna virus (MVV) in sheep, and human immunodeficiency virus (HIV) in

38 Maedi-visna virus (MVV) is a natural pathogen of sheep with a tropism for m

39 Maedi-visna virus (MVV) sequesters CypA as the noncanonical cofactor for th

40 tabilize the complex, the maedi-visna virus (MVV) Vif hijacks cyclophilin A (CypA) instead.

41 Maedi-visna virus (MVV) Vif is similarly promiscuous, degrading not only sh

42 of the intasome from the maedi-visna virus (MVV), an ovine lentivirus, revealed a large assembly con

43 novel metric named Macular Vascular Volume (MVV), were extracted to assess consistency across two-ti