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1 ominated by skin-adapted yeasts of the genus Malassezia.
2 dominated by only 1 genus, either Candida or Malassezia.
3 hes, such as Microsporidia, Pneumocystis and Malassezia.
4 kin commensal yeasts are the basidiomycetes, Malassezia.
5 coccus and a fungal genus-specific probe for Malassezia.
6 ysis indicated that most organisms resembled Malassezia.
7 d mycobiome was predominantly represented by Malassezia (43.6%), Curvularia (18.5%) and Aspergillus (
8 hile the NF mycobiome was nearly exclusively Malassezia (84.2%) with an absence of Aspergillus or dem
9 abolic genes in the altered cutaneous niche, Malassezia acquired enhanced fitness to efficiently colo
10 ive abundance in children than adults, while Malassezia and certain food-associated fungi were lower
11 ding members of the genera Pseudogymnoascus, Malassezia and Rhodotorula, which include some taxa repo
12                                              Malassezia are involved in disorders including dandruff
13  further studies needed to establish whether Malassezia are metabolically active oral commensals.
14                                              Malassezia are the most abundant fungal skin inhabitant
15             Fungi in the basidiomycete genus Malassezia are the most prevalent eukaryotic microbes re
16                           Fungi in the genus Malassezia are ubiquitous skin residents of humans and o
17 ommunity types (mycotypes), with Candida and Malassezia as the main taxa driving cluster partitioning
18 f 24 subjects, in which cultivation revealed Malassezia as viable oral mycobiome members, although th
19 gether, we show that this dominant secretory Malassezia aspartyl protease has an important role in en
20 obiome of AFRS, with a mycobiome abundant in Malassezia, Aspergillus and Curvularia.
21                         In a murine model of Malassezia colonization, we further demonstrated Mfsap1
22 e high-resolution X-ray crystal structure of Malassezia flavohemoglobin revealed features conserved w
23       In vivo pathogenesis is independent of Malassezia flavohemoglobin.
24                                              Malassezia form the dominant eukaryotic microbial commun
25                        The lipophilic fungus Malassezia furfur (M. furfur) is a commensal microbe ass
26 y, in an approach to uncover a sexual cycle, Malassezia furfur strains were engineered to express dif
27  Candida albicans, Saccharomyces cerevisiae, Malassezia furfur, and Cryptococcus neoformans.
28 act dermatitis and increased specific IgE to Malassezia furfur, but not with filaggrin gene defect.
29                                              Malassezia furfur, Malassezia globosa, and Malassezia sy
30 es were identified as CENP-A-rich regions in Malassezia furfur, which has seven chromosomes, and hist
31 hereas the most common non-Candida yeast was Malassezia furfur, which the automated system failed to
32 protease MFSAP1 in the genetically tractable Malassezia furfur.
33                                  Analysis of Malassezia genomes revealed that key adaptations to the
34                                          The Malassezia genus possesses a repertoire of secretory hyd
35 enomics and phylogenetic analyses within the Malassezia genus revealed that flavohemoglobin-encoding
36  result, in part, from metabolic activity of Malassezia globosa and Malassezia restricta, commensal b
37                                              Malassezia globosa cytochromes P450 CYP51 and CYP5218 ar
38 se (CA, EC 4.2.1.1) from the fungal pathogen Malassezia globosa has been cloned, characterized, and s
39 es, phospholipases, and sphingomyelinases of Malassezia globosa in healthy subjects and seborrheic de
40     Species-level classification showed that Malassezia globosa predominated in children.
41 Here, we characterized the dominant secreted Malassezia globosa protease in culture and subsequently
42 rotease in culture and subsequently named it Malassezia globosa Secreted Aspartyl Protease 1 (MgSAP1)
43 depleted regions in Malassezia slooffiae and Malassezia globosa with nine chromosomes each.
44                           Malassezia furfur, Malassezia globosa, and Malassezia sympodialis are most
45 lex interactions with the dominant eukaryote Malassezia globosa.
46 roteome, and expression of selected genes of Malassezia globosa.
47                                              Malassezia grows excessively in AD-like skin.
48 ically, Staphylococcus species and the fungi Malassezia had an outsized contribution to metatranscrip
49 ymnoascus, Penicillium, Meyerozyma, Lecidea, Malassezia, Hanseniaspora, Austroplaca, Mortierella, Rho
50                                   Species of Malassezia have been connected to skin and opportunistic
51 imon et al. (2019) present murine models for Malassezia/host interaction and describe a role for Mala
52 ifferential expression of metabolic genes in Malassezia in atopic and control skin was quantified.
53                    Despite the importance of Malassezia in common skin diseases, remarkably little is
54 zia/host interaction and describe a role for Malassezia in inflammatory skin and gut disease.
55 our study defines a role for the skin fungus Malassezia in inter-kingdom interactions and suggests th
56 a greater abundance of Candida in plaque and Malassezia in saliva.
57 ase activity was detected in four species of Malassezia, including M. globosa.
58 potheses about the mechanism of D/SD include Malassezia-induced fatty acid metabolism, particularly l
59                                         Host-Malassezia interactions and mechanisms to evade local im
60  as a single species residing on human skin, Malassezia is now recognized as a diverse genus comprisi
61                                              Malassezia is the dominant fungus in the human skin myco
62                                        While Malassezia is the most abundant commensal fungus of the
63 ial oral colonizers, such as lipid-dependent Malassezia, is still unclear, with further studies neede
64 ing-type genes, providing an indication that Malassezia may be capable of sex.
65                       In three subjects, the Malassezia microbiota composition appeared relatively st
66                                   Samples of Malassezia microbiota from healthy skin and psoriatic le
67 andida mycotype had lower diversity than the Malassezia mycotype and was positively correlated with c
68 wer diversity than those associated with the Malassezia mycotype, suggesting that common environmenta
69 and inflammophilic bacteria increased in the Malassezia mycotype.
70  showed low sensitivity for detection of the Malassezia mycotype.
71      These data indicate the predominance of Malassezia organisms in healthy human skin, host-specifi
72 beta-1,2-mannosyloxymannitol glycolipid from Malassezia pachydermatis 44-2, which was reported to sig
73 resent the analysis of the volatile space of Malassezia pachydermatis grown at three pH values (5.7,
74              When a cluster of patients with Malassezia pachydermatis infection was identified in an
75 ome-level genome assemblies, and an improved Malassezia phylogeny, we infer that the pseudobipolar ar
76 uding Cryptococcus, Filobasidium, Kloeckera, Malassezia, Pichia, Sporidiobolus, Rhodotorula, Zygosacc
77            However, it has become clear that Malassezia plays a multifaceted role in human health, wi
78  in healthy adults, showing lipophilic fungi Malassezia predominate in most skin sites.
79                                 Among fungi, Malassezia predominated at most skin sites in more sexua
80                                     Although Malassezia predominated on the trunk, head, and arm skin
81 es, and showed their value for understanding Malassezia reproduction by confirming four alternative a
82 ant gut mycobiome (Rhodotorula mucilaginosa, Malassezia restricta and Candida albicans) that correlat
83  etiologies were detected in 13/16 cases and Malassezia restricta in the 3 other cases.
84                                              Malassezia restricta is one of the most predominant yeas
85 rvey of the genome and secretory proteome of Malassezia restricta, a close relative implicated in sim
86 metabolic activity of Malassezia globosa and Malassezia restricta, commensal basidiomycete yeasts com
87 ne of these, the common skin resident fungus Malassezia restricta, is also linked to the presence of
88 coccus epidermidis, Cutibacterium acnes, and Malassezia restricta, with the latter 2 being the most a
89                      The surging interest in Malassezia results from development of tools to improve
90 robe interactions will be required to define Malassezia's role in human and animal health and disease
91                       The exact role of most Malassezia secreted enzymes, including those in interact
92 omosomes, and histone H3 depleted regions in Malassezia slooffiae and Malassezia globosa with nine ch
93                               All cultivated Malassezia species are lipid dependent, having lost gene
94                                              Malassezia species are lipophilic yeasts that are emergi
95                                              Malassezia species are ubiquitous residents of human ski
96 centromeres drive karyotype diversity in the Malassezia species complex through breakage and inactiva
97 bundance Malassezia sympodialis was the only Malassezia species recovered.
98                                              Malassezia species show variable karyotypes ranging betw
99  or two MAT loci on separate chromosomes, in Malassezia species studied thus far the two MAT loci are
100 asive models of PDA, and repopulation with a Malassezia species-but not species in the genera Candida
101 ies or as a result of skin colonization with Malassezia species; cutaneous infection with other funga
102                                              Malassezia-specific 5.8S/ITS2 analysis of 1,374 clones i
103 8S rDNA/internal transcribed spacer 2 (ITS2) Malassezia-specific PCR primers.
104 sicolor is common worldwide and is caused by Malassezia spp, which are human saprophytes that sometim
105 1 to 2 log(10) lower than for bacteria, with Malassezia spp. accounting for the majority of fungal ge
106 trated PDA tumours was markedly enriched for Malassezia spp. in both mice and humans.
107 us sites, where lipophilic Cutibacterium and Malassezia spp. predominate.
108 tudy, MC903-treated mice were colonized with Malassezia spp. to assess the host-fungal interactions i
109  skin is predominantly obligatory lipophilic Malassezia, suggesting that fungal communities on skin p
110  genome assembly of the skin commensal yeast Malassezia sympodialis and demonstrate how proteogenomic
111   Malassezia furfur, Malassezia globosa, and Malassezia sympodialis are most closely linked to tinea
112 stance mediated by the fungal skin commensal Malassezia sympodialis exerts evolutionary pressures on
113                  Recently, it was found that Malassezia sympodialis secretes nanosized exosome-like v
114 e observe that colonization of human skin by Malassezia sympodialis significantly reduces subsequent
115 ycobiome members, although the low-abundance Malassezia sympodialis was the only Malassezia species r
116 e deletion and functional complementation in Malassezia sympodialis, we demonstrated that bacterially
117 irmed the presence of fungi pathognomonic of Malassezia that reacted with patient sera in an auto-imm
118 reus adaptation in response to antagonism by Malassezia, we identified multiple mutations in the stri
119 ing certain fungal taxa, such as Candida and Malassezia, with cellular and molecular pathways of IBD
120 m sites were dominated by fungi of the genus Malassezia, with only species-level classifications reve
121 ward the identification of a sexual cycle in Malassezia, with possible implications for pathogenicity
122                                              Malassezia yeast exist on all humans and have long been
123                                              Malassezia yeasts are commensal microorganisms found in

 
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