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1 0.20 mg/L; 95% CI, 0.19-0.21 mg/L; P < .001; Mann-Whitney U test).
2 that of patients given the placebo (P = .02, Mann-Whitney U test).
3 elopment of nosocomial infections (p < 0.05, Mann-Whitney U test).
4 .38, 4.7 letters) in the MTX arm (P = .0435, Mann-Whitney U test).
5 nged in asthmatics than controls (p < 0.001, Mann-Whitney U test).
6 e multimodal group (5 vs. 7 days; P < 0.001, Mann-Whitney U test).
7 ian decrease 3%, range -13-16%; P = 0.029 by Mann-Whitney U test).
8 omplicated malaria (P, >0.1 for all enzymes; Mann-Whitney U test).
9 group compared with the CMV group (p <.005; Mann-Whitney U test).
10 and reduced or absent p27 levels (P = 0.02, Mann-Whitney U test).
11 o 91 copies for 31 subjects without (P=0.02, Mann-Whitney U test).
12 .1% CI difference medians 1.9-4.7, p<0.0001. Mann-Whitney U test).
13 n=4) vesus controls (8.2+/-1.3, n=5, P<0.02, Mann-Whitney U test).
14 ly longer than that in group 2 (P<0.05 using Mann-Whitney U test).
15 ptor motif, gp130 (0.833 mg ml-1) (P < 0.05, Mann-Whitney U test).
16 dopamine, respectively, p < .05 for both by Mann-Whitney U test).
17 ,256 vs. $11,234 + $12,146) costs (p < 0.01, Mann-Whitney U test).
18 nimals than in immunized animals (P = 0.014, Mann-Whitney U test).
19 ial diversity parameters between the groups (Mann-Whitney U test).
20 ce (Delta AUROC [SD], 0.44 [0.02]; P = .007, Mann-Whitney U test).
21 eplication remained asymptomatic (P < 0.001, Mann-Whitney U test).
22 nd bilateral dural sinus stenosis [p=0.837], Mann-Whitney U test).
23 istically significant difference (P < 0.001, Mann-Whitney U test).
24 between groups were significant (P = 0.001, Mann-Whitney U test).
25 minutes (SD, 18.5), respectively (P < 0.001; Mann-Whitney U test).
26 ces in activation volumes by tumor location (Mann-Whitney U test).
27 re frequent in lesion tissue (all P < 0.005, Mann-Whitney U-test).
28 MSA subjects (0.9; 0.3-2.4 mU/l; P < 0.005, Mann-Whitney U-test).
29 ere 12.28 and 23.14, respectively (P < 0.01, Mann-Whitney U-test).
30 13] to 641 (IQR 507-694) (-31.5%, P = 0.001, Mann-Whitney U-test).
31 Statistical analysis was by ANOVA or Mann Whitney U test.
32 els were compared between diagnoses with the Mann-Whitney U test.
33 exact test, two-sample unpaired t test, and Mann-Whitney U test.
34 was performed with paired Student t test or Mann-Whitney U test.
35 tor expression included the median, IQR, and Mann-Whitney U test.
36 h cross tabulation, Pearson chi(2) test, and Mann-Whitney U test.
37 Relationships were assessed by using the Mann-Whitney U test.
38 tatistical difference was analyzed using the Mann-Whitney U test.
39 re assessed with the Wilcoxon signed rank or Mann-Whitney U test.
40 ps were tested using Kruskal-Wallis test and Mann-Whitney U test.
41 nerve (AC/C) strain ratio, analyzed with the Mann-Whitney U test.
42 ween-group differences of change scores with Mann-Whitney U test.
43 Data were analyzed with the Mann-Whitney U test.
44 s in the two groups were compared by using a Mann-Whitney U test.
45 ood-based markers was investigated using the Mann-Whitney U test.
46 Differences in dose were assessed with the Mann-Whitney U test.
47 UCCA was compared between groups with the Mann-Whitney U test.
48 amyloidosis groups were compared by using a Mann-Whitney U test.
49 ing a Spearman correlation coefficient and a Mann-Whitney U test.
50 cancer and positive control groups using the Mann-Whitney U test.
51 uated using a two-tailed Student's t-test or Mann-Whitney U test.
52 lesion sizes were compared with the Wilcoxon Mann-Whitney U test.
53 1, 2, 3, and 4 years were compared with the Mann-Whitney U test.
54 ent between cancer and healthy groups by the Mann-Whitney U test.
55 ficant differences were determined using the Mann-Whitney U test.
56 ificance was computed by using the t test or Mann-Whitney U test.
57 Data were tested statistically by Mann-Whitney U test.
58 ared using the independent samples t test or Mann-Whitney U test.
59 m factors among groups were sought using the Mann-Whitney U test.
60 ltiple logistic regression analyses, and the Mann-Whitney U test.
61 ed semiquantitatively and analyzed using the Mann-Whitney U test.
62 encounters were compared between groups via Mann-Whitney U test.
63 le nonparametric data were assessed with the Mann-Whitney U test.
64 sed were Cox regression, Student t test, and Mann-Whitney U test.
65 tered treatment plans were assessed with the Mann-Whitney U test.
66 was evaluated with the independent t test or Mann-Whitney U test.
67 mpared with those of normal subjects, by the Mann-Whitney U test.
68 ns of spike-rates and HFO-rates were done by Mann-Whitney U test.
69 bution was compared between groups using the Mann-Whitney U test.
70 es were compared between groups by using the Mann-Whitney U test.
71 rading system were compared using a Wilcoxon Mann-Whitney U test.
72 Differences between groups were assessed via Mann-Whitney U test.
73 vs did not improve were evaluated using the Mann-Whitney U test.
74 s denture experience were analysed using the Mann-Whitney U test.
75 gray matter volumes were compared using the Mann-Whitney U test.
76 s without revascularization surgery by using Mann-Whitney U test.
77 ositionings, which was compared by using the Mann-Whitney U test.
78 d immunosorbent assay and compared using the Mann-Whitney U test.
79 l by census region and division by using the Mann-Whitney U test.
80 ion analysis (Pearson's coefficient) and the Mann-Whitney U test.
81 ed using two-sample t test and nonparametric Mann-Whitney U test.
82 Results were compared with Student t test or Mann-Whitney U test.
83 with that in control mice with a two-tailed Mann-Whitney U test.
84 d by chi2 test and continuous variables with Mann-Whitney U test.
85 (interquartile range) and compared using the Mann-Whitney U test.
86 neral phases of bone were compared using the Mann-Whitney U test.
87 frequency of motor seizures were done with a Mann-Whitney U test.
88 wise post hoc tests were conducted using the Mann-Whitney U test.
89 m admission to CT were compared by using the Mann-Whitney U test.
90 etween responders and nonresponders with the Mann-Whitney U test.
91 pletely responding lesions were evaluated by Mann-Whitney U test.
92 control subjects were assessed by using the Mann-Whitney U test.
93 met inhibitor after RF ablation by using the Mann-Whitney U test.
94 were analyzed using the chi-squared test and Mann-Whitney U-test.
95 to sets of unchanging control genes using a Mann-Whitney U-test.
96 chi(2) test and continuous variables by the Mann--Whitney U test.
97 to individual questions were compared using Mann-Whitney U tests.
98 nd carcinoma volumes were compared by use of Mann-Whitney U tests.
99 lysis was performed with the signed-rank and Mann-Whitney U tests.
100 scores in the two groups were compared with Mann-Whitney U tests.
101 were compared using descriptive analyses and Mann-Whitney U tests.
102 analysis was performed by Kruskal-Wallis and Mann-Whitney U tests.
103 in HPV testing were assessed using chi2 and Mann-Whitney U testing.
104 ses included: 1) Kolmogorov-Smirnov test; 2) Mann-Whitney U test; 3) Pearson chi(2) test; 4) Kruskal-
107 without an SSI, was tested using a nonpaired Mann-Whitney U test, an analysis of covariance, and a Pe
113 Between-group differences were tested by Mann-Whitney U test and correlations by Spearman's rank.
114 er of subjective and objective AEs using the Mann-Whitney U test and examined trends in the frequency
122 Demographic data were compared using the Mann-Whitney U test and NSAID groups with one-way ANOVA.
123 and nonresponders were compared by using the Mann-Whitney U test and receiver operating characteristi
128 Nonparametric statistics, including the Mann-Whitney U test and the Kruskal-Wallis analysis of v
134 for the 2 patient groups were compared with Mann-Whitney U tests and effect likelihood-ratio test.
135 heir predictive value was investigated using Mann-Whitney U tests and receiver-operating-characterist
137 Data were analyzed by Kruskal-Wallis and Mann-Whitney U tests and Spearman correlation analysis.
139 d MAIT cells between health and asthma using Mann-Whitney U tests and the Jonckheere-Terpstra test (l
141 p compared with increases of 66% (P = 0.004, Mann-Whitney U test) and 21% (P = 0.07) for patients who
143 ion exposure (0.06 versus 0.34 mSv; P=0.037, Mann-Whitney U test) and lower median costs ($934 versus
144 results were larger (68 vs. 34 mm2; P=0.08, Mann-Whitney U test) and were more likely to have papill
145 sub-100 bp nuclear genomic cfDNA (p 10(-5), Mann-Whitney U Test), and an increased relative abundanc
146 harm and procedural flow disruption scores (Mann-Whitney U test), and number of preventable failures
147 analyses were performed by Student's t-test, Mann Whitney U test, and Pearson product moment test.
148 ith intraclass correlation coefficients, the Mann Whitney U test, and the Wilcoxon signed rank test.
149 Data were analysed using Pearson chi(2), the Mann-Whitney U test, and binary logistic regression.
154 tween groups with unpaired Student t test or Mann-Whitney U test, and linear regression was performed
156 uded Fisher exact test, Kruskal-Wallis test, Mann-Whitney U test, and Spearman rank correlation.
157 For statistical analysis, Student t test, Mann-Whitney U test, and Spearman's correlation coeffici
158 cross mRECIST reads were compared by using a Mann-Whitney U test, and the difference in prevalence of
159 rank composite is typically analyzed using a Mann-Whitney U test, and the results are summarized by t
163 25th, 50th, 75th, and 90th percentile, using Mann-Whitney U-test, and association between DAOH-90 and
164 stical analysis comprised paired t tests and Mann-Whitney U tests, as well as Pearson r and Spearman
166 enic targets were compared (paired t test or Mann-Whitney U test) at enrollment and after gD/AS04 vac
168 ndicated no significant difference (P >0.05, Mann-Whitney U test) between the S or F inserts in the a
169 e that was significant at 4 years (P = .036; Mann-Whitney U test) but had a similar occurrence of sun
170 n (range), relative risk, and analyzed using Mann Whitney U test, Chi-square test, as appropriate, a
171 ients (</=55 years) were compared by t-test, Mann-Whitney U test, chi-square, or Fisher's exact test.
172 cal analysis was performed with the Wilcoxon Mann-Whitney U test, chi2 test, Wilcoxon matched-pairs s
176 ts), (c) pairwise tests between tumor types (Mann-Whitney U test), (d) relationships between fast flu
177 compared by using unpaired Student t test or Mann-Whitney U test, depending on data distribution.
178 f clinical EAE (p = 0.0002 vs control by the Mann-Whitney U test) enough to completely prevent fatal
179 used to compare paired samples, such as the Mann-Whitney U test (equivalent to the Wilcoxon rank sum
183 Fisher exact test for categorical variables, Mann-Whitney U test for continuous variables, and logist
188 ests were used for within-group comparisons; Mann-Whitney U tests for between-group differences.
190 11-514 min] vs 30 min [5-90 min]; p<0.0001, Mann-Whitney U test); for each minute delay from onset o
193 there was a significant reduction (P <0.05, Mann-Whitney U test) in the amount of contamination for
197 y markers with QODD scores were tested using Mann-Whitney U tests, Kruskal-Wallis tests, or Spearman'
198 atalase quantification.Data were analyzed by Mann-Whitney U-test, Kruskal-Wallis test and Cuzick's te
199 Comparisons were made using the t-test, Mann-Whitney U test, linear mixed models, and generalize
200 between groups was significant (P = 0.005 on Mann-Whitney U test; mean ranks 13.9 and 6.3 [of 21], fo
201 ribution, two-sided t-tests (mean +/- SD) or Mann-Whitney U tests (median[IQR]) were applied, p-value
202 g for interobserver agreement, McNemar test, Mann-Whitney U test, multiple regression analysis, Spear
204 between Mp-positive and -negative groups by Mann-Whitney U test or Fisher exact test, as appropriate
205 between Mp-positive and -negative groups by Mann-Whitney U test or Fisher's exact test, as appropria
206 n adenocarcinoma (AC), whereas VB was lower (Mann-Whitney U test or t test, P = .003, P = .036, and P
208 ysis was carried out using Student's t-test, Mann-Whitney U test, or chi-square test (significance, p
209 were compared between both groups by t test, Mann-Whitney U test, or likelihood ratio chi-square test
215 2 +/- 35 versus 73 +/- 24 nmol L(-1) d(-1) , Mann-Whitney U-test p < 0.0001), and the South Atlantic
218 basic tasks demonstrated construct validity (Mann-Whitney U test, P < 0.05), and learning curves for
222 g infarct growth in the upper tertile range (Mann-Whitney U test, p = 0.04) but not in the middle ter
224 bited significantly fewer reactive epitopes (Mann-Whitney U test; P < 0.0001) relative to subjects wi
228 were analyzed using chi-square analysis and Mann-Whitney U-tests; P < 0.05 was used to define signif
236 Kruskal-Wallis ANOVA-on-ranks with post hoc Mann-Whitney U tests showed significant pairwise between
237 s of variance (ANOVA)-on-Ranks with post-hoc Mann-Whitney U-tests showed significant pairwise between
238 and single-shot DWI were assessed using the Mann-Whitney U test; significance was defined at P < .05
239 ) points in the intervention group (P = .02, Mann-Whitney U test); similarly, mean (SD) parent-estima
240 5, multiple regression analysis; P =.25-.75, Mann-Whitney U test; Spearman correlation coefficients b
241 atistical analyses (Cohen kappa coefficient, Mann-Whitney U test, t tests, and intraclass correlation
244 categorical variables and the t test or the Mann-Whitney U test to compare continuous variables.
246 h fluctuating hearing and MRI-designated EH (Mann-Whitney U Test: U = 39, 27; p = 0.01, 0.003 respect
247 d Student's t test for continuous variables (Mann-Whitney U test used for nonnormally distributed var
258 llis test was used for significance, and the Mann-Whitney U test was used for pairwise comparison of
268 analysis of functional development, Wilcoxon-Mann-Whitney U test was used to compare the medians of 2
271 valuate the masticatory performance, and the Mann-Whitney U test was used to determine quality of lif
274 the data were not normally distributed, the Mann-Whitney U-test was employed to assess the statistic
280 npaired Student t, chi(2), Fisher exact, and Mann-Whitney U tests were applied to analyze the differe
284 tric Wilcoxon signed rank, Fisher exact, and Mann-Whitney U tests were used for statistical analysis.
285 is followed by Bonferroni-corrected post hoc Mann-Whitney U tests were used to analyze the data.
291 The chi-square test, Student's t-test, and Mann-Whitney U-test were used for statistical analysis.
293 he gene sets is performed by an extension of Mann-Whitney U test which is based on weighted rank sums
294 test, Wilcoxon's matched pairs test, and the Mann Whitney U Test with P < 0.05 considered significant
297 ible, non-hypervariable Env sites (p = 0.50, Mann-Whitney U-test) with no significant relationship be
298 versus 23 +/- 1.4 in controls; P < 0.0001 by Mann-Whitney U test), with virtually no overlap between
300 iability and differentiated VCD vs. healthy (Mann-Whitney U-test: z = -5.390, P < 0.001) and asthma (