ライフサイエンスコーパス: Mirabilis

1 condition in longjaw mudsuckers (Gillichthys mirabilis).

2 arming in the urinary tract pathogen Proteus mirabilis.

3 details of self-nonself discrimination in P. mirabilis.

4 nce of siderophore production in vivo for P. mirabilis.

5 ted from the marine sponge Siliquariaspongia mirabilis.

6 the 3' end of the mrp fimbrial operon in P. mirabilis.

7 ntly reduced in an isogenic pta mutant of P. mirabilis.

8 ra from mice experimentally infected with P. mirabilis.

9 but were detected in every population of A. mirabilis.

10 an immunogenic outer membrane protein in P. mirabilis.

11 putrescine-deficient speA mutant of Proteus mirabilis.

12 ebsiella spp., Escherichia coli, and Proteus mirabilis.

13 sertion was mapped to a speA homologue in P. mirabilis.

14 nt virulence factor of uropathogenic Proteus mirabilis.

15 o prevent urinary tract infection by Proteus mirabilis.

16 erant burrow-dwelling goby fish, Gillichthys mirabilis.

17 theterized mice to infection by wild-type P. mirabilis.

18 ods for identifying individual strains of P. mirabilis.

19 so contributes to the uropathogenicity of P. mirabilis.

20 's-eye colonies typically associated with P. mirabilis.

21 ike (MR/P) fimbriae of uropathogenic Proteus mirabilis.

22 pe on an otherwise nonhemolytic mutant of P. mirabilis.

23 nce of collagenolytic E faecalis and Proteus mirabilis.

24 the formation of crystalline biofilms by P. mirabilis.

25 arboxylase that inhibits swarming in Proteus mirabilis.

26 putrescine importer, was characterized in P. mirabilis.

27 iation of Klebsiella pneumoniae from Proteus mirabilis 16S rRNA target sequences differing by a singl

28 mmon species (68.6%), followed by Ochroconis mirabilis (21.5%).

29 Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella oxytoca (2.7%), and Citroba

30 Here we characterize PmDsbA from Proteus mirabilis, a bacterial pathogen increasingly associated

31 Proteus mirabilis, a cause of complicated urinary tract infectio

32 y, we identified a genomic island of Proteus mirabilis, a common agent of catheter-associated urinary

33 Proteus mirabilis, a common cause of nosocomial and catheter-ass

34 Proteus mirabilis, a common cause of urinary tract infections (U

35 Proteus mirabilis, a gram-negative bacterium associated with com

36 Proteus mirabilis, a gram-negative bacterium, is a frequent caus

37 Proteus mirabilis, a Gram-negative bacterium, represents a commo

38 Proteus mirabilis, a leading cause of catheter-associated urinar

39 solute tested, but the catalytic rate of G. mirabilis A4-LDH was decreased most by solutes and incre

40 To identify interactions among P. mirabilis accessory proteins, in vitro immunoprecipitati

41 Proteus mirabilis alternates between motile and adherent forms.

42 Proteus mirabilis, an etiologic agent of complicated urinary tra

43 dder colonization factor of uropathogenic P. mirabilis and also suggested that the ability to switch

44 ant, especially against the bacteria Proteus mirabilis and Antibiotic resistant Escherichia coli.

45 -negative bacterial cells, including Proteus mirabilis and Caulobacter crescentus, initiates asymmetr

46 metabolism as an adaptation strategy for P. mirabilis and contributes to better understand the ecolo

47 tedly and unlike other fliL mutants, both P. mirabilis and E. coli DeltafliL cells swarm (Swr(+)).

48 We focused on Lrp from Proteus mirabilis and E. coli, orthologs with 98% identity overa

49 The closely related Proteus mirabilis and Enterobacterlaceae plasmid-encoded urease

50 n these mice isolated K. pneumoniae, Proteus mirabilis and Enterococcus gallinarum, which were preval

51 ed and characterized DeltafliL mutants of P. mirabilis and Escherichia coli.

52 flagellar operon, in vegetative cells of P. mirabilis and found that increased flagellum density pro

53 We sampled several populations of A. mirabilis and its photosynthetic sister groups to correl

54 and antigenicity of an autotransporter in P. mirabilis and its use in vaccine development.

55 ase-negative mutant of uropathogenic Proteus mirabilis and its wild-type parent strain was assessed b

56 UTI caused by the major uropathogens Proteus mirabilis and Klebsiella pneumoniae, in addition to UPEC

57 al catheter colonization, particularly by P. mirabilis and other urease-positive bacteria.

58 usly determined that interactions between P. mirabilis and other uropathogens can enhance P. mirabili

59 We conclude that P. mirabilis and P. stuartii coinfection promotes urolithia

60 iously demonstrated that coinfection with P. mirabilis and P. stuartii increased overall urease activ

61 The urease-positive species Proteus mirabilis and Providencia stuartii are two of the leadin

62 y urease-positive organisms, such as Proteus mirabilis and Providencia stuartii, commonly occurs with

63 moter sequences of Escherichia coli, Proteus mirabilis and Salmonella typhimurium allowed determinati

64 a coli, Pseudomonas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interferi

65 e from other human pathogens such as Proteus mirabilis and Staphylococcus aureus that outcompete P. a

66 , the AT also promoted autoaggregation of P. mirabilis and this function was independent of its prote

67 ) isolates of Enterobacterales (excluding P. mirabilis) and P. aeruginosa demonstrated an unacceptabl

68 etine and thioridazine) to act as EPIs in P. mirabilis, and control crystalline biofilm formation.

69 ies, lrp genes from Vibrio cholerae, Proteus mirabilis, and E. coli were introduced into the same E.

70 es including Listeria monocytogenes, Proteus mirabilis, and Escherichia coli in various host cell typ

71 tine and thioridazine inhibited efflux in P. mirabilis, and molecular modelling predicted both drugs

72 nella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

73 siella pneumoniae, Escherichia coli, Proteus mirabilis, and Salmonella enterica serovar Typhimurium.

74 f Escherichia coli, Klebsiella spp., Proteus mirabilis, and Salmonella spp. and are associated with m

75 Thus, P. mirabilis appears to use a related mechanism to inhibit

76 ary tract infections (UTI) caused by Proteus mirabilis are associated with severe pathology in the bl

77 Fimbriae of the human uropathogen Proteus mirabilis are the only characterized surface proteins th

78 ften invasive, and, with the exception of P. mirabilis, are multiclonal.

79 Using Proteus mirabilis as a model, we investigate the role of choline

80 aracterizing trimeric autotransporters in P. mirabilis as afimbrial surface adhesins and autoagglutin

81 pplication of the mini-Tn7 system in Proteus mirabilis as an example of a bacterium with a secondary

82 These results support the hypothesis that P. mirabilis ascertains its location in the environment or

83 a pneumoniae, Klebsiella oxytoca, or Proteus mirabilis at >=50 000 colony-forming units/mL; (2) ident

84 lebsiella pneumoniae, K. oxytoca, or Proteus mirabilis at >=50,000 CFU/mL, (2) identification of an E

85 Ten strains of Lautropia mirabilis (ATCC 51599(T) and nine phenotypically similar

86 One of the six predicted Proteus mirabilis autotransporters (ATs), ORF c2341, is predicte

87 P. mirabilis bacteriuria may lead to acute pyelonephritis,

88 ed from the 50 %-identical TEM-1 and Proteus mirabilis beta-lactamases.

89 encrusted and blocked by crystalline Proteus mirabilis biofilms.

90 s eliminated collagenolytic E faecalis and P mirabilis but did not reduce tumor formation.

91 la pneumoniae, Escherichia coli, and Proteus mirabilis by acidifying the proximal colon and triggerin

92 site-specific transposition pathway into P. mirabilis by transformation, followed by selection of in

93 The crystalline biofilms of P. mirabilis can cause serious complications for patients u

94 tients revealed that a single genotype of P. mirabilis can persist in the urinary tract despite many

95 Proteus mirabilis can rapidly utilize choline to enhance growth

96 first report of carbapenem resistance in P. mirabilis caused by the acquisition of bla(KPC).

97 Recent work has elucidated how P. mirabilis causes all of these disease states.

98 Proteus mirabilis causes complicated urinary tract infections (U

99 In this study, we show that P. mirabilis CaUTI isolates initiate swarming in response t

100 clear how energetically costly changes in P. mirabilis cell morphology translate into an advantage fo

101 ntal behaviors of motile, rod-shaped Proteus mirabilis cells (3 mum in length) adsorbed to the LC int

102 The number of P. mirabilis cells adhering to bladder tissue did not appea

103 In liquid, P. mirabilis cells are 1.5- to 2.0-mum swimmer cells with 4

104 In the lab, P. mirabilis cells become long and multinucleate and increa

105 The majority of wild-type P. mirabilis cells in transurethrally infected mice produce

106 that MR/P fimbriae are expressed by most P. mirabilis cells infecting the urinary tract, dictate the

107 ility to sense a surface: e.g., DeltafliL P. mirabilis cells swarm precociously over surfaces with lo

108 A total of 63 clinical isolates of Proteus mirabilis collected over a 19-month period were typed by

109 creening for the presence of ICEPm1 among P. mirabilis colonizing isolates showed that ICEPm1 is more

110 On agar, a P. mirabilis colony grows outward in a bull's-eye pattern f

111 Proteus mirabilis commonly infects the complicated urinary tract

112 Proteus mirabilis compromises the care of many patients undergoi

113 found to significantly reduce the rate of P. mirabilis crystalline biofilm formation on catheters, an

114 dentified as important for development of P. mirabilis crystalline biofilms, highlighting the potenti

115 ibitors in the treatment or prevention of P. mirabilis crystalline biofilms.

116 Further analysis revealed that P. mirabilis DeltafliL cells also exhibit an alteration in

117 ia availability due to urease activity in P. mirabilis did not drive this gene expression.

118 We conclude that prior infection with P. mirabilis does not protect significantly against homolog

119 Etest MEV should not be used with P. mirabilis due to unacceptable analytical performance.

120 fimbriae, a surface antigen expressed by P. mirabilis during experimental urinary tract infection, v

121 Examination of a set of 55 isolates of P. mirabilis, each from a different clinical or environment

122 , is a functional homolog of MrpJ of Proteus mirabilis; ectopic expression of papX in P. mirabilis re

123 dible) (K > Cu > Zn > Se > Na > Mg); Boletus mirabilis (edible) (K > Cu > Zn > Na > Mg); Lactarius de

124 When P. mirabilis encounters a highly viscous environment, e.g.,

125 When P. mirabilis encounters a solid surface, where flagellar ro

126 l pathogens: Pseudomonas aeruginosa, Proteus mirabilis, Enterococcus faecalis, Klebsiella pneumoniae,

127 The moulting behaviour of A. mirabilis evokes that of decapods, in which the carapace

128 Overall, P. mirabilis exhibits an extraordinary lifestyle, and furth

129 ns (RIPs) were found in the storage roots of Mirabilis expansa, an underutilized Andean root crop.

130 mmunoreactivity with antibodies of RIPs from Mirabilis expansa.

131 results establish a relationship between P. mirabilis flagellum density and cell motility in viscous

132 d into the culture medium by the original P. mirabilis flgN mutant demonstrated that export of FlgK a

133 and motility, a result also observed when P. mirabilis fliL+ was expressed in Escherichia coli.

134 ates of >=80%, with the exception of Proteus mirabilis, for which clinical and challenge isolates dem

135 Proteus mirabilis forms extensive crystalline biofilms on indwel

136 Proteus mirabilis forms extensive crystalline biofilms on urethr

137 en of the bizarrely specialised Malleodectes mirabilis from middle Miocene deposits in the Riversleig

138 la pneumoniae, Escherichia coli, and Proteus mirabilis from the intestinal lumen and represents a pot

139 s study, microarrays were used to analyze P. mirabilis gene expression in vivo from experimentally in

140 In this study, we describe wosA, a Proteus mirabilis gene identified by its ability to increase swa

141 In a search for Proteus mirabilis genes that were regulated by cell-to-cell sign

142 Sequencing of the P. mirabilis genome revealed 14 additional paralogues of mr

143 idence for ancient polyploidy in Welwitschia mirabilis (Gnetales) and no evidence for polyploidy in p

144 institution, E. coli, K. pneumoniae, and P. mirabilis harbor plasmid-mediated AmpC enzymes.

145 tility by the urinary tract pathogen Proteus mirabilis has been a long-studied but little understood

146 edicts with 85% accuracy whether this "annus mirabilis" has passed, or if better days are still to co

147 ed significantly more biofilm than either P. mirabilis HI4320 (P = 0.03) or MR/P OFF (P = 0.05).

148 ract, a ureR mutation was introduced into P. mirabilis HI4320 by homologous recombination.

149 Microarray analysis of P. mirabilis HI4320 cultured under iron limitation identifi

150 After 7 days, however, P. mirabilis HI4320 formed a 65-mum-thick biofilm, while th

151 In addition, the P. mirabilis HI4320 genome possesses four tandem copies of

152 Immunogold electron microscopy of P. mirabilis HI4320 revealed that MrpH was located at the t

153 o evaluate the colonization of mutants of P. mirabilis HI4320 that were generated by signature-tagged

154 we report the complete genome sequence of P. mirabilis HI4320, a representative strain cultured in ou

155 Although in P. mirabilis HI4320, ICEPm1 is annotated as integrated into

156 th the newly completed genome sequence of P. mirabilis HI4320, was used to identify surface-exposed a

157 I null mutants from a clinical isolate of P. mirabilis, HI4320.

158 A recombinant plasmid containing cloned P. mirabilis hns was able to complement and restore repress

159 ese data underscore the importance of the P. mirabilis IgA-degrading metalloprotease in UTI.

160 A. mirabilis illuminates the moult strategy of stem-group E

161 ent work reveals that M. morganii acts on P. mirabilis in a contact-independent manner to decrease ur

162 d TaaP individually offered advantages to P. mirabilis in a murine model.

163 stal structure of a Crl homolog from Proteus mirabilis in conjunction with in vivo and in vitro appro

164 ated and important for the persistence of P. mirabilis in the host, it was selected as a vaccine cand

165 Modeling CaiT from P. mirabilis in the outward-open and closed states on the c

166 ontributes to the pathogenic potential of P. mirabilis in the urinary tract.

167 n of urease by urea, and for virulence of P. mirabilis in the urinary tract.

168 y be a significant virulence component of P. mirabilis in urinary tract infections.

169 ght into crystalline biofilm formation by P. mirabilis, including the link between biofilm formation

170 In wild-type P. mirabilis, increased expression of the flhDC operon occu

171 eae and in particular the pathobiont Proteus mirabilis, induced robust IL-1beta release that was comp

172 al. (2015) show that the pathobiont Proteus mirabilis induces NLRP3 inflammasome-dependent interleuk

173 deposits in bladder and kidney tissues of P. mirabilis-infected mice.

174 ides a clear visual early warning of Proteus mirabilis infection and subsequent blockage.

175 ey metabolic pathways as requirements for P. mirabilis infection of the urinary tract.

176 of urease-mediated urea hydrolysis during P. mirabilis infection.

177 idencia stuartii, and Morganella morganii P. mirabilis infections are particularly challenging due to

178 d the epidemiology of catheter-associated P. mirabilis infections by use of pulsed-field gel electrop

179 The enteric bacterium Proteus mirabilis is a common cause of complicated urinary tract

180 Proteus mirabilis is a common cause of urinary tract infection (

181 Proteus mirabilis is a common uropathogen in patients with long-

182 Proteus mirabilis is a dimorphic motile bacterium well known for

183 Proteus mirabilis is a dimorphic, motile bacterium often associa

184 The gram-negative enteric bacterium Proteus mirabilis is a frequent cause of urinary tract infection

185 Proteus mirabilis is a Gram-negative bacterium that exists as a

186 Proteus mirabilis is a Gram-negative bacterium that undergoes a

187 Proteus mirabilis is a Gram-negative uropathogen and frequent ca

188 Proteus mirabilis is a model organism for urease-producing uropa

189 Proteus mirabilis is a urinary tract pathogen and well known for

190 Proteus mirabilis is a urinary tract pathogen that differentiate

191 Proteus mirabilis is an opportunistic pathogen that is frequentl

192 The enteric bacterium Proteus mirabilis is associated with a significant number of cat

193 The bacterium Proteus mirabilis is capable of movement on solid surfaces by a

194 P. mirabilis is capable of swarming, a form of multicellula

195 ZapA, of the urinary tract pathogen Proteus mirabilis is co-ordinately expressed along with other pr

196 Proteus mirabilis is one of the most common causes of monomicrob

197 eding and biochemical analysis shows that P. mirabilis is unable to utilize or produce yersiniabactin

198 solates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2 A. baumannii isolates) producin

199 Swarming colonies of independent Proteus mirabilis isolates recognize each other as foreign and d

200 lebsiella sp. isolates, and 7 (100%) Proteus mirabilis isolates tested were CTX-M positive, with 15%

201 f 683 E. coli, 371 K. pneumoniae, and 232 P. mirabilis isolates tested, 13 (1.9%), 28 (7.6%), and 4 (

202 One of seven positive P. mirabilis isolates was in group II, with the remainder b

203 e CTX-M-positive isolates showed that six P. mirabilis isolates were clonal and that there were seven

204 Five of seven P. mirabilis isolates were from blood cultures.

205 pneumoniae, Klebsiella oxytoca, and Proteus mirabilis isolates, including phenotypically ESBL-positi

206 tion, is useful for the identification of L. mirabilis isolates.

207 initial epidemiologic characterization of P. mirabilis isolates.

208 ME2 showed high similarity to the Mirabilis jalapa antiviral protein, a type I RIP.

209 red beet (Beta vulgaris) and four o'clocks (Mirabilis jalapa) to identify a novel, betalain-related

210 net exchange rate of a plant-soil ecosystem (Mirabilis jalapa).

211 bilis, which is responsible for infection of Mirabilis jalapa, are adapted to protease targets unique

212 of similarity to sequences encoding Proteus mirabilis mannose-resistant fimbriae.

213 Excluding P. mirabilis, MEV Etest MEV demonstrated 95.8% EA, 99.3% CA

214 ragment was isolated from a genomic clone of mirabilis mosaic virus (MMV), a double-stranded DNA plan

215 While there are similarities between P. mirabilis MR/P and E. coli P fimbriae, there are more no

216 ted zinc present in the urinary tract and P. mirabilis must scavenge this ion to colonize and persist

217 4), Pseudomonas aeruginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Stenotrophomo

218 infection (mean log(10) number of CFU of P. mirabilis Nal(r) HI4320 per milliliter or gram in vaccin

219 s parainfluenzae, Kingella oralis, Lautropia mirabilis, Neisseria elongata, Rothia dentocariosa, Stre

220 Mice were infected with P. mirabilis or a urease mutant, P. stuartii, or a combinat

221 nal colonization with K. pneumoniae, Proteus mirabilis, or Enterobacter cloacae promoted greater recr

222 In particular, for both the E. coli and P. mirabilis orthologs, Lrp responsiveness to methionine wa

223 ection against urinary tract infection by P. mirabilis (P < 0.002).

224 from ascending urinary tract infection by P. mirabilis (P < 0.05).

225 al related Phytophthora species including P. mirabilis, P. ipomoeae, and possibly P. phaseoli.

226 Transurethral inoculation of P. mirabilis(pBAC001) resulted in ascending urinary tract i

227 P. mirabilis(pBAC001), which expresses green fluorescent pr

228 olecular analyses have revealed that Proteus mirabilis possesses two genes, flaA and flaB, that are h

229 e consolidation phase is a state in which P. mirabilis prepares for the next wave of swarming.

230 ptional specimen of the fuxianhuiid Alacaris mirabilis preserved in the act of moulting from the Camb

231 Furthermore, colonization with P. mirabilis promoted intestinal inflammation upon intestin

232 designated ICEPm1, that is common to Proteus mirabilis, Providencia stuartii, and Morganella morganii

233 This chromosome integration system in P. mirabilis provides an important tool for animal and biof

234 ncluded probes for Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Enterocococcus spp.,

235 ative bacteria are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Klebsiella pneumonia.

236 An isolate of Proteus mirabilis recovered from blood cultures of a diabetic pa

237 mirabilis; ectopic expression of papX in P. mirabilis reduces motility.

238 E. coli and P. mirabilis remained susceptible to most of the drugs test

239 hia coli, Klebsiella pneumoniae, and Proteus mirabilis remains unknown.

240 One population of A. mirabilis revealed a unique deletion of 541 bp in the ps

241 es of CaiT from Escherichia coli and Proteus mirabilis revealed an inverted five-transmembrane-helix

242 e sequence of the parasitic liverwort Aneura mirabilis revealed the loss of five chlororespiration (n

243 How P. mirabilis senses a surface is not fully understood; howe

244 d autoagglutination, and a taaP mutant of P. mirabilis showed significantly (P < 0.05) more reduced a

245 a Foley catheterised bladder infected with P mirabilis showed that when retention balloons were infla

246 In a P. mirabilis speA mutant with greatly reduced levels of int

247 colonization by common uropathogens Proteus mirabilis, Staphylococcus aureus and Escherichia coli fo

248 P. mirabilis strain HI4320 encodes two putative nickel impo

249 The isogenic mrpJ null mutant of wild-type P.mirabilis strain HI4320 is attenuated in the murine mode

250 nriched the relative proportion of a Proteus mirabilis strain in the ceca of animals not pre-treated

251 rm between swarming colonies of different P. mirabilis strains but not between colonies of a single s

252 e prevalent in urine isolates compared to P. mirabilis strains isolated from other body sites (P<0.00

253 Inactivation of aipA in P. mirabilis strains significantly (P < 0.01) reduced the m

254 o assess the relatedness of swarming Proteus mirabilis strains, was used to study 15 P. aeruginosa is

255 erichia coli, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subtilis, St

256 ar basal body protein FliL is involved in P. mirabilis surface sensing.

257 dentify rhomboid-encoding genes from Proteus mirabilis, tatA was identified as a multicopy suppressor

258 d flagellum are among virulence factors of P.mirabilis that contribute to its colonization in a murin

259 laboratory models of colonization by Proteus mirabilis, the sensor signaled encrustation at an averag

260 ids genes are involved in the ability of P. mirabilis to distinguish self from nonself.

261 In order for P. mirabilis to swarm, it first needs to detect a surface.

262 in behaviour characterization in Welwitschia mirabilis to test whether the known regulatory links bet

263 ification of 37 consistently out-competed P. mirabilis transposon mutants, 25 of which were out-compe

264 By performing primary screening of 2088 P. mirabilis transposon mutants, we identified 502 mutants

265 s swarming-associated colony expansion of P. mirabilis under anaerobic conditions on a solid surface.

266 MR/P fimbriae of uropathogenic Proteus mirabilis undergo invertible element-mediated phase vari

267 this organism, was sufficient to enhance P. mirabilis urease activity and increase disease severity,

268 esized that nickel import is critical for P. mirabilis urease activity and pathogenesis during infect

269 Other uropathogens also enhanced P. mirabilis urease activity in vitro, including recent cli

270 abilis and other uropathogens can enhance P. mirabilis urease activity, resulting in greater disease

271 Proteus mirabilis urease catalyzes the hydrolysis of urea to CO(

272 Expression of Proteus mirabilis urease is governed by UreR, an AraC-like posit

273 re not observed during coinfection with a P. mirabilis urease mutant.

274 This report suggests that in vivo, P. mirabilis UreD may be important for recruitment of UreF

275 ted the hypothesis that experimental Proteus mirabilis urinary tract infection in mice would protect

276 ot utilize citrate, the data suggest that P. mirabilis uses glutamate dehydrogenase to monitor carbon

277 d form of hemolysin A (HpmA265) from Proteus mirabilis using a series of functional and structural st

278 transconjugants/donor to ICEPm1-deficient P. mirabilis using plate mating assays with clinical isolat

279 d Enterobacterales, with the exception of P. mirabilis, using CLSI/FDA breakpoints.

280 Patients with Proteus mirabilis UTIs were more likely to have a foreign body i

281 airpins from Haemophilus influenzae, Proteus mirabilis, Vibrio fischeri, and Pasteurella multocida ar

282 We show that the P. mirabilis, Vibrio harveyi, and E. coli Crl homologs func

283 t a role of MrpJ as a global regulator of P. mirabilis virulence.

284 cherichia coli, Klebsiella spp., and Proteus mirabilis was evaluated, using incidence rates of resist

285 ence in other pathogens, its role in Proteus mirabilis was investigated by constructing a strain with

286 Wild-type P. mirabilis was usually found colonizing intact uroepithel

287 ncoding the entire urease gene cluster of P. mirabilis) was equivalent in both the H-NS(-) background

288 ng strains of Myxococcus xanthus and Proteus mirabilis, we found the rates of killing between the str

289 Swimming and swarming motilies in P. mirabilis were also significantly reduced by both EPIs.

290 a dispar, Rothia dentocariosa, and Lautropia mirabilis were more abundant in health.

291 a, Haemophilus parainfluenzae, and Lautropia mirabilis were most abundant in healthy children, while

292 lpha and an isogenic mrpH::aphA mutant of P. mirabilis were unable to produce normal MR/P fimbriae ef

293 athogens, Pseudomonas aeruginosa and Proteus mirabilis, were made bioluminescent by stable insertion

294 -LDH from a warm-temperate goby (Gillichthys mirabilis), which has lower values for kcat and the Mich

295 estans, and its sister species, Phytophthora mirabilis, which is responsible for infection of Mirabil

296 p of compounds was also confirmed in Proteus mirabilis whole-cell-based inhibition assays.

297 pneumoniae, Klebsiella oxytoca, and Proteus mirabilis with an ertapenem-susceptible extended-spectru

298 Strains of P. mirabilis with mutations in three of the corresponding g

299 croscopy, we demonstrated the presence of P. mirabilis within the urease-induced stone matrix.

300 tion studies with hemolysin-negative Proteus mirabilis WPM111 (a HpmA(-) mutant of BA6163) transforme