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1                                              Monod kinetics for utilization of acetate were relativel
2                                              Monod kinetics with competitive inhibition are used to d
3                                              Monod, Wyman, and Changeux (MWC) explained allostery in
4                                            A Monod-Wyman-Changeux co-agonist mechanism with two equiv
5  is described by either of two models: (1) a Monod-Wyman-Changeux-based model (MWC) where salt bridge
6       All our results are well-captured by a Monod consumer resource model, which also explains how U
7 heteromultimeric channels were well fit by a Monod, Wyman, and Changeux model with a concerted allost
8 wth of algal cells, with each contributing a Monod growth kinetic term in a multiplicative model.
9                      Here I show that, for a Monod-Wyman-Changeux model with independent sensors, the
10        This is based on the development of a Monod-equation based, differential equation model, which
11           We have devised a model based on a Monod-Wyman-Changeux cooperative mechanism with volume c
12                       Our results rule out a Monod-Wyman-Changeux allosteric mechanism with a central
13 ms, we propose that by differentiating along Monod parameters characterizing microbial growth rates i
14           It is now 50 years since Jacob and Monod first proposed a model for gene regulation, which
15                The papers from the Jacob and Monod groups that presented the operon model and repress
16 atory motif, originally posited by Jacob and Monod in the 1960s, consists of a single transcriptional
17 the circuit blueprints imagined by Jacob and Monod laid the foundation for the first synthetic gene n
18 e composability of these elements, Jacob and Monod linked phenotypic diversity to the architectures o
19                           In 1961, Jacob and Monod proposed the operon model of gene regulation.
20      The operon model, proposed by Jacob and Monod, provides a cogent depiction for how gene expressi
21 n is based on dual-substrate utilization and Monod growth kinetics.
22 r effect (1904) to the birth of allostery by Monod and Jacob (1961).
23 lanation of enzymatic adaptation proposed by Monod and shared by scientists for more than half a cent
24  Microbial Evolution, beginning with work by Monod.
25                     By extending the classic Monod model of resource-limited population growth to all
26          The 50th anniversary of the classic Monod-Wyman-Changeux (MWC) model provides an opportunity
27                Here, inspired by the classic Monod-Wyman-Changeux model of cooperativity(10), we inve
28 lem that has hindered the use of the classic Monod-Wyman-Changuex (MWC) allosteric model since its in
29 se, that are well described by the classical Monod, Wyman and Changeux (MWC) model.
30                                The classical Monod-Wyman-Changeux model for homogeneous allosteric pr
31 egulation is well described by the classical Monod-Wyman-Changeux model of allostery.
32 on has often been described by the concerted Monod-Wyman-Changeux and sequential Koshland-Nemethy-Fil
33                                 Conventional Monod kinetic models were unable to predict kinetic para
34                                       A dual Monod model of dehalorespiration provided a good fit to
35 ing these complex interactions into the dual Monod model.
36  mechanistic interpretation to the empirical Monod law, and demonstrates the potential of coupling lo
37 ysis of mutant activity using an established Monod-Wyman-Changeux (MWC) allosteric model indicates th
38                    PURPOSE OF REVIEW: Formal Monod-Wyman-Changeux allosteric mechanisms have proven v
39                         We use a generalized Monod-Wyman-Changeux (MWC) model that allows for variabl
40 plained consistently within this generalized Monod-Wyman-Changeux model.
41 s and their metabolism and microbial growth (Monod kinetics with decay) and tested it with three aero
42                       The model incorporates Monod kinetics, with phenotypic transition probabilities
43 nsible for the rapid acceptance of the Jacob-Monod model and inhibited suggestions for alternative me
44 pher Democritus and later adopted by Jacques Monod is "everything existing in the universe is the fru
45 st, Hungary, I was fortunate to meet Jacques Monod at the Pasteur Institute, and this became a turnin
46                                Since Jacques Monod's foundational work in the 1940s, investigators st
47                                  The Jacques Monod Conference on 'Growth and regeneration during deve
48 scinating, for I met and worked with Jacques Monod and Francois Jacob, a collaboration that culminate
49 ess is in full agreement with the well-known Monod-Wyman-Changeux model.
50 were analyzed in the framework of a modified Monod-Wyman-Changeux allosteric activation model.
51 tructure that can be explained by a modified Monod-Wyman-Changeux model.
52 ribing such transitions is the multistimulus Monod-Wyman-Changeux model, in which each stimulus inter
53                                     The MWC (Monod-Wyman-Changeux) allosteric model postulates concer
54 at maximum current density revealed a Nernst-Monod response with a half saturation potential (EKA) of
55  multiple layers of active cells, and Nernst-Monod behavior support extracellular electron transfer (
56 ng kinetic proofreading and a nonequilibrium Monod-Wyman-Changeux (MWC) model proposed for the E. col
57 and can be explained within the framework of Monod-Wymann-Changeux (MWC) theory that was originally f
58  to extend the two-state allosteric model of Monod, Wyman, and Changeux (MWC) to include geminate lig
59 sic quaternary two-state allosteric model of Monod, Wyman, and Changeux to include tertiary conformat
60  this study, we used the allosteric model of Monod, Wyman, and Changeux to simulate whole-blood oxyge
61 emoglobin: the quaternary two-state model of Monod, Wyman, and Changeux; the tertiary two-state model
62              Since the seminal 1961 paper of Monod and Jacob, mathematical models of biomolecular cir
63 ls of binding cooperativity, such as that of Monod, Wyman, and Changeux, in the limit of an infinite
64                      With the Python package Monod, we demonstrate how nascent and mature RNA counts,
65 echanisms that underpin the phenomenological Monod parameters: the maximum specific growth rate could
66 fruit of chance and necessity." While I read Monod's book Chance and Necessity as an undergraduate st
67              We show that the model recovers Monod's law for the growth of microbes and two other emp
68 centrations fall below the (half-saturation) Monod constant.
69 Methanocaldococcus jannaschii showed similar Monod growth kinetics when grown in a bioreactor at vary
70            Our data, fitted to a three-state Monod-Wyman-Changeux model, show that mutations at the s
71 f oxygenation properties under the two-state Monod-Wyman-Changeux allosteric model revealed that the
72 lculated and analyzed using global two-state Monod-Wyman-Changeux models to derive log(d) parameters
73 ubunits activate in a single concerted step (Monod-Wyman-Changeux model) or in four independent steps
74  one-dimension model includes dual-substrate Monod kinetics for a steady-state biofilm with five soli
75 nce analysis (FBA) into a multiple-substrate Monod model to perform the dynamic flux balance analysis
76                                          The Monod equation shares a similar form with the mechanisti
77                                          The Monod-Wyman-Changeux (MWC) cyclic model was described as
78                                          The Monod-Wyman-Changeux (MWC) model was conceived in 1965 t
79                                          The Monod-Wyman-Changeux (MWC) model was initially proposed
80 l and two-dimensional lattice models and the Monod-Wyman-Changeux model of isolated strongly-coupled
81         Empirical kinetic models such as the Monod equation have been widely applied to relate the ce
82 e (e.g., galactose), a paradigm known as the Monod model.
83 or of detailed multisite systems such as the Monod-Wyman-Changeux allosteric model or rule-based mode
84 ottleneck when concentrations fell below the Monod constant of microbial growth.
85 or concerted transitions as specified by the Monod-Wyman-Changeux model.
86 etically beyond what can be explained by the Monod-Wyman-Changeux/Perutz model.
87                        Here, we employed the Monod-Wyman-Changeux concerted transition model to analy
88 rtinent proteome cost; more importantly, the Monod constant (K(s)) was shown to relate to the Michael
89 yeast, we consequently are able to infer the Monod constant for growth on fructose.
90              We use omniplate to measure the Monod relationship for the growth of budding yeast in ra
91 te statistical mechanical description of the Monod-Wyman-Changeaux allosteric model for both single a
92              The acknowledged success of the Monod-Wyman-Changeux (MWC) allosteric model stems from i
93  al., which is the simplest extension of the Monod-Wyman-Changeux model to include pre-equilibria of
94 ons can be interpreted in the context of the Monod-Wyman-Changeux model.
95 ing to the inactive (T) state complex of the Monod-Wyman-Changeux two-state model.
96  T/R quaternary structural transition of the Monod-Wyman-Changeux/Perutz model.
97 and conformational changes parameters of the Monod-Wymann-Changeux allosteric model and graphed the '
98  of models of DNA accessibility based on the Monod-Wyman-Changeux (MWC) model of allostery, which pos
99             Recent experiments show that the Monod, Wyman and Changeux formalism for allosteric prote
100 i.e., approximately 400 mini-FBAs), then the Monod model provided time-dependent inflow/outflow fluxe
101 ity-velocity qualitatively is similar to the Monod equation, while providing additional information o
102                        Contradictions to the Monod-Wyman-Changeaux/Perutz allosteric model arise sinc
103 d inhibited T conformations according to the Monod-Wyman-Changeux (MWC) model, although a more comple
104                  We fit the data sets to the Monod-Wyman-Changeux model to extract microscopic parame
105 steric ligand and can be explained using the Monod-Wyman-Changeux two-state model of allostery.
106 standard logistic growth models and with the Monod-like function that governs the dependence of the g
107  simulations are largely consistent with the Monod-Wyman-Changeux model for allosteric activation but
108 f receptor coupling, but consistent with the Monod-Wyman-Changeux model of receptor coupling, suggest
109 ll culture behavior which may not conform to Monod kinetics.
110 ten-Henri (MMH) and Gene-Regulation (GRN) to Monod-Wyman-Changeaux (MWC), user defined reactions and
111         In fact, investigators even prior to Monod had identified other aspects of bacterial growth,
112  rate data fit reasonably well to a trimeric Monod-Wyman-Changeux model, suggesting a two-state confo
113                                      We used Monod kinetic models to describe substrate utilization a
114 ls of this response, such as the widely used Monod model, are generally characterized by a maximum gr
115 numerical experiments was conducted in which Monod kinetic models with and without mass loss were fit

 
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