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1 egative pathogens Haemophilus influenzae and Moraxella catarrhalis .
2 es and the universal surface protein A II of Moraxella catarrhalis.
3 egative organisms Haemophilus influenzae and Moraxella catarrhalis.
4 ylococcus aureus, Pseudomonas aeruginosa, or Moraxella catarrhalis.
5 H influenzae, Streptococcus pneumoniae, and Moraxella catarrhalis.
6 involved in the serum-resistant phenotype of Moraxella catarrhalis.
7 lows: for Streptococcus spp., 0.06/0.12; for Moraxella catarrhalis, 0.06/0.12; for Staphylococcus spp
8 ccus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis), (2) microbial function, and (3)
10 6%]), Haemophilus influenzae (70 [25%]), and Moraxella catarrhalis (65 [24%]) being most prevalent.
12 3%, Staphylococcus epidermis; 15%, 10%, 16%, Moraxella catarrhalis; 9%, 25%, 19%, and Streptococcus P
14 eria in bacterium-host cell cocultures using Moraxella catarrhalis, a respiratory tract disease-causi
15 e are no licensed vaccines available against Moraxella catarrhalis, a significant human respiratory p
17 haride (LOS) is a major surface component of Moraxella catarrhalis and a possible virulence factor in
18 ater abundance of Haemophilus influenzae and Moraxella catarrhalis and in eosinophilic asthma, Trophe
19 putum neutrophils correlated positively with Moraxella catarrhalis and negatively with Prevotella, Ne
20 he growth of the respiratory tract pathogens Moraxella catarrhalis and Neisseria meningitidis was als
21 pneumoniae and S. aureus, and Gram-negative, Moraxella catarrhalis and non-typeable Haemophilus influ
22 uence similarity to HumA, a heme receptor of Moraxella catarrhalis, and contains conserved motifs fou
23 th an increase in Haemophilus influenzae and Moraxella catarrhalis, and Haemophilus influenzae and Tr
24 ptococcus pneumoniae, Staphylococcus aureus, Moraxella catarrhalis, and Haemophilus influenzae detect
25 ride-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most recently in Acinetobacte
26 ined PF orthologs in Pseudomonas aeruginosa, Moraxella catarrhalis, and Staphylococcus aureus, bacter
30 ic bacterial strains Haemophilus influenzae, Moraxella catarrhalis, and Streptococcus pneumoniae is a
31 pathogenic bacteria-Haemophilus influenzae, Moraxella catarrhalis, and Streptococcus pneumoniae-for
33 e the acquisition of Haemophilus influenzae, Moraxella catarrhalis, and Streptococcus pneumoniae.
35 dA of Yersinia species, the UspA proteins of Moraxella catarrhalis, and the Eib proteins of Escherich
36 was identified in the human mucosal pathogen Moraxella catarrhalis, and the gene responsible for the
37 tococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, and, less frequently, group A Str
41 ccus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis are the etiologic agents of acute
42 iae, nontypeable Haemophilus influenzae, and Moraxella catarrhalis are the predominant bacteria assoc
43 cleotide open reading frame in the genome of Moraxella catarrhalis ATCC 43617 that was highly conserv
44 The nucleotide sequence from the genome of Moraxella catarrhalis ATCC 43617 was annotated and used
47 The pathogens Streptococcus pyogenes and Moraxella catarrhalis colonize overlapping regions of th
49 resence of viable Haemophilus influenzae and Moraxella catarrhalis (detected by PCR) in 106 middle-ea
52 age and density of Streptococcus pneumoniae, Moraxella catarrhalis, Haemophilus influenzae, and Staph
53 ecifically binds respiratory tract pathogens Moraxella catarrhalis, Haemophilus influenzae, and Strep
54 The high-molecular-weight UspA protein of Moraxella catarrhalis has been described as being both p
59 , depletion of Prevotella, and enrichment of Moraxella catarrhalis including drug-resistant strains.
96 usion and, with Streptococcus pneumoniae and Moraxella catarrhalis, is a causative agent of acute oti
97 e (LOS), a major outer membrane component of Moraxella catarrhalis, is a possible virulence factor in
100 that pili might be present on the surface of Moraxella catarrhalis isolates, detailed molecular and p
101 ed MAb 8E7 (immunoglobulin G3), specific for Moraxella catarrhalis lipooligosaccharide (LOS) was eval
103 ociated with Streptococcus pneumoniae and/or Moraxella catarrhalis lung coinfection (75%), living in
104 s mutants (SM), Streptococcus gordonii (SG), Moraxella catarrhalis (MC), and Porphyromonas gingivalis
105 s among diverse respiratory pathogens: NTHI, Moraxella catarrhalis (MC), Streptococcus pneumoniae (SP
106 aphylococcus aureus Cowan strain I (SAC) and Moraxella catarrhalis (MCat) stimulate human B cells by
108 he inflammation by Staphylococcus species or Moraxella catarrhalis might involve symptom appearance i
110 trate that a major outer membrane protein of Moraxella catarrhalis (Mx) strains, belonging to the ubi
111 te by the AniA protein is bactericidal for a Moraxella catarrhalis O35E norB mutant but not for wild-
113 occus pneumoniae, Haemophilus influenzae, or Moraxella catarrhalis on bacterial culture and by the pr
114 ial coinfection with Haemophilus influenzae, Moraxella catarrhalis, or Streptococcus pneumoniae (odds
115 d against the CopB outer membrane protein of Moraxella catarrhalis previously was found to enhance pu
116 yma whipplei, and Haemophilus influenzae and Moraxella catarrhalis, respectively, compared to HC.
119 s, N. gonorrhoea, Haemophilus influenzae and Moraxella catarrhalis share the property of targeting th
120 this association for Haemophilus influenzae, Moraxella catarrhalis, Staphylococcus aureus, and Pneumo
123 usly shown to be an adhesin expressed by the Moraxella catarrhalis strain O35E, which also displays e
124 seven uninoculated mother-sibling pairs, and Moraxella catarrhalis strain sharing in nine (38%) of 24
128 rways with the pathogenic bacterial species, Moraxella catarrhalis, Streptococcus pneumoniae, and Hae
129 s of nonencapsulated Haemophilus influenzae, Moraxella catarrhalis, Streptococcus pneumoniae, and Pse
130 l, nontypable Haemophilus influenzae (NTHi), Moraxella catarrhalis, Streptococcus pyogenes, and cultu
131 Previous studies have demonstrated that the Moraxella catarrhalis surface antigen UspA1 is an adhesi
132 ulum pigrum, Staphylococcus epidermidis, and Moraxella catarrhalis; the microbiome can be exploited w
133 o studies done on Neisseria meningitidis and Moraxella catarrhalis; the two other organisms with this
134 ucts that could contribute to the ability of Moraxella catarrhalis to colonize the human nasopharynx.
136 bacteria such as Haemophilus influenzae and Moraxella catarrhalis was found to be associated with th
137 posed, 81-kDa CopB outer membrane protein of Moraxella catarrhalis was found to be similar to those o
139 lococci, streptococci, Haemophilus spp., and Moraxella catarrhalis were minimal due to the high poten
141 ccus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were performed on all nasal sample
142 ccus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were significantly associated with
143 ccus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis) were identified in airway secreti
144 ane protein E (OMP E) is a 50-kDa protein of Moraxella catarrhalis which has several features that su
145 ane protein E (OMP E) is a 50-kDa protein of Moraxella catarrhalis which possesses several characteri
148 n shown to be involved in the ability of the Moraxella catarrhalis wild-type strain O35E to bind to h
149 sence of a 200-kDa protein on the surface of Moraxella catarrhalis with the ability of this organism