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1 CE ADVICE 6: In all patients undergoing BET, mucosal ablation should be applied to 1) all visible eso
2 in the tubular esophagus can be treated with mucosal ablation.
3 esophageal biopsies to rule out anatomic and mucosal abnormalities, esophageal high-resolution manome
4 d and bronchoalveolar lavage (BAL) following mucosal adenovirus (Ad)-SIV recombinant priming, intramu
5 of a broad spectrum of active compounds upon mucosal administration, mucus-penetrating and mucoadhesi
6 ese mice will be valuable for studying vagal mucosal afferent morphology, interactions with other GI
7                        Consistent with other mucosal AGM models of NiVB infection, we achieved unifor
8  alterations in gastrointestinal microbiota, mucosal and immune function, and CNS processing.
9 rointestinal microbiota, and disturbances in mucosal and immune function.
10 erns to HCMV, comparing infected human nasal mucosal and placental tissues, representing the viral en
11 luding careful inspection of the neosquamous mucosal and retroflexed inspection of the gastric cardia
12                                              Mucosal and skin bleeding scores, number of previous tre
13                                              Mucosal and skin cancers are associated with infections
14 ve-attenuated influenza vaccine induces both mucosal and systemic antibodies and systemic T-cell resp
15    Both regimens elicited strong, comparable mucosal and systemic cellular and humoral immunity.
16      The vaccine-only group developed strong mucosal and systemic humoral and cellular immunity but d
17 ies may strengthen the communication between mucosal and systemic immune compartments.
18                                 To study the mucosal and systemic immune response against norovirus,
19       Female C3H/HeN mice were vaccinated by mucosal and systemic routes with C. trachomatis serovar
20 xamination of the assessment of periodontal, mucosal, and oral health.
21                                  Respiratory mucosal antibodies and CD8+ T cells appear to contribute
22 e functions of IgA, these hitherto neglected mucosal antibodies may strengthen the communication betw
23         Our aim was to evaluate systemic and mucosal antibody responses toward SARS-CoV-2 in mild ver
24 (S) Immunoglobulin (Ig) A is the predominant mucosal antibody, which binds pathogens and commensal mi
25  This finding suggests a protective role for mucosal antibody-mediated immunity in naturally exposed
26 3 + T, CD4 + T, CD8 + T, NK, TCR-gammadelta, Mucosal associated invariant (MAIT), and NKT cells as we
27                                              Mucosal Associated Invariant T (MAIT) cells can sense in
28 R1 presents vitamin B-related metabolites to mucosal associated invariant T (MAIT) cells, which are c
29      Also recognised by the alphabeta-TCR of mucosal associated invariant T cells, MR1 interacts with
30 luding invariant natural killer T (iNKT) and mucosal-associated innate T (MAIT) cells, are a heteroge
31                              In human blood, mucosal-associated invariant T (MAIT) cells are abundant
32                                              Mucosal-associated invariant T (MAIT) cells are importan
33                                              Mucosal-associated invariant T (MAIT) cells are innate T
34                                              Mucosal-associated invariant T (MAIT) cells are innate-l
35                                              Mucosal-associated invariant T (MAIT) cells have been at
36 the cirrhosis-associated immune dysfunction, mucosal-associated invariant T (MAIT) cells, which have
37 microbial vitamin B(2) synthesis to activate mucosal-associated invariant T (MAIT) cells.
38 MR1 presents riboflavin-based metabolites to Mucosal-Associated Invariant T (MAIT) cells.
39 e natural killer (NK), invariant NKT (iNKT), mucosal-associated invariant T (MAIT), and Vdelta2(+) ga
40 nse of three TCR gammadelta cell subsets and mucosal-associated invariant T cells in healthy voluntee
41 -like B cells (MZB); gammadelta T cells; and mucosal-associated invariant T cells in RM from 69 HIV-n
42                                       Unlike mucosal-associated invariant T cells, recognition of tar
43 l T cell subsets, such as NKT, gammadelta T, mucosal-associated invariant T, and CD8alphaalpha T cell
44 ucture of mucus, the epithelial barrier, the mucosal-associated lymphatic tissues and microbiota.
45              A mutation in SPLUNC1 affecting mucosal attachment, biofilm formation, and invasion of m
46  wound repair is critical to reestablish the mucosal barrier and homeostasis.
47  mitigates gamma-irradiation-induced colonic mucosal barrier dysfunction and endotoxemia.
48 phoid cells (ILCs) play fundamental roles in mucosal barrier functionality and tissue homeostasis, IL
49 e growth of beneficial bacteria, can enhance mucosal barrier integrity, and reduce chronic inflammati
50 notype is caused by a persistently disrupted mucosal barrier, but assessments of gut mucosal immunolo
51 te obesity-associated defects in the colonic mucosal barrier, even in the presence of dietary fiber.
52 robiome and innate immune components in oral mucosal barriers indicates that a signaling hub coordina
53 l route can be utilized, however degradative mucosal barriers must be overcome.
54 ocytes that promote immunity to pathogens at mucosal barriers, but the mechanisms regulating their de
55 matory ILC2 responses and type 2 immunity at mucosal barriers.
56 option to providers to remove complex, large mucosal-based lesions in the GI tract using standard end
57 ow herpesvirus virions overcome the abundant mucosal beta-defensins during host invasion.
58  average gut mucosal microbiome composition, mucosal biopsies allowed detecting the subtle variations
59 ns are needed to elucidate the exact role of mucosal biopsies for fVCA patient management.
60 s (IECs) and mice, and we measured levels in mucosal biopsies from patients with IBD.
61       This work used shotgun metagenomics of mucosal biopsies to explore the microbial communities' c
62 ulfovibrio was enriched both in stool and in mucosal biopsies.
63                                              Mucosal biopsy specimens from patients with ulcerative c
64 ole of the microbiota in regulating not only mucosal but also systemic immune responses has led to in
65 ucosal immunity in the context of cancer and mucosal cancer clinical trials is provided.
66 serves as the main etiological agent of oral mucosal candidiasis, in which a Candida-bacteriome partn
67 contrast, the cytotoxic marker expression by mucosal CD4(+) and CD8(+) T cells differed according to
68                         Therefore, pulmonary mucosal CD8 T cells are more differentiated, activated,
69                                Activation of mucosal CD8+ T cells, particularly tissue-resident memor
70 and gene expression profiles in upper airway mucosal cells and assessed AR at age 6 years in children
71 fferentially methylated CpGs in upper airway mucosal cells at age 6 years, 792 of which formed 3 modu
72  innate and adaptive immune responses in the mucosal cells of the intestine.
73 red DNA methylation patterns in upper airway mucosal cells.
74  56% had conjunctival injection, and 27% had mucosal changes.
75 LP strains colonize the intestine, reduce Lm mucosal colonization and systemic dissemination, and pro
76 owth on mucus as a carbon source, as well as mucosal colonization of mice.
77 pathways and the functional impact of IgA on mucosal commensal bacteria.
78 and CD8(+) T cells differed according to the mucosal compartment.
79 ilored' surgical approaches, focusing on the mucosal concept in those with severe CRSwNP and on the i
80 r class of nonpeptidergic neurons innervated mucosal crypts, myenteric ganglia, and submucosa.
81 were clinical remission (clinical) and bowel mucosal damage (preclinical).
82 ciated with a significant reduction in bowel mucosal damage compared to placebo (standardized mean di
83 roliferation and regeneration in response to mucosal damage.
84 rvation periods without an increased risk of mucosal damage.
85 or doxorubicin (DXR)-induced small intestine mucosal damage.
86 oncordance of the results with the degree of mucosal damage.
87 -organ injury following the chemical-induced mucosal damage.
88 cooler months-which coincided with when host mucosal defences were most potent against Bd.
89 esis.IMPORTANCE How herpesviruses circumvent mucosal defenses to promote infection of new hosts throu
90 EoE-specific steroid formulations optimizing mucosal deposition have been developed, which has culmin
91       We investigated circulating markers of mucosal dysfunction, immune activation, mucosal Th17 and
92 es are needed to determine the mechanisms of mucosal dysregulation in patients with inflammatory bowe
93 ial events of viral infection at the primary mucosal entry site following horizontal person-to-person
94                           This pro-T(h)2 gut mucosal environment inhibited the induction of antigen-s
95 anding how antibodies are distributed in the mucosal environment is valuable for developing a vaccine
96  Experimental RV infection induces bronchial mucosal eosinophilia and neutrophilia only in patients w
97 enitis are characterized by gastrointestinal mucosal eosinophilia, chronic symptoms, impaired quality
98 infection increased the numbers of bronchial mucosal eosinophils and neutrophils only in COPD and CD8
99                                     Duodenal mucosal eosinophils, mast cells and permeability were qu
100 ease in peripheral tissues, such as skin and mucosal epithelia.
101 lates mitochondrial respiration in two human mucosal epithelial cell types: insulin-insensitive corne
102 tachment, biofilm formation, and invasion of mucosal epithelial cells is a new genetic cause of menin
103 aborate maze-like patterns on the surface of mucosal epithelial cells.
104 rum are intracellular bacterial pathogens of mucosal epithelial cells.
105                             ZIKV infects the mucosal epithelium and submucosal dendritic cells, induc
106 in a mouse model of LASV infection, in which mucosal exposure resulted in remarkably high lethality c
107 er of these routes adequately models natural mucosal exposure to NiV.
108 steum at the base of the graft; whereas, the mucosal flap margin in the control group was left free.
109 inkage when performing FSTA surgery when the mucosal flap margin is left free and unsutured when comp
110                        In the test group the mucosal flap margin was sutured apically to the perioste
111 gand [CCL]2, CCL3, CCL4, and CCL13) in nasal mucosal fluid, without causing systemic immune activatio
112  SARS-CoV-2-specific IgA and IgG in sera and mucosal fluids of 2 cohorts, including SARS-CoV-2 PCR-po
113 ody titers showed SARS-CoV-2-specific IgA in mucosal fluids with virus-neutralizing capacity in some
114 the role of endogenous galectin-9 (Gal-9), a mucosal galectin that has been linked to inflammatory bo
115                                        These mucosal gateways are not only portals of entry for poten
116         Here we construct autologous jejunal mucosal grafts using biomaterials from pediatric patient
117                                              Mucosal grass pollen allergen exposure by SLIT resulted
118 t endpoint, which we called histo-endoscopic mucosal healing (or histo-endoscopic mucosal improvement
119 ndicated the achievement of histo-endoscopic mucosal healing after induction therapy is associated wi
120     There was an inverse association between mucosal healing risk of future small bowel adenocarcinom
121  10% to 20% higher rates of histo-endoscopic mucosal healing, clinical remission, and corticosteroid-
122                     Secretor status controls mucosal histoblood group antigen expression and is assoc
123 pecifically, superior sustained systemic and mucosal HIV Gag-specific poly-functional/cytotoxic CD4(+
124  the importance of inflammatory cytokines in mucosal HIV infection, demonstrating the likely critical
125  these innate immune responses play in early mucosal HIV replication in humans.
126  and Desmocollin-2 (Dsc2) that contribute to mucosal homeostasis by strengthening intercellular adhes
127 nsic involvement of leptin in the control of mucosal homeostasis.
128 d an S1PR1+ and alpha4beta7+ emigration- and mucosal-homing competent phenotype, while this was restr
129      Measurements at equilibrium showed that mucosal hydration reduced the release of only two compou
130 ture around the topic of innate immunity and mucosal hypoxia with a focus on transcriptional response
131 h allergic disorders have an increased nasal mucosal IFN and chemokine response to the viral RNA anal
132  significantly higher titers of RSV-specific mucosal IgA antibodies.
133 an initial infection induced a serum IgM and mucosal IgA response against the toxin, but a low serum
134 xhibited microbiota-dependent differences in mucosal IgA responses to oral vaccination with cholera t
135  or intestinal acidification, influence host mucosal immune cells and enterocytes via butyrate produc
136 stricted to the colon, enterocyte damage and mucosal immune dysfunction play a role for insufficient
137  and activation (CCL3, CCL4, and CDCP1), and mucosal immune dysregulation (IL-17A, CCL20, and CCL28).
138 ound, environmental and luminal factors, and mucosal immune dysregulation, have been suggested to con
139  microbial dysbiosis within the gut, altered mucosal immune function, visceral hypersensitivity, and
140                   Humans develop an array of mucosal immune responses following S. Typhi infection.
141 owever, despite their critical importance in mucosal immune responses, very little is known about the
142  the microbiome, which is known to influence mucosal immune responses.
143 trains in driving phenotype variation in the mucosal immune system and provide a strategy to robustly
144 icobacter pylori colonization may affect the mucosal immune system through modification of microbiota
145 development of the intestinal microbiota and mucosal immune system.
146 es and involve complex interactions with the mucosal immune system.
147 ar pathways that regulate homeostasis of the mucosal immune system.
148 icrobiome early in life imprint the host gut mucosal immunity and may play a critical role in precipi
149 ion of this niche, and many are important in mucosal immunity and vaccine development.
150                                              Mucosal immunity develops in the human fetal intestine b
151                         Here, an overview of mucosal immunity in the context of cancer and mucosal ca
152               Inducing humoral, cellular and mucosal immunity is likely to improve the effectiveness
153      Vitamin D exerts a regulatory role over mucosal immunity via the vitamin D receptor (VDR).
154                          No vaccine to boost mucosal immunity, or as a therapeutic, has yet been deve
155 ting as both induction and effector site for mucosal immunity.
156 pteronyssinus had no effect on offspring gut mucosal immunity.
157 utralizing antibodies, promotes systemic and mucosal immunoglobulin A (IgA) and T cell responses, and
158 ering a surge of excitement in the fields of mucosal immunology and microbiology.
159 pted mucosal barrier, but assessments of gut mucosal immunology in different anatomical compartments
160 tory works on a wide variety of topics, from mucosal immunology to viruses, and recently she publishe
161 oscopic mucosal healing (or histo-endoscopic mucosal improvement).
162 esponses in an acute superficial respiratory mucosal infection and their implications in development
163 is in multiple U.S. cities since 2015, other mucosal infections, and cases of invasive meningococcal
164 nal epithelial barrier, commonly observed in mucosal inflammation and diseases such as inflammatory b
165  of cytokines may be important in regulating mucosal inflammation and homeostasis.
166 r to WD-fed knockout mice attenuated colonic mucosal inflammation and improved CEB.
167                                         Both mucosal inflammation and psychologic dysfunction have be
168 ia; and, 2) relationships between nausea and mucosal inflammation as defined by antral and duodenal e
169            Chronic, low-grade, systemic, and mucosal inflammation correlates with increased morbidity
170 determine the extent and nature of bronchial mucosal inflammation following experimental rhinovirus (
171                       Whether the associated mucosal inflammation is important or redundant for effec
172 clear whether virus per se induces bronchial mucosal inflammation, nor whether this relates to exacer
173 nterobacteriaceae that exacerbated low-grade mucosal inflammation, suggesting that remediating dysbio
174 CII from IECs reduces but does not eliminate mucosal inflammation.
175 ed intestinal epithelial barrier and chronic mucosal inflammation.
176 intestinal microbiotic dysbiosis and chronic mucosal inflammation.To assess phenotypic changes in pro
177                                    Bronchial mucosal inflammatory cell phenotypes were determined at
178 s locally produced in response to intestinal mucosal injury.
179 e early innate gammadelta T cell response to mucosal injury.
180 elivery of R848 enables simple assessment of mucosal innate responsiveness, revealing that patients w
181                                  Loss of gut mucosal integrity and an aberrant gut microbiota are pro
182 ediated interdomain communication at the gut-mucosal interface.
183 ated with a rapid, presymptomatic decline in mucosal interleukin-17A (IL-17A) and other mediators.
184 unofluorescence localization of TJ proteins, mucosal inulin permeability, and plasma lipopolysacchari
185  previously undescribed role for Mtb EsxA in mucosal invasion and identify SR-B1 as the airway M cell
186                                     The slug mucosal irritation assay revealed that a low level of mu
187 us was secreted by slugs indicating moderate mucosal irritation.
188 the adaptive immune system in submucosal and mucosal isolated lymphoid follicles (SM-ILFs and M-ILFs,
189 njury: complex lesions (intramural hematoma, mucosal laceration) and minor lesions (petechiae, ecchym
190 ed network intimately adherent to the entire mucosal lamina propria vasculature.
191 nd prevent microorganisms from breaching the mucosal layer, which is accomplished through crosstalk w
192 L.) infantum causes visceral, cutaneous, and mucosal leishmaniasis in humans and canine leishmaniasis
193      Because the clinical appearance of oral mucosal lesions is not an adequate indicator of their di
194  En-bloc resection of large, flat dysplastic mucosal lesions of the luminal GI tract can be challengi
195 imals (4 of 4, 100%; P=0.005): a mean of 1.5 mucosal lesions/animal (length, -21.8+/-8.9 mm; width, -
196       Additionally, the change in mid-buccal mucosal level (MBML) and approximal marginal bone level
197 he same time better maintains the mid-buccal mucosal level.
198 ocrine phenotype and contribute to increased mucosal levels of acetylcholine.
199           In contrast to the lymph nodes and mucosal lymphoid tissues with well-defined entry and exi
200 haracterized by an apical discrepancy of the mucosal margin respective to its ideal position with or
201 oup has previously identified IL-9-producing mucosal mast cell (MMC9) as the primary source of IL-9 t
202 er levels of PNA-specific IgE and intestinal mucosal mast cells and eosinophils over sham treatments.
203 philic esophagitis have increased numbers of mucosal mast cells.
204                                Patients with mucosal melanoma may be offered the same therapies recom
205     Key exclusion criteria included uveal or mucosal melanoma, previous therapy with checkpoint inhib
206 ed pressure injuries", "pressure injuries in mucosal membranes", "pressure injuries in pediatric popu
207 pneumonia, with RESTV shedding from oronasal mucosal membranes.
208                                          Gut mucosal microbes evolved closest to the host, developing
209 ided a good approximation of the average gut mucosal microbiome composition, mucosal biopsies allowed
210  the establishment and stability of the oral mucosal microbiome in mice, we evaluated oral microbiome
211    Given their significant enrichment in the mucosal microbiota, we highlight the roles of Bacteroide
212 d smokers had increased numbers of bronchial mucosal monocytes/macrophages and CD8(+) T lymphocytes b
213 ble complications derived from radiotherapy (mucosal necrosis, osteoradionecrosis, vasculopathy, cere
214 m of healthy subjects, intraductal papillary mucosal neoplasms and pancreatic ductal adenocarcinoma i
215                                              Mucosal neutrophil activation before exposure was highly
216 sis, indicating that bacteria inhabiting the mucosal niche are metabolically active.
217 ciated with M1b disease and cutaneous versus mucosal or acral primaries.
218                      Successful infection by mucosal pathogens requires overcoming the mucus barrier.
219 mmune system's first line of defense against mucosal pathogens.
220 2-one evidenced that the constituents of the mucosal pellicle influenced release kinetics differently
221 ht junction and adherens junction, increased mucosal permeability, and elevated plasma LPS; TJ disrup
222 jacent to the conducting airway and assume a mucosal phenotype (MC(T)).
223 ith thin (<=2 mm) and thick (>2 mm) vertical mucosal phenotypes.
224         The vaccine regimens, especially the mucosal primes, significantly altered the rectal microbi
225 estigated two SIV vaccine regimens combining mucosal priming immunizations and systemic protein boost
226 mm (95% CI = 0.12 to 0.59 mm, P = 0.003) for mucosal recession (MR); a WMD of 0.13 mm (95% CI = -0.11
227 ociation was observed to increased levels of mucosal redness (P = 0.026) and the full-mouth bleeding
228 equently occurs; however, the added value of mucosal rejection assessment for patient management is u
229 es to identify articles that provide data on mucosal rejection following fVCA.
230                                              Mucosal rejection frequently occurs; however, the added
231  skin rejection does not occur in absence of mucosal rejection.
232 -Cre(ERT2); Dsc2 (fl/fl)) exhibited impaired mucosal repair after biopsy-induced colonic wounding and
233                                   Endoscopic mucosal resection (EMR) is a minimally invasive procedur
234  sexual dimorphism in human small intestinal mucosal responses to IR.
235 gen induced specific IgG (systemic) and IgA (mucosal) responses against LTB, ST, and LptD epitopes.
236                      To circumvent this, the mucosal route can be utilized, however degradative mucos
237 y, we compare microbial loads in lumenal and mucosal samples along the GI tract.
238                                              Mucosal samples were collected once at the enrollment vi
239                   SARS-CoV-2 was detected in mucosal samples, including rectal swabs, as late as 15 d
240 -CoV-2-specific antibodies but may stimulate mucosal SARS-CoV-2-specific IgA secretion.
241                  We further demonstrate that mucosal secretory IgA is not recognized by FCRL4 and tha
242 ecause of their rapid cytotoxic responses at mucosal services to a widely conserved bacterial ligand.
243 ed anterogradely inside axons to the initial mucosal site of infection, rely on axonal bidirectional
244 e reproductive tract (FRT), similar to other mucosal sites, harbours a site-specific microbiome, whic
245 ment of unique microbial ecosystems in the 2 mucosal sites.
246  pathogens and the normal flora of different mucosal sites.IMPORTANCE Sialic acids (Sia) are involved
247 udy clearly demonstrates direct effects of a mucosal SIV vaccine regimen on the rectal microbiome and
248 ficacy and safety of mechanical adjuvants in mucosal-sparing, mechanical endoscopic dacryocystorhinos
249 d intestinal microbiota were analyzed in the mucosal specimen.
250 mucosa-adherent microbiota signatures in gut mucosal specimens from sigmoid colon and terminal ileum
251 s a radiation-free examination of the entire mucosal surface of the small bowel with high-quality ima
252 dant acquired immune component secreted onto mucosal surfaces and, via the process of immune exclusio
253                  Humoral immune responses at mucosal surfaces have historically focused on IgA.
254 at innervate barrier sites like the skin and mucosal surfaces like the eye.
255                                              Mucosal surfaces like those present in the lung, gut, an
256 pria of the gastrointestinal tract and other mucosal surfaces of humans and mice host a network of mo
257                                              Mucosal surfaces such as fish gills interface between th
258 icrobials, pathogenesis, and adhesion to the mucosal surfaces to colonize the host.
259 t will afford antitumor immune protection at mucosal surfaces, despite limited knowledge surrounding
260 l ganglia following the initial infection at mucosal surfaces.
261 e allergens that initiate type 2 immunity at mucosal surfaces.
262 elopment of type 2 inflammatory responses at mucosal surfaces.
263 d direct contact with patients' conjunctival mucosal surfaces.
264 mune functionality and tissue homeostasis at mucosal surfaces.
265 processes that mediate epithelial healing at mucosal surfaces.
266 ogen entry and provide optimal protection of mucosal surfaces.
267 niquely expressed in epithelial cells lining mucosal surfaces.
268 eating diseases associated with dysbiosis at mucosal surfaces.
269 d unpredictable episodes of subcutaneous and mucosal swelling that can be life threatening.
270                  On day 4 she developed oral mucosal symptoms (paresthesia of the tongue, sore and an
271 iven the presentation of fevers, rashes, and mucosal symptoms observed in many of these syndromes, pa
272 mmunogenicity and protective efficacy of two mucosal/systemic vaccine regimens and investigated their
273  murine model, we detail whether respiratory mucosal TB vaccination profoundly alters the airway inna
274 s of mucosal dysfunction, immune activation, mucosal Th17 and Th22 cells, and mucosa-adherent microbi
275 re esophagogastroduodenoscopy revealed minor mucosal thermal injury in 2 of 36 RF/PF and 0 of 24 PF/P
276 reased branching (59%; P < 0.001), increased mucosal thickness (34%; P < 0.001), and increased epithe
277  and volumetric three-dimensional changes in mucosal thickness (MT) 1 year after treatment with an ac
278 ompasses the keratinized mucosa width (KMW), mucosal thickness (MT), and supracrestal tissue height (
279 l tissue height plays a key role in MBL than mucosal thickness in tissue level implant.
280                   Stomach weight and gastric mucosal thickness were also reduced in infected Stat3(SA
281 abundantly present in genital and intestinal mucosal tissue and are among the first innate immune cel
282 greater inhibitory potencies in cellular and mucosal tissue pre-clinical models than the parent seque
283                                   To protect mucosal tissues and maintain immune tolerance, animal ho
284 uitous commensal fungus that colonizes human mucosal tissues and skin, can become pathogenic, clinica
285                                              Mucosal tissues constitute the largest interface between
286 ng proteins involved in autophagy in colonic mucosal tissues from patients with sporadic CRC colonize
287 T) cells are innate-like T cells enriched in mucosal tissues that recognize bacterial ligands.
288 overviews the complex structural features of mucosal tissues, including the structure of mucus, the e
289  the full cellular repertoire of human nasal mucosal tissues.
290 can reactivate to cause recurrent lesions in mucosal tissues.
291 of lymphocytes, and thus vaccine efficacy in mucosal tissues.
292 ntification of the microorganisms colonizing mucosal tissues.
293 tence of gut bacteria than simply protecting mucosal tissues.
294                                              Mucosal TNF transcripts showed high test reliability for
295                        However, dysregulated mucosal trafficking of PMNs and associated epithelial ti
296 faces, despite limited knowledge surrounding mucosal vaccination, particularly aided by biomaterials
297 evelopment of a safer and potent alternative mucosal vaccine to replace live vaccines against IBV and
298 al blood and dynamic adhesion to recombinant mucosal vascular addressin cell adhesion molecule (MAdCA
299 lial Dsc2 is a key contributor to intestinal mucosal wound healing in vivo.
300 ng, increased epithelial cell migration, and mucosal wound repair.

 
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