ライフサイエンスコーパス: Muller cell

1 aquaporin 4 (AQP4) water channel in retinal Muller cells.

2 loss of adhesion between photoreceptors and Muller cells.

3 Further, we show that MFAT is expressed in Muller cells.

4 tina and retinal endothelial cells (REC) and Muller cells.

5 an 11-cis-specific retinyl-ester synthase in Muller cells.

6 a of the mouse retina such as astrocytes and Muller cells.

7 sEH-dependent generation of a diol of DHA in Muller cells.

8 y TRPV4 antagonists and absent in TRPV4(-/-) Muller cells.

9 tional deletion of Crb2 in photoreceptors or Muller cells.

10 Pathogenesis involves toxicity to retinal Muller cells.

11 with the use of 11-cis-retinol supplied from Muller cells.

12 -dependent signaling pathways in bipolar and Muller cells.

13 HIF-1alpha and ANGPTL4 localize to ischemic Muller cells.

14 0eembedded shRNA against VEGFA that targeted Muller cells.

15 nt to promote vessel permeability by hypoxic Muller cells.

16 patial organization similar to those seen in Muller cells.

17 tem cells (ESMVs) have on retinal progenitor Muller cells.

18 27a1-/- retina was hypoxic and had activated Muller cells.

19 olocalized with glutamine synthetase-labeled Muller cells.

20 s were present in treated but not in control Muller cells.

21 asal levels of endogenous human Oct4 mRNA in Muller cells.

22 o gene expression by STAT3 activation in rat Muller cells.

23 lar adhesion molecule 1 and VEGF proteins in Muller cells.

24 lpha (Pdgfralpha)-Cre mice to delete Des1 in Muller cells.

25 s in comparison with unregulated promoter in Muller cells.

26 re more activated in macular than peripheral Muller cells.

27 l significantly reduced death of the retinal Muller cells.

28 o cones by using 11-cis-retinol generated in Muller cells.

29 na, and are expressed by retinal neurons and Muller cells.

30 ular location of FRalpha and PCFT in primary Muller cells.

31 ERG components and showed immunostaining of Muller cells.

32 lipase A(2) (PLA(2)) activity exclusively in Muller cells.

33 ferative diabetic retinopathy can arise from Muller cells.

34 induce GAPDH nuclear accumulation in retinal Muller cells.

35 ere assessed by Western blot analysis in rat Muller cells.

36 ocation and subsequent cell death in retinal Muller cells.

37 Instead, they found that Ad5/F37 transduced Muller cells.

38 as predominantly localized in the nucleus of Muller cells.

39 nd the amino acid serine, synthesized by the Muller cells.

40 enzyme in serine synthesis) than peripheral Muller cells.

41 ence between them may be the nature of their Muller cells.

42 Muller cells from diabetic mice with CD40(+) Muller cells.

43 not cause TNF-alpha or IL-1beta secretion in Muller cells.

44 um entry that replenishes ER stores in mouse Muller cells.

45 aldehyde binding protein (CRALBP) present in Muller cells.

46 -retinal from the RPE, possibly imported via Muller cells.

47 desaturase-1 (DES1), is expressed in RPE and Muller cells.

48 emokine and cytokine expression in activated Muller cells.

49 s mediated by the 11-cis selective CRALBP in Muller cells.

50 ehyde binding protein 1 gene for conditional Muller cell ablation and the consequences of primary Mul

51 sis entering the tricarboxylic acid cycle in Muller cells accompanied by increased glutamine consumpt

52 ted retinal ganglion cell axon regeneration, Muller cell activation and CNTF production in the retina

53 Retinal vasculature and Muller cell activation were analyzed by quantifying acel

54 vasculature degeneration, and inhibition of Muller cell activation were demonstrated in the treated

55 adherent area, stiffness, and propagation of Muller cells all are affected by matrix stiffness, but t

56 The Wnt-responsive Muller cells also exhibited long-term survival and, in s

57 so we could specifically quantify changes in Muller cell anatomy between control mice (C57Bl/6) and t

58 marker Pax2 but inconsistently expressed the Muller cell and occasional astrocyte marker CRALBP.

59 leted in colorectal carcinoma), was found in Muller cells and astrocytes.

60 FRalpha and PCFT are expressed in retinal Muller cells and colocalize in the endosomal compartment

61 OPL and HFL, indicating junctions in between Muller cells and cone axons.

62 equence of loss of control with depletion of Muller cells and exposure of the remaining retinal vesse

63 old increase in promoter activity in primary Muller cells and human Muller cell lines, respectively.

64 y reduced the reactive expression of GFAP in Muller cells and improved the morphology of astrocytes i

65 understand better the mechanosensitivity of Muller cells and its association with vitreoretinal diso

66 Muller cells and macrophages/microglia are likely import

67 MacTel zone that coincides with the loss of Muller cells and macular pigment.

68 s for two weeks resulted in proliferation of Muller cells and maintenance of some rod and cone photor

69 nced retinal disease highlighting a role for Muller cells and may inform future therapeutic strategie

70 and activity were also increased in cultured Muller cells and microglia treated with LPS.

71 glaucoma, upregulates TNFalpha production by Muller cells and microglia.

72 del, we show that TNFalpha is upregulated by Muller cells and microglia/macrophages soon after induct

73 tural change is supported by distribution of Muller cells and patterns of AQP4 expression.

74 ion suppressed the improper proliferation of Muller cells and promoted the regression of vascular ves

75 Retinal astrocytes/Muller cells and retinal ganglion cells were the source

76 ptor in angiogenic cell behaviors of retinal Muller cells and retinal microvascular endothelial cells

77 ear layer; they are present in primary mouse Muller cells and rMC-1 cells.

78 s in the regulation of potassium channels in Muller cells and subsequently in the promotion of glutam

79 n mouse, about the anatomical arrangement of Muller cells and their arbors, and how these features ar

80 wed macrophages, granulocytes (neutrophils), Muller cells, and microglial cells as TNF-alpha sources.

81 targets were ganglion cells, bipolar cells, Muller cells, and photoreceptors.

82 meshwork, ciliary body, ciliary epithelium, Muller cells, and retinal ganglion cells.

83 photoreceptors, bipolar cells, mitochondria, Muller cells, and retinal pigment epithelium (RPE) cells

84 neural retina and RPE/eyecup, primary mouse Muller cells, and rMC-1 cells for the three known folate

85 viding cells, with anti-vimentin to identify Muller cells, and with the isolectin B4 to identify micr

86 bution of glutamine synthetase, expressed by Muller cells, and zonula occludens-1, a tight-junction p

87 embranes of photoreceptor inner segments and Muller cell apical processes in the zebrafish retina.

88 the putative all-trans retinol isomerase in Muller cells, appears to be 9-cis retinol.

89 Additionally, the mislocalized Muller cells are observed in the photoreceptor layer in

90 Positively identified Muller cells are present in diabetic epiretinal tissues

91 Muller cells are principal glial cells in rat retina and

92 Muller cells are the major glia of the retina that serve

93 A subpopulation of mammalian Muller cells are Wnt responsive and, when Wnt signaling

94 toskeletal integrity, and gene regulation in Muller cells as a function of the stiffness of the subst

95 uorescent protein expression only in retinal Muller cells at P17.

96 P = 0.03, Student's t test), and gliosis in Muller cells (at 6 mo, using SPION-glial fibrillary acid

97 n of glutamine and glutamate was observed in Muller cells before established gliosis markers.

98 In addition, abnormal positioning of Muller cell bodies and bipolar cells was evident through

99 Gliosis, characterized by translocation of Muller cell bodies to the outer retina and thickening of

100 The retinal area occupied by the total Muller cell body (soma and processes) was significantly

101 ls within the retinal pigment epithelium and Muller cells, but may limit iron transport into the reti

102 oration of CRALBP expression specifically in Muller cells, but not retinal pigment epithelial (RPE) c

103 Swelling and [Ca(2+)]i elevations evoked in Muller cells by hypotonic stimulation were antagonized b

104 Knockdown of VEGFA in rat Muller cells by lentivector VEGFA-shRNA significantly re

105 Muller cell changes in culture included loss of glutamin

106 c rodents and by hyperglycemic conditions in Muller cells concomitant with increased VEGF expression.

107 ostaglandin E(2) (PGE(2)) and VEGF levels in Muller cell-conditioned medium and in retinal tissue sam

108 ed hypoxia-induced PGE(2) and VEGF levels in Muller cell-conditioned medium by 68.6% (P < 0.001) and

109 The previously reported increases in Muller cell contraction-promoting activity detected in h

110 iated with significant increases in vitreous Muller cell contraction-promoting activity that are pres

111 diabetic swine were evaluated for changes in Muller cell contraction-promoting activity, the presence

112 Our results identify Muller cell CRALBP as a key component of the retinal vis

113 ype 2 diabetic rat model, as well as REC and Muller cells cultured in normoglycemia and hyperglycemic

114 ion of insulin receptor signaling in retinal Muller cells cultured under hyperglycemic conditions and

115 Similar analyses were performed in Muller cell cultures exposed to hyperglycemic conditions

116 ow levels in the retina, and none in primary Muller cell cultures, indicating the presence of Isomera

117 In human MIO-M1 Muller cell cultures, REDD1 deletion prevented oxidative

118 induce neurogenesis in vivo and in purified Muller cell cultures.

119 lipoproteins may be implicated in apoptotic Muller cell death, acting at least partially via enhance

120 cusing on its ability to influence mammalian Muller cell dedifferentiation, proliferation, and neurog

121 ngiogenic therapies may be required to treat Muller cell deficiency in retinal diseases and in other

122 In contrast, Muller cells demonstrated robust extracellular matrix co

123 x2 in the mouse postnatal retina, leading to Muller cell depletion and retinal degeneration.

124 Muller cells derived from COX-2-null mice were treated w

125 l role in retinal inflammatory signaling and Muller cell-derived inflammatory cytokine production in

126 ription factor 4 (ATF4) in the regulation of Muller cell-derived inflammatory mediators in diabetic r

127 ntial, was similar between rd/rd and control Muller cells during cone degeneration.

128 ell ablation and the consequences of primary Muller cell dysfunction have been studied in adult mice.

129 nd vascular pathogenic pathways secondary to Muller cell dysfunction, the cause of which remains obsc

130 the slow PII component, suggesting specific Muller cell dysfunction.

131 f VEGFA by lentivector VEGFA-shRNAetargeting Muller cells efficiently reduced 50/10 OIR up-regulated

132 Muller cell electrophysiology, particularly K(+) current

133 ESMVs transferred to Muller cells embryonic stem cell (ESC) mRNAs involved in

134 inner segments, and pedicles, as well as to Muller cell endfeet.

135 or alpha (PPARalpha) is expressed in retinal Muller cells, endothelial cells, and in retinal pigment

136 ImM10 Muller cells express numerous genes associated with neur

137 In this article, we show that Muller cells express TLR2, a key sensor implicated in re

138 Retinal endothelial and Muller cells expressed CD40.

139 Macular Muller cells expressed more phosphoglycerate dehydrogena

140 We assessed cone functional dependence on Muller cell-expressed Des1 through a conditional knockou

141 entiviral-delivered shRNAs that knocked down Muller cell-expressed VEGF in the retinopathy of prematu

142 Immunohistochemistry confirmed that Muller cells followed a radial distribution around the f

143 stmortem histologic artifact of eosinophilic Muller cell foot process swelling that mimics a nerve fi

144 A focal artifact of Muller cell foot process swelling was identified in most

145 iew demonstrates an artifact of eosinophilic Muller cell foot processes swelling in postmortem examin

146 han 3 months, demonstrated diffusely swollen Muller cell foot processes with intensely eosinophilic c

147 TNF-alpha was not detected in Muller cells from diabetic mice with CD40(+) Muller cell

148 We found primary cultured Muller cells from macula and peripheral retina display s

149 thread in many retinal diseases is reactive Muller cell gliosis, an untreatable condition that leads

150 s, migration, and formation of new synapses; Muller cell gliosis, migration, and scarring; blood vess

151 y on P2RX7 caused further neurodegeneration, Muller cell gliosis, progenitor proliferation, and micro

152 show that GPR81, localized predominantly in Muller cells, governs deep vascular complex formation du

153 In cultured Muller cells, high glucose induced a time-dependent incr

154 pigment epithelial cells (HRPEs), and human Muller cells (HMCs) under elevated glucose conditions wa

155 In Muller cells, hyperglycemic conditions attenuated global

156 kening of OPLHenle in older eyes may reflect Muller cell hypertrophy associated with rod loss.

157 In Muller cells, hypoxia increased the phosphorylation of c

158 rhombencephalon at the level of the isthmic Muller cell I1 close to sulcus limitans of His.

159 retinal endothelial cells (REC) and retinal Muller cells in either 5 mM (normal) or 25 mM (high) glu

160 Furthermore, IKKbeta isolated from Muller cells in normal glucose medium was found to be gl

161 porting a potential mechanism of traction by Muller cells in the CB.

162 iated virus (AAV) variant, ShH10, transduces Muller cells in the Dp71-null mouse retina efficiently a

163 tetrahydrofolate ([(3)H]-MTF) was assayed in Muller cells in the presence/absence of thiamine pyropho

164 anatomical remodeling and gliosis of retinal Muller cells in the rd/rd mouse model of photoreceptor d

165 explores the nature of the cis retinol that Muller cells in the retina provide to cones for the rege

166 VEGF) expression were partially localized to Muller cells in the retina.

167 We found that the Muller cells in the salamander neural retina promote con

168 a cytokine up-regulated by HIF-1 in hypoxic Muller cells in vitro and the ischemic inner retina in v

169 ced expression of progenitor cell markers by Muller cells in vitro or stimulated Muller cell migratio

170 00 mum composed of densely packed cones (and Muller cells) in the central fovea.

171 oxia, anaplerotic catabolism of glutamine by Muller cells increased ammonium release two-fold.

172 gative CASP6 mutant activates astrocytes and Muller cells, increases CNTF levels in the retina and le

173 lation of hypoxia-inducible factor-1alpha in Muller cells induces the expression of VEGF, which, in t

174 duced release of inflammatory mediators from Muller cells, inhibited accumulation of mononuclear phag

175 ese results indicate that Des1 expression in Muller cells is not required for cone visual pigment reg

176 Our studies indicate that CD40 in Muller cells is sufficient to upregulate retinal inflamm

177 e hybrid retinas and expressed abundantly in Muller cells, is the enzyme that drives this reaction.

178 ssociation with KCNJ10 mutations affecting a Muller cell K+ channel.

179 lar hypertension (COH) in rat down-regulated Muller cells Kir2.1, Kir4.1, TASK-1, GS and GLAST expres

180 ive A2A AR antagonist SCH442416 up-regulated Muller cell Kir4.1, TASK-1, GS and GLAST expressions and

181 KO mice had reduced protection, although the Muller cell KO mice did not, thus gp130-induced protecti

182 r signal-regulated kinase (ERK) signaling in Muller cells, leading to Stat3 activation in photorecept

183 and specific expression of exogenous Dp71 in Muller cells leads to correct localization of Dp71 prote

184 We found that selective ablation of Muller cells led to photoreceptor apoptosis, vascular te

185 aled that sEH deletion decreased retinal and Muller cell levels of 19,20-dihydroxydocosapentaenoic ac

186 A conditionally immortalized mouse Muller cell line was cultured on laminin-coated polyacry

187 In a human Muller cell line, MIO-M1, transfection of a miR-200b mim

188 r activity in primary Muller cells and human Muller cell lines, respectively.

189 Additionally, mutant Muller cells lose apical processes and their perikarya t

190 evaluation was performed on freshly isolated Muller cells maintained in high- and low-glucose culture

191 he ganglion cell marker Thy 1.2 and with the Muller cell marker vimentin, confirming their presence i

192 Ras-MAPK) signaling plays a critical role in Muller cell maturation and function, which is necessary

193 Foveal Muller cells may play an integral role in the transmissi

194 g of RFC expression and activity in cultured Muller cells may reflect the upregulation of this protei

195 ent epithelial cells on Bruch's membrane and Muller cells (MCs) on the inner limiting membrane (ILM),

196 cells that provide a mechanical link at the Muller cell membrane by direct binding to actin and a tr

197 amine-fueled anaplerosis that reverses basal Muller cell metabolism from production to consumption of

198 margins, extended zones of damage, activated Muller cells, microglial recruitment and functional reti

199 imiting membrane by photoreceptor nuclei and Muller cell microvilli could minimize the light reflecti

200 rkers by Muller cells in vitro or stimulated Muller cell migration to the outer nuclear layer (ONL) a

201 Cultured human retinal Muller cells (MIO-M1) were treated with highly oxidized

202 ected in rabbit eyes along with fibroblasts, Muller cells (MIO-M1), or Muller cells transfected to in

203 tive inhibitor of PVR in both fibroblast and Muller cell models of PVR.

204 ofile of cavities around MHs correlates with Muller cell morphology and is consistent with the hypoth

205 croglia induced secondary gliotic changes in Muller cells, neuronal apoptosis, and decreased light-ev

206 Bergmann glia and retinal Muller cells, nonforebrain astrocytes that have not been

207 , we use ShH10 to restore Dp71 expression in Muller cells of Dp71 deficient mouse to study molecular

208 uclear layer corresponding to CRALBP-labeled Muller cells of pups in the 50/10 OIR model.

209 ate in part through an additional pathway in Muller cells of the neural retina.

210 izing enzyme, is expressed by astrocytes and Muller cells of the retina, but little is known about it

211 A second visual cycle is present in Muller cells of the retina.

212 crine cell subtypes but not significantly in Muller cells or photoreceptors.

213 lacking RDH10 either in cone photoreceptors, Muller cells, or the entire retina.

214 e experimental diabetic retinopathy and that Muller cells orchestrate inflammatory responses in myelo

215 versus low-glucose medium does not influence Muller cell phenotype changes.

216 prevented high glucose-induced cell death of Muller cells potentially by inhibiting p53 phosphorylati

217 hot spots of rod death and the remodeling of Muller cell process into zones of low density of photore

218 ha(2)delta(4) subunit is mainly localized to Muller cell processes and endfeet, photoreceptor termina

219 However, at older ages an increase in Muller cell processes was seen.

220 outer retina was considered, we found rd/rd Muller cell processes were dramatically reduced during t

221 synthetase shows that CB2R is restricted to Muller cell processes, extending from the internal limit

222 rophysiology followed by an active growth of Muller cell processes, glial seal formation, and attenua

223 At P10, weak GAT-1 immunostaining was in Muller cell processes.

224 rvative glial response involving the loss of Muller cells processes, hyperexpression of GFAP, and pre

225 Lactate-stimulated GPR81 Muller cells produce numerous angiogenic factors, includ

226 Porcine Muller cells proliferate in response to PDGF, but not IG

227 he loss of the Wnt negative regulator Axin2, Muller cells proliferated after injury and adopted the e

228 his study was to characterize its effects on Muller cell proliferation and tractional force generatio

229 ombinant protein for the ability to modulate Muller cell proliferation and tractional force generatio

230 Palomid 529 is an effective suppressor of Muller cell proliferation, glial scar formation, and pho

231 hy, we provide evidence showing that hypoxic Muller cells promote vascular permeability by stabilizin

232 bit the development of PVR in fibroblast and Muller cell rabbit models of PVR.

233 we show that the store-operated response in Muller cells, radial glia that perform key structural, s

234 g the ERG b-wave and oscillatory potentials, Muller cell reactive gliosis, and neuronal cell death, a

235 -characterized retina visual cycle, in which Muller cells recycle spent all-trans-retinol visual chro

236 Interestingly, Muller cells require HIF--but not VEGF--to promote vascu

237 TNF-alpha-only treatments of Muller cells resulted in significant decreases of tyrosi

238 Chromophore supply by the retinal Muller cells (retina visual cycle) supports the efficien

239 Drug toxicity toward Muller cells, retinal pigment epithelium, and cones was

240 NA microarrays of ESMV-treated vs. untreated Muller cells revealed the up-regulation of genes and miR

241 n VEGF and PEDF expression were evaluated in Muller cells (rMCs), primary mouse retinal pigment epith

242 f these deficiencies is that they impair the Muller cell's ability to metabolize Glc.

243 targeted knockdown of overexpressed VEGFA in Muller cells safely reduces IVNV in a relevant ROP model

244 emonstrate a complex interplay among hypoxic Muller cells, secreted angiogenic factors, and neighbori

245 , whereas retinas lacking CRB2 in developing Muller cells showed late onset retinal disorganization.

246 reticular neurons of the lamprey brainstem (Muller cells) showed ASP immunoreactivity in perikarya a

247 We found no change in the number of Muller cell somata between mice strains, indicating no c

248 Retinal Muller cells span the retina and secrete several trophic

249 Western blotting demonstrated that Muller cell-specific AQP4 was expressed at a higher leve

250 ion also promoted Cd40 mRNA association with Muller cell-specific ribosomes isolated from the retina

251 sEH was localized in Muller glia cells, and Muller cell-specific sEH deletion reproduced the sEH(-/-

252 othelial cells (ECs), retinal microglia, and Muller cells stimulated with TNF-alpha, VEGF, or LPS.

253 Stratified outer nuclear layer with many Muller cells suggests high sensitivity to dim conditions

254 Complementary in vitro studies in cultured Muller cells supported these findings and demonstrated t

255 nd accumulation by targeting siah-1 promotes Muller cell survival under hyperglycemic conditions.

256 Since Muller cells synthesize retinal serine, we propose that

257 RFC was expressed in mouse Muller cells that had been allowed to proliferate in cul

258 Edn2 has detrimental effects on the BRB and Muller cells that involve interactions with the renin-an

259 ne cytoskeletal protein, expressed mainly in Muller cells that provide a mechanical link at the Mulle

260 ogy, propagation, and expression of genes in Muller cells that were cultured on substrates of varying

261 decrease in the number of anti-BrdU-labeled Muller cells, the number and size of subretinal scars, a

262 Muller cells, the principal glia of the retina, play sev

263 eterogeneous cell population of ESMV-treated Muller cells, their expression of retinal cell markers w

264 ties of store-operated signaling pathways in Muller cells, these studies expand the current knowledge

265 in, which may exert its effects in cones and Muller cells through multiple ways of action.

266 lucose-induced GAPDH nuclear accumulation in Muller cells through production and autocrine stimulatio

267 he cycle, 11-cis-retinol is transported from Muller cells to cone inner segments, where it is oxidize

268 the endogenous Wnt signal causes a subset of Muller cells to proliferate and dedifferentiate into RPC

269 It appears that Muller cell tractional force generation in PDR is driven

270 Intact IGFBP-3 modulates Muller cell tractional force generation stimulated by IG

271 with fibroblasts, Muller cells (MIO-M1), or Muller cells transfected to increase their expression of

272 Muller cells trasfected with plasmids or virus were comp

273 Wild-type mouse Muller cells treated with increasing concentrations of L

274 COX-2-null mouse Muller cells treated with increasing concentrations of P

275 These changes occurred in both REC and Muller cells treated with pioglitazone, suggesting that

276 er cells, we identified a mechanism by which Muller cells trigger proinflammatory cytokine expression

277 CD40 ligation in Muller cells triggered phospholipase C-dependent ATP rel

278 in which CD40 stimulation of endothelial and Muller cells triggers adhesion molecule up-regulation an

279 s of K2P channel expression are observed for Muller cells (TWIK-1, TASK-3, TRAAK, and TREK-2) and ret

280 Muller cells, unlike neurons, are spread across the reti

281 During diabetes, mice with CD40 expressed in Muller cells upregulated retinal tumor necrosis factor-a

282 Muller cell uptake of [(3)H]-MTF was robust at pH 5.0 to

283 hat RGR opsin is a critical component of the Muller cell visual cycle and that regeneration of cone v

284 ntify TRPV4 channels as transducers of mouse Muller cell volume increases into physiological response

285 Furthermore, ectopic expression of REDD1 in Muller cells was sufficient to promote both increased 4E

286 sing mice with CD40 expression restricted to Muller cells, we identified a mechanism by which Muller

287 Retinal Muller cells were cultured in normal (5 mM) or high (25

288 sion in cultures of freshly isolated porcine Muller cells were evaluated by indirect immunofluorescen

289 Cultured human Muller cells were exposed to mouse ESMVs every 48 hours

290 Muller cells were isolated from normal porcine retina.

291 IL-33(+) Muller cells were more abundant and IL-33 cytokine was e

292 Muller cells were stimulated to proliferate by serum and

293 In separate experiments, Muller cells were treated with 7-ketocholesterol (7-KC,

294 f the EP(4) agonist PGE(1)-OH, and wild-type Muller cells were treated with increasing concentrations

295 s were increased in primary cultured macular Muller cells which were more susceptible to oxidative st

296 Further, macroglial Muller cells, which influence the integrity of the BRB a

297 erentiation and pluripotency in their target Muller cells, which may turn on an early retinogenic pro

298 Muller cells, which normally elongate and thicken in res

299 al membranes contained positively identified Muller cells with vimentin, GFAP, and glutamine syntheta

300 dition, zonula occludens-1 co-localized with Muller cells within the complex of OPL and HFL, indicati