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1 bud, mesonephric tubules, Wolffian duct, and Mullerian duct.
2 , represses the expression of Hoxa-10 in the Mullerian duct.
3 pothesized to occur at its target organ, the Mullerian duct.
4 equently the non-cell-autonomous loss of the Mullerian duct.
5 es that are embryologically derived from the Mullerian Duct.
6  receptor for beta-catenin activation in the Mullerian duct.
7 lffian duct does not contribute cells to the Mullerian duct.
8 atenin specifically in the mesenchyme of the Mullerian duct.
9 d for the development and maintenance of the Mullerian duct.
10 anephric tubules and caudal extension of the Mullerian duct.
11  in beta-catenin expression in the embryonic Mullerian duct.
12  neoplasms, including those arising from the Mullerian ducts.
13 ype I receptor for Amh-induced regression of Mullerian ducts.
14 e the specialized epithelia derived from the mullerian ducts.
15 sufficient MIS was produced to eliminate the MUllerian ducts.
16 allows sexually dimorphic development of the Mullerian ducts.
17 s epithelium of the urogenital sinus or from mullerian ducts.
18   Lhx1 is expressed in both the Wolffian and Mullerian ducts.
19 t is still responsible for regression of the Mullerian ducts.
20 es that are embryologically derived from the Mullerian ducts.
21 development) by the former and the uterus by Mullerian ducts.
22 two pairs of genital ducts, the Wolffian and Mullerian ducts.
23 o arise from the coelomic epithelium and the Mullerian ducts.
24 ervix and upper vagina, are derived from the Mullerian ducts, a pair of epithelial tubes that form wi
25 ian duct showed that epithelial cells of the Mullerian duct actively migrate along the anterior-poste
26                                              Mullerian duct adenocarcinomas, in particular epithelial
27 ession in males causes the regression of the Mullerian ducts, an essential process in male sexual dif
28 enital anomaly of urogenital tract involving Mullerian ducts and mesonephric ducts.
29 ien, Wolffian (mesonephric) ducts instead of Mullerian ducts and sinovaginal bulbs, give rise to the
30                On embryonic day 15.5, mutant Mullerian ducts and Wolffian ducts have elongated but th
31  male reproductive tracts are developed from Mullerian ducts and Wolffian ducts, respectively, involv
32 , including the ureteric bud, Wolffian duct, Mullerian duct, and developing tubules in the mesonephro
33          While estimates of the frequency of mullerian duct anomalies vary widely owing to different
34 signaling, which is altered in patients with Mullerian duct anomalies, inhibits the regenerative abil
35 ale reproductive tract organs categorized as Mullerian duct anomalies.
36               Robert's uterus is a very rare mullerian duct anomaly which is characterised by septate
37 iation of uterine tissues from the embryonic Mullerian duct are critical for normal fertility.
38 chanisms that regulate the elongation of the Mullerian duct are currently unclear.
39 sis reveals that the cells of the developing Mullerian duct are mesoepithelial when deposited, and su
40                                              Mullerian ducts are paired tubular structures that give
41                                          The Mullerian ducts are the anlagen of the female reproducti
42 ule Wnt-7a fail to undergo regression of the Mullerian duct as a result of the absence of the recepto
43 ts of limb patterning defects accompanied by Mullerian duct-associated sterility in both sexes.
44            There is increasing evidence that Mullerian duct cancers may exfoliate cells.
45 MRKH) syndrome is a congenital defect of the Mullerian ducts characterized by uterovaginal agenesis a
46                             We show that all Mullerian duct components derive from the coelomic epith
47 evelopment, characterized by the presence of Mullerian duct derivatives in 46,XY individuals with mal
48 elopment characterized by the persistence of Mullerian duct derivatives, uterus and tubes, in otherwi
49 ess, white belly spotting, and hypoplasia of Mullerian duct derivatives.
50 of the oviduct and uterus, both of which are Mullerian duct derivatives.
51  DNA methylation signature in easy-to-access Mullerian Duct-derived cervical cells from women with an
52 phologic features that resemble those of the mullerian duct-derived epithelia of the reproductive tra
53 inclusion cysts in normal ovaries and in the mullerian duct-derived epithelium of normal fallopian tu
54 en exhibit morphologic features of embryonic Mullerian duct-derived tissue lineages and colonize peri
55                                  In females, Mullerian ducts develop into the oviduct, uterus, cervix
56 ound that HOX genes, which normally regulate mullerian duct differentiation, are not expressed in nor
57 y and could possibly function redundantly in Mullerian duct differentiation.
58 ssion is altered by DES, leading to abnormal Mullerian duct differentiation.
59 (MIS) functions to promote regression of the Mullerian duct during male development.
60 epubertal testes, promotes involution of the mullerian ducts during normal male sexual differentiatio
61                 Programmed cell death of the Mullerian duct eliminates the primitive female reproduct
62                          In vertebrates, the Mullerian duct elongates along the Wolffian duct, a meso
63  Mullerian duct epithelium led to a block in Mullerian duct elongation and uterine hypoplasia charact
64            However, the precise mechanism of Mullerian duct elongation remains to be elucidated.
65 usly to maintain ductal progenitor cells for Mullerian duct elongation.
66 inpoint previously unknown genes involved in Mullerian duct emergence and regression and urethral can
67 deciding vaginal epithelial cell fate in the Mullerian duct epithelial cells (MDECs) via direct bindi
68 f the tip of the duct; however, migration of Mullerian duct epithelial cells proximal to the tip rema
69 ranscription factor that is essential in the Mullerian duct epithelial progenitor cells for female re
70 genesis in mesenchymal cells adjacent to the Mullerian duct epithelium and in Sertoli and granulosa c
71 3K/AKT signaling pathway is activated in the Mullerian duct epithelium and is required for elongation
72     Cervicovaginal mesenchyme induced p63 in Mullerian duct epithelium and subsequent squamous differ
73  that Lim1 is required cell autonomously for Mullerian duct epithelium formation.
74                          Loss of Lhx1 in the Mullerian duct epithelium led to a block in Mullerian du
75          Thus, p63 is an identity switch for Mullerian duct epithelium to be cervicovaginal versus ut
76 termine the role of Lhx1 specifically in the Mullerian duct epithelium, we performed a Mullerian duct
77 senchyme that culminates in apoptosis of the Mullerian duct epithelium.
78 , thereby altering the developmental fate of Mullerian duct epithelium.
79                                       Female Mullerian ducts exposed to MIS also exhibited prominent
80                                          The Mullerian ducts form in a rostral to caudal manner, guid
81 erstanding of the mechanisms contributing to Mullerian duct formation and to the general process of e
82 Recently, several genes required for initial Mullerian duct formation have been identified.
83 a mesonephric structure that is required for Mullerian duct formation.
84 define cellular and molecular mechanisms for Mullerian duct formation.
85 ed to provide information of the dynamics of Mullerian duct formation.
86                                       As the Mullerian duct forms, its growing tip is intimately asso
87  (didelphic) uterus, representing defects of Mullerian duct fusion.
88  1830, the origin of the cells making up the Mullerian duct has remained controversial.
89 beta family member, causes regression of the Mullerian duct in male embryos.
90 -beta superfamily, induces regression of the Mullerian duct in male embryos.
91 amic morphological changes in the elongating Mullerian duct in rat urogenital ridges in organ culture
92     In addition to causing regression of the Mullerian duct in the male embryo, Mullerian Inhibiting
93 response modifiers, causes regression of the Mullerian ducts in developing male embryos.
94 iological modifier that causes regression of Mullerian ducts in male embryos, is effective as a singl
95 e (AMH) is responsible for regression of the Mullerian ducts in males during embryonic development.
96  Hnf1b specifically in the epithelium of the Mullerian ducts in mice and found that this caused hypop
97 ibiting Substance (MIS) causes regression of Mullerian ducts in the mammalian embryo.
98 ian epithelial tumors arise from remnants of mullerian ducts in the vicinity of the ovary therefore m
99 enerally not maintained and extension of the Mullerian duct inhibited.
100                         Wnt4 is required for Mullerian duct initiation, whereas Wnt7a is required for
101 ed in the formation and morphogenesis of the Mullerian duct into various parts of the female reproduc
102 r, Wnt-4-mutant females are masculinized-the Mullerian duct is absent while the Wolffian duct continu
103                       Lim1 expression in the Mullerian duct is dynamic, corresponding to its formatio
104                                          The Mullerian duct is known to form by invagination of the c
105                  MIS type II receptor in the Mullerian duct is temporally and spatially regulated dur
106 own as Alk3) in the mesenchymal cells of the Mullerian ducts leads to retention of oviducts and uteri
107                                    The upper Mullerian duct (MD) is patterned and specified into two
108 gonads to testicular differentiation, causes Mullerian duct (MD) regression.
109                 Disrupted development of the Mullerian ducts (MD)/Wolffian ducts (WD) through multifa
110 on pattern of Osr1/Osr1 in murine developing Mullerian ducts (MDs) and endometrium, respectively.
111 1 (serine threonine kinase 11), in the fetal Mullerian duct mesenchyme (MDM), the caudal remnant of w
112 und that Fzd1 is one receptor present in the Mullerian duct mesenchyme and could be the putative rece
113  conditional deletion of beta-catenin in the Mullerian duct mesenchyme before postnatal differentiati
114  that male-specific Wnt4 expression in mouse Mullerian duct mesenchyme depends upon AMH signaling, im
115        At birth, loss of beta-catenin in the Mullerian duct mesenchyme disrupted the normal coiling o
116 d not affect AMH signaling activation in the Mullerian duct mesenchyme, but did block Mullerian duct
117 ignificantly elevated in male but not female Mullerian duct mesenchyme.
118 ion along the anterior-posterior axis of the Mullerian duct occurs prior to postnatal day 7 in mice.
119 bstance (MIS) causes regression of the fetal Mullerian duct on binding a heteromeric complex of types
120 substance (MIS), initiates regression of the Mullerian duct or female reproductive tract anlagen; thi
121 s which involve defects of the limbs and the Mullerian ducts or its derivatives.
122  the uterine cervix is derived from the same Mullerian duct precursor as the epithelium of the ovary,
123 hat precursor cells at the caudal tip of the Mullerian duct proliferate to deposit a cord of cells al
124 We were intrigued that both the limb and the Mullerian duct px phenotypes are similar to those caused
125 nd biological activity was tested in ex vivo Mullerian duct regression bioassays.
126                                              Mullerian duct regression can also be inhibited or accel
127 ubstance, is a protein hormone that promotes Mullerian duct regression during male fetal sexual diffe
128 that beta-catenin mediates AMH signaling for Mullerian duct regression during male sexual differentia
129  Smad proteins that are required in vivo for Mullerian duct regression have not yet been identified.
130   Here, we show that apoptosis occurs during Mullerian duct regression in male embryos beginning at E
131 erian-inhibiting substance, which stimulates Mullerian duct regression in males.
132 he coelomic epithelium to the mesenchyme and Mullerian duct regression in organ culture.
133                          We chose to examine Mullerian duct regression in the developing reproductive
134              During male gonadal development Mullerian duct regression is mediated by the actions of
135           In addition to its role in causing Mullerian duct regression, Mullerian inhibiting substanc
136 molecular and cellular mechanisms leading to Mullerian duct regression, the present findings provide
137 tivation of MMP2 promoted ligand-independent Mullerian duct regression.
138  experiments resulted in partial blockage of Mullerian duct regression.
139  the WNT pathway as a downstream mediator of Mullerian duct regression.
140 the Mullerian duct mesenchyme, but did block Mullerian duct regression.
141 ption, germ cell depletion and inhibition of Mullerian duct regression.
142 ributed to a window of MIS responsiveness in Mullerian duct regression.
143 e mesenchyme, suggesting sequential roles in Mullerian duct regression.
144 e to function as a paracrine death factor in Mullerian duct regression.
145        Mechanical division of the developing Mullerian duct showed that epithelial cells of the Mulle
146                                 By using the Mullerian duct-specific anti-Mullerian hormone receptor
147 he Mullerian duct epithelium, we performed a Mullerian duct-specific knockout study using Wnt7a-Cre m
148                               The persistent Mullerian duct syndrome (PMDS) is a 46,XY disorder of se
149                                   Persistent Mullerian duct syndrome (PMDS) is a rare difference of s
150  also be a contributing factor in persistent Mullerian duct syndrome, a form of male pseudohermaphrod
151 ngenital total lipodystrophy, and persistent mullerian duct syndrome.
152 xpressed by mesenchymal cells underlying the Mullerian duct that are thought to mediate regression of
153 expression, the epithelium of the developing Mullerian duct that gives rise to the oviduct, uterus an
154 nic) and metanephric (adult) kidneys and the Mullerian duct, the anlage of much of the female reprodu
155 e elongation and maintenance of the adjacent Mullerian duct, the anlage of the female reproductive tr
156 e fetal testes and induces regression of the Mullerian ducts, the primordia of the female reproductiv
157 tance (Mis)), which causes regression of the Mullerian ducts, the primordia of the oviducts, uterus a
158  required in both sexes for formation of the Mullerian duct, then Wnt-4 in the developing ovary appea
159 anscript levels, perhaps for influencing non-Mullerian duct tissues.
160 udies indicate that caudal elongation of the Mullerian duct towards the urogenital sinus occurs in pa
161 ate into the mesenchyme surrounding the male Mullerian duct under the influence of MIS.
162 ive from a pair of tubular structures called Mullerian ducts, which are composed of three elements: a

 
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