ライフサイエンスコーパス: Mycobacterium

1 ed highly promising in vitro potency against Mycobacterium.

2 cell Doubling Evaluation of Living Arrays of Mycobacterium), a time-lapse microscopy-based method tha

3 Mycobacterium abscessus (34%) and Mycobacterium avium co

4 rium xenopi among the slowly growing NTM and Mycobacterium abscessus among the rapidly growing NTM.

5 rcoidosis with microparticles generated from Mycobacterium abscessus cell walls.

6 Mycobacterium abscessus is an extensively drug-resistant

7 ail to treat a patient with a drug-resistant Mycobacterium abscessus suggests that phages may have co

8 ulmonary Mycobacteroides abscessus (formerly Mycobacterium abscessus) infection in an immunocompetent

9 Mycobacterium abscessus, a rapidly growing nontuberculou

10 deletion of the hflX gene in the pathogenic Mycobacterium abscessus, as well as the nonpathogenic My

11 of a new class of inhibitors against TrmD in Mycobacterium abscessus.

12 roportionally abundant microbial genera were Mycobacterium and Achromobacter at 10,000 ft; Stenotroph

13 ter, Vibrio, Shigella, Salmonella, Yersinia, Mycobacterium and Bacillus-yet are relatively non-toxic

14 Pathogenic bacteria of the genera Mycobacterium and Corynebacterium cause severe human dis

15 erent bacteria (Haemophilus, Salmonella, and Mycobacterium) as our model systems and analyzed structu

16 Mycobacterium abscessus (34%) and Mycobacterium avium complex (83%) were the most common n

17 spp., nontuberculous mycobacteria (NTM), and Mycobacterium avium complex (MAC), however, were widespr

18 ies causing pulmonary disease are members of Mycobacterium avium complex (MAC).

19 sed by the most common NTM pathogens such as Mycobacterium avium complex, Mycobacterium kansasii, and

20 Mycobacterium avium subsp. hominissuis (MAH) is increasi

21 y detect a 563 bp fragment of genomic DNA of Mycobacterium avium subspecies paratuberculosis through

22 Sera from 12 NTM-PD patients due to Mycobacterium avium, M. intracellulare, M. abscessus, or

23 ell-characterised population with an endemic Mycobacterium bovis (the causative agent of bovine/zoono

24 cient immune control of Helicobacter pylori, Mycobacterium bovis bacillus Calmette-Guerin, and Citrob

25 nd trehalose-6,6-dibehenate or infected with Mycobacterium bovis bacillus Calmette-Guerin.

26 negative human sputum spiked with 0 to 10(5) Mycobacterium bovis BCG cells/ml) underwent liquefaction

27 d lung tissue in a mouse model infected with Mycobacterium bovis BCG, as tested by real-time polymera

28 M.tb infection or whether vaccination with, Mycobacterium bovis BCG, has a similar effect.

29 of the BCG2529 gene, the Rv2509 homologue in Mycobacterium bovis BCG, was unable to grow following th

30 nd tissues of cattle naturally infected with Mycobacterium bovis Detailed postmortem and immunohistoc

31 erculosis (bTB), a zoonosis mainly caused by Mycobacterium bovis has severe socio-economic consequenc

32 Here, Whole-Genome Sequencing of Mycobacterium bovis isolated from tissues with TB-like l

33 bTB, caused by Mycobacterium bovis, also causes disease in the Eurasian

34 of bacille Calmette-Guerin but not wild-type Mycobacterium bovis, which both lack a functional nicoti

35 pticum is a rarely identified nontuberculous mycobacterium capable of causing infections in both heal

36 ptomyces exospores, and metabolically latent Mycobacterium cells.

37 Mycobacterium FVL 201832 caused BSIs in oncology clinic

38 identified species was novel and designated Mycobacterium FVL 201832.

39 Mycobacterium haemophilum is a rare and emerging nontube

40 ively identify genes essential for growth in Mycobacterium intracellulare.

41 abscessus, a rapidly growing nontuberculous mycobacterium, is increasingly prevalent in chronic lung

42 PIPS from Mycobacterium kansasii is 86% identical to the ortholog

43 Mycobacterium kansasii is a slow-growing nontuberculous

44 Here we present structures of PIPS from Mycobacterium kansasii with and without evidence of dono

45 thogens such as Mycobacterium avium complex, Mycobacterium kansasii, and Mycobacterium xenopi among t

46 - Mycobacterium tuberculosis (tuberculosis), Mycobacterium leprae (leprosy) and Treponema pallidum pa

47 However, the impossibility of growing Mycobacterium leprae in axenic media has historically im

48 Mycobacterium leprae was obtained from biopsies of 37 le

49 Mycobacterium leprae was thought to be the exclusive cau

50 prosy, which is caused by the human pathogen Mycobacterium leprae, causes nerve damage, deformity and

51 rol to reduce cases and curb transmission of Mycobacterium leprae.

52 e exclusive causative agent of leprosy until Mycobacterium lepromatosis was identified in a rare form

53 of the ESX-1 substrate pair EsxB_1/EsxA_1 in Mycobacterium marinum Although this substrate pair was h

54 We employed a collection of Mycobacterium marinum ESX-1 transposon mutants in a macr

55 ped a chemical approach to track PDIM during Mycobacterium marinum infection of zebrafish.

56 rison of intravenous and hindbrain routes of Mycobacterium marinum infection, which are indistinguish

57 crimination of wild type and DeltaRD1 mutant Mycobacterium marinum strains in a zebrafish embryo mode

58 ulosis, the cause of human tuberculosis, and Mycobacterium marinum, a nontubercular pathogen with a b

59 We infected murine RAW264.7 macrophages with Mycobacterium marinum, a surrogate model organism for M.

60 Vdelta2(+) gammadelta T lymphocytes, and of Mycobacterium-non reactive classic T(H)1 lymphocytes, wi

61 (BSIs) with a rapidly growing nontuberculous Mycobacterium (NTM) species; on 5 September 2018, 6 addi

62 ient has extremely low counts of circulating Mycobacterium-reactive natural killer (NK), invariant NK

63 iboswitch aptamers with and without glycine, Mycobacterium SAM-IV riboswitch with and without S-adeno

64 Mycobacterium septicum is a rarely identified nontubercu

65 ative HBHA with recombinant HBHA produced in Mycobacterium smegmatis (rHBHA-Ms), we could link antige

66 Their expression in Mycobacterium smegmatis confirmed that these PE/PPE prot

67 rticle cryo-electron microscopy structure of Mycobacterium smegmatis EmbB, providing insights on subs

68 We report that the Mycobacterium smegmatis gene annotated as encoding Tam,

69 teomic approaches, we established that PE of Mycobacterium smegmatis is exported to the cell envelope

70 vidually expressed in the heterologous host, Mycobacterium smegmatis mc(2)-155.

71 a cryo-EM structure of a complex between the Mycobacterium smegmatis RNAP and HelD.

72 trom cryo-electron microscopy structure of a Mycobacterium smegmatis Sdh, which forms a trimer.

73 We have previously identified a USP in Mycobacterium smegmatis that is a product of the msmeg_4

74 Titration of small ssDNA oligonucleotides to Mycobacterium smegmatis topoisomerase I with progressive

75 roteomic responses to cysteine limitation in Mycobacterium smegmatis using mass spectrometry.

76 repair pattern and genome-wide repair map of Mycobacterium smegmatis We find that M. smegmatis, which

77 When deployed in Mycobacterium smegmatis with quantitative proteomics, th

78 rium abscessus, as well as the nonpathogenic Mycobacterium smegmatis, results in hypersensitivity to

79 t in vitro models of rifampicin tolerance in Mycobacterium smegmatis.

80 es for their ability to impair the growth of Mycobacterium smegmatis.

81 Mycobacterium species, including Mycobacterium tuberculo

82 A mycobacterium-specific loop of the enzyme's rotary gamma

83 ells) and IFN-gamma production by them, with mycobacterium-specific, IFN-gamma-producing, purely adap

84 , Streptococcus spp., Bacillus subtilis, and Mycobacterium spp. have demonstrated EV production in Gr

85 gaps regarding the role of MDSCs in HIV and Mycobacterium tuberculosis (co)infections.

86 Mycobacterium tuberculosis (M. tb.) is a pervasive respi

87 Mycobacterium tuberculosis (M. tuberculosis) has coevolv

88 ncluding the notoriously persistent pathogen Mycobacterium tuberculosis (M. tuberculosis).

89 Knowing when a person was infected with Mycobacterium tuberculosis (M.tb) is critical as recent

90 Antigens from Mycobacterium tuberculosis (M.tb), have been shown to st

91 ht into the within-host genetic diversity of Mycobacterium tuberculosis (M.TB), revealing remarkably

92 We used a 24-week guinea pig vaccination-Mycobacterium tuberculosis (M.tb.) challenge model to te

93 ndered by absence of rapid tests to identify Mycobacterium tuberculosis (MTB) and detect isoniazid (I

94 Mycobacterium tuberculosis (Mtb) and human immunodeficie

95 deadliest infectious diseases, is caused by Mycobacterium tuberculosis (MTB) and remains a public he

96 nc-responsive binding of Mpy to ribosomes in Mycobacterium tuberculosis (Mtb) and show Mpy-dependent

97 s from 58 individuals latently infected with Mycobacterium tuberculosis (Mtb) and to group them accor

98 Mycobacterium tuberculosis (Mtb) can enter the body thro

99 Antimicrobial-resistant Mycobacterium tuberculosis (Mtb) causes over 200,000 dea

100 Mycobacterium tuberculosis (Mtb) continues to be a major

101 or S100A8/A9 deficiency resulted in improved Mycobacterium tuberculosis (Mtb) control during chronic

102 Automated genotyping of drug-resistant Mycobacterium tuberculosis (MTB) directly from sputum is

103 ationale: Direct evidence for persistence of Mycobacterium tuberculosis (Mtb) during asymptomatic lat

104 Mycobacterium tuberculosis (Mtb) employs plethora of mec

105 including Bacille Calmette-Guerin (BCG) and Mycobacterium tuberculosis (MTB) Erdman.

106 esis hallmarks for tuberculosis (TB) are the Mycobacterium tuberculosis (Mtb) escape from phagolysoso

107 ry of recombinant BCG strains expressing the Mycobacterium tuberculosis (Mtb) ESX-1 secretion system

108 The human pathogen Mycobacterium tuberculosis (Mtb) harbors a well-orchestr

109 ection of mice with clinical "hypervirulent" Mycobacterium tuberculosis (Mtb) HN878 induces human-lik

110 es known to play a vital role in controlling Mycobacterium tuberculosis (Mtb) infection and disease p

111 Tuberculosis (TB) is caused by Mycobacterium tuberculosis (Mtb) infection and is a majo

112 Strategies to prevent Mycobacterium tuberculosis (Mtb) infection are urgently

113 Murine models of Mycobacterium tuberculosis (Mtb) infection demonstrate p

114 Mycobacterium tuberculosis (Mtb) infection is among top

115 Ag85A expressed by Mycobacterium tuberculosis (Mtb) is a bacterial surface

116 Mycobacterium tuberculosis (Mtb) is an obligate human pa

117 The haematogenous dissemination of Mycobacterium tuberculosis (Mtb) is critical to the path

118 Mycobacterium tuberculosis (Mtb) is the leading cause of

119 mutations in lepA that are found in clinical Mycobacterium tuberculosis (Mtb) isolates phenocopy lepA

120 Tuberculosis (TB), caused by Mycobacterium tuberculosis (Mtb) latently infects approx

121 e demonstrate an unexpected role of H(2)S in Mycobacterium tuberculosis (Mtb) pathogenesis.

122 While Lrrk2 KO mice can control Mycobacterium tuberculosis (Mtb) replication, they have

123 Mycobacterium tuberculosis (Mtb) strains are classified

124 involved in the presentation of ligands from Mycobacterium tuberculosis (Mtb) to MAIT cells.

125 rios including: 1) sustained interruption of Mycobacterium tuberculosis (Mtb) transmission, 2) sustai

126 nfected individuals, the mechanisms by which Mycobacterium tuberculosis (Mtb) worsens HIV-1 pathogene

127 genic strategy of the causative agent of TB, Mycobacterium tuberculosis (Mtb), and therefore metaboli

128 pies aimed at optimizing immune responses to Mycobacterium tuberculosis (Mtb), as adjunctive treatmen

129 Pulmonary tuberculosis, a disease caused by Mycobacterium tuberculosis (Mtb), manifests with a persi

130 r of the world's population is infected with Mycobacterium tuberculosis (Mtb), the causative agent of

131 Mycobacterium tuberculosis (Mtb), the causative infectio

132 In the pathogen Mycobacterium tuberculosis (Mtb), the protective effect

133 A) modules encoded by the bacterial pathogen Mycobacterium tuberculosis (Mtb), three contain antitoxi

134 of the human population have been exposed to Mycobacterium tuberculosis (MTB), with the overwhelming

135 several inflammatory and stress responses in Mycobacterium tuberculosis (Mtb)-infected host cells.

136 In patients, PD-1 is expressed in Mycobacterium tuberculosis (Mtb)-infected lung tissue bu

137 ontent and activity are globally elevated in Mycobacterium tuberculosis (Mtb)-infected macrophages.

138 Ruhl et al. find that a Mycobacterium tuberculosis (Mtb)-specific lipid, SL-1, s

139 tion mechanism of NQ inhibitors of ThyX from Mycobacterium tuberculosis (Mtb).

140 own precisely how BDQ triggers cell death in Mycobacterium tuberculosis (Mtb).

141 of synchronously replicating populations of Mycobacterium tuberculosis (Mtb).

142 n infectious disease caused by the bacterium Mycobacterium tuberculosis (Mtb).

143 set of individuals infected with aerosolized Mycobacterium tuberculosis (Mtb).

144 losis (ATB) currently relies on detection of Mycobacterium tuberculosis (Mtb).

145 are insensitive to overexpression of InhA in Mycobacterium tuberculosis (Mtb).

146 many bacteria, including the human pathogen Mycobacterium tuberculosis (Mtb).

147 s), important lipid pathogenicity factors of Mycobacterium tuberculosis (Mtb).

148 hogens that cause chronic infections such as Mycobacterium tuberculosis (Mtb).

149 ulosis (TB), which is caused by the pathogen Mycobacterium tuberculosis (Mtb).

150 in rifampicin-resistant clinical isolates of Mycobacterium tuberculosis (Mtb).

151 BI, recent work in macaques co-infected with Mycobacterium tuberculosis (Mtb)/simian immunodeficiency

152 lly important endemic human disease agents - Mycobacterium tuberculosis (tuberculosis), Mycobacterium

153 Pathogens such as Mycobacterium tuberculosis accumulate polyphosphate, and

154 We also show that the AHAS complex of Mycobacterium tuberculosis adopts a similar structure, t

155 Genetic diversity of Mycobacterium tuberculosis affects immune responses and

156 ite that controls the redox/energy status of Mycobacterium tuberculosis Although the major steps of M

157 phylogenetic spectrum of pathogens including Mycobacterium tuberculosis and carbapenem-resistant Ente

158 -epimerase (DprE1) is an essential enzyme in Mycobacterium tuberculosis and has recently been studied

159 ns may modulate the inflammatory response to Mycobacterium tuberculosis and influence disease present

160 hase is required for growth and viability of Mycobacterium tuberculosis and is a validated clinical t

161 Mycobacterium tuberculosis and M. smegmatis form drug-to

162 more IL-35 after stimulation by antigens of Mycobacterium tuberculosis and secreted more IL-10.

163 of IL-1beta, total IgA, and IgA specific to Mycobacterium tuberculosis antigen in the exhaled breath

164 The reconstitution of Mycobacterium tuberculosis antigen-specific CD4 T cells

165 Several Wbl proteins of Mycobacterium tuberculosis are known to interact with it

166 Novel antimicrobials for treatment of Mycobacterium tuberculosis are needed.

167 thogens such as Streptococcus pneumoniae and Mycobacterium tuberculosis AtaC is monomeric in solution

168 ligand-induced ordering of the N terminus of Mycobacterium tuberculosis ATR, which organizes a dynami

169 -cell variation in morphological features of Mycobacterium tuberculosis bacilli to develop a rapid pr

170 re stimulated with Staphylococcus aureus and Mycobacterium tuberculosis before, as well as 2 weeks an

171 recycling pathways of NAD(+) biosynthesis in Mycobacterium tuberculosis but not in humans.

172 A), interrupts biosynthesis of coenzyme A in Mycobacterium tuberculosis by binding to aspartate decar

173 y T cells from humans latently infected with Mycobacterium tuberculosis By comparing native HBHA with

174 Mycobacterium tuberculosis causes tuberculosis, a diseas

175 The Mycobacterium tuberculosis cell envelope is a critical i

176 385 Mycobacterium tuberculosis clinical isolates were proces

177 The 300-kDa ClpP1P2 protease from Mycobacterium tuberculosis collaborates with the AAA+ (A

178 he human- and animal-adapted lineages of the Mycobacterium tuberculosis complex (MTBC) are thought to

179 e Xpert MTB/RIF Ultra assay for detection of Mycobacterium tuberculosis complex (MTBC) DNA in samples

180 tudy assembles DNA adenine methylomes for 93 Mycobacterium tuberculosis complex (MTBC) isolates from

181 to bedaquiline (BDQ) and delamanid (DLM) in Mycobacterium tuberculosis complex (MTBc) strains is poo

182 man tuberculosis is caused by members of the Mycobacterium tuberculosis complex (MTBC) that vary in v

183 n and analysis of drug-resistance profile of Mycobacterium tuberculosis complex (MTC), a causative ag

184 ibutes to the inability to differentiate the Mycobacterium tuberculosis Complex species, leading to a

185 opsy of the pleural lesion were positive for Mycobacterium tuberculosis complex.

186 Based on a set of 654 clinical Mycobacterium tuberculosis culture isolates with known m

187 omparison to that of a reference standard of Mycobacterium tuberculosis culture on two sputum samples

188 We analyzed Mycobacterium tuberculosis culture-positive cases report

189 Mycobacterium tuberculosis DNA can be detected from oral

190 Mycobacterium tuberculosis encodes FadR orthologs one of

191 The mechanisms by which Mycobacterium tuberculosis evades host immunity remain e

192 ods for drug susceptibility testing (DST) of Mycobacterium tuberculosis from patient sputum samples f

193 with drug-resistant TB would have culturable Mycobacterium tuberculosis from respirable, cough-genera

194 Tens of millions of children are exposed to Mycobacterium tuberculosis globally every year; however,

195 of Bacillus subtilis, Escherichia coli, and Mycobacterium tuberculosis gyrases and of heterologous e

196 and low oxygen recovery assay (LORA) against Mycobacterium tuberculosis H37Rv with minimum inhibitory

197 Mycobacterium tuberculosis has an unusual outer membrane

198 ive channel of large conductance (MscL) from Mycobacterium tuberculosis has been used as a structural

199 Over the years, Mycobacterium tuberculosis has developed a form of resis

200 Mycobacterium tuberculosis has evolved numerous type VII

201 particular action on the persistent forms of Mycobacterium tuberculosis However, no drug susceptibili

202 SK-proprietary library against intracellular Mycobacterium tuberculosis identified 1, a thioalkylbenz

203 e current diagnostic methods for identifying Mycobacterium tuberculosis in cerebrospinal fluid (CSF)

204 .-born persons who were likely infected with Mycobacterium tuberculosis in their countries of birth.

205 B) is variable among individuals with latent Mycobacterium tuberculosis infection (LTBI), but validat

206 Identification of biomarkers for latent Mycobacterium tuberculosis infection and risk of progres

207 Mycobacterium tuberculosis infection causes high rates o

208 BCG appears to reduce acquisition of Mycobacterium tuberculosis infection in children, measur

209 iming strategy could protect against aerosol Mycobacterium tuberculosis infection in mice.

210 role in the early innate immune response to Mycobacterium tuberculosis infection in the lung.

211 Screening for, and treatment of, latent Mycobacterium tuberculosis infection is routine before i

212 ne components involved in protecting against Mycobacterium tuberculosis infection is urgently needed

213 t prevalent in regions of the world in which Mycobacterium tuberculosis infection remains endemic and

214 cts from The Gambia who never develop latent Mycobacterium tuberculosis infection shows distinct tran

215 terial loads and exacerbate pathology during Mycobacterium tuberculosis infection upon GM-CSF blockad

216 -control study of patients with asymptomatic Mycobacterium tuberculosis infection, a novel 3-gene tra

217 h type 2 diabetes (T2D) have a lower risk of Mycobacterium tuberculosis infection, progression from i

218 ve disease with immunodeficiency, as well as Mycobacterium tuberculosis infection, underscoring their

219 rials including 6859 adult participants with Mycobacterium tuberculosis infection.

220 soniazid-rifapentine for treatment of latent Mycobacterium tuberculosis infection.

221 ttle is known about the physiology of latent Mycobacterium tuberculosis infection.

222 In this review, we focus on how Mycobacterium tuberculosis infects antigen-presenting ce

223 lapse imaging to reveal the dynamics of host-Mycobacterium tuberculosis interactions at an air-liquid

224 Mycobacterium tuberculosis is a global health problem in

225 Because Mycobacterium tuberculosis is an activator of cGAS-depen

226 e final step in mycolic acid biosynthesis in Mycobacterium tuberculosis is catalysed by mycolyl reduc

227 Gene rv3722c of Mycobacterium tuberculosis is essential for in vitro gro

228 and the growing prevalence of drug-resistant Mycobacterium tuberculosis is making disease control mor

229 Mycobacterium tuberculosis is one of the most hard-to-tr

230 The human pathogen Mycobacterium tuberculosis is the causative agent of tub

231 Early clearance of Mycobacterium tuberculosis is the eradication of infecti

232 Mycobacterium tuberculosis is the leading cause of death

233 ncentrations (CCs) for categorizing clinical Mycobacterium tuberculosis isolates as susceptible/resis

234 opore MinION whole-genome sequencing data of Mycobacterium tuberculosis isolates for species identifi

235 lly and genotypically characterized clinical Mycobacterium tuberculosis isolates.

236 ges with the membrane-permeabilizing microbe Mycobacterium tuberculosis or infection of target cells

237 Mycobacterium tuberculosis possesses a large number of g

238 The related Mycobacterium tuberculosis proteins were also found to i

239 acellular replication of the deadly pathogen Mycobacterium tuberculosis relies on the production of s

240 Mycobacterium tuberculosis remains a global health probl

241 cent study reported that the UvrC protein of Mycobacterium tuberculosis removes damage in a manner an

242 The Mg(2+)-dependent Mycobacterium tuberculosis salicylate synthase (MbtI) is

243 ases on a transmission network inferred from Mycobacterium tuberculosis sequencing data on extensivel

244 ply the MAC to a dataset of 1595 drug-tested Mycobacterium tuberculosis strains and show that MACs pr

245 xhibited moderate inhibitory potency against Mycobacterium tuberculosis thymidylate kinase, the targe

246 Synthesis of the cofactor is essential for Mycobacterium tuberculosis to establish and maintain chr

247 ously uncharacterized network adaptations of Mycobacterium tuberculosis to the first-line anti-tuberc

248 with trehalose-based glycolipids produced by Mycobacterium tuberculosis TPP production starts in the

249 The Mycobacterium tuberculosis virulence factor EsxA and its

250 We measured a network of genomic links using Mycobacterium tuberculosis whole-genome sequences.

251 severe human diseases such as tuberculosis (Mycobacterium tuberculosis) and diphtheria (Corynebacter

252 ifications of proteins during infection with Mycobacterium tuberculosis, a notorious intracellular pa

253 However, many persistent pathogens, such as Mycobacterium tuberculosis, actively target the very hos

254 B) patients lack microbiological evidence of Mycobacterium tuberculosis, and misdiagnosis or delayed

255 osts, including herpesviruses, retroviruses, Mycobacterium tuberculosis, and Toxoplasma gondii.

256 s, a severe infectious disease caused by the Mycobacterium tuberculosis, arouses huge concerns global

257 nvolved in the mechanism of action of PZA in Mycobacterium tuberculosis, as previously thought.

258 Tuberculosis (TB), caused by Mycobacterium tuberculosis, continues to be a major glob

259 Mycobacterium species, including Mycobacterium tuberculosis, employs atypical long (C(60-

260 As important virulence factors of Mycobacterium tuberculosis, EsxA and EsxB not only play

261 surviving bacterial pathogens that includes Mycobacterium tuberculosis, generates a salicyl-capped p

262 MtCM, the CM from Mycobacterium tuberculosis, has less than 1% of the cata

263 ofclinical isolates of the causative agent, Mycobacterium tuberculosis, has recently garnered attent

264 Many microbes, including Mycobacterium tuberculosis, have evolved to evade or exp

265 in fbiA, fbiB, fbiC, ddn, and fgd1 genes of Mycobacterium tuberculosis, knowledge about the molecula

266 bacterial phylum that includes the pathogen Mycobacterium tuberculosis, lack the canonical FtsZ-memb

267 In Mycobacterium tuberculosis, mycolic acids and their glyc

268 There were no IEps due to Mycobacterium tuberculosis, Pneumocystis jirovecii, or T

269 ersity (panallelome) of Salmonella enterica, Mycobacterium tuberculosis, Pseudomonas aeruginosa, and

270 For Klebsiella pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, and Sta

271 f 9 prevalent bacterial pathogens, including Mycobacterium tuberculosis, Staphylococcus aureus, and E

272 ir role in protection against infection with Mycobacterium tuberculosis, termed "early clearance." ME

273 Mycobacterium tuberculosis, the causative agent of pulmo

274 Glycolipids in Mycobacterium tuberculosis, the causative agent of tuber

275 Mycobacterium tuberculosis, the cause of human tuberculo

276 Mycobacterium tuberculosis, the leading cause of death d

277 tensively used to study the host response to Mycobacterium tuberculosis, their validity in revealing

278 teway to aromatic amino acid biosynthesis in Mycobacterium tuberculosis, which shows extremely comple

279 ore and after BCG vaccination, while ex vivo Mycobacterium tuberculosis- and Staphylococcus aureus-in

280 active TB (n = 16) and age- and sex-matched, Mycobacterium tuberculosis-exposed but uninfected househ

281 patients with TB disease relative to healthy Mycobacterium tuberculosis-exposed controls from Peru an

282 s in surgical lung resections and blood from Mycobacterium tuberculosis-infected (Mtb-infected) indiv

283 athic tau and during phagosome parasitism by Mycobacterium tuberculosis.

284 ences in infection with different strains of Mycobacterium tuberculosis.

285 rved in the plasma of patients infected with Mycobacterium tuberculosis.

286 tivities against ESKAPE pathogens as well as Mycobacterium tuberculosis.

287 ophagic control of membrane-damaging microbe Mycobacterium tuberculosis.

288 er scientific community: cow, pig, wheat and mycobacterium tuberculosis.

289 tabolites support innate immune responses to Mycobacterium tuberculosis.

290 PCR assay for pyrazinamide susceptibility in Mycobacterium tuberculosis.

291 lete understanding of drug susceptibility to Mycobacterium tuberculosis.

292 biotic in lung lesions of mice infected with Mycobacterium tuberculosis.

293 mechanisms at the host-pathogen interface of Mycobacterium tuberculosis.

294 We used Mycobacterium tuberculosis/simian immunodeficiency virus

295 Iron is an essential element for Mycobacterium tuberculosis; it has at least 40 enzymes t

296 Mycobacterium ulcerans cases from an observational cohor

297 tructure of mammalian Sec61 inhibited by the Mycobacterium ulcerans exotoxin mycolactone via electron

298 er is a neglected tropical disease caused by Mycobacterium ulcerans infection that damages the skin a

299 Antibiotics are highly effective in curing Mycobacterium ulcerans lesions, but are associated with

300 m avium complex, Mycobacterium kansasii, and Mycobacterium xenopi among the slowly growing NTM and My