ライフサイエンスコーパス: Mycobacterium avium

1 tic plasmids from the opportunistic pathogen Mycobacterium avium.

2 was identified in the opportunistic pathogen Mycobacterium avium.

3 of TLR2 expression following infection with Mycobacterium avium.

4 variety of intracellular pathogens including Mycobacterium avium.

5 s tested, including Mycobacterium leprae and Mycobacterium avium.

6 ase mediated by the intramacrophage pathogen Mycobacterium avium.

7 TLR) 2 mRNA was induced after infection with Mycobacterium avium.

8 s virus stimulated by in vivo infection with Mycobacterium avium.

9 llowing infection of murine macrophages with Mycobacterium avium.

10 rium bovis bacillus Calmette-Guerin (BCG) or Mycobacterium avium.

11 nt, phenol treatment, and contamination with Mycobacterium avium.

12 virulent (SmD) or virulent (SmT) variants of Mycobacterium avium 2-151.

13 ently were: Mycobacterium mucogenicum (52%), Mycobacterium avium (30%), and Mycobacterium gordonae (2

14 smegmatis and to a lesser extent pathogenic Mycobacterium avium, activate Ca(2+)-dependent calmoduli

15 restored antimycobacterial activity against Mycobacterium avium and Mycobacterium bovis Bacille Calm

16 Mycobacterium avium and Mycobacterium intracellulare are

17 The clinical significance and prevalence of Mycobacterium avium and Mycobacterium intracellulare wer

18 Mycobacterium avium and Mycobacterium tuberculosis are h

19 olipids (GPLs), a major surface component of Mycobacterium avium and other non-tuberculosis mycobacte

20 s complex and to have homology with DNA from Mycobacterium avium and other nontuberculous mycobacteri

21 R2-dependent responses were seen using whole Mycobacterium avium and Staphylococcus aureus, demonstra

22 lass II promoter (hu10Tg) were infected with Mycobacterium avium, and bacterial burdens and immune re

23 c infections caused by Pneumocystis carinii, Mycobacterium avium, and Campylobacter coli that require

24 /bg(-) mice were infected intravenously with Mycobacterium avium, and cultures of blood and brain as

25 7(-/-) and wild-type mice were infected with Mycobacterium avium, and host responses were analyzed.

26 er, Pneumocystis carinii, Toxoplasma gondii, Mycobacterium avium, and Lactobacillus casei) and showed

27 Legionella pneumophila, Mycobacterium avium, and Pseudomonas aeruginosa are oppo

28 rtunistic pathogens (Legionella pneumophila, Mycobacterium avium, and Pseudomonas aeruginosa), broade

29 Mycobacterium tuberculosis and Mycobacterium avium are facultative intracellular pathog

30 ous mycobacteria (NTM), some of which-namely Mycobacterium avium-are important opportunistic pathogen

31 ag2(-/-)gammac(-/-) mice are as resistant to Mycobacterium avium as Rag2(-/-) mice, whereas Rag2(-/-)

32 duced in response to live M. tuberculosis or Mycobacterium avium as well as certain mycobacterial pro

33 CD43-transfected HeLa cells bound Mycobacterium avium, but not Salmonella typhimurium or S

34 ues from both Mycobacterium tuberculosis and Mycobacterium avium can complement lpqM donor mutants, s

35 Previous studies have demonstrated that Mycobacterium avium can invade intestinal epithelial cel

36 Mycobacterium avium causes disseminated disease in human

37 Mycobacterium avium causes disseminated infection in pat

38 against DHFR from both Toxoplasma gondii and Mycobacterium avium compared to mammalian DHFR.

39 rophages following infection with pathogenic Mycobacterium avium compared to the activation following

40 rophages infected with pathogenic strains of Mycobacterium avium compared with infections with the fa

41 The most common species recovered were Mycobacterium avium complex (69 isolates) and Mycobacter

42 Mycobacterium avium complex (72%) and Mycobacterium absc

43 (100%), CD4 cell count less than 200 (84%), Mycobacterium avium complex (73%), and Pneumocystis cari

44 Mycobacterium abscessus (34%) and Mycobacterium avium complex (83%) were the most common n

45 We report a case of recurrent disseminated Mycobacterium avium complex (DMAC) disease with anti-gam

46 Whether infection with Mycobacterium avium complex (MAC) among patients with ac

47 e cases, 122 (64%) were culture-positive for Mycobacterium avium complex (MAC) and 69 (36%) for M. ab

48 Of 600 sets tested, 85 (14%) yielded Mycobacterium avium complex (MAC) and 9 (2%) yielded oth

49 In lymphoid tissues coinfected with Mycobacterium avium complex (MAC) and HIV-1, increased v

50 f 2,4-diamino-5-deazapteridine inhibitors of Mycobacterium avium complex (MAC) and human dihydrofolat

51 Members of the Mycobacterium avium complex (MAC) are characterized as n

52 Members of the Mycobacterium avium complex (MAC) are important environm

53 Mycobacterium avium complex (MAC) are opportunistic resp

54 type 1-infected persons with newly diagnosed Mycobacterium avium complex (MAC) bacteremia were enroll

55 y virus type 1 (HIV-1)-infected persons with Mycobacterium avium complex (MAC) bacteremia, the levels

56 reproducibility of susceptibility testing of Mycobacterium avium complex (MAC) by broth microdilution

57 r antimycobacterial therapy for disseminated Mycobacterium avium complex (MAC) could be withdrawn fro

58 n alone and in combination for prevention of Mycobacterium avium complex (MAC) disease were compared

59 in is a major drug used for the treatment of Mycobacterium avium complex (MAC) disease, but standard

60 effective drug regimens for the treatment of Mycobacterium avium complex (MAC) disease.

61 Although opportunistic infections due to Mycobacterium avium complex (MAC) have been less common

62 Persistent growth of Mycobacterium avium complex (MAC) in the lungs indicates

63 Mycobacterium avium complex (MAC) infection is the most

64 The relationship between Mycobacterium avium complex (MAC) infection of blood and

65 The Mycobacterium avium complex (MAC) is an important cause

66 The Mycobacterium avium complex (MAC) is an important group

67 Lung disease caused by Mycobacterium avium complex (MAC) is increasing in preva

68 Rapid identification of Mycobacterium avium complex (MAC) is possible by use of

69 Mycobacterium avium complex (MAC) is the most common dis

70 features and outcome of macrolide-resistant Mycobacterium avium complex (MAC) lung disease are not k

71 in prospective macrolide treatment trials of Mycobacterium avium complex (MAC) lung disease were asse

72 household water sources for 36 patients with Mycobacterium avium complex (MAC) lung disease were eval

73 cs and to evaluate relapses in patients with Mycobacterium avium complex (MAC) lung disease, but the

74 atment of noncavitary nodular bronchiectatic Mycobacterium avium complex (MAC) lung disease, supporti

75 Organisms within the Mycobacterium avium complex (MAC) may have differential

76 Mycobacterium avium complex (MAC) organisms cause dissem

77 The clinical significance of recovery of Mycobacterium avium complex (MAC) organisms from respira

78 Species identification of isolates of the Mycobacterium avium complex (MAC) remains a difficult ta

79 DR (NTM-DR) line probe assay for identifying Mycobacterium avium complex (MAC) species and Mycobacter

80 Adherence of Mycobacterium avium complex (MAC) to human respiratory e

81 Total mycobacteria and Mycobacterium avium complex (MAC) were quantified by qPC

82 Mycobacterium avium complex (MAC) were the dominant spec

83 Mycobacterium avium complex (MAC) within macrophages und

84 actors that contribute to protection against Mycobacterium avium complex (MAC), cytokine production b

85 spp., nontuberculous mycobacteria (NTM), and Mycobacterium avium complex (MAC), however, were widespr

86 genetically similar to other members of the Mycobacterium avium complex (MAC), some of which are non

87 y (HAART) on cell-mediated immunity (CMI) to Mycobacterium avium complex (MAC), we measured immune re

88 ies causing pulmonary disease are members of Mycobacterium avium complex (MAC).

89 Organisms in the Mycobacterium avium complex (MAC; M. avium, M. intracell

90 ow-growing species, including members of the Mycobacterium avium complex (MAVC).

91 s with Emberger syndrome, monocytopenia with Mycobacterium avium complex (MonoMAC), and MDS.

92 mocystis carinii (n = 26), bacteria (n = 3), Mycobacterium avium complex (n = 2), Nocardia sp. (n = 1

93 l disease (mean number of organs infected by Mycobacterium avium complex 4.1 [SD 0.8] vs 2.0 [1.1], p

94 tory pulmonary nontuberculous mycobacterial (Mycobacterium avium complex [MAC] or Mycobacterium absce

95 One hundred isolates of Mycobacterium avium complex and eight M. simiae isolates

96 Prophylaxis against Mycobacterium avium complex can safely be withdrawn or w

97 Mycobacterium avium complex cultures, CD4(+) cell counts

98 ttributable to cytomegalovirus retinitis and Mycobacterium avium complex declined over time (p=0.0058

99 Calif.) to detect Mycobacterium gordonae and Mycobacterium avium complex directly in liquid medium fl

100 Several agents are effective in preventing Mycobacterium avium complex disease in patients with adv

101 phylaxis versus withdrawal for prevention of Mycobacterium avium complex disease.

102 g the pseudocording, or loose aggregation of Mycobacterium avium complex from M. tuberculosis and the

103 safety of discontinuing prophylaxis against Mycobacterium avium complex has been uncertain.

104 yelitis (patient 1) and disseminated CMV and Mycobacterium avium complex infection (patient 2), respe

105 IS event in HIV-infected patients, unmasking Mycobacterium avium complex infection after starting ant

106 Infection with Mycobacterium avium complex is acquired from the environ

107 A major phenotypic trait of the Mycobacterium avium complex is the ability to produce ro

108 were evaluated for susceptibility testing of Mycobacterium avium complex isolates against clarithromy

109 ribosomal internal transcribed spacer of 56 Mycobacterium avium complex isolates from pediatric pati

110 g activities seem not to be risk factors for Mycobacterium avium complex lung disease in HIV-negative

111 Mycobacterium avium complex lung disease is an increasin

112 omponent of multidrug treatment regimens for Mycobacterium avium complex lung disease.

113 ia, esophageal candidiasis, and disseminated Mycobacterium avium complex or Mycobacterium kansasii in

114 Ten of 639 MGIT cultures grew Mycobacterium avium complex or Mycobacterium kansasii, h

115 hose previously treated for tuberculosis and Mycobacterium avium complex predominated (27.7% [95% CI:

116 Mycobacterium avium complex pulmonary disease (MAC-PD) i

117 new therapy for Candida esophagitis, whereas Mycobacterium avium complex therapy may be discontinued

118 Mycobacterium avium complex was concomitantly isolated i

119 Mycobacterium avium complex was seen in 75% of NTM-posit

120 Isolates of the Mycobacterium avium complex were examined for hemolysin

121 Nearly 60% of positive cultures were for Mycobacterium avium complex, although this ranged by sta

122 , 14% (1/7) for Cryptococcus, 10% (1/10) for Mycobacterium avium complex, and 4% (3/72) for PCP.

123 ifferent Mycobacterium tuberculosis complex, Mycobacterium avium complex, and Mycobacterium spp. dire

124 neumocystis jeroveci pneumonia, disseminated Mycobacterium avium complex, lymphoid interstitial pneum

125 sed by the most common NTM pathogens such as Mycobacterium avium complex, Mycobacterium kansasii, and

126 46 children (10.8%); 45.7% of isolates were Mycobacterium avium complex.

127 e closely related organisms and comprise the Mycobacterium avium complex.

128 tis carinii pneumonia [PCP] and disseminated Mycobacterium avium-complex [MAC] infection) in persons

129 HIV-infected patients with untreated Mycobacterium avium-complex diarrhea are associated with

130 ents and household water/biofilm isolates of Mycobacterium avium could be matched by DNA fingerprinti

131 Of the two common morphotypes of Mycobacterium avium, designated smooth transparent (SmT)

132 Pneumocystis carinii, Toxoplasma gondii, and Mycobacterium avium DHFR vs rat DHFR.

133 hat represent 46 genes that are expressed by Mycobacterium avium during growth in human macrophages.

134 tosporidium parvum, Enterocytozoon bieneusi, Mycobacterium avium, Entamoeba histolytica, Balantidium

135 imensional gel electrophoresis revealed that Mycobacterium avium expresses several proteins unique to

136 n binding motif of the previously identified Mycobacterium avium fibronectin attachment protein (FAP-

137 ate of MTB from a nonTB species of the genus Mycobacterium avium grown in liquid culture media.

138 ly related species Mycobacterium marinum and Mycobacterium avium harboring insertions in the ortholog

139 ating inflammation induced by infection with Mycobacterium avium in human primary macrophages.

140 vation of Janus Kinase (JAK)-STAT pathway in Mycobacterium avium-infected macrophages.

141 Here, we show that Mycobacterium avium-infected T cell-deficient mice injec

142 We used a model of disseminated Mycobacterium avium infection in mice to investigate the

143 Disseminated Mycobacterium avium infection is common in AIDS patients

144 Active disseminated Mycobacterium avium infection may significantly compromi

145 We previously reported that Mycobacterium avium infection of mouse macrophages decre

146 Disseminated Mycobacterium avium infection persisted and the patient

147 In this study, 30 AIDS patients without Mycobacterium avium infection were randomized to receive

148 ceptor (TLR) signaling in host resistance to Mycobacterium avium infection, mice deficient in the TLR

149 Here, we report on a patient with Mycobacterium avium infection, recurrent Staphylococcus

150 ere we show, using an in vivo mouse model of Mycobacterium avium infection, that an increased proport

151 Using the mouse model of Mycobacterium avium infection, we show in this study tha

152 d whether ISS-ODN could modify the course of Mycobacterium avium infection.

153 s evaluated in a mouse model of disseminated Mycobacterium avium infection.

154 sis pulmonary hypersensitive pneumonitis and Mycobacterium avium infections.

155 Isolates of Mycobacterium avium-intracellulare (MAI) form multiple c

156 that most resemble Mycobacterium simiae and Mycobacterium avium-intracellulare complex but which pos

157 These included a psoas abscess secondary to Mycobacterium avium-intracellulare, septic wrist, bacter

158 Mycobacterium avium is a common opportunistic infection

159 Invasion of intestinal mucosa of the host by Mycobacterium avium is a critical step in pathogenesis a

160 Mycobacterium avium is a facultative intracellular patho

161 Mycobacterium avium is a major opportunistic pathogen in

162 Mycobacterium avium is a major opportunistic pathogen of

163 Mycobacterium avium is abundant in the environment.

164 Mycobacterium avium is an intracellular pathogen that ha

165 Mycobacterium avium is an opportunistic pathogen in AIDS

166 Mycobacterium avium is capable of invading the intestina

167 terium tuberculosis, Mycobacterium leprae or Mycobacterium avium is correlated with strong inflammato

168 The cell wall of the environmental pathogen Mycobacterium avium is important to its virulence and in

169 Mycobacterium avium is the most frequent cause of dissem

170 The LRF patterns of Mycobacterium avium isolates recovered from potable wate

171 Mycobacterium avium (M. avium) subspecies vary widely in

172 pj), Toxoplasma gondii (T. gondii, tg), and Mycobacterium avium (M. avium, ma) are the principal cau

173 Sera from 12 NTM-PD patients due to Mycobacterium avium, M. intracellulare, M. abscessus, or

174 vide a tool to better understand the role of Mycobacterium avium-M. intracellulare complex isolates i

175 The 159 Mycobacterium avium-M. intracellulare complex isolates w

176 Additional differentiation of Mycobacterium avium-M. intracellulare complex strains in

177 cystis carinii (Pc), Toxoplasma gondii (Tg), Mycobacterium avium (Ma), and rat liver in comparison wi

178 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three life-threatening pathoge

179 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three of the opportunistic org

180 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three of the opportunistic pat

181 r activity against M. tuberculosis (Mtb) and Mycobacterium avium (MAC) are reported.

182 fected with different mycobacterial strains (Mycobacterium avium, Mycobacterium bovis BCG or Mtb), we

183 erium smegmatis, Mycobacterium tuberculosis, Mycobacterium avium, Mycobacterium gadium and Mycobacter

184 PP (Legionella spp., Legionella pneumophila, Mycobacterium avium, Mycobacterium intracellulare, Pseud

185 Mycobacterium avium, Mycobacterium tuberculosis, and Myc

186 Infection of mice with Mycobacterium avium or immunization with a novel PE gene

187 lpha) production in macrophage infected with Mycobacterium avium or M smegmatis is dependent on myelo

188 erived macrophages were infected with either Mycobacterium avium or Mycobacterium tuberculosis and th

189 Later, the mice received Mycobacterium avium or Mycobacterium tuberculosis I.T.

190 nse in the thymus following dissemination of Mycobacterium avium or Mycobacterium tuberculosis.

191 drome, and opportunistic infections, such as Mycobacterium avium or Pneumocystis carinii infections.

192 sease in humans have led to speculation that Mycobacterium avium paratuberculosis (MAP) might be a ca

193 eening of microbial VOCs and (2) apply it to Mycobacterium avium paratuberculosis; the vaccine strain

194 When tested against Mycobacterium avium, PCIH was more effective than INH at

195 A structural genomics protein from Mycobacterium avium (PDB ID 3q1t) has been reported to b

196 The role that colonization with Mycobacterium avium plays in the development of dissemin

197 hat primary murine macrophages infected with Mycobacterium avium produced lower levels of tumor necro

198 primary murine macrophages had no effect on Mycobacterium avium retention in an early endosomal comp

199 human macrophages respond to infection with Mycobacterium avium, serovar 4, by producing tumor necro

200 olipids (GPLs), a major surface component of Mycobacterium avium, showed limited acidification and de

201 cal survey of distribution in Great Britain, Mycobacterium avium ssp. paratuberculosis (MAP) was dete

202 time in macrophages infected with pathogenic Mycobacterium avium strains relative to infections with

203 say that uses a unique element (ISMap02) for Mycobacterium avium sub0:36 PMparatuberculosis that is p

204 Mycobacterium avium subsp hominissuis is associated with

205 Johnes disease (JD), caused by Mycobacterium avium subsp paratuberculosis (MAP), occurs

206 evaluated for growth stimulating behavior in Mycobacterium avium subsp paratuberculosis.

207 obacterium avium subsp. paratuberculosis and Mycobacterium avium subsp. avium are antigenically and g

208 obacterium avium subsp. paratuberculosis and Mycobacterium avium subsp. avium are antigenically and g

209 tories has shown >98% sequence identity with Mycobacterium avium subsp. avium in some regions.

210 of Mycobacterium avium subsp. silvaticum and Mycobacterium avium subsp. avium, and a single isolate e

211 Mycobacterium avium subsp. hominissuis (MAH) is increasi

212 Mycobacterium avium subsp. hominissuis is an opportunist

213 "Mycobacterium avium subsp. hominissuis" is a robust and

214 "Mycobacterium avium subsp. hominissuis" is an opportunis

215 cosa by M. avium subsp. paratuberculosis and Mycobacterium avium subsp. hominissuis, a pathogen known

216 egration loci of IS900 (loci L1 and L9), one Mycobacterium avium subsp. paratuberculosis (M. paratube

217 R) sequencing approach for the genotyping of Mycobacterium avium subsp. paratuberculosis (M. paratube

218 Presently, no drugs against Mycobacterium avium subsp. paratuberculosis (M. paratube

219 ing affinity of Ag85A, Ag85B, and Ag85C from Mycobacterium avium subsp. paratuberculosis (MAP) (K(D)

220 protocol was optimized for the isolation of Mycobacterium avium subsp. paratuberculosis (MAP) from m

221 Available diagnostic assays for Mycobacterium avium subsp. paratuberculosis (MAP) have p

222 us intestinal inflammatory disease caused by Mycobacterium avium subsp. paratuberculosis (MAP) in cat

223 Johne's disease is caused by Mycobacterium avium subsp. paratuberculosis (MAP) infect

224 r regulatory mechanisms of host responses to Mycobacterium avium subsp. paratuberculosis (MAP) infect

225 To investigate the stochastic dynamics of Mycobacterium avium subsp. paratuberculosis (MAP) infect

226 Mycobacterium avium subsp. paratuberculosis (MAP), a slo

227 = 128), Mycobacterium kansasii (n = 10), and Mycobacterium avium subsp. paratuberculosis (n = 10), ca

228 SOD], and 35-kDa protein) were purified from Mycobacterium avium subsp. paratuberculosis and evaluate

229 Little is known of protein expression in Mycobacterium avium subsp. paratuberculosis and how this

230 nce repeats (SSRs) of 211 and 56 isolates of Mycobacterium avium subsp. paratuberculosis and M. avium

231 Mycobacterium avium subsp. paratuberculosis and Mycobact

232 Mycobacterium avium subsp. paratuberculosis and Mycobact

233 The genetic similarity between Mycobacterium avium subsp. paratuberculosis and other my

234 made between PBMCs stimulated in vitro with Mycobacterium avium subsp. paratuberculosis and PBMCs st

235 = 39) were tested for the presence of viable Mycobacterium avium subsp. paratuberculosis by a novel p

236 Efficient attachment and ingestion of Mycobacterium avium subsp. paratuberculosis by cultured

237 Attachment and ingestion of Mycobacterium avium subsp. paratuberculosis by two epith

238 Mycobacterium avium subsp. paratuberculosis causes an en

239 Infection with Mycobacterium avium subsp. paratuberculosis causes Johne

240 Mycobacterium avium subsp. paratuberculosis causes Johne

241 Mycobacterium avium subsp. paratuberculosis causes Johne

242 With the genome sequence of Mycobacterium avium subsp. paratuberculosis determined,

243 ism (AFLP) to characterize the genomes of 20 Mycobacterium avium subsp. paratuberculosis field isolat

244 the isolation, separation, and detection of Mycobacterium avium subsp. paratuberculosis from milk an

245 Mycobacterium avium subsp. paratuberculosis has been inc

246 The infection biology of Mycobacterium avium subsp. paratuberculosis has recently

247 Interactions between epithelium and Mycobacterium avium subsp. paratuberculosis have not bee

248 orescent probes (molecular beacons) detected Mycobacterium avium subsp. paratuberculosis in bovine fe

249 Mycobacterium avium subsp. paratuberculosis infection of

250 Mycobacterium avium subsp. paratuberculosis infection of

251 sensitivity and reduce time to diagnosis of Mycobacterium avium subsp. paratuberculosis infection.

252 In the present study, instillation of Mycobacterium avium subsp. paratuberculosis into the ton

253 and veterinary Johne's disease suggests that Mycobacterium avium subsp. paratuberculosis is a causati

254 Infection with Mycobacterium avium subsp. paratuberculosis is associate

255 Mycobacterium avium subsp. paratuberculosis is genetical

256 Mycobacterium avium subsp. paratuberculosis is shed into

257 Mycobacterium avium subsp. paratuberculosis is the causa

258 Mycobacterium avium subsp. paratuberculosis is the causa

259 Mycobacterium avium subsp. paratuberculosis is the cause

260 Analysis of short sequence repeats of Mycobacterium avium subsp. paratuberculosis isolated fro

261 lar diversity of animal and human strains of Mycobacterium avium subsp. paratuberculosis isolated in

262 for the study of the genetic relatedness of Mycobacterium avium subsp. paratuberculosis isolates har

263 s, infection with the intracellular pathogen Mycobacterium avium subsp. paratuberculosis results in a

264 Infection of the host with Mycobacterium avium subsp. paratuberculosis results in c

265 nd/or enrichment methods on the selection of Mycobacterium avium subsp. paratuberculosis subtypes.

266 Attachment of Mycobacterium avium subsp. paratuberculosis to host tiss

267 Five pigmented isolates of Mycobacterium avium subsp. paratuberculosis were examine

268 luate whether cows that were low shedders of Mycobacterium avium subsp. paratuberculosis were passive

269 Mycobacterium avium subsp. paratuberculosis, the agent o

270 omprised of crude whole-cell preparations of Mycobacterium avium subsp. paratuberculosis.

271 ease in cattle requires antigens specific to Mycobacterium avium subsp. paratuberculosis.

272 ntrol of mycobacterial infections, including Mycobacterium avium subsp. paratuberculosis.

273 The causative agent of Johne's disease is Mycobacterium avium subsp. paratuberculosis.

274 atuberculosis isolates, two isolates each of Mycobacterium avium subsp. silvaticum and Mycobacterium

275 MAP and Mycobacterium avium subspecies avium, a closely related

276 The role of Mycobacterium avium subspecies paratuberculosis (MAP) in

277 sheep, is caused by slow replicating bacilli Mycobacterium avium subspecies paratuberculosis (MAP) in

278 omplete genome sequence of a common clone of Mycobacterium avium subspecies paratuberculosis (Map) st

279 Mycobacterium avium subspecies paratuberculosis (MAP), t

280 By the analysis of 14 SNPs Mycobacterium avium subspecies paratuberculosis isolates

281 uipment for the large-scale global typing of Mycobacterium avium subspecies paratuberculosis isolates

282 erformed using genome sequence data from 133 Mycobacterium avium subspecies paratuberculosis isolates

283 g-chain acyl-CoA carboxylase holoenzyme from Mycobacterium avium subspecies paratuberculosis revealed

284 Typing of Mycobacterium avium subspecies paratuberculosis strains

285 y detect a 563 bp fragment of genomic DNA of Mycobacterium avium subspecies paratuberculosis through

286 A number of intestinal pathogens including Mycobacterium avium subspecies paratuberculosis, adheren

287 ed possibilities for the characterization of Mycobacterium avium subspecies paratuberculosis, and who

288 acillary paratuberculosis are both caused by Mycobacterium avium subspecies paratuberculosis.

289 guinea pigs were infected with M. bovis BCG, Mycobacterium avium, the attenuated Mycobacterium tuberc

290 Mycobacterium avium, the most common opportunistic patho

291 however, even within a single subspecies of Mycobacterium avium these lipids can differ.

292 ed mouse macrophages to resist the growth of Mycobacterium avium via alpha(2)-adrenergic stimulation.

293 nce strain 104 of the opportunistic pathogen Mycobacterium avium was isolated from an adult AIDS pati

294 effect described in macrophages infected by Mycobacterium avium was not observed in our model, which

295 While Mycobacterium avium was once regarded as innocuous, its

296 Mycobacterium avium was recovered from 41 specimens; 36

297 f the fibrinolytic system and infectivity by Mycobacterium avium were analyzed.

298 pportunistic pathogens Toxoplasma gondii and Mycobacterium avium when administered via the i.p. or i.

299 s contrasted with Gram-positive bacteria and Mycobacterium avium, which activated cells via TLR2 but

300 valuated the effect of iron on the growth of Mycobacterium avium within macrophages as well as on the