ライフサイエンスコーパス: Mycobacterium tuberculosis

1 ences in infection with different strains of Mycobacterium tuberculosis.

2 rved in the plasma of patients infected with Mycobacterium tuberculosis.

3 tivities against ESKAPE pathogens as well as Mycobacterium tuberculosis.

4 ophagic control of membrane-damaging microbe Mycobacterium tuberculosis.

5 er scientific community: cow, pig, wheat and mycobacterium tuberculosis.

6 tabolites support innate immune responses to Mycobacterium tuberculosis.

7 PCR assay for pyrazinamide susceptibility in Mycobacterium tuberculosis.

8 a and bacteria, including the human pathogen Mycobacterium tuberculosis.

9 smegmatis, organisms that serve as models of Mycobacterium tuberculosis.

10 about its effects on human host responses to Mycobacterium tuberculosis.

11 from spontaneous mutations in the genome of Mycobacterium tuberculosis.

12 port system is critical for the virulence of Mycobacterium tuberculosis.

13 plate for clinical reporting NGS results for Mycobacterium tuberculosis.

14 lete understanding of drug susceptibility to Mycobacterium tuberculosis.

15 biotic in lung lesions of mice infected with Mycobacterium tuberculosis.

16 mechanisms at the host-pathogen interface of Mycobacterium tuberculosis.

17 athic tau and during phagosome parasitism by Mycobacterium tuberculosis.

18 which is a highly immunogenic polypeptide of Mycobacterium tuberculosis, a causative agent of TB.

19 diated bactericidal killing of the bacterium Mycobacterium tuberculosis, a major clinical breakthroug

20 ifications of proteins during infection with Mycobacterium tuberculosis, a notorious intracellular pa

21 In contrast, Mycobacterium smegmatis and Mycobacterium tuberculosis accumulate detectable levels

22 Pathogens such as Mycobacterium tuberculosis accumulate polyphosphate, and

23 However, many persistent pathogens, such as Mycobacterium tuberculosis, actively target the very hos

24 We also show that the AHAS complex of Mycobacterium tuberculosis adopts a similar structure, t

25 Genetic diversity of Mycobacterium tuberculosis affects immune responses and

26 activity and, unlike the previously reported Mycobacterium tuberculosis AhpC/D system, SpAhpD does no

27 ite that controls the redox/energy status of Mycobacterium tuberculosis Although the major steps of M

28 his study describes the genetic diversity of Mycobacterium tuberculosis among tuberculosis patients i

29 phylogenetic spectrum of pathogens including Mycobacterium tuberculosis and carbapenem-resistant Ente

30 -epimerase (DprE1) is an essential enzyme in Mycobacterium tuberculosis and has recently been studied

31 ns may modulate the inflammatory response to Mycobacterium tuberculosis and influence disease present

32 hase is required for growth and viability of Mycobacterium tuberculosis and is a validated clinical t

33 Mycobacterium tuberculosis and M. smegmatis form drug-to

34 ity against mycobacterial species, including Mycobacterium tuberculosis and Mycobacterium leprae in a

35 Pathogenic mycobacteria, such as Mycobacterium tuberculosis and Mycobacterium marinum, ha

36 pert assay (Cepheid, Sunnyvale, CA, USA) for Mycobacterium tuberculosis and rifampicin resistance (Xp

37 more IL-35 after stimulation by antigens of Mycobacterium tuberculosis and secreted more IL-10.

38 responses to intracellular pathogens such as Mycobacterium tuberculosis and Toxoplasma gondii.

39 severe human diseases such as tuberculosis (Mycobacterium tuberculosis) and diphtheria (Corynebacter

40 B) patients lack microbiological evidence of Mycobacterium tuberculosis, and misdiagnosis or delayed

41 osts, including herpesviruses, retroviruses, Mycobacterium tuberculosis, and Toxoplasma gondii.

42 ore and after BCG vaccination, while ex vivo Mycobacterium tuberculosis- and Staphylococcus aureus-in

43 routes would boost cellular immunity to the Mycobacterium tuberculosis antigen 85A (Ag85A).

44 of IL-1beta, total IgA, and IgA specific to Mycobacterium tuberculosis antigen in the exhaled breath

45 The reconstitution of Mycobacterium tuberculosis antigen-specific CD4 T cells

46 Several Wbl proteins of Mycobacterium tuberculosis are known to interact with it

47 Novel antimicrobials for treatment of Mycobacterium tuberculosis are needed.

48 s, a severe infectious disease caused by the Mycobacterium tuberculosis, arouses huge concerns global

49 ene isotuberculosinol, a virulence factor of Mycobacterium tuberculosis as a representative example.

50 more than 10% of controls were infected with Mycobacterium tuberculosis, as 97% were of European gene

51 nvolved in the mechanism of action of PZA in Mycobacterium tuberculosis, as previously thought.

52 132 weeks, defined by cultures negative for Mycobacterium tuberculosis at 132 weeks and at a previou

53 thogens such as Streptococcus pneumoniae and Mycobacterium tuberculosis AtaC is monomeric in solution

54 ligand-induced ordering of the N terminus of Mycobacterium tuberculosis ATR, which organizes a dynami

55 -cell variation in morphological features of Mycobacterium tuberculosis bacilli to develop a rapid pr

56 re stimulated with Staphylococcus aureus and Mycobacterium tuberculosis before, as well as 2 weeks an

57 recycling pathways of NAD(+) biosynthesis in Mycobacterium tuberculosis but not in humans.

58 A), interrupts biosynthesis of coenzyme A in Mycobacterium tuberculosis by binding to aspartate decar

59 y T cells from humans latently infected with Mycobacterium tuberculosis By comparing native HBHA with

60 Further, we demonstrate that NfuA from Mycobacterium tuberculosis can also regenerate the auxil

61 Molecular surveillance of rifampin-resistant Mycobacterium tuberculosis can help to monitor the trans

62 Mycobacterium tuberculosis causes tuberculosis, a diseas

63 The Mycobacterium tuberculosis cell envelope is a critical i

64 ferases EmbA, EmbB, and EmbC are involved in Mycobacterium tuberculosis cell wall synthesis and are r

65 After exposure to Mycobacterium tuberculosis, children aged under 5 years

66 385 Mycobacterium tuberculosis clinical isolates were proces

67 gaps regarding the role of MDSCs in HIV and Mycobacterium tuberculosis (co)infections.

68 The 300-kDa ClpP1P2 protease from Mycobacterium tuberculosis collaborates with the AAA+ (A

69 he human- and animal-adapted lineages of the Mycobacterium tuberculosis complex (MTBC) are thought to

70 e Xpert MTB/RIF Ultra assay for detection of Mycobacterium tuberculosis complex (MTBC) DNA in samples

71 tudy assembles DNA adenine methylomes for 93 Mycobacterium tuberculosis complex (MTBC) isolates from

72 The Mycobacterium tuberculosis complex (MTBC) members displa

73 to bedaquiline (BDQ) and delamanid (DLM) in Mycobacterium tuberculosis complex (MTBc) strains is poo

74 man tuberculosis is caused by members of the Mycobacterium tuberculosis complex (MTBC) that vary in v

75 n and analysis of drug-resistance profile of Mycobacterium tuberculosis complex (MTC), a causative ag

76 We evaluated WOT in persons with active Mycobacterium tuberculosis complex disease using IS-enab

77 ibutes to the inability to differentiate the Mycobacterium tuberculosis Complex species, leading to a

78 opsy of the pleural lesion were positive for Mycobacterium tuberculosis complex.

79 Tuberculosis (TB), caused by Mycobacterium tuberculosis, continues to be a major glob

80 Based on a set of 654 clinical Mycobacterium tuberculosis culture isolates with known m

81 omparison to that of a reference standard of Mycobacterium tuberculosis culture on two sputum samples

82 We analyzed Mycobacterium tuberculosis culture-positive cases report

83 The pathogenicity of Mycobacterium tuberculosis depends upon its ability to c

84 Mycobacterium tuberculosis DNA can be detected from oral

85 thought that pathogenic mycobacteria such as Mycobacterium tuberculosis do not recycle their PG.

86 Mycobacterium species, including Mycobacterium tuberculosis, employs atypical long (C(60-

87 Mycobacterium tuberculosis encodes FadR orthologs one of

88 As important virulence factors of Mycobacterium tuberculosis, EsxA and EsxB not only play

89 The mechanisms by which Mycobacterium tuberculosis evades host immunity remain e

90 active TB (n = 16) and age- and sex-matched, Mycobacterium tuberculosis-exposed but uninfected househ

91 patients with TB disease relative to healthy Mycobacterium tuberculosis-exposed controls from Peru an

92 Following Mycobacterium tuberculosis exposure, significant increas

93 diagnosis of UG-TB is achieved by culturing Mycobacterium tuberculosis from an appropriate clinical

94 ods for drug susceptibility testing (DST) of Mycobacterium tuberculosis from patient sputum samples f

95 with drug-resistant TB would have culturable Mycobacterium tuberculosis from respirable, cough-genera

96 surviving bacterial pathogens that includes Mycobacterium tuberculosis, generates a salicyl-capped p

97 The Mycobacterium tuberculosis genome harbors an unusually h

98 Tens of millions of children are exposed to Mycobacterium tuberculosis globally every year; however,

99 of Bacillus subtilis, Escherichia coli, and Mycobacterium tuberculosis gyrases and of heterologous e

100 bacterium bovis bacillus Calmette-Guerin and Mycobacterium tuberculosis H(37)R(v) burden in the lungs

101 and low oxygen recovery assay (LORA) against Mycobacterium tuberculosis H37Rv with minimum inhibitory

102 Mycobacterium tuberculosis has an unusual outer membrane

103 ive channel of large conductance (MscL) from Mycobacterium tuberculosis has been used as a structural

104 Over the years, Mycobacterium tuberculosis has developed a form of resis

105 Mycobacterium tuberculosis has evolved numerous type VII

106 MtCM, the CM from Mycobacterium tuberculosis, has less than 1% of the cata

107 ofclinical isolates of the causative agent, Mycobacterium tuberculosis, has recently garnered attent

108 Many microbes, including Mycobacterium tuberculosis, have evolved to evade or exp

109 receptor 2 (TLR2) to host resistance against Mycobacterium tuberculosis HN878, a clinical isolate bel

110 particular action on the persistent forms of Mycobacterium tuberculosis However, no drug susceptibili

111 cytokine responses in latent infection with Mycobacterium tuberculosis However, their role in modula

112 SK-proprietary library against intracellular Mycobacterium tuberculosis identified 1, a thioalkylbenz

113 e current diagnostic methods for identifying Mycobacterium tuberculosis in cerebrospinal fluid (CSF)

114 .-born persons who were likely infected with Mycobacterium tuberculosis in their countries of birth.

115 s in surgical lung resections and blood from Mycobacterium tuberculosis-infected (Mtb-infected) indiv

116 B) is variable among individuals with latent Mycobacterium tuberculosis infection (LTBI), but validat

117 Identification of biomarkers for latent Mycobacterium tuberculosis infection and risk of progres

118 Patients were tested for Mycobacterium tuberculosis infection at baseline with co

119 Mycobacterium tuberculosis infection causes high rates o

120 BCG appears to reduce acquisition of Mycobacterium tuberculosis infection in children, measur

121 iming strategy could protect against aerosol Mycobacterium tuberculosis infection in mice.

122 role in the early innate immune response to Mycobacterium tuberculosis infection in the lung.

123 reatest risk factor for transition of latent Mycobacterium tuberculosis infection into active tubercu

124 Screening for, and treatment of, latent Mycobacterium tuberculosis infection is routine before i

125 ne components involved in protecting against Mycobacterium tuberculosis infection is urgently needed

126 t prevalent in regions of the world in which Mycobacterium tuberculosis infection remains endemic and

127 cts from The Gambia who never develop latent Mycobacterium tuberculosis infection shows distinct tran

128 terial loads and exacerbate pathology during Mycobacterium tuberculosis infection upon GM-CSF blockad

129 is a chronic inflammatory disease caused by Mycobacterium tuberculosis infection which causes tremen

130 -control study of patients with asymptomatic Mycobacterium tuberculosis infection, a novel 3-gene tra

131 h type 2 diabetes (T2D) have a lower risk of Mycobacterium tuberculosis infection, progression from i

132 ve disease with immunodeficiency, as well as Mycobacterium tuberculosis infection, underscoring their

133 ttle is known about the physiology of latent Mycobacterium tuberculosis infection.

134 rials including 6859 adult participants with Mycobacterium tuberculosis infection.

135 soniazid-rifapentine for treatment of latent Mycobacterium tuberculosis infection.

136 In this review, we focus on how Mycobacterium tuberculosis infects antigen-presenting ce

137 lapse imaging to reveal the dynamics of host-Mycobacterium tuberculosis interactions at an air-liquid

138 Mycobacterium tuberculosis is a global health problem in

139 es both in vivo and in vitro analyses, since Mycobacterium tuberculosis is a highly specialized human

140 Because Mycobacterium tuberculosis is an activator of cGAS-depen

141 e final step in mycolic acid biosynthesis in Mycobacterium tuberculosis is catalysed by mycolyl reduc

142 Gene rv3722c of Mycobacterium tuberculosis is essential for in vitro gro

143 and the growing prevalence of drug-resistant Mycobacterium tuberculosis is making disease control mor

144 Mycobacterium tuberculosis is one of the most hard-to-tr

145 The major human pathogen Mycobacterium tuberculosis is rarely reported to cause d

146 The human pathogen Mycobacterium tuberculosis is the causative agent of tub

147 Mycobacterium tuberculosis is the cause of the world's m

148 Early clearance of Mycobacterium tuberculosis is the eradication of infecti

149 Mycobacterium tuberculosis is the leading cause of death

150 Mycobacterium tuberculosis is the leading worldwide caus

151 g hemagglutinin (HBHA), a surface protein of Mycobacterium tuberculosis, is an attractive vaccine can

152 nfection by pathogenic mycobacteria, such as Mycobacterium tuberculosis, is facilitated by virulence

153 ncentrations (CCs) for categorizing clinical Mycobacterium tuberculosis isolates as susceptible/resis

154 opore MinION whole-genome sequencing data of Mycobacterium tuberculosis isolates for species identifi

155 lly and genotypically characterized clinical Mycobacterium tuberculosis isolates.

156 Iron is an essential element for Mycobacterium tuberculosis; it has at least 40 enzymes t

157 in fbiA, fbiB, fbiC, ddn, and fgd1 genes of Mycobacterium tuberculosis, knowledge about the molecula

158 bacterial phylum that includes the pathogen Mycobacterium tuberculosis, lack the canonical FtsZ-memb

159 Mycobacterium tuberculosis lipid metabolism pathways fac

160 The Mycobacterium tuberculosis load in the brain of individu

161 Mycobacterium tuberculosis (M. tb.) is a pervasive respi

162 Mycobacterium tuberculosis (M. tuberculosis) has coevolv

163 ncluding the notoriously persistent pathogen Mycobacterium tuberculosis (M. tuberculosis).

164 ent progression to TB among individuals with Mycobacterium tuberculosis (M.tb) infection.

165 Knowing when a person was infected with Mycobacterium tuberculosis (M.tb) is critical as recent

166 Antigens from Mycobacterium tuberculosis (M.tb), have been shown to st

167 ht into the within-host genetic diversity of Mycobacterium tuberculosis (M.TB), revealing remarkably

168 We used a 24-week guinea pig vaccination-Mycobacterium tuberculosis (M.tb.) challenge model to te

169 Multidrug-resistant Mycobacterium tuberculosis (MDR-TB) accounts for 3.7% of

170 hile the blue benzoquinone is active against Mycobacterium tuberculosis (MIC = 4 ug/mL) and even agai

171 redoxin alkyl hydroperoxide reductase E from Mycobacterium tuberculosis (MtAhpE-SH) reacts fast with

172 ndered by absence of rapid tests to identify Mycobacterium tuberculosis (MTB) and detect isoniazid (I

173 Mycobacterium tuberculosis (Mtb) and human immunodeficie

174 deadliest infectious diseases, is caused by Mycobacterium tuberculosis (MTB) and remains a public he

175 nc-responsive binding of Mpy to ribosomes in Mycobacterium tuberculosis (Mtb) and show Mpy-dependent

176 s from 58 individuals latently infected with Mycobacterium tuberculosis (Mtb) and to group them accor

177 y, and a species-selective inhibitor of the Mycobacterium tuberculosis (Mtb) ATP synthase.

178 Mycobacterium tuberculosis (Mtb) can enter the body thro

179 Antimicrobial-resistant Mycobacterium tuberculosis (Mtb) causes over 200,000 dea

180 Mycobacterium tuberculosis (Mtb) continues to be a major

181 or S100A8/A9 deficiency resulted in improved Mycobacterium tuberculosis (Mtb) control during chronic

182 The viability of Mycobacterium tuberculosis (Mtb) depends on energy gener

183 Automated genotyping of drug-resistant Mycobacterium tuberculosis (MTB) directly from sputum is

184 (TB) but it has not been determined whether Mycobacterium tuberculosis (Mtb) directly regulates myel

185 combat rising levels of resistance, with new Mycobacterium tuberculosis (Mtb) drugs having the highes

186 ationale: Direct evidence for persistence of Mycobacterium tuberculosis (Mtb) during asymptomatic lat

187 Mycobacterium tuberculosis (Mtb) employs plethora of mec

188 hat has been established in Escherichia coli Mycobacterium tuberculosis (Mtb) encodes the RNA polymer

189 including Bacille Calmette-Guerin (BCG) and Mycobacterium tuberculosis (MTB) Erdman.

190 esis hallmarks for tuberculosis (TB) are the Mycobacterium tuberculosis (Mtb) escape from phagolysoso

191 ry of recombinant BCG strains expressing the Mycobacterium tuberculosis (Mtb) ESX-1 secretion system

192 Mycobacterium tuberculosis (Mtb) fumarate hydratase (fum

193 The human pathogen Mycobacterium tuberculosis (Mtb) harbors a well-orchestr

194 ng methods based on genetic data analysis of Mycobacterium tuberculosis (MTB) have been able to predi

195 ection of mice with clinical "hypervirulent" Mycobacterium tuberculosis (Mtb) HN878 induces human-lik

196 Mycobacterium tuberculosis (Mtb) imports and metabolizes

197 ents despite displaying poor potency against Mycobacterium tuberculosis (Mtb) in vitro.

198 es known to play a vital role in controlling Mycobacterium tuberculosis (Mtb) infection and disease p

199 Tuberculosis (TB) is caused by Mycobacterium tuberculosis (Mtb) infection and is a majo

200 Strategies to prevent Mycobacterium tuberculosis (Mtb) infection are urgently

201 Mycobacterium tuberculosis (Mtb) infection causes tuberc

202 Murine models of Mycobacterium tuberculosis (Mtb) infection demonstrate p

203 Mycobacterium tuberculosis (Mtb) infection is among top

204 temic glucose metabolism as a consequence of Mycobacterium tuberculosis (Mtb) infection, contributes

205 Ag85A expressed by Mycobacterium tuberculosis (Mtb) is a bacterial surface

206 Mycobacterium tuberculosis (Mtb) is an obligate human pa

207 The haematogenous dissemination of Mycobacterium tuberculosis (Mtb) is critical to the path

208 The stress response adaptability of Mycobacterium tuberculosis (Mtb) is still unresolved.

209 Mycobacterium tuberculosis (Mtb) is the etiological agen

210 Mycobacterium tuberculosis (Mtb) is the leading cause of

211 mutations in lepA that are found in clinical Mycobacterium tuberculosis (Mtb) isolates phenocopy lepA

212 ed retrospectively for the stored individual Mycobacterium tuberculosis (Mtb) isolates using broth mi

213 Tuberculosis (TB), caused by Mycobacterium tuberculosis (Mtb) latently infects approx

214 e demonstrate an unexpected role of H(2)S in Mycobacterium tuberculosis (Mtb) pathogenesis.

215 While Lrrk2 KO mice can control Mycobacterium tuberculosis (Mtb) replication, they have

216 Exposure to Mycobacterium tuberculosis (Mtb) results in heterogeneou

217 Mycobacterium tuberculosis (Mtb) strains are classified

218 nsmission of a limited number of predominant Mycobacterium tuberculosis (Mtb) strains.

219 involved in the presentation of ligands from Mycobacterium tuberculosis (Mtb) to MAIT cells.

220 The ability of Mycobacterium tuberculosis (Mtb) to persist in its host

221 rios including: 1) sustained interruption of Mycobacterium tuberculosis (Mtb) transmission, 2) sustai

222 nfected individuals, the mechanisms by which Mycobacterium tuberculosis (Mtb) worsens HIV-1 pathogene

223 ndered by absence of rapid tests to identify Mycobacterium tuberculosis (MTB), and detect isoniazid (

224 genic strategy of the causative agent of TB, Mycobacterium tuberculosis (Mtb), and therefore metaboli

225 pies aimed at optimizing immune responses to Mycobacterium tuberculosis (Mtb), as adjunctive treatmen

226 Iron is essential for growth of Mycobacterium tuberculosis (Mtb), but most iron in the h

227 erable component of the citric acid cycle in Mycobacterium tuberculosis (Mtb), is a metabolic target

228 Pulmonary tuberculosis, a disease caused by Mycobacterium tuberculosis (Mtb), manifests with a persi

229 The pathogen Mycobacterium tuberculosis (Mtb), the causative agent of

230 r of the world's population is infected with Mycobacterium tuberculosis (Mtb), the causative agent of

231 Mycobacterium tuberculosis (Mtb), the causative infectio

232 In the pathogen Mycobacterium tuberculosis (Mtb), the protective effect

233 A) modules encoded by the bacterial pathogen Mycobacterium tuberculosis (Mtb), three contain antitoxi

234 epitopes, ESAT6(1-20) and TB10.4(3-11) from Mycobacterium tuberculosis (Mtb), were covalently conjug

235 tuberculosis (TB) is caused by the bacterium Mycobacterium tuberculosis (Mtb), which has been evolvin

236 of the human population have been exposed to Mycobacterium tuberculosis (MTB), with the overwhelming

237 several inflammatory and stress responses in Mycobacterium tuberculosis (Mtb)-infected host cells.

238 In patients, PD-1 is expressed in Mycobacterium tuberculosis (Mtb)-infected lung tissue bu

239 ontent and activity are globally elevated in Mycobacterium tuberculosis (Mtb)-infected macrophages.

240 Ruhl et al. find that a Mycobacterium tuberculosis (Mtb)-specific lipid, SL-1, s

241 n infectious disease caused by the bacterium Mycobacterium tuberculosis (Mtb).

242 losis (ATB) currently relies on detection of Mycobacterium tuberculosis (Mtb).

243 set of individuals infected with aerosolized Mycobacterium tuberculosis (Mtb).

244 are insensitive to overexpression of InhA in Mycobacterium tuberculosis (Mtb).

245 many bacteria, including the human pathogen Mycobacterium tuberculosis (Mtb).

246 s), important lipid pathogenicity factors of Mycobacterium tuberculosis (Mtb).

247 hogens that cause chronic infections such as Mycobacterium tuberculosis (Mtb).

248 ulosis (TB), which is caused by the pathogen Mycobacterium tuberculosis (Mtb).

249 in rifampicin-resistant clinical isolates of Mycobacterium tuberculosis (Mtb).

250 tion mechanism of NQ inhibitors of ThyX from Mycobacterium tuberculosis (Mtb).

251 of synchronously replicating populations of Mycobacterium tuberculosis (Mtb).

252 own precisely how BDQ triggers cell death in Mycobacterium tuberculosis (Mtb).

253 BI, recent work in macaques co-infected with Mycobacterium tuberculosis (Mtb)/simian immunodeficiency

254 In Mycobacterium tuberculosis, mycolic acids and their glyc

255 ges with the membrane-permeabilizing microbe Mycobacterium tuberculosis or infection of target cells

256 There were no IEps due to Mycobacterium tuberculosis, Pneumocystis jirovecii, or T

257 Global Mycobacterium tuberculosis population comprises 7 major

258 Mycobacterium tuberculosis possesses a large number of g

259 l molecules might bind to the F(1) domain in Mycobacterium tuberculosis, prevent ATP synthesis, and i

260 The related Mycobacterium tuberculosis proteins were also found to i

261 ersity (panallelome) of Salmonella enterica, Mycobacterium tuberculosis, Pseudomonas aeruginosa, and

262 of mortality from a single infectious agent, Mycobacterium tuberculosis Relevant immune targets of th

263 acellular replication of the deadly pathogen Mycobacterium tuberculosis relies on the production of s

264 Mycobacterium tuberculosis remains a global health probl

265 ulosis, caused by the intracellular pathogen Mycobacterium tuberculosis, remains the world's deadlies

266 cent study reported that the UvrC protein of Mycobacterium tuberculosis removes damage in a manner an

267 century, but discovery of new niches, where Mycobacterium tuberculosis resides, continues.

268 Mycobacterium tuberculosis Rv3775 (LipE) was annotated a

269 The Mg(2+)-dependent Mycobacterium tuberculosis salicylate synthase (MbtI) is

270 For Klebsiella pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, and Sta

271 ases on a transmission network inferred from Mycobacterium tuberculosis sequencing data on extensivel

272 We used Mycobacterium tuberculosis/simian immunodeficiency virus

273 This assay utilizes the Mycobacterium tuberculosis-specific biomarker BlaC in re

274 f 9 prevalent bacterial pathogens, including Mycobacterium tuberculosis, Staphylococcus aureus, and E

275 ply the MAC to a dataset of 1595 drug-tested Mycobacterium tuberculosis strains and show that MACs pr

276 piratory syndrome coronavirus (SARS-CoV) and Mycobacterium tuberculosis (TB), and routinely used to r

277 ts were cytomegalovirus (CMV, n = 139, 41%), Mycobacterium tuberculosis (TB, n = 70, 21%), Plasmodium

278 ir role in protection against infection with Mycobacterium tuberculosis, termed "early clearance." ME

279 tter delineation of transmission clusters in Mycobacterium tuberculosis than traditional methods.

280 rystal structure of a full-length T-box from Mycobacterium tuberculosis that explains tRNA decoding a

281 Mycobacterium tuberculosis, the causative agent of pulmo

282 Glycolipids in Mycobacterium tuberculosis, the causative agent of tuber

283 Mycobacterium tuberculosis, the causative agent of tuber

284 Mycobacterium tuberculosis, the cause of human tuberculo

285 Mycobacterium tuberculosis, the leading cause of death d

286 Like its homolog from Mycobacterium tuberculosis, the T. curvata aldolase is a

287 tensively used to study the host response to Mycobacterium tuberculosis, their validity in revealing

288 xhibited moderate inhibitory potency against Mycobacterium tuberculosis thymidylate kinase, the targe

289 s (TB) therapy, as well as the propensity of Mycobacterium tuberculosis to develop drug resistance, a

290 Synthesis of the cofactor is essential for Mycobacterium tuberculosis to establish and maintain chr

291 ously uncharacterized network adaptations of Mycobacterium tuberculosis to the first-line anti-tuberc

292 with trehalose-based glycolipids produced by Mycobacterium tuberculosis TPP production starts in the

293 fic evidence indicating that the majority of Mycobacterium tuberculosis transmission to children in h

294 lly important endemic human disease agents - Mycobacterium tuberculosis (tuberculosis), Mycobacterium

295 The Mycobacterium tuberculosis type VII secretion system ESX

296 The Mycobacterium tuberculosis virulence factor EsxA and its

297 Here, we have shown that Mycobacterium tuberculosis was taken up by mesenchymal s

298 ichia coli and between 3.9 millions pairs in Mycobacterium tuberculosis We find strong coevolution fo

299 teway to aromatic amino acid biosynthesis in Mycobacterium tuberculosis, which shows extremely comple

300 We measured a network of genomic links using Mycobacterium tuberculosis whole-genome sequences.