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1 ion by chemoattractants, such as the peptide N-formyl-methionyl-leucyl-phenylalanine.
2 nules in the cremaster muscle in response to N-formyl- methionyl-leucyl-phenylalanine.
4 chemotactic responses upon stimulation with N-formyl methionyl leucyl phenylalanine and CC chemokine
5 g when cells are simultaneously treated with N-formyl-methionyl-leucyl-phenylalanine and IgE plus pol
6 of PMN to two biologically relevant agents, N-formyl-methionyl-leucyl-phenylalanine and leukotriene
7 to the chemoattractants interleukin 8, C5a, N-formyl-methionyl-leucyl-phenylalanine, and interleukin
8 ophils with phorbol-12-myristate-13-acetate, N-formyl-methionyl-leucyl-phenylalanine, arachidonic aci
9 however, P. stomatis significantly increased N-formyl-methionyl-leucyl phenylalanine (fMLF)-stimulate
10 Pak translocates to the plasma membrane upon N-formyl-methionyl-leucyl-phenylalanine (fMLF) stimulati
13 horbol 12-beta-myristate 13-alpha-acetate or N-formyl methionyl-leucyl-phenylalanine (fMLP) for stimu
14 ound that the chemotactic bacterial peptide, N-formyl- methionyl-leucyl-phenylalanine (fMLP), was abl
15 by lung isolation and ex vivo perfusion with n-formyl-methionyl-leucyl-phenylalanine (fMLP) (10(-)(7)
16 (oxidative species and elastase) exposed to N-formyl-methionyl-leucyl-phenylalanine (fMLP) and/or mu
18 mix than collagen I, and did not require an N-formyl-methionyl-leucyl-phenylalanine (fMLP) gradient
19 porally decreasing chemoattractant signal of N-formyl-methionyl-leucyl-phenylalanine (FMLP) in the ab
21 pendent decreases in secondary activation by N-formyl-methionyl-leucyl-phenylalanine (FMLP) or phorbo
22 ha (TNF-alpha)-primed human neutrophils with N-formyl-methionyl-leucyl-phenylalanine (fMLP) results i
23 kinases (MAPK)] were assessed in response to N-formyl-methionyl-leucyl-phenylalanine (fMLP) stimulati
24 Secondary stimulation of PMNs with 1 muM N-formyl-methionyl-leucyl-phenylalanine (fMLP) triggered
25 , interleukin-8 (IL-8), formylpeptides (e.g. N-formyl-methionyl-leucyl-phenylalanine (fMLP)), and pla
26 h was stimulated using N-formylated peptide (N-formyl-methionyl-leucyl-phenylalanine (fMLP)), platele
27 n by HT29-Cl.19A cells permits absorption of N-formyl-methionyl-leucyl-phenylalanine (fMLP), as occur
28 ating factor (G-CSF) and then activated with N-formyl-methionyl-leucyl-phenylalanine (FMLP), C(2)-cer
30 AA receptor in the CNS, and also reduces the N-formyl-methionyl-leucyl-phenylalanine (fMLP)-induced n
31 2alpha, was used to examine the mechanism of N-formyl-methionyl-leucyl-phenylalanine (fMLP)-mediated
36 ol myristate acetate, opsonized zymosan, and N-formyl-methionyl-leucyl-phenylalanine) induce p22(phox
37 3580, was able to abolish the TNF-alpha- and N-formyl-methionyl-leucyl-phenylalanine-induced activati
38 ing was examined by quantitative analysis of N-formyl-methionyl-leucyl-phenylalanine-induced increase
39 eutrophils were deficient in spontaneous and N-formyl-methionyl-leucyl-phenylalanine-induced polariza
40 migration in response to multiple agonists (N-formyl-methionyl-leucyl-phenylalanine, interleukin-8,
41 abeled chemotactic peptide receptor agonist (N-formyl-methionyl-leucyl-phenylalanine-lysine; N-For-ML
42 sPLA(2)-X to eosinophils under conditions of N-formyl-methionyl-leucyl-phenylalanine-mediated cPLA(2)
43 oth activities when cells were stimulated by N-formyl-methionyl-leucyl-phenylalanine or opsonized zym
44 in perforated-patch configuration with PMA, N-formyl-methionyl-leucyl-phenylalanine, or anti-IgE gre
45 L-13, in basophils stimulated with anti-IgE, N-formyl-methionyl-leucyl-phenylalanine, or phorbol 12-m
46 ly suppressed basophil activation induced by N-formyl-methionyl-leucyl-phenylalanine, phorbol 12-myri
47 the rate of pseudopod extension induced with N-formyl-methionyl-leucyl-phenylalanine, platelet activa
49 d (CD11b/CD18, the mannose receptor, and the N-formyl-methionyl-leucyl-phenylalanine receptor) did no
50 lly, it inhibited arachidonate production in N-formyl-methionyl-leucyl-phenylalanine-stimulated U937
51 g activity of alphaMbeta2 integrin following N-formyl-methionyl-leucyl phenylalanine stimulation.
52 necrosis factor-alpha (TNF-alpha) as well as N-formyl-methionyl-leucyl-phenylalanine treatment leads
53 ization method that retains coupling between N-formyl-methionyl-leucyl-phenylalanine tripeptide (FMLP
55 otaxis defects were also seen in response to N-formyl-methionyl-leucyl-phenylalanine, zymosan-activat