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1                                              NDGA analog-pretreated alpha-synuclein did not aggregate
2                                              NDGA analogs induced modest, progressive compaction of m
3                                              NDGA decreased sterol regulatory element binding protein
4                                              NDGA inhibited very low-density lipoprotein (VLDL) secre
5                                              NDGA negated the HCV-induced alteration of host lipid ho
6                                              NDGA seemed to be the most potent of the phenolic antiox
7                                              NDGA was able to inhibit FGFR3 autophosphorylation both
8                                              NDGA-mediated alterations of host lipid metabolism, LD m
9 ralizing ROS with nordihydroguaiaretic acid (NDGA) abrogated cell death induced by AF-TUSC2-erlotinib
10  and investigated nordihydroguaiaretic acid (NDGA) derivatives and have established that the reductiv
11 MP (8-Br-cAMP) or nordihydroguaiaretic acid (NDGA) had no effect on the relative level of channel act
12 CQ), NH(4)Cl, and nordihydroguaiaretic acid (NDGA) that alter discrete steps of endocytosis or Golgi-
13                   Nordihydroguaiaretic acid (NDGA) was also characterized as an inhibitor of carboxyl
14                   Nordihydroguaiaretic acid (NDGA) was observed by Ono et al. to decrease the fluores
15  COX enzymes; and nordihydroguaiaretic acid (NDGA), a lipoxygenase inhibitor on human lung cancer cel
16  novel analogs of nordihydroguaiaretic acid (NDGA), a phenolic dibenzenediol lignan, were explored us
17 s inhibited using nordihydroguaiaretic acid (NDGA), a specific inhibitor of 13-LOX.
18 lo-oxygenase, nor nordihydroguaiaretic acid (NDGA), an inhibitor of the lipoxygenase pathway.
19  the antioxidants nordihydroguaiaretic acid (NDGA), catechol, glutaryl probucol, and N-acetylcysteine
20 olipidemic agent, nordihydroguaiaretic acid (NDGA), on host lipid/fatty acid synthesis and HCV life c
21  Indomethacin and nordihydroguaiaretic acid (NDGA), potent inhibitors of the cyclooxygenase (COX) and
22 genase inhibitor, nordihydroguaiaretic acid (NDGA), the 5-lipoxygenase inhibitor, AA861, the epoxygen
23 tory properties of nordihydroguaiartic acid (NDGA), which blocks protein transport through the Golgi,
24           Neither nordihydroquararetic acid (NDGA), which inhibits metabolism of ArA by cyclooxygenas
25 oprevention (exisulind +/- retinoic acid +/- NDGA) and therapeutic (exisulind +/- paclitaxel +/- cisp
26                                     Although NDGA failed to disaggregate Abeta protofibrils, it did i
27 tent with the promoter studies, curcumin and NDGA, inhibitors of AP-1 activation, markedly inhibited
28 actions for sulindac sulfide, exisulind, and NDGA with paclitaxel, cisplatin, and 13-cis-retinoic aci
29 gnalling inhibitors Wortmannin, LY294002 and NDGA, and concluded that the four invasive pathogenic sp
30 gest that inhibitory small molecules such as NDGA that target a specific subcellular compartment may
31                                         Both NDGA and AA861 inhibited progesterone production and StA
32 ated protein kinase (MAPK) were inhibited by NDGA treatment.
33 nM total carboxylesterases was unaffected by NDGA.
34                            Further, cyclized NDGA reduced alpha-synuclein-driven neurodegeneration in
35                                 The cyclized NDGA analogs may serve as a platform for the development
36                          Oxidation-dependent NDGA cyclization was required for the interaction with m
37 when protofibrils were incubated with excess NDGA.
38 l lines expressing activated forms of FGFR3, NDGA generally resulted in a decrease in MAPK activation
39 ddition, we have determined that hydrophobic NDGA derivatives activate 15-HLO, suggesting a hydrophob
40 njection of the phospholipase A(2) inhibitor NDGA partially suppressed the induction of immediate ear
41 eline and in the presence of LOX inhibitors (NDGA, AA-861, CDC, baicalein, and PD146176) vs. vehicle-
42                  In addition, the binding of NDGA to Abeta protofibrils increased their nonspecific a
43 teration of thioflavin T upon the binding of NDGA to these aggregates.
44                               This effect of NDGA was enhanced by nonphenolic antioxidants such as N-
45                               This effect of NDGA was not due to inhibition of lipoxygenase because c
46                               The effects of NDGA on activated FGFR3 derivatives targeted either to t
47                     The antiviral effects of NDGA resulted in reduced HCV replication and secretion.
48 hdrawing substituents on the phenyl rings of NDGA.
49                                  Strikingly, NDGA-pretreated alpha-synuclein suppressed aggregation o
50 C50 = 0.003-0.150 microM); sulindac sulfide, NDGA, and 13-cis-retinoic acid had intermediate potency
51                          They concluded that NDGA could disaggregate Abeta fibrils into aggregates th
52      In the current study, we confirmed that NDGA induces a decrease in the fluorescence of thioflavi
53        Nuclear run-on studies indicated that NDGA increased eNOS transcription.
54                   These results suggest that NDGA might bind along the lateral surface of Abeta proto
55 lic hydroxyl groups completely inhibited the NDGA effect on eNOS mRNA levels.
56                         Exposure of BAECs to NDGA enhanced NO production, as measured by electron par
57 and perinuclear distribution of LDs, whereas NDGA most notably reduced the overall number and increas
58 re observed regardless of the order in which NDGA, thioflavin T, and Abeta protofibrils were added to
59 BAECs or human aortic endothelial cells with NDGA.
60 pha-synuclein did not aggregate even without NDGA-analogs in the aggregation mixture.