ライフサイエンスコーパス: NSE

1 NSE > 33 ng/ml has a high false-positive rate in patient

2 NSE > 33 ng/ml levels measured 1-3 days after cardiac ar

3 NSE and tau are better associated with NCD and less infl

4 NSE and tau protein at the 6-hour time point were both s

5 NSE can pinpoint the nanoscale dynamics changes in a hig

6 NSE directly probes coherent (i.e., pair correlated) sca

7 NSE levels were assessed in blood samples obtained 24, 4

8 NSE levels were deregulated in leukemias and were influe

9 NSE reveals that, in the cadherin-catenin complex, the m

10 NSE was a robust predictor of neurological outcome in a

11 NSE, S100beta, or BIS alone predicted neurological outco

12 NSE-apoE mice and similar models may facilitate the prec

13 NSE-Rb db/db mice were infected with T. cruzi to determi

14 = 0.983; at t + 7: R = 0.997, RMSE = 0.249, NSE = 0.994; at t + 14: R = 0.996, RMSE = 0.304, NSE = 0

15 d NO-mediated compensatory responses in 2K1C NSE-AT(1a) peripheral arteries during this later phase,

16 = 0.994; at t + 14: R = 0.996, RMSE = 0.304, NSE = 0.991).

17 ing level at t + 3: R = 0.992, RMSE = 0.426, NSE = 0.983; at t + 7: R = 0.997, RMSE = 0.249, NSE = 0.

18 are always slender and elongate, the PGP 9.5/NSE population comprise two morphological types--one sle

19 lar in overall shape to the pyriform PGP 9.5/NSE population, gustducin never colocalizes with PGP 9.5

20 W-M04, is achieved over Drammen (r = 0.9765, NSE = 0.9510, KGE = 0.8641, PI = 0.3211, and RMSE = 0.22

21 AR-M5P (R(2) = 0.954, R = 0.977, NSE = 0.954, MAE = 0.042, MSE = 0.003, and PBias = 1.466

22 rge, with evaluation criteria of CC = 0.983, NSE = 0.962, RMSE = 0.178, and MAE = 0.071.

23 r 36 degrees C does not significantly affect NSE levels.

24 get temperature did not significantly affect NSE values.

25 menting these synergistic properties affords NSE-based planar PSCs that deliver a remarkable power co

26 The AIMNet-NSE model allows to fully bypass QM calculations and der

27 Built upon the AIMNet2 framework, AIMNet2-NSE is trained on an extensive dataset comprising 20 mil

28 Overall, AIMNet2-NSE represents a significant advancement in machine lear

29 Here we present AIMNet2-NSE (neural spin-charge equilibration), a neural network

30 The neuronal expression of an NSE-Fig4(Cys486Ser) transgene in vivo prevented the neon

31 2 hours after CPR (FPR 10; 95% CI, 6-16) and NSE levels were not.

32 among patients and HC separately, IL-18 and NSE concentrations were positively correlated (p < 0.001

33 Double-label studies show that PGP 9.5 and NSE always colocalize.

34 cted C-33 LNCaP cells, ERK was activated and NSE level was elevated, and cells obtained the NE-like p

35 cted C-33 LNCaP cells, ERK was activated and NSE was elevated.

36 presence of PD98059, the ERK activation and NSE elevation were abolished, following a dose-response

37 , but also for immobilization of the CEA and NSE aptamers.

38 on in serum IL-6, IL-1beta, epinephrine, and NSE levels from baseline to 48 h post-injury (p = 0.001)

39 mice compared with those in infected FVB and NSE-Rb db/db mice.

40 the resulting inflammation in Hand2(+/-) and NSE-noggin mice was compared with that of wild-type litt

41 e robust expression of the NE genes HRH1 and NSE/ENO2 in KSHV-infected KS tissue samples and KS visce

42 mflow simulation testing gave RMSE, MAE, and NSE of 1.65, 1.21 MCM/month, and 0.80, respectively.

43 ion of botanicals, for instance NeemAzal and NSE deterred oviposition of S. litura.

44 ntin, and negative for A2B5, CNPase, NF, and NSE.

45 oncentrations of S100B, GFAP, BDNF, NFL, and NSE were assessed in 48 collegiate football players (32

46 the detection of the NSE derived peptide and NSE.

47 s approach is the multiplexing of ProGRP and NSE with the additional ability to perform fingerprintin

48 ighest concentrations in week 1 of S100B and NSE were associated with poor outcome, as were highest c

49 te small-angle neutron scattering (SANS) and NSE scattering properties.

50 64 dysregulated TF-miRNA-gene FFLs in SE and NSE, respectively.

51 d the formation mechanism of the CE, SSE and NSE is largely debated today, yet the upwelling and biol

52 UN, KLF4, PLXDC2, RND3, SPI1, and TIMP3) and NSE-specific dysregulated miRNAs (miR-367-3p, miR-519d-3

53 n support substantial immobilisation of anti-NSE agent.

54 The anti-NSE immobilized BP-TFG biosensor has been implemented to

55 uron-specific enolase promoter-driven apoE3 (NSE-apoE3) or apoE4 (NSE-apoE4) transgenic mice.

56 promoter-driven apoE3 (NSE-apoE3) or apoE4 (NSE-apoE4) transgenic mice.

57 N-LEPR-B and neuron-specific enolase-LEPR B (NSE-LEPR-B).

58 ry roles involving the TL difference between NSE and SE, and provided a better molecular understandin

59 ferent telomere length (TL) features between NSE (n = 48) and SE (n = 55).

60 Emerging CSF biomarkers NSE, VLP-1, HFABP, and YKL-40 were moderately associated

61 blot analysis was performed on bitransgenic NSE-tTA, TetOp-DeltaFosB, and single-transgenic NSE-tTA

62 e hypothesize that increased levels of blood NSE may characterize LHON carriers as a biomarker of ong

63 s of db/db mice that are hemizygous for both NSE-LEPR-B and SYN-LEPR-B transgenes (Nse+Syn db/db).

64 tals the enzyme forms the asymmetric complex NSE x Mg2 x SO4/NSE x Mg x Cl, where "/" separates the d

65 with NSE-apoE3 mice and wild-type controls, NSE-apoE4 mice showed impairments in learning a water ma

66 flexes, and motor scores 72 hours after CPR; NSE levels at admission, 12 hours after reaching target

67 or using a genetic approach (Nestin-CreERT2/NSE-DTA mice) impeded the forgetting of old memories.

68 th Alzheimer's disease, whereas those of CSF NSE, VLP-1, HFABP, and YKL-40 were moderate (average rat

69 -preferring mice that overexpress DeltaFosB (NSE-tTA x TetOp-DeltaFosB) exhibit less avoidance of the

70 molecule diffusion to the simulation-derived NSE and potential problems in their estimation are exami

71 TFG biosensor has been implemented to detect NSE biomarkers demonstrating ultrahigh sensitivity with

72 r was demonstrated successful in determining NSE in clinical serum samples.

73 a marker for the overall nerve distribution, NSE antibodies revealed abundant smooth and varicose ner

74 etic or biochemical tumor marker (MYCN, DNA, NSE, or ferritin), or by both unamplified MYCN and aneup

75 Higher diversity in the CSF during NSE was associated with greater CNS inflammation.

76 ron scattering (SANS) and neutron spin echo (NSE) spectroscopy were used to measure the tertiary stru

77 ensive approach-combining neutron spin-echo (NSE) spectroscopy, solid-state deuterium NMR ((2)H NMR)

78 , may exert beneficial non-specific effects (NSE) in reducing infant mortality.

79 gside the highest Nash-Sutcliffe efficiency (NSE) of 0.8048 and Pearson correlation coefficient (PCC)

80 ters based on the Nash-Sutcliffe Efficiency (NSE) to evaluate the TLBO's predictive skill using bench

81 : see text]), and Nash-Sutcliffe efficiency (NSE) values corresponding [Formula: see text] and [Formu

82 are error (RMSE), Nash-Sutcliffe efficiency (NSE), Kling-Gupta efficiency (KGE) and Mean bias error (

83 he models based on interval-PLS efficiently (NSE >= 0.90) predicted storage time with low errors (RSR

84 ti-zwitterionic small-molecule electrolytes (NSEs) are introduced, which act not only as charge-extra

85 30.4 %) carriers with significantly elevated NSE levels (mean = 75.8 +/- 42.3 mug/L), 9/14 (64.3%) we

86 levation (STE) and non-ST-segment elevation (NSE) groups and by the presence or absence of abnormal R

87 f 2.8 kb of the rat neuron-specific enolase (NSE) 5' flanking region fused to a cDNA encoding the ful

88 Serum levels of neuron-specific enolase (NSE) and neuron-enriched S100 beta (S100beta) were measu

89 ght chain (NfL) and neuron-specific enolase (NSE) are associated with poor neurological outcome after

90 ular recognition of neuron specific enolase (NSE) biomarker.

91 diagnosis of human neuron-specific enolase (NSE) cancer biomarkers.

92 Moreover, the neuron-specific enolase (NSE) immunohistochemistry results revealed the protectiv

93 c antigen (CEA) and neuron-specific enolase (NSE) in a clinical sample with high sensitivity and spec

94 Neuron-specific enolase (NSE) is a biomarker for neuronal stress.

95 Neuron-specific enolase (NSE) is a widely-used biomarker for prognostication of n

96 eptide (ProGRP) and neuron specific enolase (NSE) is presented, which involves coextraction by immuno

97 were mortality and neuron-specific enolase (NSE) levels on days 1 and 3.

98 Serum neuron specific enolase (NSE) measurements, brain imaging findings, somatosensory

99 ining for MCP-1 and neuron specific enolase (NSE) or glial fibrillary acidic protein (GFAP) showed th

100 Human neuron-specific enolase (NSE) or isozyme gamma has been expressed with a C-termin

101 that antibodies to neuron-specific enolase (NSE) preferentially label a subpopulation of photorecept

102 s incorporating the neuron specific enolase (NSE) promoter and either a rat GAP-43 cDNA or the corres

103 c, incorporated the neuron-specific enolase (NSE) promoter and the internal ribosome entry site (IRES

104 nder control of the neuron-specific enolase (NSE) promoter develops a FOP-like phenotype.

105 rol of 1.8-kilobase neuron-specific enolase (NSE) promoter for this purpose.

106 nic mice using the neuron- specific enolase (NSE) promoter or a modified glial fibrillary acidic prot

107 the control of the neuron-specific enolase (NSE) promoter show both markedly increased bone formatio

108 n, we have used the neuron-specific enolase (NSE) promoter to express human apoE3 or apoE4 at similar

109 nder control of the neuron specific enolase (NSE) promoter.

110 nder control of the neuron-specific enolase (NSE) promoter.

111 chemical sensor for neuron specific enolase (NSE) was developed by electrochemical polymerizing ionic

112 ogranin A (CgA) and neuron-specific enolase (NSE) were assessed monthly if elevated at baseline.

113 llometry, and serum neuron-specific enolase (NSE) were retrieved.

114 increased level of neuron-specific enolase (NSE), a classical marker of neuronal cells.

115 ed for detection of neuron-specific enolase (NSE), a traumatic brain injury (TBI) protein biomarker,

116 biomarkers [S100B, Neuron-Specific Enolase (NSE), and epinephrine].

117 neuromarkers S100B, neuron-specific enolase (NSE), and glial fibrillary acidic protein (GFAP), in add

118 ologic examination, neuron-specific enolase (NSE), and median nerve somatosensory-evoked potentials (

119 S-100b, neuron specific enolase (NSE), and tau protein were assayed as MBIs preoperativel

120 odified epitopes of neuron specific enolase (NSE), as-synthesized gold nanoparticles (AuNPs), and fun

121 these cells contain neuron-specific enolase (NSE), tyrosine hydroxylase (TH) and 5-hydroxytryptamine

122 rabbit antisera to neuron-specific enolase (NSE), tyrosine hydroxylase (TH), neuropeptide Y (NPY), v

123 Serotonin, neuron-specific enolase (NSE), ubiquitin carboxyl terminal hydrolase (PGP 9.5), a

124 r apoE4 in neurons [neuron-specific enolase (NSE)-apoE] or astrocytes [glial fibrillary acidic protei

125 o-localization with neuron-specific enolase (NSE)-IR, but is especially prominent in cell bodies of a

126 ofilament (NF), and neuron specific enolase (NSE).

127 or the detection of neuron-specific enolase (NSE).

128 ne ionic liquid and neuron specific enolase (NSE).

129 accuracy than serum neuron-specific enolase (NSE; the area under the receiver operating characteristi

130 receptor H1 [HRH1], neuron-specific enolase [NSE] [ENO2], neuronal protein gene product 9.5 [PGP9.5],

131 protein [GFAP], and neuron-specific enolase [NSE]), and information about intracranial pressure lower

132 earing a transgene (neuron-specific enolase [NSE]-Rb) expressing the B isoform of LEPR, the isoform c

133 ndings of nonspecific small bowel enteritis (NSE) on capsule endoscopy (CE) poses a clinical challeng

134 sion in children with nonsyndromic epilepsy (NSE) over the course of >/=10 years from initial diagnos

135 osal biopsies of normal squamous epithelium (NSE), BE and EAC obtained from a patient who progressed

136 sex ratio, and neritic survival equivalent [NSE]) computed from simulations better predicted each ot

137 this setting of shared physiologic erasure, NSEs harbor a malignancy-associated hypermethylation cor

138 efined as neurosymptomatic HIV-1 CSF escape (NSE).

139 phosphatase (TRAP) and nonspecific esterase (NSE) activity, which are selective markers for the osteo

140 exploration of a Natural Sources Exclusion (NSE) provision within the TMDL.

141 key nonsense-mediated RNA decay switch exon (NSE) in ATM is repressed by U2AF, PUF60 and hnRNPA1.

142 Surprisingly, most of the cells expressing NSE::LacZ outside the olfactory bulb were astrocytes.

143 apoE4 and, to a significantly lesser extent, NSE-apoE3 mice; however, no fragments were detected in G

144 ormulation NeemAzal and a neem seed extract (NSE) were evaluated in field spraying trials alongside c

145 EA (R(2) = 0.989) and 0.05-500 ng mL(-1) for NSE (R(2) = 0.944), respectively.

146 was 2 pg mL(-1) for CEA and 10 pg mL(-1) for NSE.

147 des (12 out of 30 for SE and 8 out of 32 for NSE) in the top ranked FFLs could predict subtype-classi

148 cteristic curve was 0.91 for tau vs 0.86 for NSE at 72 hours, p = 0.00024).

149 nts of 2.3 x 10(-10) M and 3 x 10(-11) M for NSE recognition using the corresponding epitope imprints

150 nstructed platform was successfully used for NSE determination in human serum samples.

151 Furthermore, NSE levels were nearly three times greater in asymptomat

152 pM (=700 pg/mL), for total ProGRP and gamma-NSE, respectively.

153 munometric assays for total ProGRP and gamma-NSE, were seen in the analyses of patient serum samples.

154 usly identified proteins (CRMP1, CRMP2, GDA, NSE, LDHA, LDHB, STIP1, and YBOX) that are highly expres

155 At 48 and 72 h, NSE predicted neurological outcome with areas under the

156 Of 613 cohort members, 347 had NSEs, of whom 294 (85%) were followed >/=10 years (maxim

157 High NSE cutoff values with false positive rates </=5% and ti

158 r-derived features, performed best with high NSE (0.86), KGE (0.80), and R (0.93) values during calib

159 C, MAE of 1.9867, RAE of 29.5440, and higher NSE of 0.8720 and R2 of 0.8720.

160 sitive participants had significantly higher NSE (3713 vs. 2200 pg/ml, p < 0.001) and IL-18 levels th

161 ) values during calibration, and the highest NSE (0.83), KGE (0.88), R (0.91), and lowest RMSE (892)

162 y exacerbation of renovascular hypertension, NSE-AT(1a) and nontransgenic (NT) mice underwent 2-kidne

163 To test this hypothesis, NSE-tTA x TetOp-DeltaFosB mice with normal or overexpres

164 r high-flow events, as evidenced by improved NSE (from 0.83 to 0.97) and reduced RMSE (from 892 to 44

165 In NSE-apoE mice, the pattern of apoE fragmentation resembl

166 dia accompanied this early increase in BP in NSE-AT(1a) mice, as did a substantial cardiovascular reg

167 rtan caused significant falls in basal BP in NSE-AT1a mice but had no effect in nontransgenic control

168 ceptors to the maintenance of baseline BP in NSE-AT1a mice, particularly effective baroreflex bufferi

169 -old NSE-apoE4 mice but barely detectable in NSE-apoE3 mice.

170 We found that TL elongation was dominant in NSE, whereas TL shortening prevailed in SE.

171 ively correlated with telomere elongation in NSE than SE: three telomerase activity-related genes (TE

172 factors might induce telomere elongation in NSE.

173 them, SOX2 and MYC were highly expressed in NSE versus SE, while POU5F1 and KLF4 had the opposite pa

174 LEPR-B transgene expression in NSE-Rb db3J/db3J mice partially corrected the increased

175 -apoE4 mice and, to a much lesser extent, in NSE-apoE3 mice but not in GFAP-apoE3 or GFAP-apoE4 mice.

176 ignificantly increased apoE fragmentation in NSE-apoE4 but not NSE-apoE3 mice.

177 The decrease in cerebellar growth in NSE(Cre/+)/CII(F/F) mice is due to reduced proliferation

178 nd2(+/-) mice and significantly increased in NSE-noggin animals.

179 vely correlated with telomere lengthening in NSE.

180 stabilized at significantly lower levels in NSE-AT(1a) mice despite marked elevations in Ang II leve

181 mplexes assessed were significantly lower in NSE-apoE4 neurons compared with NSE-apoE3 neurons.

182 so accumulated in an age-dependent manner in NSE-apoE4 mice and, to a much lesser extent, in NSE-apoE

183 monal profile was only partially restored in NSE-HPrP/mPrP(0/0) mice and was similar to that of mPrP(

184 The NREM and SWA deficit was restored in NSE-HPrP/mPrP(0/0) mice but not in GFAP-HPrP/mPrP(0/0).

185 unctional overexpression of the transgene in NSE-AT1a mice.

186 The control vector, pTR-UF4, incorporated NSE promoter-driven GFP expression only.

187 d db/db mice compared with those in infected NSE-Rb db/db and FVB wild-type mice.

188 SSPE patients and our work with MV-infected NSE-CD46(+) mice, which express the measles receptor CD4

189 comprised markers of cellular brain injury (NSE, BDNF, GFAP, S100beta, MCP1, VILIP-1, neurogranin);

190 the full complement of short LEPR isoforms (NSE-Rb db/db mice) were similar to those of NSE-Rb db3J/

191 vioral analyses, we demonstrate that NRG1-IV/NSE-tTA mice exhibit abnormal behaviors relevant to schi

192 developed a transgenic mouse model (NRG1-IV/NSE-tTA) in which human NRG1-IV is selectively overexpre

193 (MD-8), the values RMSE (m(3)/s), R(2), MAE, NSE, KGE and MBE are 4.57, 0.98, 2.56, 0.98, 0.94 and 0.

194 ike morphology and expression of NE markers (NSE).

195 Median NSE values were 18 ng/ml versus 35 ng/ml, 15 ng/ml versu

196 eceptor was constructed and transgenic mice (NSE-AT1a) were generated.

197 In genetically modified db/db mice, (NSE-Rb db/db mice), central leptin signaling is reconsti

198 petition of adjacent pseudoexons to modulate NSE levels.

199 and multinucleated cells and TRAP-negative, NSE-positive mononuclear precursors were identified.

200 l activation (NT-proCNP), neurodegeneration (NSE, tau), neuroaxonal damage (NfL, NfH, UCHL-1) and gli

201 ic and central expression of PrP in neurons (NSE-HPrP/mPrP(0/0)) or in glia cells (GFAP-HPrP/mPrP(0/0

202 rs reflecting neurodegeneration (T-tau, NFL, NSE, VLP-1, and HFABP), APP metabolism (Abeta42, Abeta40

203 clinically as seminoma (SE) and nonseminoma (NSE), the latter including the pluripotent embryonal car

204 ansgenic mice that have greater than normal (NSE-noggin mice, which overexpress noggin under the cont

205 ased apoE fragmentation in NSE-apoE4 but not NSE-apoE3 mice.

206 mononuclear/giant cells, and the occasional NSE-positive precursor, expressed an intense signal for

207 s signal sensitivity up to 0.12 ng mL(-1) of NSE with excellent signal reproducibility and electrode

208 CD occurred in 19% of NSE patients on follow up.

209 The majority (49%) of NSE patients do not develop significant small bowel dise

210 3% (4/28), 16.0% (8/50) and 12.2% (12/98) of NSE, BE, low-grade dysplasia, HGD and EAC tissues, respe

211 The appearance of NSE immunoreactivity in the different classes of retinal

212 in an age-dependent manner, in the brains of NSE-apoE4 and, to a significantly lesser extent, NSE-apo

213 nt production of beta-s-APP in the brains of NSE-APP mice and enzyme-linked immunosorbent assay analy

214 expression was reduced in the cerebellum of NSE(Cre/+)/CII(F/F) mice, thereby leading to decreased A

215 Colocalization of NSE with SV2 indicated that cone cells began to make syn

216 hey also demonstrate that the combination of NSE with MD is potentially useful for determining the fo

217 The detection of NSE, relied on its obstruction in analytical signal obse

218 the NE-like morphology and the elevation of NSE in C-33 LNCaP cells.

219 Axonal extensions of NSE-labeled cone cells were shown to interact with those

220 Patients with a finding of NSE were identified from a database.

221 To define the natural history of NSE in an Irish cohort.

222 Levels of NSE and S100beta were higher in patients with poor outco

223 suggests that the decreased fat phenotype of NSE-DeltaFosB mice is independent of the changes in the

224 great extent and allowed the recognition of NSE in human serum in a concentration range of 25-4000 p

225 The recognition of NSE was achieved by the same imprints in a linear range

226 This study investigated the role of NSE as a prognostic marker of outcome after out-of-hospi

227 well as the alpha- and the gamma-subunit of NSE isoenzymes.

228 d by ELISA specific for the gamma subunit of NSE.

229 (NSE-Rb db/db mice) were similar to those of NSE-Rb db3J/db3J mice, suggesting that the short LEPR is

230 The body weights of NSE-Rb transgenic mice that possessed the full complemen

231 e hippocampus were prominent in 21-month-old NSE-apoE4 mice but barely detectable in NSE-apoE3 mice.

232 gustducin never colocalizes with PGP 9.5 or NSE.

233 Patients with an early CgA or NSE response had a longer PFS compared with patients wit

234 olume associations were observed for GFAP or NSE, and no treatment group differences were detected.

235 erapeutic hypothermia, use of motor score or NSE, as recommended in current guidelines, could possibl

236 = 14 (16%), NSAIDs = 12 (14%) and persistent NSE = 2 (2%).

237 (IBS), NSAIDs enteritis (NSAIDs), persistent NSE and no significant disease (NAD).

238 er between and individually quantify ProGRP, NSE, and their isoform and isoenzyme variants, respectiv

239 rol of the neuron-specific enolase promoter (NSE-PDGF-A) resulted in a striking hyperplasia of retina

240 control of neuron-specific enolase promoter (NSE::LacZ) from the SVZ of adult mice into the striatum

241 f angiotensin II (Ang II) type 1a receptors (NSE-AT(1a)) to unmask the role of the brain renin-angiot

242 After removing NSE templates, a molecularly imprinted sensor was succes

243 concentrations) and brain biomarkers (S100B, NSE, NFL, tau, UCH-L1, and GFAP) were consistently found

244 nd an increasing profile over time in S100B, NSE, and GFAP.

245 Biomarkers of brain injury (S100B, NSE, NFL, tau, UCH-L1, and GFAP) were also top clinical

246 Mean (SD) NSE levels on day 1 after cardiac arrest were significan

247 in subtypes, seminoma (SE) and non-seminoma (NSE), but their molecular distinctions remain largely un

248 mors (TGCT): seminoma (SE) and non-seminoma (NSE).

249 High, serial NSE values are strong predictors of poor outcome after O

250 expressed as seropositivity/seronegativity, NSE and IL-18 were measured with immunoassays.

251 Serum NSE concentrations in carriers (27.17 +/- 39.82 mug/L) w

252 Serum NSE levels are higher in LHON carriers compared with aff

253 Serum NSE levels were determined by ELISA specific for the gam

254 le carriers had significantly elevated serum NSE levels.

255 reactivity and 4) continuity, 5) peak serum NSE within 48 hours less than or equal to 41 ug/L, and 6

256 It may be significantly better than serum NSE, which is recommended in guidelines and currently us

257 Op-DeltaFosB founder lines but not in single NSE-tTA transgenic controls.

258 forms the asymmetric complex NSE x Mg2 x SO4/NSE x Mg x Cl, where "/" separates the dimer subunits.

259 P-TFII knockout mice using a tissue-specific NSE-Cre recombinase.

260 Using neutron spin-echo spectroscopy (NSE), normal mode analysis, and a statistical-mechanical

261 omplex using neutron spin echo spectroscopy (NSE), selective deuteration, and theoretical physics ana

262 Using Neutron Spin Echo Spectroscopy (NSE), we have investigated the undulation dynamics of Sy

263 Using neutron spin echo spectroscopy (NSE), we show salt-concentration-dependent excitation of

264 plication of neutron spin-echo spectroscopy (NSE).

265 g/mL, which is 4 orders magnitude lower than NSE cut-off value of small cell lung cancer.

266 We show that NSE spectroscopy can determine the domain mobility tenso

267 Results show that NSE-AT(1a) mice developed hypertension much more rapidly

268 The NSE activation was haplotype-specific and was most promo

269 The NSE index is calculated 0.99 and 0.94 for the Wilson and

270 The NSE results provide insights into the nature of protein

271 The NSE-immunoreactive photoreceptors were identified as con

272 The NSE-Rb transgene was expressed in the brain, with low le

273 Because the NSE promoter drives expression primarily in neurons, thi

274 similar to that previously reported for the NSE-LEPR-B transgene.

275 The synthetic peptide derived from the NSE was synthesized along with its cysteine and histidin

276 most promoted by cytosine at rs609621 in the NSE 3' splice-site (3'ss), which is predominant in high

277 k of mortality/MI (hazard ratio 0.93) in the NSE group at 180 days.

278 ratio 0.79 in the STE group and 0.81 in the NSE group) and with risk of mortality/MI (hazard ratio 0

279 In the NSE group, 8152 of 19 304 (42%) had abnormal RF.

280 spite this partial rescue, later in life the NSE-Fig4(Cys486Ser) transgenic mice display significant

281 LFS was successfully employed to measure the NSE level in clinical blood plasma samples taken from de

282 confirm that a significant component of the NSE arises from internal dynamics.

283 ents and theoretical physics analyses of the NSE data reveal that the dynamics of full-length alpha-c

284 cavities were used for the detection of the NSE derived peptide and NSE.

285 The late onset phenotype of the NSE-Fig4(Cys486Ser) transgenic mice demonstrates that th

286 dates as the mediators of the effects of the NSE-Rb transgene on energy homeostasis, ingestive behavi

287 te the internal motion's contribution to the NSE signal, with the largest being classic hinge bending

288 Among the NSEs, Nse5 and Nse6 form a stable subcomplex (Nse5-6) th

289 id cyclases in that it does not contain the "NSE/DTE" motif on helix H that specifically chelates Mg(

290 ith the surfactant in the trans form through NSE measurements of the Q-dependent effective diffusion

291 wed high selectivity and sensitivity towards NSE, produced a linear response in the concentration ran

292 -tTA, TetOp-DeltaFosB, and single-transgenic NSE-tTA control mice to determine the pattern of DeltaFo

293 interfacial and bulk trap passivation using NSE modifiers is a promising strategy to overcome outsta

294 king blood vessel endothelial cells, whereas NSE-IR apparently marks the layer of smooth muscle cells

295 ignificantly elevated S-100b levels, whereas NSE and tau were not significantly influenced.

296 F-IR is more prominent in the nucleus, while NSE-IR is more prominent in the cytoplasm and neurites.

297 rus 1 IgG status showed no associations with NSE or IL-18 which may suggest TOXO specificity.

298 Compared with NSE-apoE3 mice and wild-type controls, NSE-apoE4 mice sh

299 tly lower in NSE-apoE4 neurons compared with NSE-apoE3 neurons.

300 13 bird exclusion structure, consistent with NSE.

301 occurs in over one-half of young people with NSE and generally persists.