ライフサイエンスコーパス: NaHCO3

1 0.1% NaHCO3 was used as an extraction solvent for cyanide for

2 tudies with [35S]cysteine/methionine or [14C]NaHCO3, polysomal distribution analyses and ribonuclease

3 L of derivatizing reagent (0.2M NAC and 0.2M NaHCO3 in water) and reaction time of 1h at 50 degrees C

4 served in the presence of Na2SO4, CaCl2, and NaHCO3.

5 oughput screening (HTS) methodology, DMF and NaHCO3 were rapidly identified as the most effective sol

6 er in patients treated with combined NAC and NaHCO3 (18.7% versus 19.1% versus 9.2% versus 21.3%; P=0

7 nt with NAC, NaHCO3, or the combined NAC and NaHCO3 did not reduce the rate of CIN significantly comp

8 Combined treatment with NAC and NaHCO3 may reduce the risk of renal dysfunction after 30

9 We have developed NaH and NaHCO3 shuttle base systems as the easiest and most cost

10 In aqueous NaHCO3 solutions, maximum Faradaic efficiencies for form

11 ntly accelerate the decomposition of aqueous NaHCO3, essentially CO2 desorption - the key step of Na2

12 Administering the same sodium intake as NaHCO3 instead of NaCl did not significantly alter BP, i

13 d storage as a bicarbonate, predominantly as NaHCO3.

14 cetylcysteine (NAC), (2) sodium bicarbonate (NaHCO3) infusion, (3) NAC in combination with NaHCO3, or

15 group II also received a sodium bicarbonate (NaHCO3) infusion, thus serving as their own control for

16 N-acetylcysteine (NAC), sodium bicarbonate (NaHCO3), NAC+NaHCO3, ascorbic acid, xanthine, dopaminerg

17 on protein tyrosine phosphorylation was both NaHCO3 and protein kinase A-dependent.

18 extraction was done with MeCN:citrate buffer:NaHCO3 followed by phase separation with the addition of

19 Attenuating brain acidosis by NaHCO3 or blocking ASIC1a with PcTX were both protective

20 he resultant 3-amino-4-arylchroman-2-ones by NaHCO3 in a mixture of THF and water gave alpha-(N-benzo

21 With promotion by NaHCO3 and (BnO)2PO2H or (PhO)2PO2H, symmetric benzamide

22 nd this response was partially suppressed by NaHCO3.

23 eral important factors on TiO(OH)2-catalyzed NaHCO3 decomposition were investigated.

24 5% to 3 or 9% while maintaining a constant [NaHCO3].

25 molar (7.2 mmol/220 g body wt per d for 7 d) NaHCO3 or NaCl.

26 lowing aldosterone administration or dietary NaHCO3 loading.

27 l of NHE3 protein did not change with either NaHCO3 or high NaCl intake.

28 ance was increased significantly in rats fed NaHCO3 (7.2 mmol/220 g body wt per d) for 7 d.

29 dosis, and sham-operated, control rats given NaHCO3.

30 In vivo, 200 mmol/L NaHCO3 added to the drinking water of 4-week-old TRAMP m

31 e quantity of CO2 generated from 0.238 mol/L NaHCO3 at 65 degrees C with catalyst is 800% of that gen

32 2.2 +/- 0.5 nm during electrolysis in 0.10 M NaHCO3 at -0.80 V (vs RHE).

33 iated glucose uptake regulates NHE3-mediated NaHCO3 reabsorption in the renal proximal tubule.

34 tion rate constant (kexp) found in both 1 mM NaHCO3 and 1 mM phosphate-buffered solutions suggested t

35 onate species (4.17 muM) in a sample of 1 mM NaHCO3.

36 med well at lower ionic strength (IS) (10 mM NaHCO3), and one of the rhamnolipid surface modifiers (J

37 uld be reactivated by the addition of 100 mM NaHCO3.

38 ld not be reversed by the addition of 100 mM NaHCO3.

39 Nonetheless, at the highest IS (300 mM NaHCO3) investigated, the mobility of rhamnolipid-coated

40 ally CO2 desorption - the key step of Na2CO3/NaHCO3 based CO2 capture technologies from overall CO2 e

41 eine (NAC), sodium bicarbonate (NaHCO3), NAC+NaHCO3, ascorbic acid, xanthine, dopaminergic agent, per

42 Although xanthine, NAC, NaHCO3, NAC+NaHCO3, ischemic preconditioning, and natriuretic peptid

43 The prevention treatment with NAC, NaHCO3, or the combined NAC and NaHCO3 did not reduce th

44 Although xanthine, NAC, NaHCO3, NAC+NaHCO3, ischemic preconditioning, and natriu

45 ced by various stresses such as CdCl2, NaCl, NaHCO3, and H2O2 treatments.

46 Coprecipitation of nahcolite (NaHCO3) and halite (NaCl) from surface waters in contact

47 d to the aqueous phase, specifically NaHSO3, NaHCO3, Na2SO3, Na2CO3, Na2SO4, and NaHSO4.

48 ed elevated HCO3 (marker of proximal nephron NaHCO3 retention) by specifically counteracting this com

49 d products by simply adjusting the amount of NaHCO3 applied.

50 emonstrate that increasing concentrations of NaHCO3 in the system lead to slower U(VI) reduction kine

51 anella oneidensis strain MR-1 as function of NaHCO3 concentration.

52 ous medium (MeCN/H2O 2/1) in the presence of NaHCO3, NaClO4, and an electron transfer agent (biphenyl

53 e PCO2 was changed by adjusting the ratio of NaHCO3 and NaCl in the medium (or by using HEPES buffer

54 is study also indicates that a supplement of NaHCO3 at high concentration could significantly improve

55 , arrest rhythm not asystole, no atropine or NaHCO3, fewer epinephrine doses, shorter duration of car

56 Treatment with NAC or NaHCO3 did not reduce the rate of acute CIN significantl

57 ing ARF rats given either NaCl (ARF-NaCl) or NaHCO3 (ARF-HCO3) to prevent acidosis, and sham-operated

58 d during changes to superfusate PCO2 and/or [NaHCO3].

59 The NaHCO3 concentration also strongly affects the speciatio

60 orbed onto the bacteria as a function of the NaHCO3 concentration in the experimental systems.

61 hanges to pHi and [Ca2+]i, while changes to [NaHCO3] altered only extracellular pH (pHo).

62 SV, 89.0 to 108.4), and did not change with NaHCO3 infusion in group II.

63 aHCO3) infusion, (3) NAC in combination with NaHCO3, or (4) hydration with sodium chloride infusion a

64 ; mean +/- SEM), but this was corrected with NaHCO3.

65 4 degrees C in tick cell culture medium with NaHCO3 and an organic buffer [3-(N-morpholino)-propanesu

66 The resulting solution was neutralized with NaHCO3, and the (63)Zn was then trapped on a carboxymeth

67 Acidemia was prevented in CRF rats with NaHCO3.