1 he risk of bilirubin encephalopathy (Crigler-Najjar syndrome).

2                                  For Crigler-Najjar syndrome, an inherited disorder that results in f

3  both alleles, whereas patients with Crigler-Najjar syndrome are homozygous for a defect that yields

4 lirubinemia in the Gunn rat model of Crigler-Najjar syndrome can be achieved with one injection of HD

5                                      Crigler-Najjar syndrome (CN) is a very rare genetic disorder cha

6                                      Crigler-Najjar syndrome is a recessively inherited disorder char

7                    Patients with the Crigler-Najjar syndrome lack the enzyme uridine diphosphoglucuro

8                      The Gunn rat, a Crigler-Najjar syndrome model animal lacking UDP-glucuronosyltra

9 encoding UGT1A1 in patients with the Crigler-Najjar syndrome that was being treated with phototherapy

10                                      Crigler-Najjar syndrome type 1 (CN-1) is a recessively inherited

11 c bilirubin glucuronidation, causing Crigler-Najjar syndrome type 1 or 2, respectively.

12 perbilirubinemic Gunn rats (model of Crigler-Najjar syndrome type 1) resulted in hUGT1A1 expression i

13                                      Crigler-Najjar syndrome type I is characterized by unconjugated

14  report describes a young adult with Crigler-Najjar syndrome type II in whom kernicterus developed af

15 transplantation in the management of Crigler-Najjar syndrome, type 1.

16  rat is an excellent animal model of Crigler-Najjar syndrome, type 1.

17                         In contrast, Crigler-Najjar syndrome types I and II are rare genetic disorder