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1 The mucosa is colonized with commensal Neisseria.
2 hesis (FabI) as a therapeutic target against Neisseria.
3 l tract incurs a fitness cost for pathogenic Neisseria.
4 studies in the characterization of the genus Neisseria.
5 mmensal-derived DNA by pathogenic strains of Neisseria.
7 nhibition of fatty acid synthesis in growing Neisseria, a delayed inhibition of growth phenotype, and
9 B OMV component plus 3 recombinant antigens (Neisseria adhesin A, factor H binding protein [fHbp]-GNA
10 ][G-4], Pseudoramibacter, Streptococcus, and Neisseria and fewer in Actinomyces, Selenomonas, Veillon
12 found that depletion of the commensal genus Neisseria and the species Streptococcus pneumoniae was a
13 rns of diversity for the genera Escherichia, Neisseria, and Borrelia are generally indistinguishable
17 those dominated by Haemophilus, Moraxella or Neisseria associated with enhanced pathogenesis in vitro
18 tered bacterial abundance profile, with more Neisseria, Bacteroides, and Rothia species and less Sphi
20 a chemical biology tool to determine whether Neisseria can bypass the inhibition of fatty acid synthe
21 Here, we show that the commensal species Neisseria cinerea expresses functional fHbp on its surfa
25 Consistent with these findings, commensal Neisseria elongata accelerates Ngo clearance from the mo
27 fatty acid labeling experiments showed that Neisseria encodes the ability to incorporate exogenous f
28 Rothia mucilaginosa trending to increase and Neisseria flavescens (P < 0.01) increased after nitrate
29 uely enriched in members of the Haemophilus, Neisseria, Fusobacterium, and Porphyromonas species and
30 in the upper GI tract (Gemella, Veillonella, Neisseria, Fusobacterium, Streptococcus, Prevotella, Pse
32 itulated in EPs from the divergent pathogens Neisseria gonorrheae and Escherichia coli Our results su
33 -negative species Neisseria meningitidis and Neisseria gonorrheae and improved activity against the G
34 y loss of capsule and gene conversion of the Neisseria gonorrheae norB-aniA cassette promoting anaero
35 With the examples of Escherichia coli and Neisseria gonorrheae, we present an electrochemical bios
36 -of-care tests for Chlamydia trachomatis and Neisseria gonorrhoea (nucleic acid amplification tests).
38 a rates are rising, and antibiotic-resistant Neisseria gonorrhoeae (AR-Ng) is an urgent public health
39 men who have receptive anal intercourse for Neisseria gonorrhoeae (GC) and Chlamydia trachomatis (CT
40 rectal Chlamydia trachomatis (chlamydia) and Neisseria gonorrhoeae (gonorrhea) infections in women.
42 accurately identify those with extragenital Neisseria gonorrhoeae (NG) and Chlamydia trachomatis (CT
44 lines recommend the systematic screening for Neisseria gonorrhoeae (NG) and Chlamydia trachomatis (CT
46 antibiotic susceptibility testing (AST) for Neisseria gonorrhoeae (Ng) is critically needed to count
47 ous detection of Chlamydia trachomatis (CT), Neisseria gonorrhoeae (NG), and an internal control in t
48 h a network-based mathematical model of HIV, Neisseria gonorrhoeae (NG), and Chlamydia trachomatis (C
50 cillus crispatus, Gardnerella vaginalis, and Neisseria gonorrhoeae All vaginal microbiota and N. gono
53 irth outcome (OR, 0.24; 95% 0.09, 0.66), and Neisseria gonorrhoeae and/or Chlamydia trachomatis had 9
54 pic pregnancy, and Chlamydia trachomatis and Neisseria gonorrhoeae are recognized microbial causes.
58 Microbe, Muenzner and Hauck (2020) find that Neisseria gonorrhoeae blocks exfoliation by producing ni
65 we performed a meta-analysis of seven large Neisseria gonorrhoeae datasets, as well as Klebsiella pn
67 if Nanopore sequencing can detect sufficient Neisseria gonorrhoeae DNA to reconstruct whole genomes d
69 in female mice to study mechanisms by which Neisseria gonorrhoeae evades host-derived antimicrobial
71 ased potency and showed its efficacy against Neisseria gonorrhoeae in a mouse vaginal infection model
72 -sensitive and cefixime-resistant strains of Neisseria gonorrhoeae in MSM in England, which was appli
73 n is associated with bacterial burden during Neisseria gonorrhoeae infection and alters the infection
74 point-of-care Gram stain testing to diagnose Neisseria gonorrhoeae infection and nongonococcal urethr
75 he main consequences of sexually transmitted Neisseria gonorrhoeae infection and probably involve an
79 acid amplification tests (NAATs) to diagnose Neisseria gonorrhoeae infections complicates the perform
82 , understanding the evolutionary dynamics of Neisseria gonorrhoeae is a significant public and global
88 chanisms of colonization and pathogenesis of Neisseria gonorrhoeae is required to aid development of
90 Rising azithromycin nonsusceptibility among Neisseria gonorrhoeae isolates threatens current treatme
94 rmed by laboratory isolation or detection of Neisseria gonorrhoeae only from a clinical specimen, and
95 ontal gene transfer between bacteria, and in Neisseria gonorrhoeae plasmids can mediate high-level an
97 BD ProbeTec Chlamydia trachomatis Q(x) (CTQ)/Neisseria gonorrhoeae Q(x) (GCQ), Hologic Aptima Combo 2
98 on MIC QC range for zoliflodacin against the Neisseria gonorrhoeae QC strain ATCC 49226 was defined a
103 Previous studies have demonstrated that Neisseria gonorrhoeae sialylates the terminal N-acetylla
104 etect Chlamydia trachomatis AC2 also detects Neisseria gonorrhoeae Storage and temperature conditions
106 hea is expected to rise due to the spread of Neisseria gonorrhoeae strains with decreased susceptibil
108 e A (gyrA) genotypic assay for prediction of Neisseria gonorrhoeae susceptibility to ciprofloxacin.
109 5 different antibiotics in 1,102 isolates of Neisseria gonorrhoeae that were confirmed in a second da
110 against multidrug-resistant bacteria such as Neisseria gonorrhoeae The first structure of BamA, the c
111 reasing azithromycin usage and resistance in Neisseria gonorrhoeae threatens current dual treatment.
112 fferent populations in the susceptibility of Neisseria gonorrhoeae to antimicrobials, and the reasons
114 rapid AST platform using RNA signatures for Neisseria gonorrhoeae Transcriptome sequencing (RNA-seq)
115 I (HepI) of the lipooligosaccharide (LOS) of Neisseria gonorrhoeae undergoes positive selection durin
117 on antimicrobial resistance determinants in Neisseria gonorrhoeae was developed and is publicly acce
118 r the detection of Chlamydia trachomatis and Neisseria gonorrhoeae was established in a multisite, pr
119 he force-dependent velocity of DNA uptake by Neisseria gonorrhoeae We found that the DNA uptake veloc
120 Independent risk factors for oropharyngeal Neisseria gonorrhoeae were assessed among MSM routinely
122 resistance and diagnostic escape variants in Neisseria gonorrhoeae, a pathogen associated with a high
123 on effect on incident Chlamydia trachomatis, Neisseria gonorrhoeae, and Mycoplasma genitalium infecti
124 fluoroquinolone-resistant Campylobacter spp, Neisseria gonorrhoeae, and Salmonella typhi were include
125 Positivity rates for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis DNA, de
126 e urine specimens for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis via com
127 d method to detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no commercial tests are clear
128 has been used to investigate transmission of Neisseria gonorrhoeae, but to date, most studies have no
129 ria species two, Neisseria meningitidis, and Neisseria gonorrhoeae, cause invasive disease: the other
131 mophilus influenzae, Neisseria meningitidis, Neisseria gonorrhoeae, Helicobacter pylori, Moraxella ca
133 ng the concurrence of Chlamydia trachomatis, Neisseria gonorrhoeae, Mycoplasma genitalium, and Tricho
135 also more prevalent among HIV-infected MSM (Neisseria gonorrhoeae, P = .03; Mycoplasma genitalium, P
136 thogenic species, Neisseria meningitidis and Neisseria gonorrhoeae, straddle the border between comme
137 , Mycoplasma genitalium, Mycoplasma hominis, Neisseria gonorrhoeae, Streptococcus agalactiae, Chlamyd
141 nce to azithromycin complicates treatment of Neisseria gonorrhoeae, the etiologic agent of gonorrhea.
142 parallel those of Chlamydia trachomatis and Neisseria gonorrhoeae, the mechanisms by which this path
144 Salmonella enterica, Haemophilus ducreyi and Neisseria gonorrhoeae, together with BamA's homolog, Tam
145 ed for M. genitalium, Chlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis, Mycoplasma
159 a was dominated by Haemophilus, Moraxella or Neisseria (HMN) were at 1.5 times higher risk of CLD tha
164 Here, we report that commensal species of Neisseria kill Ngo through a mechanism based on genetic
165 ge for Neisseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.002) (2-sided Fisher's exact
166 observed in four individuals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT e
167 did observe an influx of oral taxa, such as Neisseria lactamica, Streptococcus, Prevotella nanceiens
169 FA) class II-IV disease, vaccination against Neisseria meningitides, and previous treatment with at l
172 ed cases of meningitis, 5590 were confirmed: Neisseria meningitidis ([Nm] 85%), Streptococcus pneumon
173 as oropharyngeal carriage of disease-causing Neisseria meningitidis (group A, B, C, W, X, or Y) in st
175 (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus) was performed by
176 Streptococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus
177 ing Streptococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus
178 of Streptococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus
188 le dynamic regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7
190 of human umbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum
192 eficiency and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the tw
193 t activity against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and impr
194 pathology, while the two pathogenic species, Neisseria meningitidis and Neisseria gonorrhoeae, stradd
199 dentified as a ligand for these molecules on Neisseria meningitidis As with N. meningitidis NspA (Nm-
200 es of the substrate-bound ClpXP complex from Neisseria meningitidis at 2.3 to 3.3 angstrom resolution
203 , and AcrIIC3 proteins were found to inhibit Neisseria meningitidis Cas9 (NmeCas9) activity in bacter
206 e (SPR) based biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configu
208 ningococcal disease (IMD) due to serogroup Y Neisseria meningitidis emerged in Europe during the 2000
209 antibodies raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) a
210 s 3 main recombinant proteins, including the Neisseria meningitidis factor H binding protein (fHbp),
211 ta that the class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, the
212 H binding protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the
213 specific protection against capsular group B Neisseria meningitidis infections, but the full breadth
223 duals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acq
225 he availability of antibiotics and vaccines, Neisseria meningitidis remains a major cause of meningit
226 ing in 2010, the burden of meningitis due to Neisseria meningitidis serogroup A (NmA) has substantial
228 ecially in the African meningitis belt where Neisseria meningitidis serogroup A historically caused l
229 the countries of the meningitis belt, where Neisseria meningitidis serogroup A historically caused l
231 rrelate of protection against infection with Neisseria meningitidis serogroup A, we use an assumed SB
234 Acetylated sialic acid has been found in the Neisseria meningitidis serogroup W (NmW) capsular polysa
236 he United Kingdom due to a sublineage of the Neisseria meningitidis serogroup W ST-11 clonal complex
237 des of recombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA)
239 ve meningococcal disease is mainly caused by Neisseria meningitidis serogroups A, B, C, X, W, and Y.
240 age prevention against antigenically diverse Neisseria meningitidis strains and to compare this prote
241 erized a TE6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray cr
243 tor H binding protein (FHbp) is an important Neisseria meningitidis virulence factor that binds a neg
244 NmLgtB-B beta1-4 galactosyltransferase from Neisseria meningitidis we demonstrate fast and robust ac
245 egative pathogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation o
248 ry-confirmed Streptococcus pneumoniae (Spn), Neisseria meningitidis, and Haemophilus influenzae menin
249 igen) and qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.
250 ten human-restricted Neisseria species two, Neisseria meningitidis, and Neisseria gonorrhoeae, cause
251 h intact, heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or G
252 lipid A phosphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.
253 eillance targeted meningitis cases caused by Neisseria meningitidis, Haemophilus influenzae, and Stre
254 terial meningitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Stre
255 tococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacte
256 ajor human pathogens Haemophilus influenzae, Neisseria meningitidis, Neisseria gonorrhoeae, Helicobac
257 rs of bacterial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Esche
258 uman pathogens, Streptococcus pneumoniae and Neisseria meningitidis, revealed both previously identif
260 mophilus influenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Strept
261 ccus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis, the 3 most common bacteria causi
263 Gram-negative bacterial pathogens including Neisseria meningitidis, Vibrio cholerae, and Salmonella
265 ere we expressed the gene encoding LpxA from Neisseria meningitidis, which specifically attaches 3OH-
266 ed Cas9s to identify a compact ortholog from Neisseria meningitidis-Nme2Cas9-that recognizes a simple
272 dent responses except that made with group C Neisseria meningitidis; in the latter case, only peptide
275 pecies (N. meningitidis; N. gonorrhoeae; and Neisseria polysaccharea) and genomes of isolates unassig
278 le genomes has indicated that some commensal Neisseria species also contain genes that potentially en
279 ial susceptibility in NG, we categorized the Neisseria species and compared mean MIC levels between d
284 lore this question, we focused on pathogenic Neisseria species harboring a genomic island in their di
285 the past month was strongly associated with Neisseria species having increased MICs to cefixime, cef
290 f the LT, an outer membrane lipoprotein from Neisseria species with a disordered active site helix (a
292 We collected pharyngeal specimens, cultured Neisseria species, and measured minimum inhibitory conce
293 12 taxa associated with gum health including Neisseria spp. and a significant decrease in 10 taxa ass
294 ken (n = 112) to ascertain the prevalence of Neisseria spp. following the eighth case of invasive men
295 at provide insight into the evolution of the Neisseria, the epidemiology of meningococcal and gonococ
296 noculum in which Streptococcus, Haemophilus, Neisseria, Veillonella and Prevotella species predominat
298 252 remained an effective antibacterial when Neisseria were supplemented with exogenous fatty acids.
299 bacter, Corynebacterium, Actinobacteria, and Neisseria were the signature taxa of C environment-assoc