ライフサイエンスコーパス: Newman

1 Newman in 1950, as deduced then from interpretation of e

2 Newman-Keuls post hoc comparisons indicated variation in

3 e doxorubicin and LTSL respectively, p<0.05, Newman-Keuls).

4 Consideration of a Newman plot of dihedral angles for proteinaceous tryptop

5 alpha-toxin and PSM-positive phenotypes of a Newman sarA mutant.

6 mpared, using an analysis of variance with a Newman-Keul's post-test.

7 (LL3-1, 8325-4, and SH1003) or SigB active (Newman and SH1000).

8 revealed that strains Newman Delta(hlg) and Newman Delta(hla), although virulent, caused significant

9 s of networks (Volz et al., 2011; Karrer and Newman, 2010), e.g. networks composed of lines and non-o

10 nalysis demonstrated that strains Newman and Newman Delta(hlg), but not Newman Delta(hla), produced a

11 cued by a plasmid encoding gamma-toxin), and Newman Delta(hla) (alpha-toxin-deficient) were intrastro

12 er than those caused by wild-type USA300 and Newman strains.

13 We measured the adhesion of S aureus Newman and a vWbp-deficient mutant (vwb) to VWF, collage

14 and was exposed on the surface of S. aureus Newman and RN4220.

15 S. aureus Newman cells expressing ClfA adhered to microtitre wells

16 ere that under selective pressure, S. aureus Newman generates proline-prototrophic variants at a freq

17 S. aureus Newman lacking all four prophages was unable to cause di

18 s observed in mice challenged with S. aureus Newman strain, makes the 5-combo-OMVs a promising vaccin

19 S. aureus Newman variants with mutations in rocF (arginase), rocD

20 /6J and BALB/c mice, >10(9) CFU of S. aureus Newman were needed to produce a lethal outcome in 90% of

21 lyze the transcription profiles of S. aureus Newman, its isogeneic mgrA mutant, and an MgrA-overprodu

22 transducible into both ISP479 and S. aureus Newman.

23 -iron-induced biofilm formation of S. aureus Newman.

24 olysaccharide (CP5) of Staphylococcus aureus Newman is covalently attached to the glycan strands of p

25 ified in the genome of Staphylococcus aureus Newman, a human clinical isolate.

26 ANOVA followed by Newman-Keuls post-hoc analyses were used to test for dif

27 tion algorithms (k-means clustering, Clauset Newman Moore, and Density-Based Spatial Clustering of Ap

28 . aureus, including isogenic ClfA+ and ClfA- Newman strains, in the presence of purified rabbit fibri

29 In a recent issue of Molecular Cell, Enquist-Newman et al. demonstrate that Acm1 is ubiquitinated by

30 ic and SLEs revealed that all strains except Newman Delta(hla) produced corneal erosions.

31 Finally, via Doyle-Fuller-Newman (DFN) simulations, we investigate the K-ion and L

32 Finally, we present a Doyle-Fuller-Newman model of a KIB full cell with realistic geometry

33 greater explanatory power than the Ginsburg-Newman model.

34 The Ginsburg-Newman three-compartment model explains the offset in te

35 Girvan-Newman algorithm-based community maps of the kinase doma

36 relative edge order generated by the Girvan-Newman (G-N) algorithm.

37 We apply the Girvan-Newman clustering algorithm to these networks to reveal

38 pt of mutual information and used the Girvan-Newman method to partition PKA into structurally contigu

39 as performed with ANOVA followed by post hoc Newman-Keuls multiple comparison test.

40 ClpC, leading to capsule activation both in Newman and in UAMS-1.

41 This contrasts with MSSA strains, including Newman, SH1000, RN6390, and 8325-4, where autolysis is a

42 The Japanese beetle, Popillia japonica Newman, an introduced scarab, has become the most widesp

43 The Miyazaki-Newman-Kwart rearrangement of (dimethylcarbamothioyl)oxy

44 or PNAG production in S. aureus strains Mn8, Newman, and NCTC 10833 resulted in mutant strains with s

45 In this issue of Neuron, Newman et al. (2017) image calcium events at single syna

46 trains Newman and Newman Delta(hlg), but not Newman Delta(hla), produced alpha-toxin.

47 nalyzed by comparing CYL11481 (derivative of Newman) and its isogenic regulatory mutants in vitro.

48 gr loci of three laboratory strains (RN6390, Newman, and S6C) and four clinical isolates (UAMS-1, UAM

49 The gene NWMN2274 in S. aureus strain Newman is annotated as an oxidoreductase of the diverse

50 Mice challenged with S. aureus strain Newman or its isogenic DeltaccpE derivative revealed inc

51 ically, deletion of ccpE in S. aureus strain Newman revealed that CcpE affects transcription of virul

52 Inactivation of ccpE in S. aureus strain Newman revealed that CcpE is a positive transcriptional

53 stem, we defined a role for S. aureus strain Newman surface proteins and secreted exotoxins in pneumo

54 ve screened a collection of S. aureus strain Newman transposon mutants for lysostaphin resistance.

55 S. aureus strain Newman variants lacking the capacity for synthesizing po

56 copper resistance operon in S. aureus strain Newman.

57 ubsequently challenged with S. aureus strain Newman.

58 cific regulatory mutants of S. aureus strain Newman.

59 d by cloning the cap5P homologue from strain Newman and complementing an Escherichia coli rffE mutant

60 ite pathways in capsule regulation in strain Newman but functions as a positive activator in strain U

61 , we screened a transposon library in strain Newman expressing the transcriptional fusion norA-lacZ f

62 on in a transmembrane domain, SaeS in strain Newman has a constitutive kinase activity.

63 th points, and that the Ess system in strain Newman is inactive under these conditions.

64 body infection, the rbf mutations in strain Newman, but not in strain UAMS-1, reduced the bacterial

65 clumping factor (encoded by clfA) in strain Newman.

66 t are potential substrates of ClpC in strain Newman.

67 ction by activating the sae operon in strain Newman.

68 mutations of clfA, coa, and agr into strain Newman to obtain single, double, and triple mutants of t

69 phenotype in the arlRS mutant of MSSA strain Newman could be rescued by a mutation in either atl or l

70 the arlRS mutant of MW2 than the MSSA strain Newman.

71 Our microarray analyses of strain Newman also revealed that CodY, a repressor of capsule p

72 ults illustrate that the virulence of strain Newman involves both alpha- and gamma-toxin, with alpha-

73 By microarray analyses of strain Newman, we found that ClpC strongly activates transcript

74 ial load compared to wild-type parent strain Newman.

75 SM phenotypes of the commonly studied strain Newman, which is known to have a mutation in saeS that r

76 In the strain Newman and some other S. aureus strains, the sensor hist

77 n compared to those for the wild-type strain Newman, suggesting that the genes are involved in DNA re

78 observation to other strains, we used strain Newman, a weak biofilm producer, and strain UAMS-1, an o

79 pithelial erosions when infected with strain Newman.

80 S. aureus strains Newman (expressing gamma-toxin), Newman Delta(hlg) (defi

81 Staphylococcus aureus, for example, strains Newman and N315, cannot grow in the absence of proline,

82 ene accelerated biofilm formation in strains Newman and UAMS-1.

83 tern blot analysis demonstrated that strains Newman and Newman Delta(hlg), but not Newman Delta(hla),

84 hours after infection revealed that strains Newman Delta(hlg) and Newman Delta(hla), although virule

85 In the strains Newman and USA300, the abundance of 33 proteins were alt

86 way analysis of variance (ANOVA) and Student Newman Keuls's post hoc test at alpha = 0.05 significanc

87 025% or 0.01% latanoprost (P < 0.05, Student-Newman-Keuls test) and the largest decrease (14% +/- 8%)

88 n serum vs. plasma samples (p > .05; Student-Newman-Keuls' test).

89 ving normal saline (p > .05; n = 12; Student-Newman-Keuls' multiple comparison test) or those subject

90 infused with D5RL (p > .05; n = 12; Student-Newman-Keuls' multiple comparison test).

91 or lithium heparin (p > .05; n = 19; Student-Newman-Keuls' multiple comparisons test).

92 er anticoagulants (p < .001; n = 19; Student-Newman-Keuls' multiple comparisons).

93 the same specimen (p < .05; n = 42; Student-Newman-Keuls' multiple comparisons test).

94 antly lower tooth deformation (ANOVA/Student-Newman-Keuls post hoc, p = 0.05).

95 The Student-Newman-Keuls test has been applied to ascertain possible

96 and statistically analyzed with the Student-Newman-Keuls test.

97 icipants were recruited from two clinics (TC Newman and Mbekweni).

98 In every strain other than Newman, including RN6390, simultaneous mutation of sarA

99 The Newman Kwart Rearrangement (NKR) offers an efficient and

100 The Newman projection of the E(3) conformer indicated that n

101 The Newman-Kwart rearrangement is perhaps the quintessential

102 was also increased in abundance, causing the Newman-specific cell-aggregation phenotype.

103 Conditions for the Newman-Kwart rearrangement of phenols into thiophenols w

104 points along the reaction coordinate of the Newman Kwart rearrangement, which transforms an O-arylth

105 The kinetic profile of the Newman-Kwart rearrangement has been evaluated using micr

106 Overall, this re-evaluation of the Newman-Kwart rearrangement has shown the reaction rate o

107 In this context, we have studied the Newman-Kwart rearrangement of O-arylthiocarbamates to th

108 We apply our approach to the Newman-Girvan modularity, widely used for "community" de

109 pared for both sets of patients by using the Newman-Keuls pairwise multiple sample comparison test.

110 eus strains Newman (expressing gamma-toxin), Newman Delta(hlg) (deficient in gamma-toxin), Newman Del

111 ewman Delta(hlg) (deficient in gamma-toxin), Newman Delta(hlg)/pCU1 hlg(+) (chromosomal gamma-toxin-d

112 performs better than the most commonly used Newman-Girvan (NG) modularity in detecting the best (phy

113 ed by the primary current distribution using Newman's formalism.

114 It was recently reported that the venerable Newman-Kwart rearrangement (1-->2) proceeds via mixed fi

115 The exceptions were Newman, which formed a poor biofilm under all conditions

116 aluated using a repeated measures ANOVA with Newman-Keuls multiple comparisons.