ライフサイエンスコーパス: O3

1 O3 application promoted the removal of more than 80% of

2 O3 exposure and lack of SP-D reduced NK cell IFN-gamma a

3 O3 formation from NO oxidation is several times more eff

4 O3 increases occurred across larger areas for the season

5 a mixed ionic-electronic SrCe0.7 Zr0.2 Eu0.1 O3-delta thin film ( approximately 20 mum) supported on

6 e)-bearing bridgmanite (Mg0.9Fe0.1Si0.9Al0.1)O3 measured using high-pressure Brillouin spectroscopy a

7 sotopologue interference corrections on W(16)O3(-) species.

8 2(-), W(17)O2(16)O(-), W(18)O(16)O2(-), W(17)O3(-), W(17)O(18)O(16)O(-), and W(18)O2(16)O(-) isotopol

9 tability compared to Ba0.5 Sr0.5 Co0.8 Fe0.2 O3- delta (BSCF), superior to many well-developed bulk/n

10 um-doped Pr0.5 (Ba0.5 Sr0.5 )0.5 Co0.8 Fe0.2 O3- delta (Pr0.5BSCF) exhibits dramatically enhanced HER

11 ace of a one-end capped porous SrCe0.8 Zr0.2 O3-delta tube.

12 f energy harvested from (1-y-x)Pb(In1/2Nb1/2)O3-(y)Pb(Mg1/3Nb2/3)O3-(x)PbTiO3 (PIN-PMN-PT) crystals u

13 We derived time series of 2006-2008 O3 concentrations consistent with 50% and 75% NOx emissi

14 rom (1-y-x)Pb(In1/2Nb1/2)O3-(y)Pb(Mg1/3Nb2/3)O3-(x)PbTiO3 (PIN-PMN-PT) crystals under high strain rat

15 mulations of the prototypical Pb(Mg1/3,Nb2/3)O3-PbTiO3 relaxor material to examine its structure and

16 res grown on LSAT [(La0.3Sr0.7)(Al0.65Ta0.35)O3].

17 vely, on perovskites La0.6 Sr0.4 Co0.2 Fe0.8 O3-delta and (La0.8 Sr0.2 )0.95 MnO3+/-delta , using gas

18 appear in ultrathin epitaxial PbZr0.2 Ti0.8 O3 /SrTiO3 /PbZr0.2 Ti0.8 O3 ferroelectric sandwich stru

19 xial PbZr0.2 Ti0.8 O3 /SrTiO3 /PbZr0.2 Ti0.8 O3 ferroelectric sandwich structures due to the interpla

20 near room temperature in (1-x)Ba(Zr0.2Ti0.8)O3-x(Ba0.7Ca0.3)TiO3 compounds has directed attention to

21 ct in epitaxial La0.7Sr0.3MnO3/Pb(Zr0.2Ti0.8)O3/La0.7Sr0.3MnO3 heterostructures have been performed u

22 The lead-free Ba(Zr0.2,Ti0.8)O3 films also show excellent dielectric and energy stora

23 on is easily saturated, these Ba(Zr0.2,Ti0.8)O3 films display a much delayed saturation of the electr

24 sed on ferroelectric films of Ba(Zr0.2,Ti0.8)O3 which display high-energy densities (up to 166 J cm(-

25 Two Pb(Zr0.20Ti0.80)O3 samples of different thickness and domain configurati

26 y similar between the two wastewaters across O3/BAC conditions.

27 To model acute O3 exposure, female Balb/c mice were exposed to 3 ppm O3

28 ly KC/CXCL1 was increased in blood 6 h after O3 exposure.

29 e proteins in the circulation increase after O3 exposure and systemically convey signals of O3 exposu

30 Al2 O3 capping is then introduced to improve the carrier mob

31 h dielectric substrates such as SiO2 and Al2 O3 , or in powder samples by mixing with the strongly di

32 of inorganic electrides, such as 12 CaO7 Al2 O3 :e(-) and Ca2 N.

33 as a simply loaded Pd cube (6 nm)-CeO2 /Al2 O3 sample were used as catalysts to investigate the effe

34 f catalytic NO reduction by CO, Pd@CeO2 /Al2 O3 samples based on Pd cubes (6, 10, and 18 nm), Pd octa

35 ctures can be composited well with gamma-Al2 O3 to create a heterogeneous catalyst.

36 By introducing Al2 O3 capping, carrier field effect mobilities (41 for hole

37 The selective growth of Al2 O3 islands over defect sites on the surface of carbon na

38 d electrical properties of ZrO2 films on Al2 O3 and InGaAs substrate at the atomic scale.

39 ssisted methane (CH4 ) oxidation over Pd/Al2 O3 was investigated by direct monitoring of the X-ray ab

40 r of 0.62 mW/(m K(2) ) at 684 K on rigid Al2 O3 substrate and 0.46 mW/(m K(2) ) at 664 K on flexible

41 hene framework and the nanolayers of the Al2 O3 ceramic (NAC), the GCM demonstrates a sequence of mul

42 Here, for the first time, we tag all O3 precursors (i.e., nitrogen oxides (NOx), carbon monox

43 Ambient O3 and NOx concentrations, but not PM2.5 concentrations,

44 Ambient O3 concentrations, but not other pollutants, at baseline

45 sis, which suggests that declines in ambient O3 concentrations ([O3 ]) in the United States may have

46 the effect of long-term changes in ambient [O3 ] using 20 years of observations at Harvard forest.

47 In May 2012, we observed an O3 increase of 2.0-8.5 ppbv in monthly average maximum d

48 Here, we report an O3-NaCr(2/3)Ti(1/3)S(2) cathode that delivers a high rev

49 o derived unrelated arrangements (O3+4+1 and O3+4+7) are nearly fixed for several amino acid substitu

50 ature mortalities) attributable to PM2.5 and O3 from RC and EGU emissions by precursor species, sourc

51 surements, in particular secondary PM2.5 and O3, have some level of contribution from other sources w

52 g state-specific contributions to PM2.5- and O3-related health burden from residential combustion and

53 itrate, on the modeling of NOx abundance and O3 formation.

54 composition in response to elevated CO2 and O3 (eCO2 and eO3) the endosphere, rhizosphere and soil w

55 Rising atmospheric concentrations of CO2 and O3 are key features of global environmental change.

56 erience rising levels of atmospheric CO2 and O3.

57 are mainly H(+), nitrate, nitrite, H2O2 and O3.

58 capturing the spatial variability in NO2 and O3 and are very convenient to use, having great potentia

59 rom 6 to 12 times, for 20 min, using NO2 and O3 sensors manufactured by Aeroqual.

60 Accurate measurements of NO, NO2, and O3 mole fractions allow the calculation of ozone absorpt

61 mework, to describe the chemistry of NOx and O3 in the remote marine boundary layer at Cape Verde.

62 r air pollutants, including PM2.5, PM10, and O3, did not significantly affect the occurrence of CRAO

63 e the heterogeneous kinetics of squalene and O3 were independent of relative humidity (RH), the RH si

64 l for basic sites of different strength, and O3 and NO2 for reducing groups.

65 The temporal variations of VOCs and O3 coincided with the surface wind, implying that the fo

66 asculine grid resolution) to estimate annual O3 exposures, and estimated total respiratory deaths in

67 0 that were attributable to long-term annual O3 exposure based on the updated relative risk estimates

68 Two derived unrelated arrangements (O3+4+1 and O3+4+7) are nearly fixed for several amino ac

69 patial filter was analyzed and quantified as O3.9 mum, which balances the suppression of the silica b

70 Arsenic trioxide (ATO, As2 O3 ) is currently used to treat acute promyelocytic leuk

71 al that the most stable radical is formed at O3-atom upon scavenging the free radicals.

72 otif and its regioselective glycosylation at O3.

73 xy for the relative abundance of atmospheric O3 and HOx) from a Greenland ice core over the most rece

74 ical inputs rather than the only atmospheric O3 concentration.

75 Under these conditions, the daily average O3 mixing ratio increased to approximately 44 and approx

76 the percentage of days when the 8-h average O3 mixing ratio (MDA8) exceeds 75 ppbv and smoke is pres

77 present 10-20% of the days when 8-h average O3 mixing ratios exceed 75 ppbv.

78 monthly average maximum daily 8-hour average O3 mixing ratio (MDA8 O3) at MBO and numerous other site

79 daily and interannual variations in baseline O3.

80 e is considerable research to be done before O3 -tolerant germplasm is available to growers for most

81 ed significant positive associations between O3, PM2.5, and NO2 concentrations and all-cause and caus

82 protection, and the differentiation between O3 and O4 can be well-controlled by the N2 functionality

83 hown that it is the unique single atomic Bi2 O3-x layer at the surface that leads to the enhanced pol

84 r the generation of the additional SrTi(BiPb)O3-peak.

85 urce photochemical model predictions of both O3 and secondary PM2.5 species.

86 responsive transcription factor forkhead box O3 (FoxO3) in mediating injury-induced proximal tubular

87 tly repressed the expression of forkhead box O3 (FOXO3), and FOXO3 negatively regulated pathogenic T(

88 istically, transcription factor forkhead box O3 (Foxo3a) physically interacts with Tet2 and regulates

89 JNK3 acts through Forkhead box O3 (Foxo3a) to suppress the activity of Egr1/Creb1 trans

90 nt in shaping future strategies for breeding O3 -tolerant cultivars.

91 This portion of GPP remains unaffected by [O3 ], thus helping to buffer the changes of total photos

92 ion followed by biological activated carbon (O3/BAC) is being considered as a key component of revers

93 have improved understanding of the cellular O3 sensing, signaling and response mechanisms.

94 dence-specific air pollutant concentrations (O3, PM2.5, NOx, and black carbon) were estimated by vali

95 that declines in ambient O3 concentrations ([O3 ]) in the United States may have helped increase gros

96 enting further measures aimed at controlling O3 concentrations.

97 ciations and potential links among diabetes, O3, and lung inflammation and remodeling are currently u

98 tiple types of organic matter with different O3 and HO(*) reactivity.

99 g advanced oxidation processes (AOPs) (i.e., O3/H2O2 and UV/H2O2) was investigated.

100 cursors emissions and, thus, greatly enhance O3 attribution to source region.

101 ay 2012 contributed to the observed enhanced O3 across the western U.S.

102 tedly, both RIS and RBS can lead to enhanced O3 formation in a polluted marine environment under vola

103 n the model system Gd0.1 Ce0.9 O2-delta /Er2 O3 to set and tune the property of "memristance." The mo

104 We used a photochemical model to estimate O3 response to large NOx reductions.

105 The model estimated O3 production from these sources but often was lower tha

106 compatible magnesium shallow doped gamma-Fe2 O3 (Mg0.13 -gammaFe2 O3 ) SPNPs with exceptionally high

107 in octahedral site Fe vacancies of gamma-Fe2 O3 instead of well-known Fe3 O4 SPNPs.

108 built-in potential as large as 0.8 eV in Fe2 O3-Cr2O3 SLs.

109 the pollutant as well as to breed crops for O3 tolerance.

110 late nitrate photolysis in future models for O3 and for the photolysis rate of particulate nitrate to

111 nd to identify more promising phenotypes for O3 tolerance are needed.

112 cal 95th percentile threshold, by +7 ppb for O3, +6 microg m(-3) for PM2.5, and +1.7 degrees C for TX

113 Quantitative trait loci (QTL) for O3 tolerance have been identified in model and crop spec

114 associations with all CHDs, VSD, and TF for O3 were generally consistent compared to the models that

115 on of plant phenology in the models used for O3 risk assessment.

116 d neutrophils in bronchoalveolar lavage from O3-exposed mice.

117 Ex vivo analysis of serum from O3-treated rats revealed an augmented microglial proinfl

118 anding of the effects of current and future [O3 ] on global crop productivity, and experimental advan

119 shallow doped gamma-Fe2 O3 (Mg0.13 -gammaFe2 O3 ) SPNPs with exceptionally high intrinsic loss power

120 nduction characteristics of Mg0.13 -gammaFe2 O3 are primarily due to the dramatically enhanced out-of

121 hyperthermia studies using Mg0.13 -gammaFe2 O3 nanofluids are conducted to estimate bioavailability

122 Mg0.13 -gammaFe2 O3 nanofluids show promising hyperthermia effects to com

123 II) cohort have been used to estimate global O3-attributable mortality in adults.

124 increases the bicarbonate permeability (P HC O3/ Cl ) of anion channels by reducing energy barriers o

125 increases the bicarbonate permeability (P HC O3/ Cl ) of CFTR, ANO1 and GlyR.

126 ow O3 concentrations would increase and high O3 concentrations would decrease in response to NOx redu

127 Air parcels with mixing ratios of high O3 and low H2O (HOLW) are common features in the tropica

128 tudied under varying environments (humidity, O3, and NOx) using both an indoor chamber and an outdoor

129 Smoke-impacted O3 mixing ratios are most elevated in locations with the

130 as emission inventory and the most important O3-depleting substance emitted in this 21st century.

131 Strategies to improve O3 tolerance in crops have been hampered by the lack of

132 Biotechnological strategies for improving O3 tolerance are also being tested, although there is co

133 nd biotechnological approaches for improving O3 tolerance in crops.

134 ications of climate change-driven changes in O3 concentrations, are larger than previously thought.

135 Fibrosis in O3-exposed KKAy lungs was confirmed with immunohistochem

136 ted with a 3.6-parts-per-billion increase in O3 exposure.

137 We hypothesize that the observed increase in O3/HOx in cold climates is driven by enhanced stratosphe

138 orological conditions, synoptic variation in O3 at MBO can be observed at other surface sites in the

139 is considerable within-species variation in O3 tolerance in crops, which has been used to create map

140 owth of atomically sharp In2 O3 /ZnO and In2 O3 /Li-doped ZnO (In2 O3 /Li-ZnO) heterojunctions via sp

141 arge transport properties of the isotype In2 O3 /Li-ZnO heterojunctions as well as on the operating c

142 In the case of In2 O3 /ZnO heterojunctions, presence of the n-doped ZnO lay

143 m its conduction band minimum to that of In2 O3 over the interface, in a process similar to modulatio

144 he controlled growth of atomically sharp In2 O3 /ZnO and In2 O3 /Li-doped ZnO (In2 O3 /Li-ZnO) hetero

145 By judicious optimization of the In2 O3 /Li-ZnO interface microstructure, and Li concentratio

146 rp In2 O3 /ZnO and In2 O3 /Li-doped ZnO (In2 O3 /Li-ZnO) heterojunctions via spin-coating at 200 degr

147 n in BALB/c mice might mediate the increased O3 responsiveness.

148 and tetralogy of fallot (TF) with increasing O3 exposure.

149 t at the air-water interface with increasing O3(g) during tauc approximately 1 mus contact time and c

150 lysis could be an important source of indoor O3.

151 In addition to neutrophil influx, O3 inhalation elicited the appearance of eosinophils and

152 monitor location and spatially interpolated O3 to census-tract centroids.

153 at ozonolysis occurs near the surface and is O3-transport limited.

154 hotolysis rate constants [e.g., J(NO2) and J(O3)] by attenuating solar flux.

155 With an apparent rate constant of k(O3) = (1.8 +/- 0.7) x 10(3) M(-1) s(-1) at pH 8, STG can

156 At the benchmark dose of 3.4 kg O3/kg initial TPH, TPH carbon was reduced by nearly 6 gC

157 ack of correlation between visual leaf-level O3 damage and yield loss to O3 stress.

158 ng them, Arabidopsis MILDEW RESISTANCE LOCUS O3 (MLO3) was identified as a previously unidentified po

159 We predicted that low O3 concentrations would increase and high O3 concentrati

160 Herein, a novel Li-rich cathode material, O3-type Li(0.6) [Li(0.2) Mn(0.8) ]O(2) , is developed wi

161 Our findings indicate that maternal O3 exposure over pregnancy is associated with stillbirth

162 m daily 8-hour average O3 mixing ratio (MDA8 O3) at MBO and numerous other sites in the western U.S.

163 This analysis shows that (i) the mean O3 abundance measured on smoke-impacted days is higher t

164 providing a biochemical mechanism mediating O3-derived formation of oxidized lipids in the airways a

165 effect of different ozone dosages (0-1.0 mg O3/mg dissolved organic carbon) and BAC empty bed contac

166 bromate at specific ozone doses of </=0.4 mg O3/mg DOC, while the bromate yields were almost linearly

167 f water at the (001) surfaces of Sr(n+1)Ru(n)O3(n+1) (n = 1, 2) using low-temperature scanning tunnel

168 or allergic conjunctivitis, but not for NO2, O3, or temperature.

169 s County, Texas) with historic nonattainment O3 levels, we obtained birth and fetal death records fro

170 l evolution without perturbing the nonlinear O3 chemistry.

171 ent concentrations of PM2.5 and NOx, but not O3, decreased substantially during follow-up.

172 aily metrics of 12 pollutants (CO, NO2, NOx, O3, SO2, PM10, PM2.5, and five PM2.5 components) over th

173 tively, regioselective monotriflation at O2, O3, and O4 of l-rhamnose/l-fucose allowed selective inve

174 In both the presence and the absence of O3 and NOx, gammaSO4(2-),light and gammaSO4(2-),dark gre

175 Ambient concentrations of O3, PM2.5, NOx, and black carbon at study baseline were

176 OCs and other factors in the contribution of O3 formation in China.

177 14, we find strong, positive correlations of O3 with multiple biomass burning tracers in these HOLW s

178 tween CH3O2 and OH leads to a 6% decrease of O3.

179 Production and destruction of O3 are also influenced by these changes, and the model i

180 Considering the acute effects of O3 exposure, approximately 670 cardiovascular and 300 re

181 sensitive to the proinflammatory effects of O3 serum.

182 We obtained estimates of O3 concentrations at the participant's residence from a

183 AQMS) analyses indicate that a large flux of O3 from the UT/LS in May 2012 contributed to the observe

184 surface wind, implying that the formation of O3 was impacted by both exports of plumes upwind and loc

185 The increase in the level of O3 was associated with an increased mixing ratio of hydr

186 The increase in the level of O3 was partially induced by enhanced ClNO3 formation for

187 Although the lungs of O3-exposed C57BL/6J and KK mice had minimal centriacinar

188 gical changes without fibrosis, the lungs of O3-exposed KKAy mice contained marked epithelial hyperpl

189 oduce a large uncertainty in the modeling of O3 formation.

190 vations, LiDAR and satellite observations of O3 could provide key data on daily and interannual varia

191 onding to the minimum or fifth percentile of O3 exposure in the earlier study population.

192 ds set at the minimum or fifth percentile of O3 exposure in the most recent CPS-II analysis.

193 rine regions can improve model prediction of O3 concentrations near the coast.

194 SAA could be an important systemic signal of O3 exposure to the CNS.

195 exposure and systemically convey signals of O3 exposure to the CNS.

196 ded the galactosamine derivative and that of O3 yielded allosamine.

197 eling in the soil improved mass transport of O3 to TPH bound to soil and increased TPH removal.

198 ced stratosphere-to-troposphere transport of O3, and that reactive halogen chemistry is also enhanced

199 A decline of [O3 ] over the measurement period resulted in moderate pr

200 selves and indirectly via their influence on O3 and HOx.

201 the effects of activities of RBS and RIS on O3 mixing ratios in the polluted MBL.

202 le organic compounds (VOCs) and the roles on O3 pollution have been investigated in a typical industr

203 nt compared to the models that included only O3, with the strongest aORs observed for exposures durin

204 high-ozone window and likely has an opposite O3-NOx response to what would otherwise be expected, wit

205 ally linked to pulmonary inflammation in our O3 exposure model and that A-SAA could be an important s

206 at night with lower NOx and higher oxidants (O3) and oxidized reactive nitrogen (N2O5) aloft.

207 of the most important atmospheric oxidants, O3, NO3, and OH, plays a central role in regulating atmo

208 Ozone (O3 ) damage to leaves can reduce plant photosynthesis, w

209 ), nitrogen dioxide (NO2) (2006), and ozone (O3) (2002-2004) concentrations were linked to the partic

210 f fine particulate matter (PM2.5) and ozone (O3).

211 nce of tropospheric oxidants, such as ozone (O3) and hydroxyl (OH) and peroxy radicals (HO2 + RO2), d

212 hrough NO conversion to NO2 in excess ozone (O3) and subsequent NO2 collection in a 20% triethanolami

213 cid (HONO), hydrogen peroxide (H2O2), ozone (O3), formaldehyde (HCHO), and acetaldehyde (CH3CHO).

214 When inhaled, ozone (O3) interacts with cholesterols of airway epithelial cel

215 end in concentrations of ground-level ozone (O3 ) - a common air pollutant and phytotoxin - currently

216 Ground-level ozone (O3), harmful to most living things, is produced from bot

217 nary responses to air pollutants like ozone (O3).

218 context, the role of iodide-mediated ozone (O3) deposition over seawater and marine halogen chemistr

219 nets including molecular oxygen (O2), ozone (O3), water vapor (H2O), carbon dioxide (CO2), nitrous ox

220 aims to evaluate the effectiveness of ozone (O3) gas treatment on the degradation of residual bifenth

221 d IFN-gamma, as well as the effect of ozone (O3) inhalation, were studied on recirculation of pulmona

222 chemical method using the reaction of ozone (O3) with nitrogen monoxide (NO) resulting in nitrogen di

223 The present study aimed to optimize ozone (O3) treatments, as gas and dissolved in water, to remove

224 associated with time-varying prenatal ozone (O3) exposure over pregnancy.

225 dioxide (NO2), sulfur dioxide (SO2), ozone (O3), and particulate matter with aerodynamic diameter </

226 Level of surface ozone (O3) has been increasing continuously in China in recent

227 nded summer (April-September) surface ozone (O3), fine particulate matter (PM2.5), and maximum temper

228 Relative risk estimates for long-term ozone (O3) exposure and respiratory mortality from the American

229 ometry is used to investigate how the ozone (O3) concentration, relative humidity (RH), and particle

230 mistry and causing sharp tropospheric ozone (O3) depletion in polar regions and significant O3 reduct

231 Tropospheric ozone (O3) is potentially associated with cardiovascular diseas

232 Tropospheric ozone (O3) produces harmful effects to forests and crops, leadi

233 en oxides (NOx) produces tropospheric ozone (O3), and NOx is traditionally considered to be directly

234 Concentrations of ground-level ozone ([O3 ]) over much of the Earth's land surface have more th

235 x and CO were enhanced twofold during PCAPs, O3 concentrations were approximately threefold lower.

236 We evaluated how gas-phase O3 interacts with residual petroleum hydrocarbons in soi

237 action mechanisms and kinetics for gas-phase O3, NO3, and OH when they impinge on organic surfaces.

238 ot give any information on the physiological O3 uptake into the leaves since it does not include any

239 not mRNA, was increased in the CNS 24 h post-O3 exposure.

240 re, female Balb/c mice were exposed to 3 ppm O3 or forced air for 2 h and were studied after 6 or 24

241 ltered air (FA) or repetitive ozone (0.5 ppm O3, 4 h/d, for 13 consecutive weekdays).

242 o fertilized agricultural soils could reduce O3 by up to 2.4 ppb and PM2.5 by up to 0.15 mug/m(3) in

243 Following repetitive O3 exposure, higher bronchoalveolar lavage fluid inflamm

244 2008-2013 and estimated maternal residential O3 concentrations from conception until delivery using i

245 ped with lanthanide ions, both ScOOH and Sc2 O3 can be utilized for high-temperature probing and ligh

246 Using a laboratory-scale O3/BAC system treating two nitrified wastewater effluent

247 s most abundant mineral, (Mg,Fe,Al)(Al,Fe,Si)O3 bridgmanite (also known as silicate perovskite), has

248 ) depletion in polar regions and significant O3 reduction in the marine boundary layer (MBL).

249 e sensitivity approaches capture near-source O3 titration by fresh NO emissions, in particular subgri

250 ter downwind peak estimates of single source O3 impacts.

251 (i.e., tracking ozone produced from specific O3 precursors emitted from one region).

252 onversely, risk assessment based on stomatal O3 uptake shows different spatial patterns compared to o

253 on by volume (ppbv) seasonal average surface O3 over North America can be attributed to East Asian an

254 Predicted reductions in surface O3 concentrations occur mainly along the coast where CMA

255 to the largest reductions in modeled surface O3 concentrations.

256 nmental Protection Agency Air Quality System O3 monitors are influenced by smoke.

257 h benefits of air quality policies targeting O3, health co-benefits of climate mitigation policies, a

258 piratory mortality attributable to long-term O3 exposure in adults >/=30y of age using updated effect

259 model, however, suggest an average long-term O3 inhibition of 10.4% for 1992-2011.

260 or the aqueous reaction of alpha-terpineol + O3 to be 9.9(+/-3.3) x 10(6) M(-1) s(-1).

261 Considering that O3 is toxic to humans, plants, and animals and is a gree

262 Overall, these data demonstrate that O3-derived oxysterols have pro-inflammatory functions an

263 MRO3 between 22 and 32 ppb, suggesting that O3 was the dominant oxidant in this indoor setting.

264 Accumulating evidence suggests that O3 exposure may contribute to CNS dysfunction.

265 The O3/BAC-treated wastewaters met regulatory levels for tri

266 For this reason, the use of PODY in the O3 risk assessment for vegetation is becoming recommende

267 ve to climate change with an increase in the O3/HOx ratio in cold climates, the opposite of current e

268 he calculated DBP-associated toxicity of the O3/BAC-treated chloraminated effluents were comparable o

269 tin CRD calcium site by interacting with the O3' and O4' hydroxyls alongside additional specific inte

270 shade, where stomatal conductance and thus [O3 ] deposition is lower than for sunlit leaves.

271 f a (011)-oriented ferroelectric Pb(Mg,Nb,Ti)O3 substrate intimately coupled to an epitaxial ferromag

272 a heterostructure of piezoelectric Pb(Zr,Ti)O3 (PZT) film deposited on magnetostrictive Metglas (FeB

273 larization field of a ferroelectric Pb(Zr,Ti)O3 (PZT) gate, nonvolatile resistance modulation in the

274 Co40Fe40B20 nanomagnet on top of a Pb(Zr,Ti)O3 film as an example).

275 susceptibility of human asthmatic airways to O3 exposure.

276 5 million respiratory deaths attributable to O3 exposures based on the earlier CPS-II risk estimate a

277 respiratory deaths in adults attributable to O3 exposures using the updated relative risk estimate an

278 not include any environmental constraints to O3 uptake through stomata.

279 vern the sensitivity of soybean cultivars to O3 will be important in shaping future strategies for br

280 hylation in LINE1, whereas later exposure to O3 was associated with higher LINE1 methylation levels i

281 isual leaf-level O3 damage and yield loss to O3 stress.

282 ion and microglial activation in response to O3 Further, aging glia were more sensitive to the proinf

283 iables governing the efficacy of treatments: O3 concentration, temperature and treatment time.

284 Although first-trimester O3 exposure was not associated with CIMT and systolic BP

285 of oxidants, resulting in lower tropospheric O3 in cold climates while HOx (= OH + HO2 + RO2) remains

286 perature-dependent emissions of tropospheric O3 precursors and water vapour abundance determine the c

287 n distribution or by exposure to ultraviolet/O3, which generates structural or chemical disorder, res

288 erence observed in the dimer abundance under O3- versus OH-dominant environments underlines the compe

289 imer was removed from the Pt DENs using a UV/O3 treatment, and this provided direct contact between t

290 anges of total photosynthesis due to varied [O3 ].

291 The mechanism by which O3-derived oxysterols affect molecular function is unkno

292 SO2 exposure and an inverse association with O3 in week 34.

293 were nonzero and exhibited correlations with O3 and NO3, consistent with previous studies that ozonol

294 5 and NO2 and in three-pollutant models with O3.

295 rom autoxidation and not from reactions with O3 or (1)O2.

296 10mgL(-1), respectively, in treatments with O3 as gas and dissolved in water.

297 tudied under simulated flue gas with/without O3.

298 elations above TC in underdoped BaPb1-x Bi x O3, and provide information on the dynamical interplay b

299 sient terahertz conductivity of BaPb1-x Bi x O3--a material for which superconductivity is "adjacent"

300 increases in percent emphysema per 10 years (O3: 0.13 per 3 parts per billion [95% CI, 0.03-0.24]; PM