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1                                              ONJ appears to be time-dependent with higher risk after
2                                              ONJ associated with IV bisphosphonate therapy is extreme
3                                              ONJ occurred posterior to the cuspids (n = 20) mostly in
4                                              ONJ prevents the jawbone from healing properly, leading
5                                              ONJ resolved in 60 patients (62%), resolved and recurred
6 e, 1.14 AFF, 0.21 esophageal cancer and 0.09 ONJ events per 1,000 person-years in the alendronate coh
7                                  We found 16 ONJ cases among 572,606 cohort members; seven additional
8                              We enrolled 191 ONJ cases and 573 controls in 119 dental practices.
9  prospectively for a minimum 3.2 years after ONJ.
10 geal cancer (HR 0.95, 95% CI 0.53-1.70), and ONJ (HR 1.62, 95% CI 0.78-3.34).
11     Overall, 90 patients developed confirmed ONJ, with cumulative incidence of 0.8% (95% CI, 0.5%-1.1
12            Cumulative incidence of confirmed ONJ, defined as an area of exposed bone in the maxillofa
13  guide stratification of risk for developing ONJ in patients with MBD receiving zoledronic acid.
14       The variables predictive of developing ONJ were dental extraction (P = .009), treatment with pa
15 ered profile in wound-healing markers during ONJ development.
16 ith recurrent/nonhealing than single-episode ONJ (84% v 62%; P = .02).
17 either ZA or OPG-Fc in rats with established ONJ did not lead to ONJ resolution.
18  development or in patients with established ONJ to accelerate healing.
19 ctice-Based Research Network study estimated ONJ incidence and odds ratios from bisphosphonate exposu
20  than 5 weeks were more likely to experience ONJ than patients with planned dosing intervals of 5 wee
21                    In the second experiment, ONJ was established and antiresorptives were discontinue
22                             Risk factors for ONJ were also examined.
23 xtractions were independent risk factors for ONJ.
24                        Treatment options for ONJ range from management of symptomology to surgical re
25 ate with physicians to minimize the risk for ONJ.
26 ons and suggests that the absolute risks for ONJ from oral bisphosphonates is low.
27 were 15.5 (CI, 6.0-38.7) more likely to have ONJ than non-exposed patients; however, the sparse numbe
28 ce of dental disease and tooth extraction in ONJ pathogenesis and help delineate an altered profile i
29  were 62 men; the median age was 61 years in ONJ patients and 58 years among the rest.
30 esorptives or saline, after which we induced ONJ using periapical disease and tooth extraction.
31                    Osteonecrosis of the jaw (ONJ) affects patients with cancer and metastatic bone di
32   The incidence of osteonecrosis of the jaw (ONJ) in the population is low, but specifics are unknown
33                    Osteonecrosis of the jaw (ONJ), a side-effect of bisphosphonate therapy, is charac
34 al fracture (AFF), osteonecrosis of the jaw (ONJ), and esophageal cancer.
35 lation, bone pain, osteonecrosis of the jaw (ONJ), atypical fractures, and osteosarcoma.
36 ral fractures, and osteonecrosis of the jaw (ONJ), the pathophysiological mechanisms involved are not
37 n in patients with osteonecrosis of the jaw (ONJ).
38 nal development of osteonecrosis of the jaw (ONJ).
39 de a discussion of osteonecrosis of the jaw (ONJ).
40 weigh the risk for osteonecrosis of the jaw (ONJ).
41                   Osteonecrosis of the jaws (ONJ) is a rare but severe complication of antiresorptive
42                   Osteonecrosis of the jaws (ONJ), a severe side effect of antiresorptive medications
43 onate therapy and osteonecrosis of the jaws (ONJ), but neither causality nor specific risks for lesio
44 a "drug holiday," has been used for managing ONJ patients.
45 ients with single, recurrent, and nonhealing ONJ (P = .2).
46 , 60.7 to 84.2 months), 33 possible cases of ONJ were reported, all in the zoledronate-treated patien
47 se and bone metabolism on the development of ONJ-like lesions.
48 rmed as being consistent with a diagnosis of ONJ, representing a cumulative incidence of 2.1% (95% CI
49 al, radiographic, and histologic features of ONJ.
50                We report on the frequency of ONJ and investigate oral health-related quality of life
51 r studies of the pathogenesis and healing of ONJ are needed.
52 einitiating bisphosphonates after healing of ONJ is a reasonable option in patients experiencing rela
53 e findings show, the cumulative incidence of ONJ after 3 years was 2.8% in patients receiving zoledro
54  trial is associated with a low incidence of ONJ but does not seem to adversely affect Oral-QoL.
55 ogate for rodents, OPG-Fc, in a rat model of ONJ.
56 osed patients; however, the sparse number of ONJ cases limits firm conclusions and suggests that the
57                 All potential occurrences of ONJ were reported as serious adverse events and centrall
58                             A higher rate of ONJ was associated with fewer total number of teeth (HR,
59                               Higher risk of ONJ began within 2 years of bisphosphonate initiation an
60 y of dosing were associated with the risk of ONJ.
61                         More than a third of ONJ patients also suffered from long bone fractures (n =
62                          Dental treatment of ONJ should be conservative and provide relief to the pat
63  serious jaw condition called osteonecrosis (ONJ) increases significantly.
64                   Dental procedures preceded ONJ in 46 patients (47%) and were more common in those w
65 edures, such as tooth extraction, to prevent ONJ development or in patients with established ONJ to a
66                                    Recurrent ONJ followed reinitiation of bisphosphonates in six of 1
67 r infections and four patients had recurrent ONJ.
68  Committee also discusses measures regarding ONJ.
69          Incidence of bisphosphonate-related ONJ in osteoporosis patients is unclear, but several stu
70                    Patients with spontaneous ONJ have a higher risk of nonhealing and recurrence.
71 r to tooth extraction ameliorated subsequent ONJ development.
72 tal extraction, including 12 patients in the ONJ group.
73             Median time from MM diagnosis to ONJ was 8.4 years for the whole group.
74 in rats with established ONJ did not lead to ONJ resolution.
75  Oral and intravenous bisphosphonate-treated ONJ sites had reduced numbers of basal epithelial progen
76 phosphonate use was strongly associated with ONJ (odds ratios [OR] 299.5 {95%CI 70.0-1282.7} for intr
77 l disease have been strongly associated with ONJ development.
78 R = 6.6 {1.6-26.6}) remained associated with ONJ.
79 hosphonate use were strongly associated with ONJ.
80 osis therapies and many adverse events, with ONJ an exception.
81 sphosphonate use and other risk factors with ONJ.
82  examined human tissue from individuals with ONJ and non-bisphosphonate-treated control individuals t
83 portantly, human biopsies from patients with ONJ showed similar findings.