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1 c stimuli produce different responses across OVLT neurons and may represent distinct cellular process
2                              Na(+) activates OVLT neurons via a paracrine mechanism involving sodium
3                                     Although OVLT neurons are intrinsically osmosensitive and shrink
4                                        Among OVLT neurones with axons projecting directly to the PVN
5  positive cells in the SFO (42% greater) and OVLT (100% greater).
6 ative single-cell oscillators in the SFO and OVLT are strongly rhythmic and require action potential-
7 cally as a result of inputs from the SFO and OVLT, which have themselves been activated directly by a
8 te load tended to amplify the FLI in SFO and OVLT.
9 een distant parts of the third ventricle and OVLT.
10 osum of the lamina terminalis-preoptic area (OVLT-POA) continuum.
11 ute to long-term osmosensory transduction by OVLT neurons and might therefore participate in the elev
12 cute and chronic osmosensory transduction by OVLT neurons may be mediated by distinct mechanisms.
13 reflects reduced somal stores of GnRH in DBB/OVLT and MS, suggesting that these subpopulations promot
14 num vasculosum of the lamina terminalis (DBB/OVLT) and medial septum (MS) in adults as compared to ju
15                                     However, OVLT neurons in CLP rats were hyperpolarized significant
16                                           In OVLT neurons OSR1/SPAK activates Kv3.1 to increase actio
17                Knockdown of Kv3.1 channel in OVLT by shRNA reproduced the phenotypes.
18 eptor, and AngII increases Fos expression in OVLT neurons.
19 uced increases in action potential firing in OVLT neurons was blunted by Wnk1 deletion or pharmacolog
20 uced increases in action potential firing in OVLT were blunted by Osr1 and Spak deletion.
21 oduced a concentration-dependent increase in OVLT cell discharge, lumbar SNA and ABP.
22 hypertonic NaCl evokes a greater increase in OVLT neuronal discharge frequency than equi-osmotic sorb
23  produced significantly greater increases in OVLT discharge and ABP than icv infusion of equi-osmotic
24 rosecretions to reach nearby target sites in OVLT, avoiding dilution in the systemic blood.
25 ovide the first identification of individual OVLT neurons that respond to both elevated NaCl and AngI
26  The present study tested whether individual OVLT neurons sensed both NaCl and AngII to regulate thir
27 at intracerebroventricular infusion or local OVLT injection of hypertonic NaCl increases lumbar sympa
28               Stimulating glutamatergic MnPO/OVLT neurons induced water consumption, whereas stimulat
29 stimulation of glutamatergic neurons in MnPO/OVLT drives voracious water consumption, and that optoge
30  that glutamate and GABA neurons in the MnPO/OVLT reciprocally regulate water consumption.
31  subfornical organ, suggesting that the MnPO/OVLT serves as a key link in regulating drinking respons
32       Both kinases were present in the mouse OVLT area.
33 -clamp recordings indicate 66% (23 of 35) of OVLT neurons were excited by bath application of both hy
34 l recordings demonstrate that 50% (18/36) of OVLT neurons display an increased discharge to both hype
35 t recordings revealed that 52% (23 of 44) of OVLT neurons displayed an increased discharge to intraca
36       Whereas acute hypertonic activation of OVLT neurons critically depends on TRPV1 channels, studi
37  performed to quantify osmotic activation of OVLT-PVN neurones.
38  We found that the intrinsic excitability of OVLT neurons was not affected significantly 18-24 h afte
39              Next, optogenetic excitation of OVLT neurons stimulated thirst but not salt appetite.
40        Conversely, optogenetic inhibition of OVLT neurons attenuated thirst stimulated by hypernatrem
41  PVN-projecting neurones in the DC and LM of OVLT could participate in behavioural, neuroendocrine, a
42  dorsal cap (DC) and lateral margins (LM) of OVLT.
43 -cell recordings demonstrate the majority of OVLT neurons are responsive to hypertonic NaCl or mannit
44  recordings, and optogenetic manipulation of OVLT neurons, was used in adult, male Sprague Dawley rat
45  the DC and LM contained a similar number of OVLT-PVN neurones, the proportion of such neurones that
46 mined the electrophysiological properties of OVLT neurons and magnocellular neurosecretory cells (MNC
47                The different subdivisions of OVLT received glial processes of different origins.
48 ly, these novel data suggest that subsets of OVLT neurons respond differently to hypertonic NaCl vers
49      Renin induced Fos-IR in SFO, MnPO, peri-OVLT region, SON and PVN.
50 ipheral origin activates the SFO and/or peri-OVLT region and contributes to sodium appetite.
51 n which OVLT neurones projecting to the PVN (OVLT-PVN) were retrogradely labelled with cholera toxin
52                                      Second, OVLT microinjection (20 nl) of 1.0 m NaCl significantly
53    Furosemide-induced activation in the SFO, OVLT, SON and PVN does not depend on renal innervation.
54 ults suggest that the activation of the SFO, OVLT, SON and PVN may be via a different mechanism than
55 the number of Fos-positive cells in the SFO, OVLT, SON and PVN, but not in the caudal thoracic spinal
56 ation of the pathway that occurs in the SFO, OVLT, SON, and magnocellular region of the paraventricul
57 erminalis (OVLT), these observations suggest OVLT neurons may sense or respond differently to hyperto
58 organum vasculosum of the lamina terminalis (OVLT) and lateral hypothalamus (LH) of rats with coronar
59 organum vasculosum of the lamina terminalis (OVLT) and median preoptic nucleus (MnPO).
60 organum vasculosum of the lamina terminalis (OVLT) and subfornical organ (SFO) became infected.
61 rgans, organum vasculosum lamina terminalis (OVLT) and subfornical organ (SFO), are potential sites t
62 organum vasculosum of the lamina terminalis (OVLT) and the preoptic area (POA).
63 organum vasculosum of the lamina terminalis (OVLT) are known to regulate fluid/electrolyte homeostasi
64 organum vasculosum of the lamina terminalis (OVLT) contains NaCl-sensitive neurons to regulate thirst
65 in the organum vasculosum lamina terminalis (OVLT) region of the preoptic area (POA).
66 organum vasculosum of the lamina terminalis (OVLT) sense changes in extracellular osmolarity and NaCl
67 the vascular organ of the lamina terminalis (OVLT) that contains a subpopulation of gonadotropin rele
68 organum vasculosum of the lamina terminalis (OVLT) were compared between the two developmental stages
69 organum vasculosum of the lamina terminalis (OVLT), a circumventricular organ.
70 organum vasculosum of the lamina terminalis (OVLT), a region that has also been implicated in fluid a
71 organum vasculosum of the lamina terminalis (OVLT), begging the question of the direction of blood fl
72 (VMH), organum vasculosum lamina terminalis (OVLT), CA1 field of the hippocampus, striatum or cortex.
73 organum vasculosum of the lamina terminalis (OVLT), medial preoptic nucleus (MNPO), subfornical organ
74 organum vasculosum of the lamina terminalis (OVLT), median preoptic nucleus (MNPO), hypothalamic para
75 (SFO), organum vasculosum lamina terminalis (OVLT), supraoptic nuclei (SON), and magnocellular region
76 (SFO), organum vasculosum lamina terminalis (OVLT), supraoptic nucleus (SON), magnocellular region of
77  organum vasculosm of the lamina terminalis (OVLT), the median preoptic nucleus (MnPO) and/or the sub
78 organum vasculosum of the lamina terminalis (OVLT), these observations suggest OVLT neurons may sense
79 the vascular organ of the lamina terminalis (OVLT).
80 in the organum vasculosum lamina terminalis (OVLT; which drives thirst) and attenuates that of neuros
81  in the vascular-organ-of-lamina-terminalis (OVLT) nuclei of the brain mediates the hypertonicity-ind
82 sing on vascular-organ-of-lamina-terminalis (OVLT) nuclei, we showed that WNK1 kinase was activated b
83 brain organum vasculosum laminae terminalis (OVLT) and hypothalamic paraventricular nucleus (PVN) eac
84 d the organum vasculosum laminae terminalis (OVLT) are two sensory circumventricular organs (sCVOs) t
85 rs of organon vasculosum laminae terminalis (OVLT) in three planes.
86 brain organum vasculosum laminae terminalis (OVLT) play a pivotal role in triggering hyperosmotic act
87 y the organum vasculosum laminae terminalis (OVLT), on the midline of the POA, by the presumptive act
88 n the organum vasculosum laminae terminalis (OVLT), which then activates downstream neurons that indu
89 d the organum vasculosum laminae terminalis (OVLT).
90 a the organum vasculosum laminae terminalis (OVLT).
91 n the organum vasculosum lateral terminalis (OVLT), DOC pretreatment augmented cFos expression.
92                       Notably, we found that OVLT neurons are hyperpolarized and electrically silence
93                                          The OVLT also expresses the angiotensin II (AngII) type1 rec
94 le (AV3V), which destroys cell bodies in the OVLT and MnPO, as well as efferent projections from the
95  effects on the properties of neurons in the OVLT and supraoptic nucleus.
96  also increased the number of neurons in the OVLT expressing AngII-induced cFos.
97      Double deletion of Osr1 and Spak in the OVLT in mice caused polyuria with relative hypotonic uri
98 tinct patterns of Fos-positive nuclei in the OVLT largely restricted to the dorsal cap versus vascula
99 by synaptic density were not observed in the OVLT nor on GnRH dendrites in either brain region.
100 ion of the constitutively active OSR1 in the OVLT resulted in increased AVP release and inappropriate
101         Deletion of the Kv3.1 channel in the OVLT showed a similar phenotype.
102 ted at 14:00 h and 20:00 h (P < 0.01) in the OVLT-POA of young females.
103 dance of phosphorylated OSR1 and SPAK in the OVLT.
104 bstantial presence of oxytocin fibers in the OVLT.
105                       Injecting TTX into the OVLT completely blocked the lactate-induced response, wh
106 jections of lactate (100 or 500 nl) into the OVLT elicited robust anxiety-like responses in these rat
107 rminate means of neurons projecting into the OVLT from the brain.
108 cannula was implanted into the region of the OVLT, SFO, or an adjacent control site, the median preop
109               These results suggest that the OVLT may be the primary site that detects lactate infusi
110 sive neurons were distributed throughout the OVLT.
111  as efferent projections from the SFO to the OVLT and MnPO, abolishes DOCA-salt hypertension in the r
112                                Caudal to the OVLT, l-GnRH-III-positive neurons were also observed dor
113                         One is linked to the OVLT, SFO, and anteroventral third ventricular (AV3V) re
114 d flows unidirectionally from the SCN to the OVLT, that blood flow rate displays daily oscillations w
115 ossibly relayed directly from the PVN to the OVLT.
116  were delayed relative to projections to the OVLT.
117 fy molecularly defined cell types within the OVLT and MnPO that are activated by fluid imbalance and
118  hyperosmolality activate specifically those OVLT neurones that form a monosynaptic pathway to the PV
119 na terminalis, including organum vasculosum (OVLT) and subfornical organ (SFO), as well as in the mag
120 , PVH, SFO and hippocampus, but not the VMH, OVLT and striatum or cortex.
121 ns were performed in conscious rats in which OVLT neurones projecting to the PVN (OVLT-PVN) were retr

 
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