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1 inook salmon or in tissues of rainbow trout (Oncorhynchus mykiss).
2 tide") and growth in juvenile rainbow trout (Oncorhynchus mykiss).
3 the surface, was examined in rainbow trout (Oncorhynchus mykiss).
4 (HR) and low-responsive (LR) rainbow trout (Oncorhynchus mykiss).
5 ects the freshwater production of steelhead (Oncorhynchus mykiss).
6 er S9 fractions isolated from rainbow trout (Oncorhynchus mykiss).
7 have gained high virulence in rainbow trout (Oncorhynchus mykiss).
8 and skin mucosal surfaces of rainbow trout (Oncorhynchus mykiss).
9 umulation of selenium (Se) in rainbow trout (Oncorhynchus mykiss).
10 reported a homolog to CCR7 in rainbow trout (Oncorhynchus mykiss).
11 ) have been identified in the rainbow trout (Oncorhynchus mykiss).
12 SalHV-1) is a pathogen of the rainbow trout (Oncorhynchus mykiss).
13 tailed genetic linkage map of rainbow trout (Oncorhynchus mykiss).
14 an acute, lethal infection in rainbow trout (Oncorhynchus mykiss).
15 ated toxicities in vivo using rainbow trout (Oncorhynchus mykiss).
16 ures on the fillet quality of rainbow trout (Oncorhynchus mykiss).
17 n 18 has been identified from rainbow trout, Oncorhynchus mykiss.
18 y during embryogenesis in the rainbow trout, Oncorhynchus mykiss.
19 s using an in vitro digestion model based on Oncorhynchus mykiss.
23 BPA deposition in the eggs of rainbow trout (Oncorhynchus mykiss), an ecologically and economically i
24 er subgroups (IFN-e and -f) in rainbow trout Oncorhynchus mykiss and analyzed the expression of all s
25 genes were identified in rainbow trout (rt) Oncorhynchus mykiss and are classified into two groups b
26 f widely introduced salmonids rainbow trout (Oncorhynchus mykiss) and brook trout (Salvelinus fontina
29 -anchored genome assembly for rainbow trout (Oncorhynchus mykiss) and characterize a 55-Mb double-inv
30 ed in two previous studies of rainbow trout (Oncorhynchus mykiss) and common carp (Cyprinus carpio).
31 hemicals in two fish species: rainbow trout (Oncorhynchus mykiss) and fathead minnow (Pimephales prom
32 out-migration success in juvenile steelhead (Oncorhynchus mykiss) and focuses on the application of m
33 o bioassays using rainbow trout hepatocytes (Oncorhynchus mykiss) and in vivo studies with Japanese m
34 isulfide polymerization of IgM in the trout (Oncorhynchus mykiss) and its effect on its half-life wer
35 luding two sensitive species, rainbow trout (Oncorhynchus mykiss) and lake trout (Salvelinus namaycus
36 lamin-binding proteins of the rainbow trout (Oncorhynchus mykiss) and to compare their properties wit
37 angliosides found in sperm of rainbow trout (Oncorhynchus mykiss) and was shown to be present promine
38 tlantic salmon (Salmo salar), rainbow trout (Oncorhynchus mykiss), and Arctic char (Salvelinus alpinu
39 tion mixtures in coho salmon, rainbow trout (Oncorhynchus mykiss), and fathead minnow (Pimephales pro
40 hic invertebrates, juvenile steelhead trout (Oncorhynchus mykiss), and water striders (Gerris remigis
41 mologue has been identified in rainbow trout Oncorhynchus mykiss, and its biological activities have
44 ntic salmon (Salmo salar) and rainbow trout (Oncorhynchus mykiss) are two of the most susceptible sal
47 vely affects muscle growth in rainbow trout (Oncorhynchus mykiss), but the mechanisms directing with
48 ocal anadromous (ocean-run) steelhead trout (Oncorhynchus mykiss) by blocking their migration route a
50 tracting sulfated polysaccharides (SPs) from Oncorhynchus mykiss byproducts using alkaline/acid solub
52 unctional characterization of rainbow trout (Oncorhynchus mykiss) CD4-1(+) T cells and the establishm
53 hether a relevant model fish (rainbow trout, Oncorhynchus mykiss) could detect OSPW using its olfacto
54 eta2m of a salmonid fish, the rainbow trout (Oncorhynchus mykiss), does not conform to the mammalian
55 r vitelline envelope (VE), of rainbow trout (Oncorhynchus mykiss) eggs consists of three proteins, ca
56 members within the available rainbow trout (Oncorhynchus mykiss) EST gene index, we identified a uni
57 of salmonid fishes, including rainbow trout (Oncorhynchus mykiss), experienced a whole genome duplica
59 o examine stress responses in rainbow trout (Oncorhynchus mykiss) exposed to five distinct environmen
61 ss (Dicentrarchus labrax) and rainbow trout (Oncorhynchus mykiss), fed diets at 25%, 50% and 60% inse
62 the rancidity development in rainbow trout (Oncorhynchus mykiss) fillets during refrigerated storage
63 scle and edible skin parts of rainbow trout (Oncorhynchus mykiss) fillets, sampled at two growth stag
64 bile samples was validated in rainbow trout (Oncorhynchus mykiss) following short-term laboratory exp
65 her vertebrate taxa including rainbow trout (Oncorhynchus mykiss), frog (Xenopus laevis), chicken (Ga
66 mentation on a wild population of steelhead (Oncorhynchus mykiss) from the Hood River, Oregon, by mat
70 expression was studied in the rainbow trout (Oncorhynchus mykiss) gonad (RTG) (fibroblast) cell line.
71 vacuum packaged low processed rainbow trout (Oncorhynchus mykiss) gravad during storage at 7 +/- 1 de
72 vacuum packaged low processed rainbow trout (Oncorhynchus mykiss) gravad during storage at 7 1 C for
74 ry and sequence analysis of a rainbow trout (Oncorhynchus mykiss) IL-17A/F2 molecule and an IL-17RA r
76 th of the young of the year steelhead trout (Oncorhynchus mykiss) in the recipient tributary over the
77 al barrier model built on the rainbow trout (Oncorhynchus mykiss) intestinal cell line, RTgutGC and t
78 RTgutGC cells, derived from rainbow trout (Oncorhynchus mykiss) intestine, were used to evaluate th
79 this study, PFAS exposure on rainbow trout (Oncorhynchus mykiss) is examined at the molecular level,
83 thermal tolerance of two populations of wild Oncorhynchus mykiss near the species' southern range lim
85 n of plasmablasts and plasma cells in trout (Oncorhynchus mykiss) peripheral blood and splenic and an
86 ial infections of a natural vertebrate host, Oncorhynchus mykiss (rainbow trout), with variants of a
87 d first-generation hatchery steelhead trout (Oncorhynchus mykiss) reared in a common environment.
88 not benzocaine or MS-222; and rainbow trout (Oncorhynchus mykiss) showed no avoidance to the three ag
89 ss<30kDa (PF30) isolated from rainbow trout (Oncorhynchus mykiss) skin gelatin hydrolysates was encap
90 ly life stages of ammonotelic rainbow trout (Oncorhynchus mykiss), suggesting that the urea cycle may
91 (Salvelinus fontinalis), and rainbow trout (Oncorhynchus mykiss), suggesting that tolerant species m
92 erated recombinant C5a of the rainbow trout, Oncorhynchus mykiss (tC5a), and used fluoresceinated tC5
94 ark dose (BMD) assay for rainbow trout (RBT; Oncorhynchus mykiss) to derive transcriptomic points-of-
95 nerve of a teleost fish, the rainbow trout (Oncorhynchus mykiss) to determine what types of somatose
96 ranscriptomics on the CNSS of rainbow trout (Oncorhynchus mykiss) to establish: (1) how the CNSS resp
97 ur studies have revealed that rainbow trout (Oncorhynchus mykiss) use a novel strategy for the genera
98 ith early life stages of rainbow trout (RBT; Oncorhynchus mykiss) using benzo[a]pyrene (B[a]P) as the
99 sory receptors on the head of rainbow trout, Oncorhynchus mykiss, using extracellular recording from
104 aquatic ecosystems, juvenile rainbow trout (Oncorhynchus mykiss) were separately exposed to a mixtur
105 ress this, adult and juvenile Rainbow Trout (Oncorhynchus mykiss) were, respectively, exposed for 96
106 n (Acipenser fulvescens), and rainbow trout (Oncorhynchus mykiss), were selected to evaluate TFM redu
107 ed to the controversy is that rainbow trout (Oncorhynchus mykiss), which have served as the primary m
108 her blastomeres isolated from rainbow trout (Oncorhynchus mykiss) will incorporate and continue to de