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1 losses to the water mold Phytophthora spp. (Oomycetes).
2 cialized intercellular structures (fungi and oomycetes).
3 rolegnia spp. which are basal members of the oomycetes.
4 ies and catalogues the effector secretome of oomycetes.
5 in avirulence proteins from three different oomycetes.
6 t appear to be widespread and diverse in the oomycetes.
7 ent in a few eukaryotic lineages such as the Oomycetes.
8 ommunities that include bacteria, fungi, and oomycetes.
9 y used chemical fungicide for the control of oomycetes.
10 bacteria (including phytoplasma), fungi, and oomycetes.
11 on other microorganisms, including fungi and oomycetes.
12 sively extracellular and unique to fungi and oomycetes.
13 o new methods to suppress diseases caused by oomycetes.
14 gen-associated molecular patterns (PAMPs) in oomycetes.
15 ced by plant pathogenic bacteria, fungi, and oomycetes.
16 t the fungal kingdom, and in the fungus-like oomycetes.
17 e are shared with plant-associated fungi and oomycetes.
18 from the same or closely related species of oomycetes.
19 , SWEETs had not been identified in fungi or oomycetes.
20 ially destructive plant-associated fungi and oomycetes.
21 or future P450 annotations in newly explored oomycetes.
22 and 31 P450 subfamilies were newly found in oomycetes.
23 ee hundred and fifty-six P450s were found in oomycetes.
24 homology data revealed P450 family blooms in oomycetes.
25 ution of phytopathogenic traits in fungi and oomycetes.
26 oactivity against plant pathogenic fungi and oomycetes.
27 ribution; they occur in bacteria, fungi, and oomycetes.
28 independently in plant pathogenic fungi and oomycetes.
29 , protects plants against diseases caused by oomycetes.
30 infection structures of pathogenic fungi and oomycetes.
31 production and bioactivity against fungi and oomycetes.
32 ted for RxLR-effectors from plant pathogenic oomycetes.
33 ttern of cross-kingdom HGT between fungi and oomycetes.
34 to pathogen Phytophthora infestans and other oomycetes.
35 s is not the case for several subfamilies in oomycetes.
36 ved in the perception of bacteria, fungi and oomycetes.
37 e encoded by the genomes of plant pathogenic oomycetes.
38 e population structure within these obligate oomycetes.
39 or delivery are uncharacterized in fungi and oomycetes.
40 hogen of potato and a model organism for the oomycetes, a distinct lineage of fungus-like eukaryotes
41 diseases of plants and animals are caused by oomycetes, a group of eukaryotic pathogens important to
46 earch on eukaryotes such as animals, plants, oomycetes and fungi has shown that P450s profiles in the
47 her than the so-called Crinkler effectors of oomycetes and fungi, these effectors are encoded by othe
48 abidopsidis, which represents the kingdom of oomycetes and is phylogenetically distant from fungi, em
49 e monooxygenases (LPMOs) in plant pathogenic oomycetes and its role in plant infection by P. infestan
50 onal secreted proteins from plant pathogenic oomycetes and its similarity to a host-targeting signal
52 d nematodes), common host-targeting signals (oomycetes and protozoans) and specialized intercellular
53 d economical impact of the animal pathogenic oomycetes and review the recent advances in this emergin
55 The resource is focused on fungi, protists (oomycetes) and bacterial plant pathogens that have genom
57 acteria (including phytoplasmas), fungi, and oomycetes, and this tool should also be capable of gener
58 olite not previously shown to be produced by oomycetes, and two proteins with homology to vertebrate
61 sequences similar to those seen in fungi and oomycetes are also found in the animal kingdom, but rath
67 e, we report the identification of SWEETs in oomycetes as well as SWEETs and a potential SemiSWEET in
68 toplasm, consistent with the hypothesis that oomycetes, as is the case with bacteria and fungi, activ
69 keleton confers resistance against fungi and oomycetes, AtADF4 is not involved in resistance against
70 ggests a molecular "arms race" as plants and oomycetes attempt to achieve and evade detection, respec
71 by pathogenic microorganisms, such as fungi, oomycetes, bacteria, viruses, phytoplasma, and nematodes
72 are consistent with the hypothesis that some oomycetes became successful plant parasites by multiple
74 and Saccharomycotina, and in phytopathogenic Oomycetes, but neither other eukaryotes nor prokaryotes.
75 icals to control plant and animal pathogenic oomycetes cannot be used anymore; due to resistance in t
81 nce and composition of fungal, bacterial and oomycetes communities, as well as the prevalence of plan
82 rizontal gene transfers (HGTs) from fungi-to-oomycetes contributed to the evolution of plant-pathogen
84 challenge by pathogenic bacteria, fungi, and oomycetes, for whom they provide a resource of living sp
90 ve major pathogen groups (viruses, bacteria, oomycetes, fungi, and nematodes), has contributed to our
96 only form of extracellular SOD in fungi and oomycetes, in stark contrast to the extracellular Cu/Zn-
100 Current research is helping us learn how oomycetes interact with host and environment, understand
101 tion of RxLR effectors from plant pathogenic oomycetes into the cytoplasm of their host is currently
102 nt that the effector secretome of pathogenic oomycetes is more complex than expected, with perhaps se
103 largest group of translocated proteins from oomycetes is the RxLR effectors, defined by their conser
104 TIR-NBS-LRR R genes specifying resistance to oomycetes, is dependent on a functional EDS1 allele for
105 te having limited secondary metabolism, many oomycetes make chemicals for communicating within their
107 of LPMOs as virulence factors in pathogenic oomycetes opens up opportunities in crop protection and
111 on between Phytophthora pathogens, which are oomycetes, phylogenetically distinct from fungi, has bee
112 ased leaf susceptibility to infection by the oomycetes Phytophthora infestans and Phytophthora palmiv
120 rium catenoides, a free-living sister of the oomycetes, shows that these transfers largely converge w
124 ellular algae, the fish and plant pathogenic oomycetes, such as the potato blight Phytophthora, and t
125 , Chytridiomycetes, Thecofilosea (Cercozoa), Oomycetes, Syndiniales (Dinoflagellata) and Labyrinthulo
129 nse (ORR), which promotes resistance against oomycetes that infect through the epidermis, and the int
130 erved effector, secreted by plant pathogenic oomycetes, that co-opts a host Rab GTPase-activating pro
132 icroorganisms ranging from fungi, algae, and oomycetes to testate amoebozoans, and even cyanobacteria
133 ed by eukaryotic microbes, such as fungi and oomycetes, to host plants and contribute to the establis
135 ically diverse: viruses, bacteria, protozoa, oomycetes, true fungi, parasitic plants, and many types
138 ning and biochemical studies have shown that oomycetes, which belong to the kingdom Stramenopila, sec
140 quences and other sequenced plant pathogenic oomycetes with 91% of the hybrid assembly derived sequen
141 ering effectors, have emerged from comparing oomycetes with different genome characteristics, parasit
143 lacking chromalveolates such as ciliates and oomycetes would be explained by plastid loss in these li