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1 Osler ended his address on a more somber note, seemingly
2 Osler was not the only one on the verge of leaving as th
3 Osler's On Chorea uniquely captures the transition perio
4 n: Hodges and Smith, 1849), Bucknill (1882), Osler and Boller and Forbes reveal the changing attitude
5 in most instances, Osler and Charcot agreed, Osler used data from the new fields of genetics and bact
7 entists and clinicians including Haldane and Osler, and published important physiologic discoveries.
9 remarkable masterpiece, "The Agnew Clinic." Osler had been on the faculty of the University for 5 ye
11 Hereditary hemorrhagic telangiectasia (HHT; Osler-Weber-Rendu disease) affects 1 in 5000 persons, ma
12 classic dermatological manifestations of IE (Osler nodes, Janeway lesions, purpura, and conjunctival
15 duced excessive daytime sleepiness; the mean Osler change was +7.9 minutes (SEM 2.9), when compared w
16 tations (1894), and in this pithy monograph, Osler offered a particularly useful evaluation of Charco
17 Hereditary haemorrhagic telangiectasia, or Osler-Rendu-Weber (ORW) syndrome, is an autosomal domina
18 ic telangiectasia (HHT), also known as Rendu-Osler-Weber syndrome is a bleeding disorder that can aff
19 editary hemorrhagic telangiectasia, or Rendu-Osler-Weber disease, is an autosomal dominant disorder c
20 y hemorrhagic telangiectasia (HHT), or Rendu-Osler-Weber disease, is an autosomal dominant disorder o
21 ry hemorrhagic telangiectasia (HHT, or Rendu-Osler-Weber syndrome), clinical evaluations and genetic
23 l-time clinical professorships, an idea that Osler opposed but that eventually became the prevailing
26 this sesquicentennial of his birth, William Osler continues to serve as a model of excellence for th
28 inciples and Practice of Medicine by William Osler while on a summer vacation at Lake Liberty, New Yo
29 9, which was marked by an address by William Osler, the retirement of the renowned Professor of Surge
30 and not an art." The thoughts of Sir William Osler in 1892 reflect the view of medicine over the past
35 uding 39 (8.0%) with purpura, 13 (2.7%) with Osler nodes, 8 (1.6%) with Janeway lesions, and 3 (0.6%)