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1                                              P. intermedia alone could induce a similar neutrophil ph
2                                              P. intermedia-induced proliferation was T-cell specific
3                    Antibodies raised against P. intermedia type C fimbriae and against whole cells in
4 P. gingivalis, A. actinomycetemcomitans, and P. intermedia, was associated with pronounced volume los
5 importance, P. gingivalis, T. denticola, and P. intermedia were the microorganisms significantly asso
6 , and with coinfections of P. gingivalis and P. intermedia (OR 4.03); P. gingivalis and B. forsythus
7 ut inhibited the growth of P. gingivalis and P. intermedia after 72 hours.
8 indings indicate that both P. gingivalis and P. intermedia express heme-repressible proteinaceous hem
9 A. actinomycetemcomitans, P. gingivalis, and P. intermedia in subgingival plaque, at levels above the
10 A. actinomycetemcomitans, P. gingivalis, and P. intermedia.
11 intermedia 27, another clinical isolate, and P. intermedia 25611, the type strain, were not found to
12 esent results suggest that periodontitis and P. intermedia are associated with severe asthma.
13 domonas maltophilia, Strept, pneumoniae, and P. intermedia were the predominant organisms detected an
14                  Anti-P. gingivalis and anti-P. intermedia antibody concentrations were positively as
15               Serum anti-P. gingivalis, anti-P. intermedia, and anti-F. nucleatum antibody concentrat
16 d as much hemin in the absence of RSA as did P. intermedia.
17 y and greater heme storage capacity than did P. intermedia, suggesting that the former would be ecolo
18 nomycetemcomitans, and 14% were positive for P. intermedia.
19 ections with P. gingivalis, T. forsythensis, P. intermedia, and C. rectus were associated with an inc
20 ycetemcomitans, P. gingivalis, T. forsythia, P. intermedia, and total bacteria significantly decrease
21 elaninogenicus, F. nucleatum, P. gingivalis, P. intermedia, S. mutans, S. sanguis, Selenomonas sputig
22                               P. gingivalis, P. intermedia, T. forsythia, and T. denticola were more
23 n teeth for Parvimonas micra, P. gingivalis, P. intermedia, T. forsythia, and Treponema denticola.
24  the groups were observed for P. gingivalis, P. intermedia, Veillonella, and Capnocytophaga, with the
25 tion of eukaryotic cells with AdpF increased P. intermedia 17 internalization by 5- and 10-fold for H
26 la, A. actinomycetemcomitans, P. nigrescens, P. intermedia, B. forsythus, and P. gingivalis in 38% to
27  any orange-complex pathogens (F. nucleatum, P. intermedia, and C. rectus), total cancer risk (HR = 1
28                               The ability of P. intermedia to stimulate CD4(+)-T-cell proliferation w
29 rAdpF plays a role in the internalization of P. intermedia 17 by a variety of host cells.
30 4 degreesC) inhibited the internalization of P. intermedia 17.
31                               High levels of P. intermedia were found in association with the presenc
32 orse periodontal status and higher levels of P. intermedia, E. corrodens, F. nucleatum, and IL-1beta
33 ens MH1 and 2) the cell-free cell lysates of P. intermedia MH2.
34 ed from the index teeth, and the presence of P. intermedia, P. gingivalis, A. actinomycetemcomitans,
35 that AdpC is an important invasin protein of P. intermedia 17.
36 rovides evidence that at least one strain of P. intermedia can invade an oral epithelial cell line an
37 collectively suggest that certain strains of P. intermedia can activate Vbeta-specific T cells in a m
38                        Streptococcus oralis, P. intermedia, and Selenomonas noxia were elevated in sa
39  that the endodontic pathogens, particularly P. intermedia, can efficiently disable and kill infiltra
40 tion of A. actinomycetemcomitans, C. rectus, P. intermedia/nigrescens, E. corrodens, P. micros, Capno
41 control E. coli BL21 cells expressing a sham P. intermedia 17 protein.
42         Under these conditions, heme-starved P. intermedia cells (two strains) expressed a single bin
43          Among the bacterial strains tested, P. intermedia strain 17, a clinical isolate, induced the
44                    Finally, cells exposed to P. intermedia 17 internalized the bacteria more readily
45 Vbeta12, and Vbeta17 expanded in response to P. intermedia strain 17.
46 uman oral epithelial cell line (KB), whereas P. intermedia 27, another clinical isolate, and P. inter
47 te a new virulence mechanism associated with P. intermedia.