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1                                              P. multocida and A. actinomycetemcomitans are among the
2       The ompH gene was distributed among 15 P. multocida serotypes and strain CU.
3                                            A P. multocida strain that lacked a functional pnhA gene,
4                                     Although P. multocida lacks the genes for the two earliest steps
5  porin of strain X-73 and is conserved among P. multocida somatic serotypes.
6 90 was similar to that for H. influenzae and P. multocida as well as for other known homologues.
7 ida, i.e., P. multocida subsp. multocida and P. multocida subsp. septica.
8 stimated the median duration of H. somni and P. multocida carriage at 14.8 (CI(95%): 10.6-20.9) and 5
9 us 17%, respectively) (P < 0.05, Z test) and P. multocida subsp. multocida more often associated with
10                   All strains categorized as P. multocida subsp. multocida displayed either the group
11                    All strains identified as P. multocida subsp. septica were positive for alpha-Glu
12 to identify putative OMPs from two available P. multocida genomes: those of avian strain Pm70 and por
13 lon ( o ), fifty percent sensitive bacteria (P. multocida) killing effect ( Ls50 ), fifty percent of
14 isons of 1,197 orthologous sequences between P. multocida, Haemophilus influenzae, and Escherichia co
15 imilar to haemorrhagic septicaemia caused by P. multocida.
16 the severity of turbinate atrophy induced by P. multocida compared with that occurring in pigs kept i
17                    Neuraminidase produced by P. multocida A:3 was purified by a combination of salt f
18               The 965-residue enzyme, called P. multocida chondroitin synthase (pmCS), is 87% identic
19 nome sequence contains many of the canonical P. multocida virulence factors associated with a range o
20         Overall, the scenarios that decrease P. multocida, including resistant P. multocida did not c
21  days respectively, and found higher density P. multocida carriage was associated with slower clearan
22 -weight neuraminidases produced by different P. multocida serotypes are quite similar.
23 y provide reliable means for differentiating P. multocida subsp. multocida from P. multocida subsp. s
24 viously shown to be a major immunogen during P. multocida infection in rabbits.
25 l-negative subspecies of P. multocida, i.e., P. multocida subsp. multocida and P. multocida subsp. se
26  10 ppm or greater, in the absence of either P. multocida or Bordetella bronchiseptica, induced a mil
27            A majority of these genes encoded P. multocida proteins that were involved in either trans
28 g that, upon exposure to a rich environment, P. multocida immediately begins to turn on many energy-i
29 phates, may also play a role in facilitating P. multocida pathogenicity in the host.
30 ubsp. septica; the 16S rDNA is identical for P. multocida subsp. multocida and Pasteurella multocida
31 using, high carriage rates were observed for P. multocida (95%), and H. somni (75%), while fewer calv
32 entiating P. multocida subsp. multocida from P. multocida subsp. septica, particularly in strains tha
33 evel to the hyaluronan synthase, pmHAS, from P. multocida Type A.
34             In summary, the HA synthase from P. multocida, a Gram-negative bacterium, has kinetic opt
35 ces an effective response against homologous P. multocida challenge.
36 ponse to low iron conditions was analyzed in P. multocida using whole-genome microarrays.
37  primary sialic acid cytidylyltransferase in P. multocida.
38  and transport increased 2.1- to 7.7-fold in P. multocida during the first 2 h of growth under iron-l
39 ed the number of putative OMPs identified in P. multocida and allowed these OMPs to be identified wit
40 d to inorganic or organic sources of iron in P. multocida.
41 anisms of iron acquisition and metabolism in P. multocida and other gram-negative bacteria.
42 ified, undergo transcriptional regulation in P. multocida in response to growth in minimal medium and
43  h apart) showed low economic costs, minimal P. multocida, and moderate effects on resistant E. coli.
44 fied by photoaffinity labeling of the native P. multocida HA synthase with azido-UDP sugar analogs.
45              We studied 35 dulcitol-negative P. multocida isolates from infected dog and cat bite wou
46 hat a HA synthase, PmHAS, from Gram-negative P. multocida bacteria polymerizes the HA chain by the ad
47                   Toxigenic and nontoxigenic P. multocida isolates cannot be differentiated by morpho
48                           On average, 12% of P. multocida genes were differentially expressed under a
49  (REP-PCR) to characterize 20 strains (14 of P. multocida subsp. multocida and 6 of P. multocida subs
50 was also found in a high proportion (36%) of P. multocida strains isolated from non-PDNS cases.
51 14 of P. multocida subsp. multocida and 6 of P. multocida subsp. septica; the 16S rDNA is identical f
52 unction and may contribute to the ability of P. multocida to colonize and persist on vertebrate mucos
53 hesins that are important in the adhesion of P. multocida to fibronectin.
54 etate (PMA) further enhanced the adhesion of P. multocida to TPBM.
55                    Cryopreserved aliquots of P. multocida were administered via an endotracheal tube.
56 n is on the cell surface and that binding of P. multocida to fibronectin is almost completely inhibit
57 ace proteoglycan (CD44) decreased binding of P. multocida.
58 on, each group received an i.n. challenge of P. multocida.
59                          The exacerbation of P. multocida colonization by ammonia was restricted to t
60 (PMT) and a potassium thiocyanate extract of P. multocida (CN) in combination and (ii) to evaluate th
61 h either exotoxin or thiocyanate extracts of P. multocida induces partial protection in rabbits.
62 LP techniques enable rapid fingerprinting of P. multocida isolates from multiple avian species and en
63 h sera from 62 rabbits from colonies free of P. multocida, was 92%.
64 e gene, nanH, from a fowl cholera isolate of P. multocida.
65                             Most isolates of P. multocida produce sialidase activity, which may contr
66 tor of genetic relatedness among isolates of P. multocida.
67                              The kinetics of P. multocida colonization were established by testing sa
68 ted by studying the colonization kinetics of P. multocida enhanced by ammonia and comparing them with
69 otype was observed for the limited number of P. multocida isolates.
70 eriod of ammonia exposure, and the number of P. multocida organisms colonizing the upper respiratory
71  of turbinate degeneration and the number of P. multocida organisms isolated from the nasal epitheliu
72 correlation was found between the numbers of P. multocida organisms isolated from the nasal cavity an
73  was used to capture a 37-kDa polypeptide of P. multocida serotype A:12 in an EIA to detect antibodie
74 ined from cell-free membrane preparations of P. multocida.
75 e most the treatment costs and prevalence of P. multocida resistance in the lungs, while 5 mg/kg as t
76 embrane proteins of the membrane proteome of P. multocida were identified.
77 data provide evidence of host specificity of P. multocida clones.
78 ques, we cloned from a toxinogenic strain of P. multocida the entire toxA gene, encoding the 1,285-am
79 mucopolysaccharide of serogroup A strains of P. multocida recognizes an isoform of CD44 expressed on
80 r results demonstrate that unique subsets of P. multocida genes are expressed in response to differen
81  distinction between different subspecies of P. multocida can be made more easily and accurately.
82 ween the two dulcitol-negative subspecies of P. multocida, i.e., P. multocida subsp. multocida and P.
83 ia facilitates the growth and/or survival of P. multocida within the upper respiratory tract of the p
84 nual real-time reproductive number (R(t)) of P. multocida, previously calculated for eiders at Mitivi
85 da subsp. gallicida but differs from that of P. multocida subsp. septica) isolated from various anato
86             Isolation of a single variant of P. multocida from tissues of pigs with PDNS warrants fur
87 ntrast, ammonia had only a limited effect on P. multocida colonization at the tonsil.
88  compared for their effects on resistance on P. multocida and commensal E. coli: 12.5 mg/kg and 7.5 m
89                                     Purified P. multocida A:3 neuraminidase was employed to immunize
90 e effectiveness of vaccination with purified P. multocida toxin (PMT) and a potassium thiocyanate ext
91                      Exposure to recombinant P. multocida toxin (rPMT) causes phospholipase C-mediate
92 t decrease P. multocida, including resistant P. multocida did not coincide with those that decrease r
93   Collectively, these findings indicate that P. multocida adhesion to TPBM is mediated by capsular HA
94 nfluenzae, and Escherichia coli suggest that P. multocida and H. influenzae diverged approximately 27
95 from the B. bronchiseptica alcA gene and the P. multocida toxA gene.
96 oson insertional mutagenesis to identify the P. multocida HA synthase, the enzyme that polymerizes HA
97 ulting antiserum reduced the activity of the P. multocida A:3 enzyme by 40.3%.
98 e apparent Michaelis constants, K(M), of the P. multocida HA synthase for UDP-N-acetylglucosamine and
99                                 Overall, the P. multocida sequence is not very similar to the other k
100 e it had a low G + C content relative to the P. multocida genome.
101 and acyl-CoA binding affinities, whereas the P. multocida and H. influenzae proteins showed only weak
102 rt 3 patients who developed life-threatening P. multocida respiratory tract infections after providin
103 otype A:12 in an EIA to detect antibodies to P. multocida.
104 s in inducing protective mucosal immunity to P. multocida in rabbits.
105 munity approximated using antibody titers to P. multocida in both sexes was likely a key driver for t
106 D were inoculated with 1.4 x 10(8) toxigenic P. multocida organisms given by the intranasal route.
107 detection of B. bronchiseptica and toxigenic P. multocida that can be performed with a single colony
108 for a rapid direct specimen assay, toxigenic P. multocida was recovered efficiently from inoculated s
109 acteriophages from three different toxigenic P. multocida strains had similar but not identical restr
110 ification protocol is specific for toxigenic P. multocida and can detect fewer than 100 bacteria.
111  demonstrated in spent medium from toxigenic P. multocida isolates.
112 results show that PCR detection of toxigenic P. multocida directly from clinical swab specimens shoul
113 R for accurate, rapid detection of toxigenic P. multocida from swabs was investigated.
114 rked effect on the colonization of toxigenic P. multocida in the nasal cavities of pigs, which result
115  found to be 60% less than that of wild-type P. multocida, but the growth rate of ACP13 and its sensi
116 l presentation and the taxonomic group, with P. multocida subsp. septica more often associated with w
117 n curves of sera from rabbits immunized with P. multocida serotype A:3 or A:12 coincided, indicating
118 ined with sera from 56 rabbits infected with P. multocida, was 98%.
119 capability to identify rabbits infected with P. multocida.

 
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