ライフサイエンスコーパス: P. syringae

1 P. syringae 508 was also surveyed for the presence of ef

2 P. syringae carrying hopW1-1 have restricted host range

3 P. syringae effector proteins encounter a pH gradient as

4 P. syringae pv. tomato DC3000 HrpP has a C-terminal, put

5 P. syringae was able to attach and extensively colonize

6 P. syringae was defective in porous-paper colonization w

7 Both Sanger sequencing of 50 P. syringae pv. atrofaciens mutant clones for each genot

8 A P. syringae hopD1 mutant and ETI-inducing P. syringae st

9 A P. syringae pv tomato DC3000 mutant lacking about one-th

10 In 1991, a P. syringae pathovar tomato (Pst) strain, DC3000, was re

11 In addition, a P. syringae strain defective in coronatine synthesis sho

12 abidopsis eukaryotic activator of AvrRpt2, a P. syringae effector that is a cysteine protease.

13 d P. fluorescens heterologously expressing a P. syringae TTSS and AvrPto1(PtoJL1065).

14 ome of Por(1_6) is the first sequenced for a P. syringae isolate that is a pathogen of monocots, and,

15 ctive oxygen burst, and enhances growth of a P. syringae hrpA mutant that fails to secrete effectors.

16 oB-mediated anti-PCD activity, and abrogates P. syringae pathogenesis of susceptible tomato plants.

17 amount of NAD(P) leaking into the ECC after P. syringae pv. tobacco DC3000/avrRpt2 infection is suff

18 howed significantly reduced expression after P. syringae interference.

19 nce gene-mediated inducible defenses against P. syringae (and possibly other pathogens) by playing a

20 ent that orchestrates plant defenses against P. syringae.

21 t not Pto possessed greater immunity against P. syringae than tomatoes lacking Prf.

22 stemic resistance response in plants against P. syringae pv tomato DC3000.

23 oteins and contributed to resistance against P. syringae pv tomato.

24 ce of PRR2 in plant immune responses against P. syringae and suggest a novel function for this partic

25 NLR induces strong defense responses against P. syringae and X. campestris The P. syringae T3SE HopZ1

26 Plants in turn defend themselves against P. syringae by activating the salicylic acid (SA)-mediat

27 a constitutive GFP marker to account for all P. syringae cells on a leaf.

28 that RhpRS is a global regulator that allows P. syringae to sense and respond to environmental change

29 s, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A restored HR elicitati

30 a gene in P. syringae pv. syringae B728a and P. syringae pv. tomato DC3000, termed phcA, that has hom

31 inding protein (MBP) in Escherichia coli and P. syringae pv. syringae.

32 Pseudomonas syringae pv maculicola (Psm) and P. syringae pv tomato (Pst) but not the avirulent strain

33 2 is transcriptionally upregulated by SA and P. syringae, enhances SA biosynthesis and SA signalling

34 -host pathogens P. syringae pv. syringae and P. syringae pv. tabaci.

35 The interaction between A. thaliana and P. syringae DC3000 highly induced the secretion of sever

36 athogens Pseudomonas syringae pv. tomato and P. syringae pv. maculicola.

37 athogens Pseudomonas syringae pv. tomato and P. syringae pv. maculicola.

38 onella enterica, it was effective in another P. syringae strain and Ralstonia solanacearum.

39 ore Pseudomonas genome and 365 ORFs that are P. syringae specific.

40 s a selective advantage for microbes such as P. syringae that are adapted to maximally exploit cholin

41 The HopS2 effector possessed atypical P. syringae TTSS N-terminal characteristics and was tran

42 of cytosolic calcium triggered by avirulent P. syringae was compromised in crt1-2 crh1-1 plants, but

43 were compromised for resistance to avirulent P. syringae and more susceptible to virulent strains tha

44 t DC3000) and avirulent (Pst DC3000 AvrRPM1) P. syringae strains, conserving typical hypersensitive r

45 ulture and injected into plant cells by both P. syringae and Q8r1-96 T3SSs.

46 WRKY60 made plants more susceptible to both P. syringae and B. cinerea.

47 AvrPtoB did not prevent the HR activated by P. syringae pv. tomato DC3000 + avrB, avrRpm1, avrRps4 o

48 f exogenous auxin enhances disease caused by P. syringae strain DC3000.

49 ssion at specific promoter configurations by P. syringae.

50 Expression of HDA19 is also induced by P. syringae, and the stability of its induced transcript

51 ishing plant protection against infection by P. syringae acting on the activity of the SA pathway.

52 resistance signaling following infection by P. syringae expressing the Cys protease Type III effecto

53 he main characterized surfactant produced by P. syringae.

54 The protein HrpJ is secreted by P. syringae and is required for a fully functional T3SS.

55 A tyrosine phosphatase, HopAO1, secreted by P. syringae, reduces EFR phosphorylation and prevents su

56 s being the only siderophores synthesized by P. syringae pv. syringae B728a.

57 arched for homologs to 44 known or candidate P. syringae type III effectors and two effector chaperon

58 resistance of Arabidopsis against compatible P. syringae, possibly by protecting leaves from the path

59 t increased susceptibility toward compatible P. syringae and possess heightened levels of markers of

60 Here, we show that a conserved P. syringae virulence protein, HopM1, targets an immunit

61 COR and the critical P. syringae type III effector HopM1 target distinct sign

62 In counter defense, P. syringae pathovars evade host immunity by using BGAL1

63 gher in leaves challenged with COR-deficient P. syringae or in the more resistant JA receptor mutant

64 eins restores virulence of a HopM1-deficient P. syringae mutant, providing a link between HopM1 and t

65 y divergent protein in the T3SS of different P. syringae pathovars, hrpP from P. syringae pv. syringa

66 r this tail; this function was confirmed for P. syringae BetT using deletion derivatives.

67 t high temperature, HopI1 is dispensable for P. syringae pathogenicity, unless excess Hsp70 is provid

68 HopO1-1 is required for P. syringae to spread locally to neighboring tissues dur

69 3000 effectors and a central requirement for P. syringae pathogenesis.

70 The primary role for P. syringae type-III effectors is the suppression of pla

71 gesting an epidemic population structure for P. syringae.

72 of effectors and related T3SS substrates for P. syringae pv. tomato DC3000 and three other sequenced

73 ins from P. syringae pv. tomato, and 19 from P. syringae pv. phaseolicola race 6.

74 contributes to virulence after delivery from P. syringae in leaves of susceptible soybean plants, and

75 We previously identified hopA1 from P. syringae pv syringae strain 61 as an avirulence gene

76 f different P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseoli

77 0 PFPs along with pDC3000A and pDC3000B from P. syringae pv. tomato encoded a type IVB T4SS (tra syst

78 from P. syringae pv. syringae, pPh1448B from P. syringae pv. phaseolicola, and pPMA4326A from P. syri

79 yringae pv. phaseolicola, and pPMA4326A from P. syringae pv. maculicola encoded a type IVA T4SS (VirB

80 Twelve PFPs along with pPSR1 from P. syringae pv. syringae, pPh1448B from P. syringae pv.

81 l criteria defined 29 type III proteins from P. syringae pv. tomato, and 19 from P. syringae pv. phas

82 omparative genomic analyses to elucidate how P. syringae subverts the attack and defense responses of

83 a detailed mechanistic understanding of how P. syringae transitions from reliance on exogenously der

84 nome, high-throughput screen for identifying P. syringae type III effector genes.

85 rnitine/choline family transporter (BCCT) in P. syringae pv. tomato strain DC3000 that mediates the t

86 homologues occur at very low frequencies in P. syringae populations on A. thaliana.

87 Here we characterize a gene in P. syringae pv. syringae B728a and P. syringae pv. tomat

88 The fitness contributions of 4,296 genes in P. syringae pv. syringae B728a were determined by genome

89 zed the hopO1-1, hopS1, and hopS2 operons in P. syringae pv. tomato DC3000; these operons encode thre

90 * hopAS1 is broadly present in P. syringae strains, contributes to virulence in tomato,

91 Three alleles are known to be present in P. syringae, with HopZ1a most resembling the ancestral a

92 d stringent response plays a central role in P. syringae virulence and survival and indicated that pp

93 e data indicate that HrpJ has a dual role in P. syringae: inside bacterial cells HrpJ controls the se

94 enes coregulated with the Hrp T3SS system in P. syringae pv. tomato DC3000 have predicted lytic trans

95 loring multiple aspects of the Hrp system in P. syringae.

96 To identify additional RhpR targets in P. syringae, we performed chromatin immunoprecipitation

97 e global role of rppH in thermoregulation in P. syringae, RNA sequencing was used to compare the tran

98 1 supported increased growth of ETI-inducing P. syringae strains compared with wild-type Arabidopsis.

99 A P. syringae hopD1 mutant and ETI-inducing P. syringae strains exhibited enhanced growth on Arabido

100 In contrast, ETI-inducing P. syringae strains were unable to overcome PTI-induced

101 Much existing research into P. syringae-plant interactions has focused on the molecu

102 -LRR genes RPS2, RPM1, and RPS5 and isogenic P. syringae strains expressing single corresponding avir

103 In the nonpathogenic isolate P. syringae 508 the genomic region that in pathogenic P.

104 a variety of additional genes encoding known P. syringae virulence factors.

105 r TNL class is represented by a single known P. syringae resistance gene, RPS4.

106 phcA is conserved in many P. syringae strains, but is absent in one of the major c

107 Moreover, P. syringae mutants that were deficient in the uptake of

108 cks early MAMP signaling and enables nonhost P. syringae growth.

109 We determined the charge state of nonviable P. syringae as a function of pH by monitoring the degree

110 Analyses of P. syringae pv. tomato DC3000 mutants indicated that bot

111 at is supported by the common association of P. syringae with plants and the widespread production of

112 In the case of P. syringae, growth on a nata1 mutant is reduced compare

113 and is carried in the functional cluster of P. syringae pv. syringae 61 hrp genes cloned in cosmid p

114 open reading frames (ORFs) within the EEL of P. syringae pv. tomato DC3000.

115 :acyl carrier protein transacylase (FabD) of P. syringae was overproduced and shown to catalyze malon

116 vrRpt2 may be among the virulence factors of P. syringae that modulate host auxin physiology to promo

117 tion was visualized via dual fluorescence of P. syringae cells harboring a transcriptional fusion of

118 erexpressing plants supported more growth of P. syringae and developed more severe disease symptoms t

119 us, flagellin perception restricts growth of P. syringae strains on N. benthamiana.

120 Conversely, growth of P. syringae strains was reduced in plants expressing a c

121 g serine(s) in two other effectors, HopZ3 of P. syringae and PopP2 of Ralstonia solanacerum, also abo

122 fector as reporter revealed the inability of P. syringae 508 to translocate effectors into plant cell

123 gh-frequency mutations allowing infection of P. syringae pv. atrofaciens.

124 By analyzing the interaction of P. syringae mutants with Arabidopsis thaliana mutants, w

125 tor-triggered immunity in the interaction of P. syringae pv tomato DC3000 and N. benthamiana.

126 lenge inoculation with a virulent isolate of P. syringae.

127 A shotgun library of P. syringae was screened in the mutant E. coli by growin

128 The localization of P. syringae bioreporter cells to the surface and interce

129 The enzymatic activities of most of P. syringae effectors and their targets remain obscure.

130 resulted in the loss of swarming motility of P. syringae pv. tomato DC3000 on medium containing a low

131 onas fluorescens, a TTSS-deficient mutant of P. syringae pv. tabaci, or flg22 (a flagellin-derived pe

132 The pangenome of P. syringae encodes 57 families of effectors injected by

133 ed and eight known PFPs from 12 pathovars of P. syringae, which belong to four genomospecies.

134 III effector proteins from two pathovars of P. syringae.

135 ular traits and characteristic phenotypes of P. syringae Lz4W that enable life at low temperatures.

136 However, the presence of P. syringae carrying avrPphB is probably insufficient to

137 ce factor, coronatine, during progression of P. syringae infection of Arabidopsis thaliana.

138 rther work needed on the psychrotolerance of P. syringae.

139 role in Arabidopsis NHR to a broad-range of P. syringae strains.

140 esistance signaling following recognition of P. syringae DC3000-AvrRpt2 by Arabidopsis.

141 of the effectors comprising the secretome of P. syringae pv tomato DC3000 led to the identification o

142 s study, we report on the genome sequence of P. syringae pv. phaseolicola isolate 1448A, which encode

143 as coinoculated with the avirulent strain of P. syringae pv phaseolicola into tobacco leaves.

144 roteins were delivered by the RW60 strain of P. syringae pv. phaseolicola.

145 by PstAvr, infection by a virulent strain of P. syringae, and low temperature.

146 ced disease severity to a virulent strain of P. syringae, suggesting a role of ATT1 in disease resist

147 In basal resistance to virulent strains of P. syringae and H. arabidopsidis, PAD4 functions togethe

148 nces basal resistance to virulent strains of P. syringae and the oomycete Phytophthora sojae.

149 al phenotypic differences between strains of P. syringae is the range of plant hosts they infect.

150 -depleted plants to nonpathogenic strains of P. syringae supports a defense-promoting role for Hsp70.

151 ible, non-host and non-pathogenic strains of P. syringae.

152 ere we report the existence of a subgroup of P. syringae isolates that do not cause disease on any pl

153 been determined and is compared with that of P. syringae pv. tomato DC3000 (Pst DC3000).

154 n the temperature-dependent transcriptome of P. syringae, affecting the expression of 569 genes at ei

155 in of the Xcv AvrBs2 protein via the TTSS of P. syringae.

156 t (p)ppGpp is required for full virulence of P. syringae.

157 GABA may have multiple effects on P. syringae-plant interactions, with elevated levels inc

158 culation with P. syringae DC3000(avrRpm1) or P. syringae DC3000(avrRpt2) induces differential decreas

159 by RLs following challenge by B. cinerea or P. syringae pv tomato.

160 iotic and biotic stresses such as drought or P. syringae infection induced similar increase.

161 defenses against Manduca sexta herbivory or P. syringae pv tomato DC3000 infection rates.

162 influence resistance against virulent Pst or P. syringae pv. maculicola (Psm) pathogens.

163 produced by immunization with Shewanella or P. syringae cells bound to B. anthracis spores but not t

164 common N-terminal characteristic from other P. syringae type III secreted substrates increased HopS2

165 between BAK1 and HopF2 and between two other P. syringae effectors, AvrPto and AvrPtoB, was confirmed

166 or repertoire of the sequenced bean pathogen P. syringae pv. syringae (Psy) B728a using bioinformatic

167 lso enhanced the growth of the host pathogen P. syringae pv tabaci by increasing nutrient efflux into

168 enes in the repertoire of the model pathogen P. syringae pv. tomato DC3000 were deleted to produce po

169 rains closely related to the tomato pathogen P. syringae pv. tomato (Pto), including strains isolated

170 ic and most likely evolved from a pathogenic P. syringae ancestor through loss of the T3SS.

171 Pseudomonas syringae sustains but pathogenic P. syringae suppresses early MAMP (microbe-associated mo

172 etions of avrPto and avrPtoB from pathogenic P. syringae reduce its virulence.

173 ae 508 the genomic region that in pathogenic P. syringae strains contains the hrp-hrc cluster coding

174 d are sufficient to transform non-pathogenic P. syringae strains into virulent pathogens in immunodef

175 ot affect plant susceptibility to pathogenic P. syringae bacteria; conversely, expression of the cons

176 ere highly susceptible to non-host pathogens P. syringae pv. syringae and P. syringae pv. tabaci.

177 with a phylogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a strong degree

178 ified a subset of putatively phytopathogenic P. syringae in a manner causally consistent with observe

179 toB acts as a virulence protein by promoting P. syringae pv. tomato growth and enhancing symptoms ass

180 es to disease resistance in response to Pto (P. syringae pathovars tomato) DC3000(avrB), but not agai

181 This purified P. syringae protein was determined to catalyze the epoxi

182 (localized to chloroplasts) greatly reduces P. syringae-induced PCD, suggesting a pro-PCD role for m

183 Repression of auxin signaling restricts P. syringae growth, implicating auxin in disease suscept

184 induce systemic susceptibility to secondary P. syringae infection in the host plant Arabidopsis thal

185 cmaL is found in all other sequenced P. syringae strains with coronatine biosynthesis genes.

186 type III effector suites from two sequenced P. syringae pathovars and show that type III effector pr

187 Several P. syringae effectors require accessory proteins called

188 cens was used to test the ability of several P. syringae pv. tomato DC3000 effectors for their abilit

189 ype plants; however, responses to A. solani, P. syringae, or M. sexta were similar to the wild-type p

190 Intriguingly, some P. syringae strains also secrete the virulence factor sy

191 While some P. syringae type III effectors were acquired recently, o

192 s from the closely related pathogenic strain P. syringae pv. syringae B728a, but none were detected.

193 Luminescence of luxCDABE-tagged P. syringae allows rapid and convenient quantification o

194 Strikingly, all tested P. syringae strains that are pathogenic in Arabidopsis c

195 We conclude that P. syringae strains may have evolved large effector repe

196 In this study, we found that P. syringae pv. tomato strain DC3000 was distinct from m

197 ical evidence supporting the hypothesis that P. syringae pv. syringae B728a produces both of these si

198 We show that P. syringae-elicited SIS is caused by the production of

199 ssing at low temperature, and speculate that P. syringae Lz4W can also synthesize glycerol to maintai

200 These results suggest that P. syringae has evolved to survive in relatively choline

201 Our results suggest that P. syringae has the potential to utilize phcA to acquire

202 These results suggest that P. syringae type III effectors and coronatine act by aug

203 This suggests that P. syringae 508 supplemented with a T3SS could be used t

204 The P. syringae effector AvrRpt2, which targets RPM1 INTERAC

205 The P. syringae pv. tomato DC3000 effector HopF2 suppresses

206 The P. syringae pv. tomato DC3000 HopK1 type-III effector wa

207 The P. syringae pv. tomato OpuC transporter had a high affin

208 The P. syringae pv. tomato OpuC transporter was more closely

209 The P. syringae TTSS is encoded by hrp-hrc genes that reside

210 The P. syringae TTSS is encoded by the hrp-hrc gene cluster.

211 The P. syringae-specific HopI1 effector has a putative chlor

212 Adding a plasmid-encoded T3SS and the P. syringae pv. syringae 61 effector gene hopA1 increase

213 n somewhat differently than YscP because the P. syringae T3SS pilus likely varies in length due to di

214 ahlR regulon presence within and beyond the P. syringae pan-genome.

215 st that chloroplast Hsp70 is targeted by the P. syringae HopI1 effector to promote bacterial virulenc

216 ure and translocated into plant cells by the P. syringae pv. tomato DC3000 TTSS.

217 order to be effectively translocated by the P. syringae T3SS.

218 es against P. syringae and X. campestris The P. syringae T3SE HopZ1a is an acetyltransferase that ace

219 three protein classes cooperate to form the P. syringae T3SS translocon.

220 rotein (PSPTO_2145), which is located in the P. syringae pyoverdine cluster.

221 one of the major clades, which includes the P. syringae pathovar phaseolicola.

222 peron from Photorhabdus luminescens into the P. syringae chromosome under the control of a constituti

223 I effector genes, which are orthologs of the P. syringae effector genes hopAA1-1 and hopM1, as well a

224 e complex is activated by recognition of the P. syringae effectors AvrPto and AvrPtoB.

225 Informatic analysis of the P. syringae genome suggests only one putative non-heme i

226 for locations associated with binding of the P. syringae IS sigma factor PSPTO_1203.

227 Por(1_6) helps to define an expansion of the P. syringae pan-genome, a corresponding contraction of t

228 Here, the functional significance of the P. syringae T3SS substrate compositional patterns was te

229 two distinct levels in the regulation of the P. syringae TTSS: regulation of assembly of the secreton

230 anslationally modified after delivery of the P. syringae type III effectors AvrRpm1, AvrB, or AvrRpt2

231 structural and regulatory components of the P. syringae type III secretion system (T3SS), essential

232 We further show that the P. syringae is able to use N. crassa as a sole nutrient

233 We also show that the ability of this P. syringae strain to block antimicrobial exudation is d

234 nduced resistance to H. arabidopsidis and to P. syringae pv tomato whereas jasmonic acid is essential

235 ell as to the fungus Botrytis cinerea and to P. syringae.

236 B only rarely confers a virulence benefit to P. syringae on A. thaliana.

237 g the importance of extracellular choline to P. syringae on leaves.

238 for the superior osmoprotection conferred to P. syringae by choline over glycine betaine when these c

239 HopD1 contributes to P. syringae virulence in Arabidopsis and reduces effecto

240 * HopD1 contributes to P. syringae virulence in Arabidopsis and reduces effecto

241 HopD1 contributes to P. syringae virulence in part by targeting NTL9, resulti

242 * HopD1 contributes to P. syringae virulence in part by targeting NTL9, resulti

243 uced accumulation of Pip in leaves distal to P. syringae inoculation, they display a considerable sys

244 tibility of WRKY33-over-expressing plants to P. syringae is associated with reduced expression of the

245 re more susceptible than wild-type plants to P. syringae.

246 gae pv maculicola 1 (RPM1) and Resistance to P. syringae 2 (RPS2) disease resistance proteins.

247 nd flg22 PAMP/DAMPs, including resistance to P. syringae and B. cinerea, production of reactive oxyge

248 RIN4b abrogates RPG1-B-derived resistance to P. syringae expressing AvrB.

249 nea) mediates species-specific resistance to P. syringae expressing the avirulence protein AvrB, simi

250 Arabidopsis increased disease resistance to P. syringae Expression of CRK28 in Nicotiana benthamiana

251 ced plant defenses, conferring resistance to P. syringae infection.

252 ng male sterility and enhanced resistance to P. syringae infection.

253 ana benthamiana did not confer resistance to P. syringae pv. tabaci (Pta) expressing avrPto or avrPto

254 vely, of the variance of basal resistance to P. syringae pv. tomato DC3000 in the Col-0 x Fl-1 F(2) p

255 expression of WRKY18 enhanced resistance to P. syringae, its coexpression with WRKY40 or WRKY60 made

256 expression results in enhanced resistance to P. syringae.

257 upted also compromised disease resistance to P. syringae.

258 ural variation that conditions resistance to P. syringae/hopW1-1.

259 s overexpressing IOS1 were more resistant to P. syringae and demonstrated a primed PTI response.

260 mutant were substantially more resistant to P. syringae but more susceptible to B. cinerea than wild

261 ed FLS2 signaling, and are more resistant to P. syringae.

262 reduced in the pbs3-1 mutant in response to P. syringae (avrRpt2) infection, free SA was elevated.

263 nerate a full disease resistance response to P. syringae expressing this type III effector.

264 es in Arabidopsis tissues and in response to P. syringae infection.

265 SA signalling responses; e.g. in response to P. syringae, PRR2 induces the production of SA and the a

266 sis of quantitative variation in response to P. syringae.

267 f three aspects of A. thaliana's response to P. syringae: symptom severity, bacterial population size

268 ween defense pathways mediating responses to P. syringae and necrotrophic pathogens.

269 we analyzed Arabidopsis defense responses to P. syringae through differential coexpression analysis.

270 play a negative role in defense responses to P. syringae.

271 ins result in an increased susceptibility to P. syringae, whereas overexpression of these genes alter

272 of Bti9 and SlLyk13 were more susceptible to P. syringae.

273 mmune deficient and were more susceptible to P. syringae.

274 similarity to Escherichia coli BetT than to P. syringae BetT.

275 mutants were more susceptible than the WT to P. syringae infection.

276 ations that may be peculiar to cold-tolerant P. syringae, including increase of unsaturated fatty aci

277 s of differences in the osmotolerance of two P. syringae strains, B728a and DC3000.

278 effectors and for host susceptibility to two P. syringae pathogens.

279 staining effect was suppressed by wild-type P. syringae pv. tabaci and P. fluorescens heterologously

280 HopI1 is a ubiquitous P. syringae virulence effector that acts inside plant ce

281 cell death in susceptible leaves undergoing P. syringae infection.

282 Upon P. syringae infection, ACD2 levels and localization chan

283 ica effector protein ATR13 was delivered via P. syringae by fusing the ATR13 gene with the avrRpm1 ty

284 Virulent P. syringae also has the potential to induce net systemi

285 Virulent P. syringae strains were able to overcome a PAMP pretrea

286 bivory caused by prior infection by virulent P. syringae.

287 exhibits enhanced susceptibility to virulent P. syringae strains, suggesting it may impact basal dise

288 WIN2 or WIN3 confers resistance to virulent P. syringae, which is consistent with these proteins bei

289 iation in growth of the universally virulent P. syringae pv. maculicola ES4326 among more than 100 Ar

290 the wild type when challenged with virulent P. syringae.

291 d to defense soon after initial contact with P. syringae, but these proteins were not secreted in the

292 fers a benefit when plants are infected with P. syringae carrying avrPphB2 but also incurs a large co

293 e was observed in irAOX plants infected with P. syringae, which correlated with higher levels of sali

294 ase in free IAA levels during infection with P. syringae pv. tomato strain DC3000 (PstDC3000).

295 t induced when M. sativa was inoculated with P. syringae DC3000.

296 reased significantly when co-inoculated with P. syringae pv. tomato but not when co-inoculated with a

297 tive response when challenge inoculated with P. syringae pv. tomato DC3000.

298 Inoculation with P. syringae DC3000(avrRpm1) or P. syringae DC3000(avrRpt

299 ion in N. caerulescens, but inoculation with P. syringae did not elicit the defensive oxidative burst

300 susceptibility upon surface inoculation with P. syringae, wider stomatal apertures, and enhanced plas