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1 toma cells stably transfected with the human P2Y4 receptor.
2  receptor's being a species homologue of the P2Y4 receptor.
3 lecule that markedly activated the expressed P2Y4 receptor.
4 ponsive receptors, the P2Y6 receptor and the P2Y4 receptor.
5 nt coupling of this release to activation of P2Y4 receptors.
6 ons as an agonist or antagonist at mammalian P2Y(4) receptors.
7 minal nucleotides, mediated by P2X(2) and/or P2Y(4) receptors.
8 ls in the aged cochlea upregulate P2Y(2) and P2Y(4) receptors and display purinergic-induced Ca(2+) r
9 2Y6 receptor), selectively activated by UTP (P2Y4 receptor), and activated by UTP and ATP but not by
10  erythrocytes, at recombinant human P2Y2 and P2Y4 receptors, and at recombinant rat P2Y6 receptors.
11                                 No selective P2Y4 receptor antagonist has been described so far.
12 e-2-sulfonate) represents the most selective P2Y4 receptor antagonist known to date.
13        Therefore, we developed and optimized P2Y4 receptor antagonists based on an anthraquinone scaf
14 enopus oocytes expressing P2X(2), P2Y(2,) or P2Y(4) receptors, but not in oocytes expressing other P2
15 mologues, we expressed a series of human/rat P2Y(4) receptor chimeras in 1321N1 human astrocytoma cel
16 uman astrocytoma cells stably expressing the P2Y4 receptor, combined addition of these nucleotides re
17               Pharmacological studies of the P2Y4 receptor expressed in 1321N1 human astrocytoma cell
18        The expression of P2Y(1) , P2Y(2) and P2Y(4) receptors in the supporting cells decreased durin
19 refore, we cloned and expressed the P2Y6 and P2Y4 receptors in 1321N1 human astrocytoma cells and com
20 UDP but weakly by UTP, ATP, and ADP; and the P2Y4 receptor is activated most potently by UTP, less po
21  the N terminus and 7TM, whereas that of the P2Y4 receptor is present in its C-terminal tail.
22                         That is, whereas the P2Y4 receptor is selectively activated by UTP and is not
23                                          The P2Y4 receptor is selectively activated by UTP.
24  homology to the previously cloned mammalian P2Y4 receptor, its pharmacological selectivity was not c
25 ituted with the corresponding regions of the P2Y4 receptor lacking its targeting signal.
26          In the absence of dDAVP, P2Y(1) and P2Y(4) receptors localized to the apical membrane, where
27 ) receptor mRNA level was unchanged, and the P2Y(4) receptor mRNA was undetectable.
28 on of the uridine nucleotide-activated human P2Y4 receptor (P2Y4-R) and P2Y6 receptor (P2Y6-R) was st
29                      Expression of the human P2Y(4) receptor, rat M(1)-muscarinic receptor, and human
30            Additional studies indicated that P2Y(4) receptors stimulate both TSP-1 expression and rel
31 l carcinomas for P2X5, P2X7, P2Y1, P2Y2, and P2Y4 receptors was performed, accompanied by detailed an
32 al tissues, while the expression of P2Y2 and P2Y4 receptors was up-regulated in GC cells.
33                                   P2Y(2) and P2Y(4) receptors were primarily responsible for the ATP-
34                                              P2Y4 receptors were found in basal cell carcinomas but n
35  in HUVEC was mediated by P2Y1, P2Y2, and/or P2Y4 receptors, whereas P2Y6, P2Y11, and P2X receptors w
36  5'-triphosphate, indicating the presence of P2Y4 receptors, whereas the P2Y1-specific agonist 2-meth
37                                     P2Y2 and P2Y4 receptors, which have 52% sequence identity, are bo
38                  In contrast, stimulation of P2Y(4) receptors with UTP or P2Y(6) receptors with UDP p
39 t at human P2Y2 receptors and also activated P2Y4 receptors with an EC(50) of 85 nM.