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1                                              PABP1 has been proposed to participate at various steps
2                                              PABP1 relocalization was sharply decreased in cells infe
3 Here we show that poly(A)-binding protein 1 (PABP1) binds preferentially to INS-1 within the p17(gag)
4 t the human polyadenylate binding protein 1 (PABP1) binds to the 5'UTR of the viral mRNAs.
5 r localization of poly(A)-binding protein 1 (PABP1) by indirect immunofluorescence as well as by tagg
6 d CARM1 substrate poly(A)-binding protein 1 (PABP1) were covalently linked to an adenosine moiety as
7 l protein, RPS17, poly(A)-binding protein 1 (PABP1), and the elongation factors, eEF1A and eEF2.
8 ion of G3BP1 with poly(A)-binding protein 1 (PABP1), but its RNA-independent interactions with caprin
9 ludes cytoplasmic poly(A) binding protein 1 (PABP1).
10 nonhistone (polyadenylate-binding protein 1, PABP1) substrates induced by coactivator-associated argi
11                                Additionally, PABP1 bound to the viral 5'UTR can recruit eIF4G in an e
12 some established substrates (e.g. BAF155 and PABP1), only phospho-CARM1 methylates the RUNX1 transcri
13 included RNA metabolism proteins hnRNPA1 and PABP1 and the glycolysis enzyme GAPDH.
14   MTAR1 enhanced binding between IGF2BPs and PABP1, thereby promoting MYC mRNA stability and increase
15 n between two translation factors, Paip1 and PABP1.
16  starvation-induced PABP1-SIRT1 association, PABP1 deacetylation, and poly(A)RNA nuclear retention.
17 nuclear proteins revealed costaining between PABP1 and markers of nuclear speckles.
18                 In the low expressing cells, PABP1 was localized in the cytoplasm, whereas in the hig
19 This establishes a new role for the cellular PABP1 inhibitor Paip2 as an innate defense that restrict
20                                 In contrast, PABP1 mRNA expression is minimal in oocytes and early em
21 ation, SIRT1 interacts with and deacetylates PABP1 and deactivates its poly(A)RNA binding, leading to
22 ing protein 2, which is known to destabilize PABP1 binding to poly(A) and inhibit steady-state transl
23 nia, the chain of interactions 5'cap4-eIF4E4-PABP1-poly(A) bridges the mRNA 5' and 3' ends.
24 ng that active transcription is required for PABP1 export.
25             These results suggest a role for PABP1 in the inhibition of gag expression mediated throu
26                    Consistent with roles for PABP1 and eIF2alpha in the pioneer round of translation,
27        However, CARM1 methylates histone H3, PABP1, AIB1, and a number of splicing factors, which str
28 similarity to the entire 5' UTR of the human PABP1 mRNA, but there is no similarity upstream of the t
29 of the transcription start site of the human PABP1 mRNA, indicating that the Pabp2 gene lacks 5' flan
30  it illustrates how a stress-induced rise in PABP1 triggered by virus infection can counter and surpa
31 synthesis and replication without increasing PABP1.
32 on is required for energy starvation-induced PABP1-SIRT1 association, PABP1 deacetylation, and poly(A
33 AR1 is essential for recruiting IGF2BPs into PABP1-mediated liquid-liquid phase separation (LLPS) com
34 Second, mRNAs encoding somatic PABP isoform, PABP1, are present at high levels in meiotic and haploid
35 calized to cytoplasmic puncta with SG marker PABP1 in CRPC.
36  enhancing MYC translation through mediating PABP1/IGF2BPs liquid-liquid phase separation.
37 localize efficiently with Vif(IIIB) and mRNA-PABP1 complexes in stress granules in a manner that is p
38 3F imply that it occurs associated with mRNA-PABP1 in translationally active polysomes and to a lesse
39                                      Nuclear PABP1 observed either after overexpression or after tran
40                               The ability of PABP1 to shuttle between nucleus and cytoplasm was also
41  binding, leading to nuclear accumulation of PABP1 and poly(A)RNA and thus facilitating eukaryotic ce
42                              Accumulation of PABP1 in the nuclei was observed upon transcription inhi
43 ent cell lines and found that the binding of PABP1 to INS-1 RNA is significantly diminished in glial
44  We found that Unr stimulated the binding of PABP1 to mRNA, and that Unr was required for the stable
45 gag correlate with the absence of binding of PABP1 to the INS-1 RNA in cellular extracts.
46 olymers demonstrated preferential binding of PABP1 to the INS-1-containing RNA.
47                        Coordinate control of PABP1, Paip2, and EDD1 required the virus-encoded UL38 m
48                        The nuclear import of PABP1 is an energy-dependent process since PABP1 fails t
49 ll lines with EZM2302 leads to inhibition of PABP1 and SMB methylation and cell stasis with IC50 valu
50 r was required for the stable interaction of PABP1 and eIF4G in cells.
51                           The interaction of PABP1 with the viral 5'UTR makes the translation of vira
52           Our results suggest involvement of PABP1 in nuclear events associated with the formation an
53  populations producing physiologic levels of PABP1 and increased in cells with reduced levels of PABP
54 of cells producing high versus low levels of PABP1, we found that the percentage of RVFV N-positive c
55 nd increased in cells with reduced levels of PABP1.
56  mutagenesis showed that the minimal part of PABP1 retaining the ability to shuttle consists of the f
57 cells demonstrated a gross relocalization of PABP1 to the nucleus late in infection.
58            Here, we investigated the role of PABP1 during RVFV infection of HeLa cells.
59 ing RVFV infection leads to sequestration of PABP1 in the nuclear speckles, creating a state within t
60                              Transfection of PABP1 or PABP1-GFP resulted in heterogeneity of intracel
61                     Transfection of PABP1 or PABP1-GFP resulted in heterogeneity of intracellular dis
62 ity of polyadenylate-binding protein PABPC1 (PABP1) to stimulate translation is regulated by its repr
63  lacks 5' flanking sequences of the parental PABP1 gene.
64 was dependent on PABP accrual, as preventing PABP1 accumulation suppressed viral replication and inhi
65 n CARM1 substrates, poly(A)-binding protein (PABP1) and the transcriptional cofactor p300, was abolis
66 lasmic somatic cell poly(A)-binding protein (PABP1) is not expressed until later in embryogenesis.
67 via deacetylating a poly(A)-binding protein, PABP1.
68  proteins as baits: poly(A)-binding proteins PABP1 and PABP2, mRNA export receptor MEX67, and the nuc
69                                        Since PABP1 binds to all polyadenylated mRNAs, and is involved
70 f PABP1 is an energy-dependent process since PABP1 fails to enter the nucleus upon ATP depletion and
71                       In addition, the SIRT1-PABP1 association is not specific to energy starvation b
72 th small interfering RNAs (siRNAs) targeting PABP1.
73                  These results indicate that PABP1 bound to the viral 5'UTR may promote eIF4E-indepen
74  changed by siRNA treatment, indicating that PABP1 was not required for RVFV infection.
75                                 We show that PABP1 is able to enter the nucleus.
76  that the Pabp2 promoter is derived from the PABP1 5' UTR.
77 y 330 bases downstream of the capsite of the PABP1 mRNA, indicating that the Pabp2 promoter is derive
78 ions to a partly folded form upon binding to PABP1, whereby this interaction is not modulated by poly
79 ction is not modulated by poly(A) binding to PABP1.
80 tein (eIF4E4) that binds via a PAM2 motif to PABP1.
81 aradigm, binds tightly to eIF4E4, but not to PABP1.
82                         To determine whether PABP1 was required for RVFV infection, we measured the p
83 2 synthesis, stability, and association with PABP1.
84 ts ability to bind RNA, and to interact with PABP1.
85 hibits 72% identity to mammalian and Xenopus PABP1 and is the predominant poly(A)-binding protein exp