ライフサイエンスコーパス: PFC

1 PFC activation was measured using optical neuroimaging.

2 PFC-NAcc gene network topological analyses, following co

3 BAergic phenotype necessary to sustain adult PFC functions.

4 and 8 weeks of treatment was plotted against PFC and ACC TSPO V(T), showing a significant nonlinear r

5 nclear whether relative coding occurs in all PFC regions and how it is affected by feedback informati

6 of spatial position was coherent for CA1 and PFC ensembles, exhibiting correlated position representa

7 the changes in dendritic function in CA1 and PFC neurons based on the presence or absence of FMRP.

8 Alpha/beta power in LIP, FEF, and PFC inhibited spiking in deep layers of V4.

9 ndings demonstrate that (1a) hippocampus and PFC play complementary roles in the implicit, unconsciou

10 ssion, synchrony between the hippocampus and PFC, and cognition.

11 HCN-TRIP8b complexes in both hippocampus and PFC.

12 factors affecting cortical interneurons and PFC function.

13 rays to record neural activity in the MD and PFC simultaneously.

14 ngs delineate unique contributions of MD and PFC to distributed processing for cognitive control and

15 rved that functional coupling between MD and PFC was strongest when the decision as to which of the t

16 simultaneous neural recordings in the MD and PFC while monkeys perform a cognitive control task trans

17 tive GSK3 via Akt inhibition in the vHip and PFC.

18 AT) while activity in the bilateral anterior PFC was monitored using functional Near Infrared Spectro

19 xpected reliance on portions of the anterior PFC (aPFC) also involved in exploration, relational reas

20 ne, there were amplitude differences between PFC and PAR TEPs across a wide time range (15-250 ms), h

21 osed rats showed precocial maturation of BLA-PFC innervation, with females affected earlier than male

22 scence may accelerate the development of BLA-PFC plasticity, probably due to BLA hyperactivity, which

23 long-term, THC-induced dysregulation of both PFC and VTA DAergic activity states, a neuroprotective e

24 thways, and normalized dysregulation of both PFC and VTA DAergic activity, demonstrating powerful and

25 tive control are preferentially performed by PFC and MD, respectively.

26 labeling and metabolic profiling to capture PFCs and demonstrate their functions.

27 es that labile plant flavonoid carbocations (PFCs) play vital roles in the biosynthesis of proanthocy

28 f short-term memory and supplement classical PFC-based models with an emerging thalamus-centric frame

29 icting results with primary fascial closure (PFC) versus bridged repair during laparoscopic ventral h

30 Conversely, suppression of constitutive PFC CRF activity improved working memory.

31 choice in cued tasks, whereas contralateral PFC-projecting neurons most potently encoded reward cont

32 whether TSPO V(T) in the prefrontal cortex (PFC) and anterior cingulate cortex (ACC) predicts reduct

33 proper functioning of the prefrontal cortex (PFC) and establishment of appropriate adult social behav

34 rocesses dependent on the prefrontal cortex (PFC) and extended frontostriatal circuitry.

35 ctural changes within the prefrontal cortex (PFC) and functional changes in its communication with di

36 antly reduced in both the prefrontal cortex (PFC) and induced pluripotent stem cell-derived neuronal

37 circuitry, including the prefrontal cortex (PFC) and mesolimbic dopamine (DA) pathway are particular

38 al gene expression in the prefrontal cortex (PFC) and nucleus accumbens (NAc) of mice exposed to mult

39 hyperactivity across the prefrontal cortex (PFC) and striatum in the neuropathology of obsessive com

40 ng and memory such as the prefrontal cortex (PFC) and the hippocampus (HPC).

41 Regulatory aspects of the prefrontal cortex (PFC) are especially relevant to human psychopathology, a

42 it persistent activity in prefrontal cortex (PFC) as a primary neural correlate, but emerging views s

43 associated with amygdala-prefrontal cortex (PFC) circuits.

44 level, but lower GABA in prefrontal cortex (PFC) detected by chemical exchange saturation transfer a

45 re critically involved in prefrontal cortex (PFC) functions, especially in working memory and neurode

46 like effects by enhancing prefrontal cortex (PFC) glutamate transmission; however, the receptor subty

47 g medial and dorsolateral prefrontal cortex (PFC) in default mode network (DMN) associated with TD in

48 o examine the role of the prefrontal cortex (PFC) in neural oscillatory activity associated with proa

49 ateral amygdala (BLA) and prefrontal cortex (PFC) inputs revealed a hierarchy of synaptic inputs that

50 The prefrontal cortex (PFC) integrates incoming information to guide our action

51 limbic connection of the prefrontal cortex (PFC) involved in emotional processing may be rendered dy

52 Human prefrontal cortex (PFC) is associated with broad individual variabilities i

53 The prefrontal cortex (PFC) is one region in which chronic drug exposure change

54 vidence indicate that the prefrontal cortex (PFC) is particularly sensitive to, and afflicted by, exp

55 Modulation by the prefrontal cortex (PFC) is thought to be key for both processes, but the pr

56 The prefrontal cortex (PFC) is thought to learn the relationships between actio

57 Prefrontal cortex (PFC) is thought to support the ability to focus on goal-

58 ronal function and in the prefrontal cortex (PFC) it may contribute to compulsive cocaine intake.

59 h and differentiated into prefrontal cortex (PFC) lineages to efficiently test early-developmental hy

60 tanding how disruption of prefrontal cortex (PFC) maturation during adolescence is crucial to reveal

61 00473 as downregulated in prefrontal cortex (PFC) of depressed females but not males.

62 excitatory DREADDs in the prefrontal cortex (PFC) of mice.

63 ectively increased in the prefrontal cortex (PFC) of Shank3-deficient mice and autistic human postmor

64 ateral amygdala (BLA) and prefrontal cortex (PFC) onto identified medium spiny neurons (MSNs) in the

65 iven maturation of either prefrontal cortex (PFC) or hippocampus (HP) to these deficits is still unkn

66 e mouse astrocytes of the prefrontal cortex (PFC) or the hippocampus would produce behavioral abnorma

67 The prefrontal cortex (PFC) partly implements this process by regulating thalam

68 between the thalamus and prefrontal cortex (PFC) play a critical role in cognitive function and arou

69 The prefrontal cortex (PFC) plays a critical role in curbing impulsive behavior

70 rk has suggested that the prefrontal cortex (PFC) plays a key role in context-dependent perceptual de

71 scriptomic types of mouse prefrontal cortex (PFC) projection neurons relate to axonal projections and

72 hippocampus (vHPC) to the prefrontal cortex (PFC) regulate cognition, emotion, and memory.

73 hat carbachol delivery to prefrontal cortex (PFC) restored wakefulness despite continuous administrat

74 owed that hippocampus and prefrontal cortex (PFC) were involved in the task and furthermore revealed

75 The prefrontal cortex (PFC)'s functions are thought to include working memory,

76 ronal excitability in the prefrontal cortex (PFC), a brain region critical for social and cognitive f

77 tnatal development of the prefrontal cortex (PFC), allowing for optimal DA-mediated maturation of exc

78 with deactivation of the prefrontal cortex (PFC), an area important for executive functions.

79 ocally connected with the prefrontal cortex (PFC), and although the MD has been implicated in a range

80 cture and function of the prefrontal cortex (PFC), and increases the risk for adult depression and an

81 naptic loss in the medial prefrontal cortex (PFC), and this leads to behavioral and cognitive impairm

82 V expression in the adult prefrontal cortex (PFC), contributing to a behavioral phenotype that mimics

83 Prefrontal cortex (PFC), hippocampus (HPC), and spleen were then collected

84 We focused on the prefrontal cortex (PFC), particularly rich in GR, early altered in AD and i

85 In the prefrontal cortex (PFC), PV expression appears last and continues to substa

86 have found, in the adult prefrontal cortex (PFC), substantial differences in the structure and conne

87 us and right dorsolateral prefrontal cortex (PFC), which in turn was negatively correlated to stress-

88 gulation in the blood and prefrontal cortex (PFC), which is accompanied by depression-like behavior,

89 king resulted in enhanced prefrontal cortex (PFC)-driven excitation of prodynorphin-containing neuron

90 decision-making, engages prefrontal cortex (PFC)-striatal circuitry and is impaired in both manifest

91 -silent mechanisms in the prefrontal cortex (PFC).

92 ed levels of HDAC6 in the prefrontal cortex (PFC).

93 quivalent is known as the prefrontal cortex (PFC).

94 the hippocampus and right prefrontal cortex (PFC).

95 entations in ventromedial prefrontal cortex (PFC).

96 tical areas including the prefrontal cortex (PFC).

97 abnormal function in the prefrontal cortex (PFC).

98 pyramidal neurons of the prefrontal cortex (PFC).

99 (NAcc), and ventromedial prefrontal cortex (PFC)] predicted cognitive-behavioral therapy (CBT) and s

100 nted eight Utah arrays in prefrontal cortex (PFC; area 46) of two male macaque monkeys, recording >50

101 ntal eye field [FEF], and prefrontal cortex [PFC]) while monkeys performed a task that modulated visu

102 or parietal lobule [IPL], prefrontal cortex [PFC], and cingulate), and aimed to visualize this using

103 ads (N = 48) to measure prefrontal cortical (PFC) activities while they listened to infant and adult

104 the lateral and medial prefrontal cortices (PFC) during task anticipation and performance on the wor

105 ed on the structure and function of critical PFC-limbic circuits, linking alterations in the processi

106 Autopsy-derived PFC samples show elevated MMP-9 levels in antidepressant

107 results in an overactive but desynchronized PFC before adolescence.

108 ventral prefrontal cortex (VPFC) and dorsal PFC (DPFC), insula and their mesial temporal, striatal a

109 Significant dorsolateral PFC (DLPFC) activations were observed during self-paced

110 functional connectivity to the dorsolateral PFC (pgACC-dlPFC), and mediated of the association betwe

111 tional connectivity between the dorsolateral PFC and caudate in pre-HD participants.

112 with FC between the hCd and the dorsolateral PFC, hippocampus, and precuneus.

113 another hub of the social brain (dorsomedial PFC), increased the effect of body position during persp

114 or (CRF) receptors in the caudal dorsomedial PFC (dmPFC) impairs higher cognitive function, as measur

115 hereas activation in the insula, dorsomedial PFC and DLPFC increased over time.

116 Here we show that a subset of dorsomedial PFC (dmPFC) layer 5 pyramidal neurons, which project to

117 tified to receive either rTPJ or dorsomedial PFC anodal high-definition transcranial direct current s

118 Stimulation to the dorsomedial PFC had no effect on perspective-tracking or taking.

119 nized involvement for Rap1 in novelty-driven PFC engagement.

120 Dysregulated PFC co-expression networks common to both the THC-treate

121 it while class 2 mutations (5 of 27) enhance PFC neurogenesis.

122 ived projections from multiple types, except PFC->PAG (periaqueductal gray).

123 trate for the first time a circuit motif for PFC function wherein anatomically non-overlapping output

124 Our findings show a novel role for PFC astrocytes in the DA modulation of cognitive perform

125 These results support integral roles for PFC astrocytes in the behavioral actions of ethanol that

126 s suggest that early TEP peaks (<80 ms) from PFC and PAR reflect stimulation site specific activity t

127 Recordings from PFC brain slices revealed that MK-801 exposure during ad

128 inant excitatory pathway onto D1 MSNs (BLA > PFC = vHipp) while PFC inputs dominate D2 MSNs (PFC > vH

129 omical connections predicting hippocampus -> PFC and DG -> CA1, i.e., theta transmission is unidirect

130 on in mice, and decreases fear-associated HC-PFC synchrony, suggesting that Bdnf transcription from p

131 tion from promoter IV plays a key role in HC-PFC function during fear memory retrieval.

132 (2+) activity of the molecularly homogeneous PFC->PAG type against two heterogeneous classes in sever

133 owever, whether PFCs exist in plants and how PFCs function remain unclear.

134 al magnetic stimulation applied to the human PFC between successive trials enhanced serial biases, th

135 n that the infralimbic prefrontal cortex (IL-PFC) is important for processing social information and,

136 iliar conspecific induces activity in the IL-PFC as evidenced by increased immediate early gene (IEG)

137 that a social neural ensemble within the IL-PFC may contribute to social buffering of fear.

138 zed that socially active cells within the IL-PFC may integrate social information to modulate fear re

139 inhibitor UNC0642 or knockdown of EHMT1/2 in PFC induced a robust rescue of autism-like social defici

140 -and-error task, that population activity in PFC is persistently changing, regardless of learning.

141 hiodide while recording cellular activity in PFC of male rhesus monkeys performing a delayed decision

142 Restoring Npas4 expression in PFC of 16p11.2(dp/+) mice ameliorated the social and cog

143 measured by (1)H-MRS; and hypomyelination in PFC as evidenced by relevant cellular and molecular chan

144 We estimated information in PFC about saccades as a function of ensemble size.

145 tivation was found in parietal areas, nor in PFC, whereas microstimulation in posterior parietal cort

146 Activating G(q)-GPCR signaling in PFC astrocytes increased drinking in ethanol-naive mice,

147 set of genes involved in neural signaling in PFC of Shank3-deficient mice.

148 preponderance of mature dendritic spines in PFC neurons.

149 decarboxylase-65 and glutamine synthetase in PFC; reduced fractional anisotropy in various brain regi

150 optimal encoding of vast ranges of values in PFC: neuronal response to value depends on the choice co

151 are prominent in layer 5 (L5) of infralimbic PFC.

152 tory cells in deep layers of the infralimbic PFC, highlighting unexpected roles for both CCK+ interne

153 , a behavioral paradigm that requires intact PFC-ventral hippocampus connectivity.

154 temporal regions, as well as stronger intra PFC connectivity, in males compared to females.

155 t in Fragile X syndrome, in adult CA1 and L5 PFC neurons regulates the number of functional dendritic

156 (-/y) mouse model of FXS, but reduced in L5 PFC dendrites.

157 ally modulates HCN channels in CA1 versus L5 PFC dendrites.

158 , active targeting of EpiSCs by EP9-labelled PFCs was able to outcompete the strong phagocytic uptake

159 hs (range: 9-42), patients treated with LVHR-PFC had on average a 12-point higher improvement in QoL

160 To learn how the MD-PFC thalamocortical network is engaged to mediate forms

161 positron emission tomography scan to measure PFC and ACC TSPO V(T).

162 ositive interneurons (PVIs) in dorsal-medial PFC (dmPFC) prior to an active bout, or a bout initiated

163 rhythmic activity within and over the medial PFC ("midfrontal theta") has been identified as a distin

164 ased mRNA expression profiling in the medial PFC (mPFC) of maternally separated (MS) pups identified

165 rain affects synaptic function in the medial PFC (mPFC).

166 dorsal, prelimbic (PL) region of the medial PFC aids active avoidance in situations requiring flexib

167 a-mediated neuronal remodeling in the medial PFC, and subsequent behavioral and cognitive consequence

168 ces aberrant neuronal activity in the medial PFC, and that neuronal hyperactivity increases CSF1 sign

169 ony-stimulating factor (CSF)-1 in the medial PFC.

170 S-induced dendritic spine loss in the medial PFC.

171 a-mediated neuronal remodeling in the medial PFC.

172 transfer to express LINC00473 in adult mouse PFC neurons, we mirrored the human sex-specific phenotyp

173 Gpr56 knockdown in mouse PFC is associated with depressive-like behaviors, execut

174 intermittent stress increases Rap1 in mouse PFC synapses, results in cognitive impairments, and redu

175 A-specific knockdown of circHomer1a in mouse PFC, we show that it modulates the expression of numerou

176 llular compartments in layer 1 (L1) of mouse PFC.

177 inct outputs from a subdivision of the mouse PFC, the anterior cingulate cortex (ACC).

178 -evoked feed-forward inhibition in the mouse PFC.

179 = vHipp) while PFC inputs dominate D2 MSNs (PFC > vHipp > BLA).

180 ing of EP9 to perfluorocarbon nanoemulsions (PFCs) resulted in the efficient delivery of (19)F cargo

181 und that Disc1-KD in the hippocampus but not PFC impaired trace fear conditioning in adult mice.

182 Moreover, activation of PFC alpha1 receptors was sufficient to improve sustained

183 ffect is explained in part by enhancement of PFC-driven cognitive control.

184 To do so, we tested the impact of PFC-targeted tDCS on behavioral and electrophysiological

185 z) functional connectivity in deep layers of PFC to the other areas.

186 2 in astrocytes postnatally produces loss of PFC DA homeostasis, leading to defective synaptic transm

187 gulation of DAergic activity states, loss of PFC GABAergic inhibitory control and affective and cogni

188 However, across a broad population of PFC neurons, we found no evidence of current-goal-specif

189 g noise correlations in large populations of PFC neurons.SIGNIFICANCE STATEMENT Recent theoretical wo

190 ugh the MD has been implicated in a range of PFC-dependent cognitive functions (Watanabe and Funahash

191 is unclear, because impaired recruitment of PFC has also been observed in OCD patients during paradi

192 at model in which cholinergic stimulation of PFC produced wakefulness despite continuous exposure to

193 lts identify a projection-defined subtype of PFC pyramidal neurons as key mediators of impulse contro

194 phological and transcriptional trajectory of PFC pyramidal neurons, which could enhance vulnerability

195 o outcompete the strong phagocytic uptake of PFCs by circulating monocytes.

196 of the HPA axis and a feed-forward effect on PFC GR sensitivity could participate in the etiology of

197 teral vmPFC lesion, 10 with a lesion outside PFC and 10 healthy participants (each group included bot

198 e subgroups of autism mutations that perturb PFC neurogenesis and are correlated to abnormal WNT/beta

199 This avoidance requires prelimbic (PL) PFC, basolateral amygdala (BLA), and ventral striatum (V

200 apses in the prelimbic prefrontal cortex (PL-PFC).

201 ns may largely reflect circuit defects in PL-PFC networks communicating through endocannabinoid-regul

202 polarizing response to NMDA in deep-layer PL-PFC neurons analyzed by current-clamp recordings.

203 activity of NMDA receptors in the mature PL-PFC.

204 lectron microscopic immunolabeling in the PL-PFC of adult mice that had received Delta9-THC only duri

205 inhibition of mGlu(2) or mGlu(3) potentiates PFC excitatory transmission and confers antidepressant e

206 teers, resting EEG and TEPs from prefrontal (PFC) and parietal (PAR) cortex were measured before and

207 2 months old APP/PS1 mice in the prefrontal (PFC), somatosensory (SS2), and primary motor cortex (M1)

208 ions in the prelimbic prefrontal cortex (PrL-PFC).

209 int-potentiated cocaine seeking required PrL-PFC CB1R activation.

210 seeking in females was localized to the PrL-PFC and both CORT- and restraint-potentiated cocaine see

211 iological recordings from female layer V PrL-PFC pyramidal neurons revealed CB1R-dependent CORT-induc

212 e stress potentiates cocaine seeking via PrL-PFC CB1R in both sexes, sensitivity to cocaine priming i

213 ated in a randomized controlled trial (RCT), PFC compared to bridged repair would improve patient qua

214 ivation on the synaptic strength of specific PFC inputs.

215 Thus, counter to a strictly PFC-driven decision process, in this spatial selective l

216 es revealed that Gpr12 enables high thalamus-PFC synchrony to support memory maintenance and choice a

217 This is the first RCT demonstrating that PFC with LVHR significantly improves patient QoL.

218 These findings suggest that PFC activation during working memory task anticipation a

219 The PFC plays a critical role in the identification of relev

220 The PFC, through its high degree of interconnectivity with c

221 y correlated with FC between the hCd and the PFC.

222 ntrast, slower maturing regions, such as the PFC, have been suggested to be the neurobiological locus

223 ly relevant to human psychopathology, as the PFC, in addition to its functions, is more recent from a

224 However, because the PFC does not directly project to sensory TRN subnetworks

225 ve functions thought to be controlled by the PFC dose-dependently.

226 eking competes with avoidance of danger, the PFC likely plays a role in selecting the optimal choice.

227 The depletion of PNNs from the PFC has also a potent effect on the connectivity of PV+

228 edforward inhibition of projections from the PFC to the ventrolateral PAG region and its downstream t

229 We recorded neural activity in the PFC and HPC of the trisomic Ts65Dn mouse model of DS dur

230 llations and cross-frequency coupling in the PFC and HPC while prefrontal-hippocampal synchronization

231 vels of excitatory-inhibitory balance in the PFC and its control of behavior.

232 proper excitatory-inhibitory balance in the PFC and its control of behavioral responses.SIGNIFICANCE

233 synaptic AMPAR/GluR composition both in the PFC and the nucleus accumbens (NAcc).

234 pathways as most prominently affected in the PFC and with lesser significance in the NAc.

235 changes in the cerebrovascular system in the PFC contribute to cocaine self-administration, and wheth

236 gy to directly downregulate PV levels in the PFC during adolescence and examined its impact on prefro

237 est is the gain of GABAergic function in the PFC during adolescence and its susceptibility to the imp

238 indicate the PV upregulation observed in the PFC during adolescence is necessary for refinement of pr

239 crease in GABAergic activity observed in the PFC during this period.

240 for changes in mitochondrial outputs in the PFC following chronic stress that are indicative of mito

241 veal that the hyper-induction of Rap1 in the PFC is sufficient to drive stress-relevant cognitive and

242 genome-wide transcriptional aberrance in the PFC of 16p11.2(dp/+) mice, including downregulation of t

243 also show that GPR56 is downregulated in the PFC of individuals with depression that died by suicide.

244 measured normal oscillatory activity in the PFC of late conditional KO (late-cKO) mice, in which Arc

245 ry using iontophoresis of antagonists in the PFC of monkeys remembering the location of a visual stim

246 y, revealing that rotational dynamics in the PFC preserve sensory choice information for the duration

247 -inhibitory synaptic activity emerges in the PFC that correlates linearly with the GABAergic deficit.

248 te of excitatory-inhibitory imbalance in the PFC that limits its functional capacity to regulate beha

249 mere 25% reduction of adult PV levels in the PFC was sufficient to reduce local GABAergic transmissio

250 h patterns do not hold working memory in the PFC when information must be employed flexibly.

251 decreases and neuronal Ca(2+) changes in the PFC, and decreased cocaine intake and blocked reinstatem

252 diazepam normalized Csf1 and C3 mRNA in the PFC, and prevented increases in Csf1r and Cd11b in front

253 re expressed on glutamatergic neurons in the PFC, optogenetic stimulation of which elicited activatio

254 ) and Akt signaling pathways directly in the PFC, two biomarkers previously associated with cannabis-

255 In the PFC, we also find that WIN preexposure blunts the typica

256 ductions in cerebral blood flow (CBF) in the PFC, which were exacerbated with chronic exposure and wi

257 tly controlled by inhibitory networks in the PFC, whose disruption is thought to contribute to mental

258 different populations of interneurons in the PFC.

259 and inhibitory synaptic transmission in the PFC.

260 hosphorylation complexes were altered in the PFC.

261 RI to predict local neurotransmitters in the PFC.

262 races inferred from spiking synchrony in the PFC.

263 fewer plaques in M1 and SS2, but not in the PFC.

264 Instead, the PFC grouped locations representing behaviorally equivale

265 itive allosteric modulator Indiplon into the PFC before extinction testing reduced the level of freez

266 begun to elucidate the contributions of the PFC and its circuitry to depression- and anxiety-like be

267 xcitability in L2/3 pyramidal neurons of the PFC and may thereby modulate the effects of stress on de

268 Whereas the variation of the PFC has been well documented in different mammalian orde

269 ts functional homology, the expansion of the PFC in human and NHPs, and most importantly how they can

270 jective values, highlighting the role of the PFC in representing a belief about the current state of

271 the BNST and chemogenetic inhibition of the PFC-BNST pathway restored abnormal responses to predator

272 In animals over 12 months, only the PFC region continued to significantly accumulate plaques

273 k during behavior.SIGNIFICANCE STATEMENT The PFC is thought to represent our knowledge about what act

274 Specifically, we show that the PFC has a multidimensional code for context, decisions a

275 These findings suggest that the PFC region is selectively susceptible to Abeta depositio

276 Thus, our study shows that comparable to the PFC in mammals, the NCL in birds varies considerably acr

277 showed a shift in theta band activity to the PFC.

278 c nuclei differentially communicate with the PFC through distinct L1 micro-circuits.

279 Various nodes of this PFC-BNST dynorphin-related circuit may serve as potentia

280 Largely through this PFC dysfunction, stress has a characterized role in faci

281 e tracing from basolateral amygdala (BLA) to PFC to identify sex-specific innervation trajectories th

282 efulness induced after carbachol delivery to PFC.

283 wakefulness induced by carbachol delivery to PFC.

284 rectional in both cases: from hippocampus to PFC and from DG to CA1 along the tri-synaptic pathway wi

285 computed tomography scan) were randomized to PFC versus bridged repair.

286 ) NAM rapidly activated biophysically unique PFC pyramidal cell ensembles.

287 minutes of warm ischemia in oxygenated UW + PFC solution, grafts showed 63% higher levels of ATP (P

288 uring self-paced suturing, and ventrolateral PFC (VLPFC) deactivations were identified during time-pr

289 ional connectivity between the ventrolateral PFC and ventral striatum in healthy controls and to func

290 tivity in the rostrolateral and ventromedial PFC, regions associated with abstract cognitive inferenc

291 ionality reduction within human ventromedial PFC during learning.

292 learning through compression in ventromedial PFC.

293 Some accounts of ventromedial PFC (vmPFC) suggest that it has a narrow role limited to

294 idence for baseline amygdala or ventromedial PFC volume serving as treatment predictors across the tw

295 with stronger engagement of the ventromedial PFC during valuation.

296 Moreover, the ventromedial PFC exhibited higher connectivity with the right temporo

297 However, whether PFCs exist in plants and how PFCs function remain unclea

298 with a 4-parameter sigmoidal model in which PFC and ACC TSPO V(T) accounted for 84% and 92% of the v

299 d BLA inputs were strongest at D1 MSNs while PFC inputs dominate D2 MSNs.

300 thway onto D1 MSNs (BLA > PFC = vHipp) while PFC inputs dominate D2 MSNs (PFC > vHipp > BLA).