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1                                              PGG also inhibited cell proliferation in both cell lines
2                                              PGG also inhibited IkBKE and MAPK1 genes and the protein
3                                              PGG binds specifically to arterial elastin and, in doing
4                                              PGG glucan-treated animals showed increases in total leu
5                                              PGG inhibited growth of both CRAB and susceptible A. bau
6                                              PGG is a gallotannin produced by a variety of medicinal
7                                              PGG.SNV archives 265 million SNVs across 220,147 present
8 nic acid is converted to prostaglandin G(2) (PGG(2)) by the cyclooxygenase activities of prostaglandi
9 e arachidonic acid (AA), prostaglandin G(2) (PGG(2)), and cyclooxygenase 2 (COX-2).
10 the PGHS-cyclooxygenase, prostaglandin G(2) (PGG(2)), by elevating the concentration of either enzyme
11         We report here the purification of a PGG-glucan-binding element from human leukocytes and its
12 taglandin precursors, arachidonic acid (AA), PGG(2) and PGH(2).
13 or, whereas alpha-pentagalloylglucose (alpha-PGG) acts as a mixed-type inhibitor.
14 etween PRO and PGO and eight between PGO and PGG.
15 o using cytotoxicity, elastin stability, and PGG-elastin interaction assays.
16 fferences from WT behavior occur in both apo PGG/GGG and in the form bound to the reaction-intermedia
17 g this time (and were often hotly debated at PGG meetings).
18 gest that the protective effect exhibited by PGG glucan in the rat peritonitis model is mediated, at
19                               In conclusion, PGG exhibits antimicrobial activity against CRAB, but du
20 st neighbours on a cyclic network after each PGG round.
21 the fluid space was reduced to 5 A at an EPC:PGG mole ratio of 5:1.
22 keratinocytes (HaCaTs) yielded an IC(50) for PGG of 256 ug/mL.
23 n chelation as a possible mode of action for PGG's activity against CRAB.
24 espectively); similarly, the K(m) values for PGG(2) and 15-HETE formation by V349L oPGHS-1 were diffe
25      With native enzyme, the K(m) values for PGG(2), 11-HETE, and 15-HETE formation were each differe
26 3-proS hydrogen from arachidonate which, for PGG(2) formation, is followed by insertion of O(2) at C-
27 whether protection could be transferred from PGG glucan-treated animals to naive recipients via splee
28 peration in a large-scale public goods game (PGG).
29 -beta-glucotriosyl-(1-3)-beta-glucopyranose (PGG) glucan, a biological-response modifier, in protecti
30 -beta-glucotriosyl-(1-3)-beta-glucopyranose (PGG)-glucan and Bacteroides fragilis polysaccharide A (P
31 -anomers of penta-O-galloyl-D-glucopyranose (PGG), 2 and 3, act as insulin mimetics that bind to and
32 3,4,6-penta- O-galloyl-beta-d-glucopyranose (PGG), binds to capillary morphogenesis gene 2 (CMG2) wit
33 enols, only penta-O-galloyl-alpha-D-glucose (PGG) was able to completely mimic the effects of TA by c
34 enols was tested using pentagalloyl glucose (PGG) as a standard, an important representative element
35 e the natural compound pentagalloyl glucose (PGG) effects on TNF-a activated TNBC cell lines, MDA-MB-
36 tringency expressed in pentagalloyl glucose (PGG) units in concentrations ranging from 1 to 140mumol/
37 ng to the isolation of pentagalloyl glucose (PGG), a bioactive gallotannin.
38 of aortic elastin with pentagalloyl glucose (PGG), an elastin-binding polyphenol, would interfere wit
39 ravenous injections of pentagalloyl glucose (PGG)-loaded nanoparticles conjugated with elastin antibo
40                                     goeldii (PGG) and elucidates the chromosomal rearrangements in th
41 e since the first Population Genetics Group (PGG) meeting in January 1968.
42                                      Imprime PGG (Imprime) is an i.v. administered, yeast beta-1,3/1,
43  and evolutionary parameters is available in PGG.SNV, a unique feature compared with other databases.
44  this in terms of altered loop-6 dynamics in PGG/GGG.
45 ical aspects were monitored and evaluated in PGG-treated aortas compared with saline-treated control
46  ranged from 0.17 to 4.7 uM, as expressed in PGG units.
47          Overall, concentrations > 1.0 uM in PGG units are signaling electrochemical impedance, provi
48                      These findings indicate PGG anti-cancer ability in inhibiting tumor cell prolife
49                             In response, low PGG contributors increase their contributions if both ne
50                             With this model, PGG was delivered periadventitially at 2 separate time p
51                                    Moreover, PGG induced apoptosis, modulating caspases, and TNF supe
52 f wild-type (WT) TIM and a quintuple mutant (PGG/GGG).
53                     The single-hinge mutants PGG and GGG had k(cat) values 200-fold lower than that o
54 , participants do not know their neighbours' PGG contribution and thus cannot link play in the PD to
55 portion of the in vivo antitumor activity of PGG may be the result of antiangiogenic activity mediate
56 on of acetaminophen, as does the addition of PGG(2) itself.
57            Periadventitial administration of PGG hinders the development of AAA in a clinically relev
58 y, oral or intraperitoneal administration of PGG inhibits angiogenesis in the mouse corneal micropock
59 a linear correlation of the concentration of PGG(2) with an LOD of 0.227microM.
60            We show that targeted delivery of PGG could reverse the aortic dilation, ameliorate the in
61  In this work, we synthetized derivatives of PGG with different sugar cores and phenolic substituents
62                     The protective effect of PGG glucan was abrogated by treatment with indomethacin,
63                       Safety and efficacy of PGG treatment were first tested in vitro using cytotoxic
64         For in vivo studies, the efficacy of PGG was evaluated within a well-established AAA model in
65  a reduction of the 15-hydroperoxyl group of PGG(2) to form PGH(2) catalyzed by the peroxidase activi
66 ventitial delivery of noncytotoxic levels of PGG inhibits elastin degeneration, attenuates aneurysmal
67 ified prostaglandin extract from the sera of PGG glucan-treated animals protected against mortality i
68              Therefore, simple iv therapy of PGG loaded nanoparticles can be an effective treatment o
69 calability of this effect: in a 1,000-person PGG, participants in the treatment condition successfull
70 ble activity (t-Boc-PGP-OMe, N-acetyl-P, PG, PGG, GP, GG and gly-pro-hyp).
71               V349L oPGHS-1 formed primarily PGG(2), 15S-HETE, and 15R-HETE but only trace amounts of
72   Native forms of oPGHS-1 produced primarily PGG(2) but also several monohydroxy acids, which, in ord
73  for CMG2 than the natural glucose prototype PGG and proved to be a potent angiogenesis inhibitor.
74            The immunomodulator Betafectin(R) PGG-glucan is a homopolymer of glucose derived from yeas
75                                 Radiolabeled PGG-glucan bound several other neutral glycosphingolipid
76                  The binding of radiolabeled PGG-glucan to lactosylceramide was not inhibited by glyc
77                  The binding of radiolabeled PGG-glucan to purified lactosylceramide was saturable, s
78 ere found to support binding to radiolabeled PGG-glucan.
79                                  Remarkably, PGG induced a 154-fold increase in TNF expression in MDA
80              Previous work demonstrated that PGG/GGG has a tenfold higher Km value and 10(3)-fold red
81  now report the serendipitous discovery that PGG (1,2,3,4,6-penta-O-galloyl-beta-D-glucopyranose) is
82                                 We find that PGG inhibits CMG2 with a submicromolar IC50 and it also
83                         These data show that PGG/GGG exists in multiple conformations that are not fu
84             The results obtained showed that PGG reduced the expression of the cytokine GRO-a/CXCL1.
85 bserve a sharp decline of cooperation in the PGG, while in the treatment condition global cooperation
86 with neighbours who contribute little in the PGG.
87 -iso-prostaglandin F2alpha, an isomer of the PGG/H synthase (cyclooxygenase or COX) enzyme product, p
88 n and thus cannot link play in the PD to the PGG.
89 s to a much more international one (with the PGG meetings having made important contributions to the
90                                        Thus, PGG use may be recommended as an adjunct therapy for TNB
91        The PRP-biosensor was able to bind to PGG with higher sensitivity, displaying lower limit of t
92 ounds on PRP and on alpha-amylase binding to PGG were tested (gallic acid, catechin, ethanol, glucose
93 he cyclooxygenase site of oPGHS-1 leading to PGG(2), 11R-HETE, and 15S-HETE and/or 15R-HETE, respecti
94 H(2), but does have a marked activity toward PGG(2) converting it to PGE(2) and 15-keto-PGE(2).
95 minal hinge was mutated to P166/V167G/W168G (PGG), and the C-terminal hinge was mutated to K174G/T175
96 eukocytes or serum from animals treated with PGG glucan.
97 initial oxygenation of arachidonate to yield PGG(2) catalyzed by the cyclooxygenase activity of the e
98  the molecule is optimally arranged to yield PGG(2) versus monohydroperoxy acid products (Val-349, Tr