ライフサイエンスコーパス: PHA

1 PHA induces accumulation of cyclin T1 mRNA and protein,

2 PHA results from failure of apoptosis during gestation.

3 PHA was calculated using the non-laboratory Framingham C

4 PHA was not reported.

5 PHA-665752 also exhibited >50-fold selectivity for c-Met

6 PHA-665752 was identified as a small molecule, ATP compe

7 PHA-665752 was identified as a small molecule, ATP-compe

8 PHA-L-labeled fibers were observed in the ipsilateral po

9 PHA-L-labeled trigemino- and spinothalamic (TSTT) termin

10 Systemic pseudohypoaldosteronism type 1 (PHA-1) is a severe salt-losing syndrome caused by loss-o

11 containing a pseudohypoaldosteronism type 1 (PHA-1)-causing missense point mutation.

12 LIN-15B, LIN-39, MAB-5, MDL-1, MEP-1, PES-1, PHA-4, PQM-1, SKN-1, and UNC-130) at diverse development

13 ted, HT of excess sludge with moderate (13%) PHA content can produce about 50 kg of alkenes per tonne

14 -3-yl]furo[2,3-c]pyridine-5-carboxamide (14, PHA-543,613), a novel agonist of the alpha7 neuronal nic

15 script cause pseudohypoaldosteronism type 2 (PHA II), characterized by hypertension and hyperkalemia.

16 The cocrystal structure of 3 (PHA-665752), bound to c-MET kinase domain, revealed a no

17 o three MET kinase inhibitors (JNJ-38877605, PHA-665752, crizotinib) and one antagonistic anti-MET an

18 urse of this work, we have also identified a PHA polymerase (CapPhaEC) from activated sludge from was

19 uenza A virus (Flu) or phytohemagglutinin A (PHA), but had no effect on CD8(+) T cells.

20 Though much is known about PHA synthesis, little is known about inclusion structure

21 When peat humic acid (PHA) was added to a NOM-deficient sediment concentration

22 pment, extinguish PHA-4 and fail to activate PHA-4 target genes.

23 ive sediment but not in sediments with added PHA, suggesting that the native NOM and the PHA respond

24 In addition, PHA-665752 inhibited HGF-stimulated or constitutive phos

25 on alone, RF ablation combined with adjuvant PHA-665752 or semaxanib reduced distant tumor growth, pr

26 ze the thickness and coverage of an adsorbed PHA layer in a natural nanocolloid is also presented.

27 We examined predicted heart/vascular age (PHA) in six LMICs and the United States.

28 ltaneous application of alpha7 nAChR agonist PHA-543613 and PAM NS-1738 resulted in additive increase

29 mpared local effects of alpha7 nAChR agonist PHA-543613 and PAMs PNU-120596 and NS-1738 on the sponta

30 ls, but proliferated in response to CMV Ags, PHA, and third party cells.

31 Although PHA-E lectin has a strong binding affinity for bisected

32 ora kinase inhibitors (AZD1152, MK-0457, and PHA-739358), at low nanomolar concentrations.

33 of pha-4 function, suggesting that PEB-1 and PHA-4 have common functions in some tissues where they a

34 liferation stimulated by dendritic cells and PHA were 3.9 microM and 2.9 microM, respectively, that i

35 cted, other c-MET inhibitors, crizotinib and PHA-665752, suppressed the growth of c-MET-addicted canc

36 d T lymphocytes were stimulated with LPS and PHA, respectively.

37 the anterograde tracers BDA, neurobiotin and PHA-L in the host.

38 cetyl cysteine, as well as by plumericin and PHA-408, inhibitors of the NF-kappaB pathway.

39 10 responses to EBV lytic antigens, PPD, and PHA correlated inversely with malaria exposure regardles

40 id A, Ppg, and PHA; farming mothers: Ppg and PHA), influenced by maternal farming.

41 ation (nonfarming mothers: lipid A, Ppg, and PHA; farming mothers: Ppg and PHA), influenced by matern

42 s of the shared nervous system (URY, PQR and PHA) to produce mate-searching behavior.

43 ainst VGluT2, tyrosine hydroxylase (TH), and PHA-L.

44 reduction, phosphate release and uptake, and PHA dynamics for all systems, suggesting the validity an

45 a complication of persistent hyaloid artery (PHA).

46 ssion of G1-S cyclins but often as potent as PHA in inducing RNAP II cyclin/CDK complexes.

47 converting most (>70% w/w) of the bacterial PHA stored inside microbial cells into alkene/CO2 gas mi

48 he catalyzed chemical synthesis of bacterial PHAs has been developed, using the metal-catalyzed stere

49 EPHA) was defined as the differences between PHA and chronological age >5 years.

50 ed extensive regulatory interactions between PHA-4, SKN-1, and miRNAs and points to two aging-associa

51 um was indicated by the relationship between PHA content and growth capabilities of the genetically m

52 xybutyrate] (PHB), a renewable biodegradable PHA polymer with potential commercial applications in pl

53 d a pathway to produce alpha-methyl-branched PHA.

54 is also found in T cells upon activation by PHA.

55 regulators and cell signaling events, and by PHA-1, an essential cytoplasmic protein of unknown funct

56 genes and activation of pharyngeal genes by PHA-4.

57 proliferation and IL-2 production induced by PHA, concanavalin A (conA), and anti-TCR MAb.

58 tetraethylammonium), decynium-22, carnitine, PHA (p-aminohippurate), alanine, or inosine.

59 llin 3 (Cul3)--mutations of which all caused PHA-II phenotypes.

60 In contrast, PHA/IL-2, which is widely used to prime cells for HIV in

61 We cultured PHA-activated human T lymphocytes in varying concentrati

62 e expression, mediated by pharynx defective (PHA)-4/FoxA in combination with additional, largely unid

63 examined under a light microscope to detect PHA-L-labeled fibers.

64 lpa1 mutation in combination with different PHA alleles affected the stability of PHA-L lectin.

65 The dominant PHA-II mutation in KLHL3 impaired claudin-8 binding, ubi

66 lectins that bind Gal, including L-PHA and E-PHA.

67 the PhaC2 protein is not the most efficient PHA polymerase biocatalyst.

68 appears as early as 4 years of age enabling PHA to whirl more freely.

69 lity, even though both of these genes encode PHA polymerases that are more efficient enzymes.

70 s established that each gene product encodes PHA polymerase.

71 n, and Phaseolus vulgaris Erythroagglutinin (PHA-E) are used to identify fucose, galactose, alpha2-6-

72 High excess PHA (HEPHA) was defined as the differences between PHA a

73 subsequently arrest development, extinguish PHA-4 and fail to activate PHA-4 target genes.

74 209 binds the pan-pharyngeal Forkhead factor PHA-4 in vitro and responds to ectopic pha-4 expression

75 gans foregut development, the pioneer factor PHA-4/FoxA binds promoters and recruits RNA polymerase I

76 e forkhead box A (FOXA) transcription factor PHA-4 and that autophagy is required to extend longevity

77 orhabditis elegans, the transcription factor PHA-4 has an essential role in the embryonic development

78 at the DR-specific FOXA transcription factor PHA-4 transcriptionally regulates the genes required for

79 s regulated by the FoxA transcription factor PHA-4.

80 R) pathway and the Foxa transcription factor PHA-4.

81 ed miRNAs connected to transcription factors PHA-4/FOXA and SKN-1/Nrf, which are both necessary for D

82 Decreasing MCL-1 levels with flavopiridol, PHA 767491, or shRNA restored sensitivity to ABT-737 res

83 r the control group (median cpm: 107,049 for PHA, 2,111 for Flu, and 2,068 for VLP).

84 [median counts per minute (cpm): 72,849 for PHA, 1,241 for Flu, and 727 for VLP] than for the contro

85 ewly discovered, adult-specific function for PHA-4 in the regulation of diet-restriction-mediated lon

86 igh homology, but TFO71 has unique genes for PHA synthesis, gene regulation and granule management.

87 more efficient and economical processes for PHA production, isolation, purification and improvement

88 Compared to previous protocols for PHA injection in amphibians, this method induced up to 2

89 ma up-regulation in Tempus purified RNA from PHA stimulated cells while only IL2 was up-regulated usi

90 rynx) cells in response to the selector gene PHA-4/FoxA.

91 e TBX-38 and express the organ selector gene PHA-4/FoxA.

92 human ENaC that contained subunits harboring PHA-1-causing substitutions within an absolutely conserv

93 phosphate-accumulating organisms (PAO), have PHA metabolism-related genes with high homology, but TFO

94 characterizations indicate that heteromeric PHA polymerases composed of mixtures of different PhaC p

95 vealed by the occurrence of three homologous PHA polymerase genes (phaC1, phaC2, and phaC3).

96 However, PHAs need to have tunable hydrophilicity, chemical funct

97 romosomes (and human orthologs) PHA1 (HSA1), PHA 2 (HSA3), PHA4 (HSA6), PHA11 (HSA12), PHA13 (HSA2),

98 13 (HSA2), PHA16 (HSA17), and PHA17 (HSA13) (PHA, P. hamadryas; HSA, Homo sapiens).

99 produce fluorinated poly(hydroxyalkanoate) (PHA) bioplastics with fluorine substitutions ranging fro

100 n syndrome--pseudohypoaldosteronism type II (PHA-II)--features hypertension, hyperkalemia, and metabo

101 KS-WNK1 (kidney-specific, KS) is altered in PHA II is not known.

102 ted whole PBMC lysate by Western blot and in PHA-activated CD56 and CD19 subsets by immunohistochemis

103 MDA-7/IL-24 was detected in PHA- and LPS-stimulated whole PBMC lysate by Western blo

104 PRP4 is expressed in PHA-stimulated human T lymphocytes from days 1 and 7 wit

105 Hyperkalemia in PHA II patients with PHA II mutations may be caused, at

106 , and interferon-gamma (IFN-gamma) levels in PHA-stimulated peripheral blood mononuclear cell superna

107 xplanation for the collecting duct's role in PHA-II.

108 Voltage sensitivity in PHA-1 mutants stems from the disruption of critical stru

109 roscopy determined the presence of VGluT2 in PHA-L- or WGA-positive terminals.

110 Pb exposure increased PHA response in both seasons, and decreased T-independen

111 -6 knockout mice (n = 24) or c-met inhibitor PHA 665752 (n = 15), to elucidate the key factors facili

112 e (AMPPNP) or the pyrrolo-pyrazole inhibitor PHA-680626 at 2.4 and 2.1 A resolution, respectively.

113 arily susceptible to the selective inhibitor PHA-665752.

114 was combined with either a c-Met inhibitor (PHA-665752) or VEGF receptor inhibitor (semaxanib) and c

115 line, SNU638, and two related MET inhibitors PHA-665752 and PF-2341066.

116 Injecting PHA-L into the visual, but not into the auditory AI, rev

117 high-molecular-weight, crystalline isotactic PHA copolymers that are hard, ductile, and tough plastic

118 Specifically, transcripts for several known PHA-inducible genes, including IFNgamma, IL13, IL2, IL3,

119 l transformations were observed: at 255 mg/L PHA aggregation of the nanocolloid was actually enhanced

120 lloid was actually enhanced, but at 380 mg/L PHA disaggregation and colloidal stability were promoted

121 ed stability of lectin phytohemagglutinin L (PHA-L) compared to the wild type (wt).

122 antification of p24 in phytohemagglutinin-L (PHA)-stimulated CD4(+) T cells from individuals under ef

123 of other lectins that bind Gal, including L-PHA and E-PHA.

124 of activated T cells by Concanavalin A or L-PHA was also reduced in Fng tKO mice.

125 labeled tissue, we found that double-labeled PHA-L (+)/VGluT2 (+) axon terminals formed synaptic cont

126 ated by a non-LPS stimulus, the plant lectin PHA.

127 ted with Phaseolus vulgaris leucoagglutinin (PHA-L) and labeled dextrans.

128 e tracer Phaseolus vulgaris leucoagglutinin (PHA-L) and the retrograde tracer FluoroGold in specific

129 beled by Phaseolus vulgaris leucoagglutinin (PHA-L) injections, whereas tyrosine hydroxylase (TH) was

130 ction of Phaseolus vulgaris leucoagglutinin (PHA-L) or an adeno-associated virus encoding wheat germ

131 e tracer Phaseolus vulgaris leucoagglutinin (PHA-L) was injected into one inferior colliculus of 10 a

132 BDA) and Phaseolus vulgaris-leucoagglutinin (PHA-L), into four subdivisions of OFC.

133 tracers Phaseolus vulgaris-leucoagglutinin (PHA-L; for outputs) and cholera toxin B subunit (CTB; fo

134 nsport of Phaseolus vulgaris leucoagglutinin(PHA-L) or carbocyanine dyes, we characterize the POm tha

135 odium loss, in the ENaC-mediated salt-losing PHA-1 phenotype.

136 mcl-PHAs were then isolated from the cells and demonstrated

137 ies to conventional carbohydrate-derived mcl-PHAs, which have applications as bioplastics.

138 p to 1.5 g L(-1) of medium chain length, mcl-PHAs, together with an efficient conversion (80% yield)

139 dium chain-length polyhydroxyalkanoates (mcl-PHAs).

140 further demonstration of their utility, mcl-PHAs were catalytically converted to both chemical precu

141 kept on a regular-salt diet, thus mimicking PHA-1.

142 Given the conservation of miRNAs, PHA-4, and SKN-1 across phylogeny, these interactions ar

143 rt that treatment of PBLs with two mitogens, PHA and PMA, results in accumulation of cyclin T1 via di

144 tronger responses to ConA (373+/-174 ng/mL), PHA (498+/-196 ng/mL) and EBV (152+/-179 ng/mL), when co

145 VL or to HVL patients (ConA 185+/-114 ng/mL, PHA 318+/-173 ng/mL, and EBV 33+/-42 ng/mL).

146 te to feed aerobic bacteria and produce more PHA.

147 o stimuli that mimic TCR activation, namely, PHA and PMA.

148 Simultaneously, most of non-PHA biomass was converted into water-soluble compounds (

149 enzenes) and higher order products observed (PHA, PHQ, and LMW oxo- and dicarboxylic acids).

150 Approximately 95% of PHA-L-labeled terminals from the central lateral, midlin

151 The accumulation of significant amount of PHA inside aerobic microbial cells occurs when a surplus

152 d firing rate of the neurons, application of PHA-543613 resulted in almost equal distribution of faci

153 Thus, in case of PHA, we advocate FA to be performed and if connection wi

154 ion of recombinant human TL1A to cultures of PHA-stimulated lamina propria mononuclear from CD patien

155 In in vivo studies, a single dose of PHA-665752 inhibited c-Met phosphorylation in tumor xeno

156 nt with the predicted immunologic effects of PHA stimulation.

157 markable occurrence of inhibitory effects of PHA-543613 (possibly originating from receptor desensiti

158 , NS-1738 counteracted inhibitory effects of PHA-543613 in 5 out of 6 neurons, resulting in a synergi

159 n and, most recently, protein engineering of PHA biosynthetic enzymes.

160 hromatin opening, is an important feature of PHA-4 pioneer factor activity.

161 Herein, hydrothermal treatment (HT) of PHA-containing sludge at 300 and 375 degrees C was demon

162 etabolic alkalosis, an exact mirror image of PHA-II.

163 , isolation, purification and improvement of PHA material properties.

164 rial antigens, and resulted in inhibition of PHA-induced proliferation.

165 Swelling induced by single injection of PHA or killed bacteria was not significantly affected by

166 swelling caused by a secondary injection of PHA, was significantly reduced by B. dendrobatidis super

167 Our current knowledge of PHA production and utilization in vitro and in vivo as w

168 ctivity was detected in the vast majority of PHA-L- or WGA-positive terminals forming asymmetric syna

169 To determine if the major surface protein of PHA inclusions, PhaP, is involved in the structure of th

170 Finally, the physiological role of PHA accumulation in enhancing the fitness of R. rubrum w

171 The results indicated a substantial role of PHA consumption in N2O accumulation due to the relativel

172 to describe N2O dynamics and the key role of PHA consumption on N2O accumulation during the denitrify

173 The role of PHA-4 in lifespan determination is specific for dietary

174 In this work, the selection of PHA storing bacteria was integrated with the side stream

175 ine conditions accomplished the selection of PHA storing biomass and nitrogen removal via nitrite.

176 The results showed that the selection of PHA storing biomass was successful in both configuration

177 ferent PHA alleles affected the stability of PHA-L lectin.

178 cent progress in the synthetic strategies of PHA-based water soluble polymers, including the function

179 ) was employed to elucidate the structure of PHA inclusions at the nanoscale level, including the cha

180 Suppression of PHA-induced T-cell proliferation by dexamethasone was as

181 The use of PHA may offer a useful avenue to communicate CVD risk.

182 Our results expand the utility of PHA-based pathways and provide biosynthetic access to al

183 However, the biosynthesis of PHAs is slow and expensive, limiting their broader appli

184 oups and the block/graft copolymerization of PHAs with hydrophilic components in various polymeric ar

185 polymers, including the functionalisation of PHAs with polar functional groups and the block/graft co

186 The versatility of PHAs has made them good candidates for the study of thei

187 nd the regulation of these miRNAs depends on PHA-4 and SKN-1.

188 polyphosphate and four-step anoxic growth on PHA using nitrate, nitrite, nitric oxide (NO), and N2O c

189 sistant 1.89 vs. sensitive 0.96, P =0.02) or PHA (1.66 vs. 0.96, respectively, P =0.02).

190 DTH responses induced by killed bacteria or PHA in the presence of B. dendrobatidis supernatants.

191 duction in response to lipopolysaccharide or PHA was lower in the salmon group (all P </= 0.045).

192 eduction in proliferation with either PPD or PHA at 2 hours compared with 0 hours.

193 ulated with lipid A, peptidoglycan (Ppg), or PHA.

194 ter stimulation with lipid A, peptidoglycan, PHA, house dust mite (Der p 1), or Der p 1 plus lipid A.

195 In combination with phytohaemagglutin (PHA) lymphocyte-conditioned medium, the number and size

196 out prior incubation in phytohaemagglutinin (PHA) - a process commonly used in immune population expe

197 Phytohemagglutinin (PHA), purified protein derivative (PPD), and T cell epit

198 ein derivative (PPD) and phytohemagglutinin (PHA).

199 oncanavalin A (ConA) and phytohemagglutinin (PHA; 190+/-86 ng/mL, 328+/-163 ng/mL) and detectable EBV

200 eptide loaded autologous phytohemagglutinin (PHA) blasts.

201 3/4 and NS5) and control phytohemagglutinin (PHA) was monitored prospectively and was correlated with

202 MMG eliminated phytohemagglutinin (PHA)-stimulated proliferation of PBMCs.

203 In phytohemagglutinin (PHA)-stimulated CD8-depleted peripheral blood mononuclea

204 ature dendritic cells or phytohemagglutinin (PHA) but did not induce apoptosis.

205 concanavalin A (ConA) or phytohemagglutinin (PHA) stimulated canine peripheral blood mononuclear cell

206 opolysaccharide (LPS) or phytohemagglutinin (PHA) stimulation, respectively.

207 polysaccharide (LPS), or phytohemagglutinin (PHA), in the presence or absence of dexamethasone (Dex).

208 on of killed bacteria or phytohemagglutinin (PHA).

209 4 (TLR4), and suppressed phytohemagglutinin (PHA)-mediated proliferation of normal human T lymphocyte

210 tor-alpha in response to phytohemagglutinin (PHA) and of IL-2 in response to Dermatophagoides pterony

211 oliferative responses to phytohemagglutinin (PHA), influenza virus (Flu), and HPV16 virus-like partic

212 ne or IL-2 combined with phytohemagglutinin (PHA) in CD8-depleted PBMCs.

213 Plant lectins such as phytohemagluttinin (PHA) can activate the T cell receptor (TCR) and other ce

214 regulation of the MIP-1alpha promoter by PMA/PHA stimulation in Jurkat T-cells.

215 the proximal RUNX site is essential for PMA/PHA-stimulated activation of the MIP-1alpha promoter.

216 zed polyacrylamide, polyhydroxamicalkanoate (PHA), which mimics the performance of the acetylcholines

217 Polyhydroxyalkanoate (PHA) inclusions are polymeric storage inclusions formed

218 uptake, glycolysis and polyhydroxyalkanoate (PHA) synthesis were conserved in all these Accumulibacte

219 dation of glycogen and polyhydroxyalkanoate (PHA), which function as energy and carbon storage compou

220 M effects on bacterial polyhydroxyalkanoate (PHA), specifically polyhydroxybutyrate (PHB), biosynthes

221 torage polymers (e.g., Polyhydroxyalkanoate (PHA)) could be produced and consumed dynamically.

222 s based on the natural polyhydroxyalkanoate (PHA) pathway offers an attractive route for producing su

223 o produce a variety of polyhydroxyalkanoate (PHA) biopolymers with desirable structures and material

224 review then focuses on polyhydroxyalkanoate (PHA) synthases that generate polyoxoesters.

225 Polyhydroxyalkanoates (PHA) are a key constituent of excess sludge produced by

226 al polyesters such as polyhydroxyalkanoates (PHA) are produced by a large fraction of the community a

227 Polyhydroxyalkanoates (PHAs) are excellent candidate biomaterials due to their

228 Polyhydroxyalkanoates (PHAs) from activated sludge and renewable organic materi

229 Bacterial polyhydroxyalkanoates (PHAs) are a unique class of biodegradable polymers becau

230 Production of polyhydroxyalkanoates (PHAs) has been investigated for more than eighty years b

231 n the biosynthesis of polyhydroxyalkanoates (PHAs) in the Rhodospirillum rubrum genome revealed by th

232 t-based production of polyhydroxyalkanoates (PHAs), silk, elastin, collagen, and cyanophycin with an

233 domonas resinovorans) polyhydroxyalkanoates (PHAs) producing cell factories.

234 cellular free iron and polyhydroxylakanoate (PHA), a bacterial energy storage polymer.

235 death in a significant population of primary PHA-activated T cells (72%) and lymphoid tumor cell line

236 r cells, using flow cytometry, without prior PHA treatment.

237 C2 contributes the major capacity to produce PHA, even though the PhaC2 protein is not the most effic

238 The properties associated with the produced PHA suggest that they are suitable for thermoplastic pro

239 Pseudohypoaldosteronism (PHA) types I and II are curious genetic disorders that s

240 gulated genes were related to N2O reduction, PHA synthesis and acetyl-CoA formation.

241 er transcription factor and master regulator PHA-4/FoxA, followed by the cytoskeletal regulator and k

242 gulated by miR-228, whereas miR-71 represses PHA-4.

243 gh a dietary restriction mechanism requiring PHA-4.

244 ristics, and the properties of the resulting PHAs have been examined.

245 f mono- and polyhydroxylated aromatic rings (PHA) and chromophoric mono- and polyhydroxylated quinone

246 escue diet reinstated the symptoms of severe PHA-1 syndrome and significantly reduced NCC phosphoryla

247 In addition, water soluble PHA monomer production will be briefly introduced, with

248 These chemically modified water soluble PHAs have significant impact on materials engineering an

249 The applications of water soluble PHAs in controlled drug release, cancer therapy, DNA/siR

250 roposed biomimetic biosensor, denoted as SPE/PHA/mPEG, represents a significant advance in the field,

251 At a later developmental stage, PHA-4 promotes chromatin opening.

252 an be highly induced by the T cell-stimulant PHA, suggesting it is a particularly important TNF-alpha

253 In cellular studies, PHA-665752 potently inhibited HGF-stimulated and constit

254 tive CD4 T cell lines, as well as short-term PHA-activated CD4 T cells, can express NKG2C, the activa

255 We studied induced immunity by testing PHA and humoral responses.

256 We also found that PHA and anti-CD3 Abs induce the expression of both the c

257 We found that PHA-L- or WGA-positive terminals from tagged VTA cells m

258 We hypothesized that PHA twisting led to torsion of the residual primordial c

259 henotypic evidence to support the model that PHA-1, a novel protein, and UBC-18, a ubiquitin-conjugat

260 In turn, we show that PHA-4 and SKN-1 are negatively regulated by miR-228, whe

261 We show that PHA-4 directly activates mRNA expression of a broad coho

262 Our data suggest that PHA-4 and DAF-12 endow the pharynx with transcriptional

263 ic pha-4 expression in vivo, suggesting that PHA-4 directly initiates ceh-22 expression through de209

264 The PHA bears functional groups inserted along its backbone

265 The PHA selection degree was evaluated by the volatile fatty

266 The PHA turnovers play important roles in nitrous oxide (N2O

267 based interfering species did not affect the PHA performance, which endorsed its superior behavior.

268 atty acid (VFA) uptake rate (-qVFAs) and the PHA production rate (qPHA), which were 239 +/- 74 and 89

269 PHA, suggesting that the native NOM and the PHA respond differently to changes in ionic strength.

270 L-7, as compared with those activated by the PHA/IL-2 treatment.

271 gsae comparison, CompareProspector found the PHA-4 motif and the UNC-86 motif.

272 nd phaC1, the PHA polymerase situated in the PHA biosynthetic operon, plays a minor role in this capa

273 The monomers in the PHA polymer produced by these strains establish that Pha

274 strains that express different levels of the PHA polymer.

275 -37/ZTF-12 as an additional component of the PHA-1 network regulating pharyngeal development.

276 tributor to PHA productivity, and phaC1, the PHA polymerase situated in the PHA biosynthetic operon,

277 Experimental immunostimulation using the PHA (phytohaemagglutinin assay) challenge technique did,

278 te occurred in the same reactor in which the PHA selection process occurred, while in configuration 2

279 Thereby, PHA was immobilized on screen printed electrodes (SPE) t

280 st, phaC3 is an insignificant contributor to PHA productivity, and phaC1, the PHA polymerase situated

281 of testing were as follows: proliferation to PHA, 77%; maternal engraftment, 35%; and genotype, 79% (

282 n mutants were characterized with respect to PHA production and growth capabilities on acetate or hex

283 trating CD4(+)CD25(-) T cells in response to PHA stimulation.

284 b mesylate, they proliferated in response to PHA, demonstrating that inhibition is reversible.

285 uL) also had markedly decreased responses to PHA (typical SCIDs).

286 diversity, and 3) proliferative responses to PHA and streptococcal superantigen, streptococcal pyroge

287 In vitro responses to PHA were recorded in 88 patients, of whom 68 had a genet

288 ns of flow cytometry), in vitro responses to PHA, and TREC levels, all measured at presentation, were

289 In contrast, IFN- gamma responses to PHA, PPD, and Plasmodium falciparum MSP-1 peptides did n

290 e relatively low N2O reduction rate by using PHA during denitrifying phosphorus removal.

291 HIV-1 viral infectivity both in vitro using PHA plus IL-2 activated PBL and in vivo using the human

292 similarly to vincristine treatment, whereas PHA-665752 or crizotinib treatment markedly induced G(0)

293 maximal proliferative response compared with PHA-activated stationary cultures.

294 ddition, the voltage dependence of ENaC with PHA-1 substitutions is akin to that which results from s

295 at de209 also binds factors functioning with PHA-4 to specifically activate ceh-22 expression in phar

296 By interacting with PHA-4 and SKN-1, miRNAs transduce the effect of dietary-

297 Stimulation of human T lymphocytes with PHA resulted in a strong downregulation of 5-LO mRNA exp

298 Hyperkalemia in PHA II patients with PHA II mutations may be caused, at least partially, by i

299 n head kidney leukocytes by stimulation with PHA or poly(I:C) and in kidney and spleen of fish inject

300 Treatment with PHA-665752 triggers massive apoptosis in 5 of 5 gastric

301 D14(+)/CD16(-)) compared to controls without PHA.