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1 PIH's experience in Sierra Leone is one of multiple part
8 ation in the pathogenic mechanism of PHH and PIH and highlight promising targets for therapeutic inte
9 arget inflammatory mediators in both PHH and PIH could address the multiple drivers of disease, inclu
13 d contrast to the cellular uptake of (1)(4)C-PIH and (1)(4)C-Bp4eT, which were linear as a function o
14 C-pyridoxal isonicotinoyl hydrazone ((1)(4)C-PIH) and the thiosemicarbazone ((1)(4)C-2-benzoylpyridin
15 cium intake, and women at risk of developing PIH, may benefit from consuming additional dietary calci
16 onversely, the PIH1D1 phospho-binding domain PIH-N is required for association with MRE11 phosphoryla
19 cribe how, in the aftermath of the epidemic, PIH is partnering with the public sector to strengthen t
22 eight showed a U-shaped relation to risk for PIH, with the highest risks associated with very low and
23 ncluded studies involved laser treatment for PIH with the degree of pigmentation as a measure of outc
25 ow the international NGO Partners In Health (PIH) partnered with the Government of Sierra Leone and W
26 rs of the pyridoxal isonicotinoyl hydrazone (PIH) class can restrict the growth of clinically signifi
27 and 59Fe-pyridoxal isonicotinoyl hydrazone (PIH) to determine whether the decrease in iron uptake by
28 estigation of post-infectious hydrocephalus (PIH) and post-hemorrhagic hydrocephalus (PHH) models rev
29 vors developed postinfectious hydrocephalus (PIH) and 1 (3%) additional survivor had neurodevelopment
31 tion for postinflammatory hyperpigmentation (PIH) but can pose a clinical dilemma given the risk for
32 tion for postinflammatory hyperpigmentation (PIH) but can pose a clinical dilemma given the risk for
35 ter risk for pregnancy-induced hypertension (PIH), a disorder associated with insulin resistance and
36 litus (GDM), pregnancy-induced hypertension (PIH), emergency cesarean delivery, low birthweight (LBW)
38 al of pharmacologically induced hypothermia (PIH) by the novel neurotensin receptor 1 (NTR1) agonist
40 ined this as prolonged intermittent hypoxia (PIH) because the duration of hypoxia in skeletal muscle
42 similar age and body weight but less than in PIH women (P<0.001) (146+/-23.5 impulses/100 beats).
47 cise factors involved in the pathogenesis of PIH are unclear, but several alterations in calcium meta
55 or pharmacological immunomodulation prevents PIH and PHH by antagonizing SPAK-dependent CSF hypersecr
56 mune-epithelial cell cross talk, and reframe PIH and PHH as related neuroimmune disorders vulnerable
63 oke are reduced by approximately 30-40% when PIH therapy is initiated either immediately after stroke