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1                                              PM ARF6 is polarized and colocalized with RPH3A, RAB21,
2                                              PM receptors can be dynamically sorted into membrane dom
3                                              PM(10) emissions were sampled on a 47 mm-diameter polyte
4                                              PM(2.5) levels of lead, arsenic, chromium, and zinc were
5 seekers on Manus Island (36.4%) between 4:00 PM and 7:59 PM (p = 0.02), and among asylum seekers in o
6 rs in onshore detention (20.4%) between 8:00 PM and 11:59 PM (p < 0.001).
7  05.30 hours to 21.00 hours (5:30 AM to 9:00 PM) on television in the UK.
8     Sixteen PM boys (hyper/hypo: 7/9) and 10 PM girls (hyper/hypo: 2/5, 3 both) displayed aberrant LG
9 ty (CDVA), and glare CDVA was performed at 4 PM (afternoon) and the following day at 8 AM (morning).
10  detention (20.4%) between 8:00 PM and 11:59 PM (p < 0.001).
11 anus Island (36.4%) between 4:00 PM and 7:59 PM (p = 0.02), and among asylum seekers in onshore deten
12 ronic exposure to particulate matter < 2.5u (PM(2.5)) has been linked to cardiopulmonary disease.
13 bjects were significantly more active from 7 PM to midnight and less active from 3 to 6 AM.
14 NG filters exhibit high efficiency (>99.995% PM(0.3) removal), low air resistance (<0.09% atmosphere
15 re anticipated to incorporate and accumulate PM(1) in their bodies.
16 dantly present in both ambient fine aerosol (PM(2.5)) and laboratory-generated isoprene SOA.
17 t Acod1/endogenous itaconate does not affect PM-induced inflammation or NRF2 activation in macrophage
18 st-exposure was significantly elevated after PM-exposure.
19 d attenuation of antioxidant potential after PM exposure.
20 ression of epidermis treated with coarse air PM.
21                     Even without any ambient PM(2.5) information, the developed models still demonstr
22 for the other models when regulatory ambient PM(2.5) data were used as predictors.
23  surface area of up to 220 mum(2)/cm(3), and PM(0.1) up to 16 mug/m(3)) with complex PM(0.1) chemistr
24              Levels of metals in PM(2.5) and PM(10) exceeded health guidelines at multiple sites.
25 a- and beta-activities for daily PM(2.5) and PM(10) filters collected at the Harvard Supersite in dow
26 potential were conserved between PM(2.5) and PM(10), although considerable temporal variations existe
27 : LM (lateromedial), AL (anterolateral), and PM (posteromedial)) in mice of both sexes.
28                      Cooperation of ARF6 and PM PtdIns4P direct RPH3A polarization, which is importan
29 mory decline, and that the effects of AT and PM FTP, but not EC, were driven by Abeta+ individuals.
30 ferent cognitive consequences of EC, AT, and PM tau.
31 uced combined monetized damages from GHG and PM(2.5)-related emissions by 42% and 57%.
32 nd higher visual areas LM (lateromedial) and PM (posteromedial) of mice.
33 o higher visual areas, LM (lateromedial) and PM (posteromedial).
34 olocalized with RPH3A, RAB21, PIP5K1C90, and PM PtdIns4P in mouse and human neutrophils upon integrin
35 cer, and chronic cancer HAPs health risk and PM in smoke decrease as a function of smoke age by up to
36  interactions between FA supplementation and PM exposures, after controlling for individual character
37 his study showed that the addition of TP and PM enhanced quality of cooked sausages produced with red
38 er concentrations (PNC; a proxy for UFP) and PM(2.5) (R(2) of 0.38 and 0.41, respectively).
39       Additionally, expression of artificial PM-PER tethers is sufficient to restore retention in inp
40 an framework: the Poisson-multinomial (ASHIC-PM) model and the zero-inflated Poisson-multinomial (ASH
41                                           At PM, 31% of patients had visually detectable FLs (2% extr
42                                           At PM, 98% of patients retained residual BM diffuse uptake,
43  survival (PFS) and overall survival (OS) at PM (OS: hazard ratio [HR], 0.6 and 0.47, respectively; P
44 PM(10) and further size-resolved atmospheric PM samples in the size range from 15 nm to 10 mum collec
45 exposed to very unhealthy air (daily average PM(2.5) >= 150.5 mug/m(3)).
46                            All-India average PM(2.5) is expected to increase from 41.4 +/- 26.5 mug m
47  novel findings that the association between PM and MI incidence is robust to adjustment for road tra
48 er diabetes mediates the association between PM(2.5) and CKD.
49 s minimally mediated the association between PM(2.5) and kidney outcomes.
50  functional potential were conserved between PM(2.5) and PM(10), although considerable temporal varia
51                               Levels of both PM and the total in-cylinder PAHs decreased following a
52 low release of specific lipids into the bulk PM.
53 ed polymer (NIP) films were characterized by PM-IRRAS and UV-vis spectroscopy and imaged with AFM.
54 xylase 1) as one of the top genes induced by PM in macrophages.
55  with the introgressed junctions mediated by PM resistance R proteins (R genes) that may involve mult
56  in vivo induction of Treg cells promoted by PM without altering the capacity to induce functional al
57   During some cooking activities, calculated PM(2.5) mass concentrations (assuming a density of 1 g c
58 tabolomic profiling of premutation carriers (PM) who, as part of an ongoing longitudinal study, emerg
59 n decreased the rigidity of the colonic cell PM.
60                     We aimed to characterize PMs and associate their activation with outcomes of pati
61 ensing peptide(s) in the ER that can cluster PM-derived cholesterol into transient detergent-resistan
62                               By comparison, PM from low elevation contained mixtures of organic and
63  and PM(0.1) up to 16 mug/m(3)) with complex PM(0.1) chemistry that included 40-60 wt % organic carbo
64 r, the intracellular machinery that controls PM accumulation of FLS2 remains largely undefined.
65 erior insular cortex (AIC), premotor cortex (PMd), and inferior parietal lobule (IPL) were modulated
66                                        Daily PM(2.5) levels were estimated at 1-kmx1-km grid cells fr
67 rk presents results from more than 300 daily PM(10) and further size-resolved atmospheric PM samples
68 e gross alpha- and beta-activities for daily PM(2.5) and PM(10) filters collected at the Harvard Supe
69 , each interquartile range increase in daily PM(2.5) was associated with an 48% higher odds of a VA e
70  considered mass concentration only, despite PM(2.5) being a heterogeneous mixture.
71 ate matter <=10 mum in aerodynamic diameter (PM(10)) during each trimester, 0-6 months, 7-12 months (
72 ticulate air pollution <2.5 mum in diameter (PM(2.5)) is a major environmental threat to global publi
73 culate matter <=2.5 um aerodynamic diameter; PM(2.5)) and particle radioactivity.
74 cal or nonclinical setting) for differential PM(2.5) exposure and outcome classification.
75 s show that these mutations severely disrupt PM association of fibroblast growth factor receptor subs
76                                     Elevated PM(2.5) concentrations frequently cause severe air pollu
77 hermore, quantitative proteomics of enriched PM fractions revealed that EPS1 was required for proper
78                         Many functions of ER-PM contacts have been proposed, including nonvesicular l
79 a LAM/StARkin-family protein localized to ER-PM contact sites.
80 -luminal calcium decrease, relocalizes to ER-PM junctions, and recruits and gates ORAI1 channels.
81 l gaseous and particulate pollutants (except PM(2.5)) were 1.1- to 4.8-fold higher than when the resi
82 he filtered air group (FA), one-day-exposure-PM(2.5) did not affect adiposity.
83 e data demonstrated that short-term exposure-PM(2.5) increases hypothalamic inflammation, similar to
84                           Long-term exposure-PM(2.5) is even worse, leading to leptin resistance, hyp
85 ssion were found after twelve-weeks-exposure-PM(2.5).
86          Finally, visual inspection of (18)F-PM-PBB3-PET images was indicated to facilitate individua
87  children (age 8-12 years, 19/14 male/female PM carriers, 15/13 male/female controls).
88 icantly influence the health effects of fine PM.
89 ising supplements to regulatory monitors for PM(2.5) exposure assessment.
90         Comparable results were observed for PM(2.5) interventions.Conclusions: We estimated that sub
91  evidence of a sensitive exposure period for PM(10) from road traffic.
92                   Temperature thresholds for PM transition from low to high organic carbon content we
93 ; T5: 50 mg of sodium nitrite and 0.150 uL/g PM.
94 sodium nitrite, 0.075 uL/g TP and 0.075 uL/g PM; T5: 50 mg of sodium nitrite and 0.150 uL/g PM.
95 harmful agents, notably combustion-generated PM.
96 paclitaxel in PEG-b-PLA micelles, as Genexol-PM(R), permits dose escalation over Taxol(R), enhancing
97  of the virtual flows of water, energy, GHG, PM(2.5), labor, and value added embodied in internationa
98 lated between HC and the premutation groups (PM).
99 ug/m(3) in summer, and 107 mug/m(3) and 24 h PM(2.5) in winter.
100 mum peak 8 h ozone level of 162 ppb and 24 h PM(2.5) of 42.7 mug/m(3) in summer, and 107 mug/m(3) and
101 -square error (RMSE) of the predicted hourly PM(2.5) was 1.73 mug/m(3) for the LSTM model and in the
102                    It remains unclear on how PM(2.5) interacts with other air pollutants and meteorol
103 imate the standard deviation of hypothetical PM(2.5) concentration distributions defined at 0.1 mug/m
104 although they were already first detected in PM in the mid-1970s.
105                  We show that differences in PM(2.5) between more and less polluted areas declined su
106 al arsenic and all species known to exist in PM.
107 ratio [95% CI] for a 10 mug/m(3) increase in PM(10): for mild: 1.20 [0.88-1.64], moderate: 1.53 [1.07
108                      Short-term increases in PM(2.5) from traffic and other combustion sources appear
109 PADs) have been used for detecting metals in PM but require different devices and methods for differe
110                          Levels of metals in PM(2.5) and PM(10) exceeded health guidelines at multipl
111 simultaneous detection of multiple metals in PM.
112                                   Neurons in PM, compared with V1 or the other HVAs, have significant
113  reactive oxygen species (ROS) production in PM-activated DCs, impairing their capacity to generate f
114  rigidity was associated with a reduction in PM free cholesterol levels and the intracellular accumul
115 nization stages: area metrics play a role in PM(2.5) trends of small-sized cities at the early urban
116  involved during compatible and incompatible PM-watermelon interactions, we inoculated PM susceptible
117 hat garbage burning emissions could increase PM(2.5) concentrations by nearly 30% in India and Nepal,
118                    Regulations on the indoor PM(2.5) level and measurement schemes are not well estab
119 le PM-watermelon interactions, we inoculated PM susceptible (USVL677-PMS) and resistant (USVL531-MDR)
120 Here, we estimate daily average ground-level PM(2.5) concentrations at a 1 km resolution during the O
121 tdoor activities of the public were limited, PM(2.5) concentrations in China and South Korea between
122                       We find that the lower PM(2.5) in the mitigation pathway (34.2% and 32.6%, resp
123                                    The LPS + PM group showed increased BALF leukocytes, characterized
124                      Peritoneal macrophages (PMs) regulate inflammation and control bacterial infecti
125 s (IF), longissimus dorsi (LD), psoas major (PM) and supraspinatus (SS) were obtained from ten Boer c
126  pathological lesions and placental malaria (PM).
127 Polymerized allergoids conjugated to mannan (PM) are suitable vaccines for allergen-specific immunoth
128 l concentrations in fine particulate matter (PM(2.5)) are important for understanding potential healt
129 average decrease of fine particulate matter (PM(2.5)) concentrations of ~14% in Beijing and surroundi
130         Exposure to fine particulate matter (PM(2.5)) from fuel combustion significantly contributes
131 approach for quantifying particulate matter (PM(2.5)) health impacts.
132 exposure to ambient fine particulate matter (PM(2.5)) is associated with increased risk of chronic ki
133 ects independent of fine particulate matter (PM(2.5)) mass is inconclusive.
134 nts of decadal-long fine particulate matter (PM(2.5)) trends in all 626 Chinese cities at the county
135     Data on ambient fine particulate matter (PM(2.5)) was obtained from the National Aeronautics and
136 s such as ozone and fine particulate matter (PM(2.5))(4).
137 we measured outdoor fine particulate matter (PM(2.5)), species of polycyclic aromatic hydrocarbons (P
138 impact of inhalable fine particulate matter (PM(2.5), aerodynamic diameter <2.5 mum) on public health
139          Our focus is on particulate matter (PM) and the associated low molecular weight polycyclic a
140 with exposure to ambient particulate matter (PM) are a major concern around the world.
141 pheric ozone (O(3)), and particulate matter (PM) can be reduced by urban trees through processes of a
142 patiotemporally resolved particulate matter (PM) estimates are essential for reconstructing long and
143 ciation between maternal particulate matter (PM) exposure and preterm delivery (PTD) by folic acid (F
144              Analysis of particulate matter (PM) is important for the assessment of human exposures t
145                          Particulate matter (PM) pollution has become a serious public health issue,
146 h effects of atmospheric particulate matter (PM) using mass-based measurements as exposure surrogates
147 rogen dioxide (NO2); and particulate matter (PM) with aerodynamic diameter < 2.5 mum (PM2.5).
148  copollutants, including particulate matter (PM) with an aerodynamic diameter of <= 10 mum or 2.5 mum
149 g nitrogen oxides (NOX), particulate matter (PM), and components of PM were estimated at participants
150 able on total arsenic in particulate matter (PM), but little is known about the relative contribution
151  of approximately 1 mum (particulate matter, PM(1)) are released from fossil fuel combustion into the
152        Our approach utilizes the annual mean PM(2.5) concentration and corresponding standard deviati
153 position in AT than in the posterior-medial (PM) memory network.
154 ate levels of PI within the plasma membrane (PM) and endosomes.
155  curvature connected to the plasma membrane (PM) by a neck region with opposing curvature.
156 erol in LEL and had reduced plasma membrane (PM) cholesterol.
157 interact primarily with the plasma membrane (PM) for its biological activity.
158 y protein engagement at the plasma membrane (PM) is crucial to a broad range of cellular functions, i
159  endoplasmic reticulum (ER)-plasma membrane (PM) junctions and STIM1 translocation to these junctions
160 TIM1 translocates to the ER-plasma membrane (PM) junctions to interact with ORAI1, the pore subunit o
161                         The plasma membrane (PM) provides a critical interface between plant cells an
162 MD then oligomerizes in the plasma membrane (PM) to form pores that increase membrane permeability, l
163 doplasmic reticulum (ER) to plasma membrane (PM) transport.
164 of the octameric TPC at the plasma membrane (PM), we performed state-of-the-art dual-color live-cell
165 rough crosstalk between the plasma membrane (PM), where most cellular cholesterol resides, and the en
166 is a calcium channel in the plasma membrane (PM).
167 t of the inner layer of the plasma membrane (PM).
168 ophysical properties of the plasma membrane (PM).
169 anchoring or docking to the plasma membrane (PM).
170 ysis of isolated mammalian plasma membranes (PMs).
171 te matter with diameter of <2.5 micrometers (PM(2.5)) particulate exposure) stratified by tertiles of
172                              Powdery mildew (PM) caused by Podosphaera xanthii is one of the most imp
173 rticulate matter of a diameter of <=2.5 mum (PM(2.5)) in mice fed with a chow diet leads to similar m
174 tter with an aerodynamic diameter <=2.5 mum [PM(2.5)]) and other important covariates.
175 ess than 10 mum, and between 2.5 and 10 mum; PM(2.5) absorbance (a measurement of the blackness of PM
176 cles with an aerodynamic diameter <=2.5 mum; PM(2.5)) have generally considered mass concentration on
177  printers emit high levels of nanoparticles (PM(0.1)) during operation.
178                Here we directly set national PM(2.5) mortality cost reduction targets within a global
179 bsorbance (a measurement of the blackness of PM(2.5) filters); and nitrogen oxides levels.
180  may fully characterize the health burden of PM(2.5).
181 , particulate matter (PM), and components of PM were estimated at participants' homes during pregnanc
182                          Direct detection of PM lipids in the 'periprotein space' supports the conclu
183 els of particle radioactivity, the effect of PM(2.5) on VAs is reduced.
184 revent the adverse cardiovascular effects of PM(2.5), guide the use of the most proven/viable approac
185                     The B[a]P equivalence of PM in the engine cylinder increased during the period of
186                The bioaccumulation factor of PM(1) in sea anemones was approximately 5-7 orders of ma
187 stimate that filtration removed ~130 mg/h of PM(0.01-0.3).
188              CCC1 functions independently of PM depolarization and negatively regulates pathogen-asso
189 rates that AP-2-dependent internalization of PM proteins via the recognition of functional Tyr motifs
190 between 2012 and 2016 with highest levels of PM(2.5) in the Department of Lima.
191 e traveling to locations with high levels of PM(2.5).
192 ary aerosol contributes the vast majority of PM(2.5) mass.
193  an average of 24.1 +/- 12.8% of the mass of PM.
194 cle surfaces, may be responsible for part of PM toxicity.
195                     In fact, the presence of PM(1) in marine biota has never been reported.
196                   Considerable quantities of PM(1) eventually enter the oceans as suspended particula
197 lly specific, suggesting that suppression of PM altered sensory integration or the decision-making pr
198 ond ROS) upon exposure to different types of PM in the future.
199 is poses limitations on our understanding of PM health effects.
200 mpanied by a contemporaneous ~15% upsurge of PM(2.5) concentrations over large areas in southern Chin
201                       We combine 36 years of PM(2.5) concentrations measured over ~8.6 million grid c
202 rban form evolution has long-term effects on PM(2.5) level, but the dominant factors shift over the u
203 ty exists as to whether the current focus on PM(2.5) mass or individual components may fully characte
204 result of interventions that reduce NO(2) or PM(2.5) concentrations.
205 ypothetical interventions on either NO(2) or PM(2.5).
206 ferentially disrupts in V1, but not in LM or PM.
207                           Changes in outdoor PM(2.5) exposure due to migration drove 137.1 (95%CI: 93
208 nts experienced 10-fold increases in outdoor PM(2.5) exposure in comparison with non-migrants.
209                           Changes in outdoor PM(2.5) exposure were observed between 2012 and 2016 wit
210   The overall geometric mean (GM) of outdoor PM(2.5), BaP, NAP, and NO(2) were 45.3 ug/m(3), 9.7 ng/m
211 Fe), are toxic components of fine particles (PM(2.5)).
212 TP) and essential oil of organic peppermint (PM) on pH, color, residual nitrite content, lipid oxidat
213 he chemicals pyridostigmine (PB), permetrim (PM) and N,N-diethyl-m-toluamide (DEET) used as protectan
214                  In plants, the plasmodesmal PM is a discrete microdomain that hosts specific recepto
215 GBS, the sensitivities of NAAT, ChromID plus PM CDM at 48 h, and ChromID alone at 48 h were 96.8%, 95
216 le of fine particulate matter air pollution (PM(2.5), or particulate matter <=2.5 mum in diameter) in
217 banicity, with particulate matter pollution (PM(25)) inversely related to home value, walkability, an
218 isproportionately much from large coal power PM(2.5) and SO(2) emission reductions.
219 before starting maintenance (premaintenance [PM]).
220 elopment but directly influenced by prenatal PM(2.5).
221 s revealed that EPS1 was required for proper PM abundance of a discrete subset of proteins with diffe
222                  Therefore, disrupting K-Ras PM interaction is a tractable approach to block oncogeni
223  locations that most cost-effectively reduce PM(2.5) mortality.
224 ired flg22 signaling correlated with reduced PM accumulation of FLS2 and its coreceptor BRASSINOSTERO
225                The spatiotemporally resolved PM estimations produced by the suggested model can be ap
226 P2, another truncated variant, also restored PM cholesterol in ORP1-null cells.
227                          At the daily scale, PM(2.5) was positively correlated with O(3), daily maxim
228            At both daily and monthly scales, PM(2.5) was positively correlated with CO and negatively
229           Compared to other indoor settings, PM(2.5) prediction models for large office buildings are
230                                      Sixteen PM boys (hyper/hypo: 7/9) and 10 PM girls (hyper/hypo: 2
231 ults, the contribution of fossil fuel source PM(1) was 8-24%.
232 atively characterize uncertainty at specific PM(2.5) concentrations, which is often needed in the con
233 ined the association between source-specific PM and the rate of hospitalizations and emergency depart
234                        Using source-specific PM(2.5) concentrations at six urban sites in New York St
235   We mathematically modeled the steady-state PM distributions of all analyzed markers to better under
236                               More stringent PM(2.5) reduction targets expedite the phaseout of high
237 y aeolian vibration, enabling self-sustained PM adhesion.
238 ches is positively associated with long-term PM(2.5) level increases.
239                    Here, we demonstrate that PM induces Acod1 and itaconate, which reduced mitochondr
240    Structural equation models estimated that PM(2.5) and mitochondrial respiration accounted for 34%
241                             We observed that PM(2.5) was positively associated with AMI incidence and
242 tainty at different concentrations along the PM(2.5) concentration distribution.
243  a D4H probe, between late endosomes and the PM.
244 ORC2), ESCRT-III/Vps4 assemblies form at the PM and help maintain membrane integrity.
245 diffused while overall protein levels at the PM increased; thus allowing enhanced association of NRas
246 gRNA complexes and their accumulation at the PM, we compared, in living and fixed cells, the interact
247 nd flotillin1-containing nanoclusters at the PM.
248 ns exhibited simultaneous recruitment at the PM.
249 venting or breaking interactions between the PM and the remaining nearby insulin granules.
250 H3A and enhances the interaction between the PM PtdIns4P and RPH3A.
251 racellular transport of cholesterol from the PM to the ER is believed to be activated by a lipid-sens
252 lar cholesterol and its trafficking from the PM to the ER.
253 ing IQSec1, ORP3 also extracts PI4P from the PM, in exchange for phosphatidylcholine.
254  the amount of accessible cholesterol in the PM exceeds a threshold level, the excess cholesterol mov
255 increased accessible cholesterol pool in the PM was accompanied by reduced cholesterol movement to th
256 e derived inhalation bioaccessibility in the PM(10) size fraction is found to be ~5% of the total amb
257 ence that EPS1 contributes to modulating the PM abundance of defense proteins for effective immune si
258                       The performance of the PM CDM was compared to manual culture review of the digi
259 scribed the overarching contributions of the PM network, but less is known about how episodic content
260  the first molecular characterization of the PM-PER tether and show it anchors peroxisomes at the mot
261 ated relatively high skill in predicting the PM(2.5) levels in well-mixed indoor air.
262                          We propose that the PM-anchored Rsp5/Rcr1 ubiquitin ligase-adaptor complex c
263 c tracer FM4-64 is also recycled back to the PM after internalization.
264 miting the amount of insulin proximal to the PM and preventing or breaking interactions between the P
265 ither ZF delayed the delivery of gRNA to the PM but did not prevent Gag-gRNA interactions in the cyto
266                     Rcr1 is delivered to the PM by the exomer complex using a newly uncovered PM sort
267 f peripheral membrane protein binding to the PM in living cells.
268 ce of reduced delivery of cholesterol to the PM in ORP1-null cells, cholesterol was diverted to the E
269 ate that HIV-1 MA binds cooperatively to the PM with a dissociation coefficient of K(d) ~16 muM and H
270 nsulin-stimulated GLUT4 translocation to the PM, although where it intersects GLUT4 traffic was unkno
271 otors prevents their stable anchoring to the PM.
272 tifs, transports cholesterol from LEL to the PM.
273 3, a previous study found that WS-Fe was the PM(2.5) species most associated with adverse cardiovascu
274                                     When the PM cholesterol exceeds a threshold, excess cholesterol m
275 nsitivity of ChromID in combination with the PM CDM was similar to the sensitivity of molecular detec
276 lin granules and their interactions with the PM within single cells.
277  surface are preferentially aligned with the PM.
278 f monophosphorylated PPIn species within the PM, Golgi complex, and endosomal compartments.
279 hanges and emission control policies on this PM(2.5) concentration change.
280 buted as high as 50-75% (80-120 mug/m(3)) to PM(2.5) in Delhi, highlighting the importance of both ex
281 s trained on the IGFR samples was applied to PM samples from woodsmoke and diesel exhaust, the model
282 th external transport and local emissions to PM(2.5) pollution in Delhi.
283  of PTD associated with maternal exposure to PM in ambient air during pregnancy.
284                           Higher exposure to PM(10) was associated with higher rhinitis severity (adj
285 al-level approaches that reduce exposures to PM(2.5) can lead to improvements in health endpoints.
286 echanism by which RPH3A is recruited only to PM PtdIns4P rather than Golgi PtdIns4P remains unclear.
287 ntrol measures contributed ~60% of the total PM(2.5) reductions.
288 matic hydrocarbons (PAHs) found in ultrafine PM have been linked to cardiovascular diseases and carci
289 ate matter with a diameter less than 2.5 um (PM(2.5)).
290 y the exomer complex using a newly uncovered PM sorting motif.
291 udy time-resolved single-cell responses upon PM exposure.
292 the composite reference for positivity, when PM CDM was used to detect GBS from ChromID, the sensitiv
293 sed model of episodic construction, in which PM regions flexibly share and manipulate event informati
294                      On days with a wildfire PM(2.5) contribution >10 mug/m(3), exposure accounted fo
295 y may lessen the risk of PTD associated with PM exposure during pregnancy among primiparas of Han eth
296    Subsequently, C2B readily associates with PM via a polybasic cluster and a Ca(2+)-binding loop.
297       Subcutaneous immunization of mice with PM plus alum inhibits in vivo induction of Treg cells pr
298  patients with AM (38%) and 78 patients with PM (62%).
299 mice oropharyngeally aspirated 200 ug of WTC-PM(53) or phosphate-buffered saline (PBS) (controls).
300                                    The yeast PM is segregated into the Micro-Compartment-of-Can1 (MCC

 
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