ライフサイエンスコーパス: PMS

1 PMS algorithms are typically evaluated on certain instan

2 PMS cases reported a new clinician-made diagnosis of PMS

3 PMS contains significant amounts of cellulose that can b

4 PMS is a relatively common monogenic and highly penetran

5 PMS is associated with faster retinal atrophy independen

6 PMS is known to be NP complete.

7 PMS liquid and solid media stimulated very low or non-de

8 PMS receives as input n strings and two integers l and d

9 PMS symptoms may therefore be attributable, in part, to

10 PMS, PMDD, and each QOL dimension were also examined sep

11 PMS-derived iNSCs displayed increased glucose-dependent

12 0.03), RRMS (AUC = 0.73 vs 0.59; p < 0.001), PMS (AUC = 0.77 vs 0.53; p < 0.001), and patients with d

13 %; RRMS: -1.74 +/- 2.57% vs -1.74 +/- 4.02%; PMS: -2.29 +/- 2.40% vs -1.29 +/- 3.20%) and healthy con

14 104 PMS enrolled at four sites participated in the CogEx MRI

15 178 RRMS, 186 PMS, and 66 control participants were followed with seri

16 than CSA (CIS: 106 vs 830; RRMS: 95 vs 335; PMS: 44 vs 215; power = 80%; alpha = 5%).

17 by activating peroxymonosulfate (HSO(5)(-), PMS) or peroxydisulfate (S(2)O(8)(2-), PDS) with Fe(H(2)

18 II + III and IV activity was measured in 51 PMS participants.

19 Like 5MPCA, PMS induced fungal red pigmentation and killing.

20 n ca. 9.0 eV can be oxidized in the CPPy-F-8/PMS system.

21 itor is used to provide the input power to a PMS.

22 demonstrates that (*)OH in Co(2+)-activated PMS can play a significant role in contaminant transform

23 er sulfonate (6:2 FTS) with Co(2+)-activated PMS.

24 The nitrogen-doped carbocatalyst activated PMS through a nonradical pathway.

25 However, independent of age, PMS was associated with faster pRNFL (-0.34 +/- 0.09%/yr

26 wed the rate of disability progression among PMS patients who had inflammatory disease activity, evid

27 cess (GOP) was developed to separate BPA and PMS into two half cells.

28 n complexes of different mobility on GMS and PMS solid and liquid media.

29 ing leukocytes into the CNS in both RRMS and PMS and suggest that blocking DICAM with a monoclonal an

30 nducted separately in patients with RRMS and PMS using propensity score-weighted logistic regression.

31 h D-arginine or D-histidine as substrate and PMS as the electron acceptor established a ping-pong bi-

32 fter 10 years, 1,057 women were confirmed as PMS cases and 1,968 as controls.

33 in chronic demyelinating disorders, such as PMS.

34 iciency revealed a close correlation between PMS abundance and microtubule regulation, consistent wit

35 ables an efficient electron transfer between PMS and the catalyst.

36 Direct oxidation of As(III) by PMS avoids the formation of nonselective reactive radica

37 As(III) is rapidly oxidized by PMS with a utilization efficiency larger than 90%.

38 The kinetics of oxidation of phen by PMS features a complex pH dependence.

39 ion Surveillance-Post-Marketing Study [CASES-PMS]; NCT00231231).

40 The CASES-PMS (Carotid Artery Stenting With Emboli Protection Surv

41 al stroke between 31 and 360 days with CASES-PMS (5.4%) was similar to the rate seen with the SAPPHIR

42 Results indicated that the obtained CNF(PMS) in paper coating shows 52% decrease in porosity, pr

43 nsfer between the adsorbed BPA and complexed PMS as the mechanism.

44 We present a monolithic low-power-consuming PMS integrated circuit (IC) chip capable of dynamic maxi

45 times as likely as never smokers to develop PMS over the next 2-4 years (95% confidence interval: 1.

46 S at baseline, including 1,057 who developed PMS over 10 years and 1,968 reporting no diagnosis of PM

47 osis year had a 35% lower risk of developing PMS than did those in the lowest quintile (relative risk

48 o solve the problem that many pattern-driven PMS algorithms present execution time instability.

49 Pattern-driven PMS algorithms usually use k out of t input sequences as

50 ement of the state-of-the-art pattern-driven PMS algorithms.

51 ie the reinforcing effects of alcohol during PMS, when eye saccade responses to low doses of alcohol

52 Late juvenile males had a CY spigot on each PMS, whereas adult males either had a CY spigot or, more

53 proteins from C. albicans grown with either PMS or P. aeruginosa were also red and demonstrated abso

54 Reducing conditions greatly enhanced PMS uptake by C. albicans and killing.

55 g an individualized therapeutic approach for PMS.

56 all, this study developed a new catalyst for PMS activation and unveiled the advantages and potential

57 t, Co-Black TNT is an effective catalyst for PMS activation, leading to the degradation of selected o

58 ly, Al doping strengthened chemisorption for PMS (from -2.615 eV to -2.623 eV) and shortened Co-O bon

59 nner side of ring cells and is necessary for PMS formation and CR inflation.

60 -OH)) and the reactivity of AgFeO(2) NPs for PMS activation.

61 exhibited the best catalytic performance for PMS activation, with 97% phenol degradation efficiency i

62 h PMS and provide catalytic active sites for PMS activation.

63 siology mechanisms and treatment targets for PMS patients.

64 All participants were free from PMS at baseline (1991).

65 Participants were free from PMS at baseline.

66 he efficient sulfate radical generation from PMS.

67 eural stem/progenitor cell (iNSC) lines from PMS patient fibroblasts, we studied their senescent phen

68 edoxin or flavodoxin, P700+ was reduced from PMS only on the first flash and was reduced from F(X)- o

69 lash, thus allowing P700+ to be reduced from PMS.

70 Extrapolating results from PMS studies of carotid artery stenting to larger real-wo

71 SASP from PMS iNSC lines induced neurotoxicity in mature neurons,

72 work, the production of radical species from PMS induced by a magnetic CuFe(2)O(4) spinel was studied

73 Fe(2)O(4), the radical production yield from PMS was determined to be near 1 mol/mol.

74 Functionally, PMS iNSCs induced paracrine senescence and inflammation

75 arried out in PubMed to identify oral health PMSs published in dental, epidemiologic, and biostatisti

76 e generation of Fe(aq)(IV) by the Fe(aq)(II)-PMS/PDS systems without HCO(3)(-).

77 In PMS subgroup analysis, anti-CD20 exposure interacted neg

78 In PMS we are given two integers l and d and n biological s

79 In PMS, CR modulated grey matter (GM) volume and insular ac

80 (3) O(4) exhibits high intrinsic activity in PMS activation and sulfamethoxazole (SMX) degradation, h

81 not impact rates of retinal layer atrophy in PMS.

82 vestigated the effectiveness of CR and EX in PMS: here, we present MRI substudy volumetric and task-r

83 that contributes to remyelination failure in PMS, which may impact how this disease is modeled and in

84 nd ONL thinning were independently faster in PMS, as compared to controls (INL:-0.09 +/- 0.04%/yr, p

85 xCon and WexInc were significantly higher in PMS vs RRMS (p < 0.001), and significantly associated wi

86 it is unknown whether smoking is involved in PMS etiology.

87 be novel biomarkers of neurodegeneration in PMS that appear to be unaffected by conventional DMTs.

88 Participants in PMS studies for carotid artery stenting have different c

89 mpared with nonparticipants, participants in PMS studies had lower rates of symptomatic carotid arter

90 unclear whether retinal atrophy persists in PMS, exceeds normal aging, or can be distinguished from

91 ipogenic state was found to induce a SASP in PMS iNSCs via cholesterol-dependent transcription factor

92 The independent predictors of EDSS score in PMS were cervical spinal cord GM CSA and brain GM volume

93 o identify 29 studies of the use of SSRIs in PMS.

94 e mortality after carotid artery stenting in PMS study participants and nonparticipants.

95 adial glia-like cell (DARG) subpopulation in PMS cell lines exhibiting senescence and potent IFN resp

96 ty, may explain some phenotypic variation in PMS individuals.

97 malities may explain phenotypic variation in PMS symptoms.

98 No significant associations between incident PMS and dietary intakes of niacin, vitamin B-6, folate,

99 were each inversely associated with incident PMS.

100 Increasing PMS concentrations and pH accelerate oxidation of As(III

101 (phen) is oxidized by peroxomonosulfate ion (PMS) in a neutral aqueous solution.

102 work offers a sustainable approach to manage PMS for use in paper coatings as a high-value-added mate

103 ree facial sutures-the pre-maxillomaxillary (PMS), the nasofrontal (NFS), and the zygomaticotemporal

104 hoxy-5-methylphenazinium methylsulfate ((MeO)PMS) to yield product, albeit at only ~50% of the maximu

105 Phenazine methosulfate (PMS) was used as a mediator which acts as an artificial

106 fast electron donor (phenazine methosulfate (PMS)).

107 tetrazolium (TNBT), phenazine methosulfate (PMS), NAD(+), and 6-phosphogluconate was carefully poure

108 d pyocyanin analogs, phenazine methosulfate (PMS+) and phenazine ethosulfate (PES+).

109 using its analogue phenazine methosulphate (PMS).

110 erone-withdrawal paradigm, designed to mimic PMS and post-partum syndrome in a rat model.

111 , the collaborative mechanics of MSH and MLH/PMS proteins have not been resolved in any organism.

112 d MutS homologs (MSH) and MutL homologs (MLH/PMS) are the fundamental components of mismatch repair (

113 Highly conserved MutS (MSH) and MutL (MLH/PMS) homologues initiate mismatch repair and, in higher

114 The function(s) of MLH/PMS proteins is less clear, although they too bind ATP a

115 heir blood concentrations in progressive MS (PMS) are limited, and there are substantial discrepancie

116 hite matter lesions of human progressive MS (PMS) autopsy brain tissues and iPS-derived NPCs from pat

117 itting MS [RRMS] and 62 with progressive MS [PMS]) and 30 age- and sex-matched healthy control partic

118 sing-remitting MS [RRMS], 34 progressive MS [PMS]), and 82 controls from 8 MAGNIMS (Magnetic Resonanc

119 ing-remitting MS-RRMS and 117 progressive MS-PMS) were recruited.

120 umerating the number of live cells using MTS/PMS kit and of dead (apoptotic) cells using 4',6-diamidi

121 of oxidative stress producing chemicals (MV, PMS and H(2)O(2)), suggested its protective role against

122 l system, NADH and phenazine methosulfate; N/PMS).

123 lationship to O2(-) generated using the NADH/PMS method (R(2)=0.859).

124 discuss potential implications of this novel PMS function along axon shafts for axon maintenance and

125 Complex activity was abnormal in 59% of PMS participants.

126 Co-Black TNTs induce the activation of PMS by itself and stabilized oxygen vacancies that enhan

127 efficient, stable, and reusable activator of PMS for the degradation of organic pollutants.

128 The addition of PMS enables As(III) to oxidize completely to As(V) withi

129 (3+) cycles facilitated the decomposition of PMS to generate ROSs.

130 offee, and tea intake and the development of PMS in a case-control study nested within the prospectiv

131 take may not help prevent the development of PMS.

132 s associated with the initial development of PMS.

133 10 years and 1,968 reporting no diagnosis of PMS and only minimal menstrual symptoms during this time

134 s reported a new clinician-made diagnosis of PMS on biennial questionnaires between 1993 and 2005, an

135 and maximizing the utilization efficiency of PMS.

136 With an excess of PMS, four consecutive oxidation steps were found in near

137 Participants were free of PMS at baseline (1991).

138 s were US women aged 27-44 years and free of PMS at baseline, including 1,057 who developed PMS over

139 ly ordered feature of axons, the function of PMS is unknown.

140 The subcellular localization of PMS proteins is also regulated during DNA damage, which

141 erization of a Shank3-deficient rat model of PMS, with a genetic alteration similar to a human SHANK3

142 ment slightly improved the elastic nature of PMS samples.

143 The underlying neurobiology of PMS is not fully known and pharmacological treatments fo

144 esterol-related, hypermetabolic phenotype of PMS iNSCs that leads to neurotoxic signaling and is resc

145 in host cells, for enhancing the potency of PMS disinfection.

146 We observed a significantly lower risk of PMS in women with high intakes of thiamine and riboflavi

147 f nonheme iron intake had a relative risk of PMS of 0.64 (95% confidence interval (CI): 0.44, 0.92; P

148 intake also was not associated with risk of PMS or specific symptoms, including breast tenderness (O

149 ents was not associated with a lower risk of PMS.

150 dependently associated with a higher risk of PMS.

151 d reduced axon numbers, suggesting a role of PMS in microtubule organization.

152 and fluorescence spectra similar to that of PMS covalently linked to either amino acids or proteins

153 vel therapeutic targets for the treatment of PMS and other neurodegenerative diseases.

154 earchers in the development and reporting of PMSs, but their application has been limited.

155 ation, and neurodegeneration with a focus on PMS.

156 y outcome measure was a reduction in overall PMS symptoms.

157 This paper presents a fast exact parallel PMS algorithm called PMS8.

158 Peroxymonosulfate (PMS)-based advanced oxidation processes generate highly

159 ous catalysts to activate peroxymonosulfate (PMS) for the removal of Orange I (OI) in the water.

160 sing commercial available peroxymonosulfate (PMS).

161 alternative catalysts for peroxymonosulfate (PMS) activation to avoid drawbacks of conventional trans

162 s excellent catalysts for peroxymonosulfate (PMS) activation, and the reported mechanisms are mostly

163 For peroxymonosulfate (PMS), we developed a new approach to determine Phi*OHPMS

164 te powerful radicals from peroxymonosulfate (PMS) for recalcitrant pollutant removal.

165 eration is challenging in peroxymonosulfate (PMS)-based AOPs because the high 3d-orbital occupancy of

166 lt-mediated activation of peroxymonosulfate (PMS) has been widely investigated for the oxidation of o

167 on-like reaction based on peroxymonosulfate (PMS) activation was adopted to evaluate its activity.

168 peptide-mediated magnetic separation-phage (PMS-phage) assay.

169 which is stoichiometrically equal to PhiSO4*-PMS (0.57 +/- 0.02).

170 different manufacturers containing Plantower PMS, Alphasense OPC-N3, and AVO-Sensor sensors by colloc

171 We evaluated three Plantower PMS A003 sensors when exposed to eight particulate matte

172 A liquid crystal (LC) polymethylsiloxane (PMS) with rod-like aromatic side-groups attached via an

173 dietary minerals may be useful in preventing PMS.

174 benzodiazepines and ethanol can also produce PMS-like withdrawal symptoms.

175 The proposed PMS continuously detects the maximum power point (MPP) o

176 ent with microscopy-derived models proposing PMS as specialized cortical actin.

177 Another formulation is quorum PMS (qPMS), where the motif appears in at least q% of th

178 cell respiration, liberated electrons reduce PMS, which is then oxidized on the electrode surface, an

179 Controls did not report PMS and confirmed experiencing no symptoms or few mild s

180 a hallmark of relapsing-remitting MS (RRMS), PMS is associated with chronic, tissue-restricted inflam

181 red carbon source) in peptone minimal salts (PMS) media stimulated only low levels of aflatoxin accum

182 Progressive multiple sclerosis (PMS) causes slow accumulation of neurologic disability a

183 velopment in progressive multiple sclerosis (PMS) has been hampered by a lack of reliable biomarkers

184 Progressive multiple sclerosis (PMS) involves a persistent, maladaptive inflammatory pro

185 Progressive multiple sclerosis (PMS) is an immune-initiated neurodegenerative condition

186 cognition in progressive multiple sclerosis (PMS) remains limited.

187 people with progressive multiple sclerosis (PMS).

188 The planted (l, d) motif search (PMS) is an important yet challenging problem in computat

189 otif search problem or Planted Motif Search (PMS).

190 (l,d)-motif search or Planted Motif Search (PMS).

191 version is called the Planted Motif Search (PMS)or (l, d)-motif Search.

192 ismatch results in post-meiotic segregation (PMS).

193 101 subjects met defined criteria for severe PMS, remained eligible after 1 month of single-blind pla

194 e an effective first-line therapy for severe PMS.

195 theories for the underlying causes of severe PMS, and describe two main methods of treating it: one t

196 ood, may increase risk of moderate to severe PMS.

197 Some patients with severe PMS experience significant and sustained improvement wit

198 were 1,257 women with clinically significant PMS (1991-2005) and 2,463 age-matched comparison women w

199 rings termed the periodic membrane skeleton (PMS).

200 Y nubbin on each posterior median spinneret (PMS) in males (5(th) stadium and later) of the spider Au

201 In the present study, proso millet starch (PMS) was treated with SHS (120-160 degrees C for 1-5 min

202 Palisade-shaped Membrane-building Structure (PMS) around the rumen side of ring cells.

203 Here we studied PMS abundance, organization, and function, combining ver

204 hers conducting prediction modeling studies (PMSs).

205 ompanies conduct postmarketing surveillance (PMS) studies of approved stent systems.

206 abolomic data from Phelan McDermid Syndrome (PMS) patients (who are heterozygous for the Shank3 gene)

207 Individuals with Phelan-McDermid syndrome (PMS) present with a wide range of developmental, medical

208 q13.3 often causes Phelan-McDermid Syndrome (PMS), a genetically defined form of autism with profound

209 Phelan-McDermid Syndrome (PMS), which is defined by a deletion within 22q13, demon

210 disorder known as Phelan-McDermid syndrome (PMS).

211 linically significant premenstrual syndrome (PMS) affects 15-20% of premenopausal women, substantiall

212 Moderate to severe premenstrual syndrome (PMS) affects as many as 20% of premenopausal women.

213 ers (PMDs), including premenstrual syndrome (PMS) and premenstrual dysphoric disorder (PMDD), are ass

214 using a rat model of premenstrual syndrome (PMS) in which 1-3 mM alcohol preferentially enhanced GAB

215 ct the development of premenstrual syndrome (PMS) through multiple mechanisms, but few studies have e

216 tion of patients with premenstrual syndrome (PMS) who were randomly assigned in controlled treatment

217 ht also contribute to premenstrual syndrome (PMS), but whether women with PMS have a higher risk of s

218 Symptoms of premenstrual syndrome (PMS), such as anxiety and seizure susceptibility, are as

219 he pathophysiology of premenstrual syndrome (PMS).

220 ne therapy for severe premenstrual syndrome (PMS).

221 ability in women with premenstrual syndrome (PMS).

222 us suffer from severe premenstrual syndrome (PMS); most of these women also meet criteria for premens

223 In this research, a power management system (PMS) has been developed to charge a cell phone battery b

224 FC) integrated with power management system (PMS) was developed as a disposable self-support real-tim

225 Power management systems (PMSs) can overcome this limitation by boosting the MFC o

226 a stronger association with reduced QOL than PMS.

227 It is concluded that PMS is a stable syndrome that may best be viewed as part

228 Mass spectrometry analyses demonstrated that PMS can be covalently modified by amino acids, a process

229 and three actin-targeting drugs suggest that PMS contains short actin filaments that are depolymeriza

230 These results suggest that PMS might be associated with future development of hyper

231 The PMS also efficiently managed the power output of a lower

232 The PMS decomposition involved an inner-sphere complexation

233 The PMS problem is NP-complete.

234 The PMS study participants had lower unadjusted rates of com

235 In the model-based approach, we adjusted the PMS of each DoC group to a causal whole-brain model.

236 the HMGCR inhibitor simvastatin altered the PMS iNSC SASP, promoting cytoprotective qualities and re

237 41,111 PFU/g of feces were indicated by the PMS-phage assay.

238 As a demonstration, the PMS connected to a 240 mL two-chamber MFC (generating 0.

239 e surface with ATR-FTIR and Raman during the PMS decomposition suggested that surface Cu(II)-Cu(III)-

240 By examining the PMS patterns in yeast strains heterozygous for a mutant

241 e possible involvement of ROSs; however, the PMS decomposition tests with and without BPA and the com

242 64% of 106 individuals in the PMS foundation registry who did not have complex activit

243 stoichiometry of oxalate degradation in the PMS/CuFe(2)O(4), the radical production yield from PMS w

244 After CR inflation, the PMS disappears, with partially inflated cells displaying

245 med at 140 degrees C for 3 min to modify the PMS.

246 he MFC and matches the load impedance of the PMS for maximum efficiency.

247 The average elastic modulus of the PMS was 1.46 +/- 0.24 MPa (mean +/- SD), significantly h

248 energy stored into the supercapacitor of the PMS, was 30%.

249 The efficiency of the PMS/CuFe(2)O(4) was highest at neutral pH and decreased

250 s and had the same distribution pattern: the PMS (2.07 +/- 0.24 MPa) significantly higher than both t

251 ct (1) makes the tested algorithms solve the PMS problem steadily in an efficient way, (2) particular

252 ls exhibiting smooth PM, suggesting that the PMS serves as the reservoir for membrane-building materi

253 -power producing MFC, demonstrating that the PMS works efficiently at various MFC power output level.

254 and provided genetic information through the PMS foundation registry.

255 This PMS increases and regulates the input voltage of stack S

256 ants were examined via an enzyme-linked TNBT-PMS colorimetric assay.

257 The ratio of gene-conversion to PMS events reflects the efficiency of DNA repair.

258 polated: the catalyst transfers electrons to PMS through active nitrogen species and becomes a metast

259 trophy, which may contribute to evolution to PMS.

260 stack SMFCs, only module-5 provides power to PMS for long periods (13 min).

261 , magnesium, and manganese were unrelated to PMS risk.

262 As per SEM images, SHS-treated PMS had rough and irregular polygon surfaces with small

263 All patients underwent PMS implantation with intraoperative MMC (0.04% for 2.5

264 Upon PMS addition into one cell, BPA was quickly oxidized in

265 The simulation of power harvest using PMS indicated that PMMFC could accomplish more frequent

266 esequencing analysis of USVL531-PMR, USVL677-PMS and four introgressed PM resistant recombinant inbre

267 tions, we inoculated PM susceptible (USVL677-PMS) and resistant (USVL531-MDR) watermelon plants with

268 ant inbred lines (RIL, USVL531-PMR x USVL677-PMS) were performed to identify the region of PM resista

269 radical is the primary oxidant generated via PMS activation on Co-Black TNT.

270 cate that great caution should be taken when PMS or other agents that generate (*)OH are used for the

271 Whether PMS studies are representative of carotid artery stentin

272 regulation, consistent with a model in which PMS-dependent microtubule polymerization contributes to

273 418 patients with RRMS (43.0%) and 226 with PMS (52.7%) were treated with anti-CD20 therapies.

274 .80 years]; 737 [76.1%] female) and 429 with PMS (median age, 54.21 years [IQR, 48.42-60.14 years]; 2

275 m supplements was marginally associated with PMS (for intake of >/=25 mg/day vs. none, relative risk

276 The risk associated with PMS was not modified by use of oral contraceptives or an

277 that caffeine intake is not associated with PMS, and that current recommendations for women to reduc

278 otal caffeine intake was not associated with PMS.

279 ygen vacancies that enhance the bonding with PMS and provide catalytic active sites for PMS activatio

280 s exhibited less axonal damage compared with PMS astrocytes.

281 frequency of coffee and tea consumption with PMS.

282 ciations in a cohort of 170 individuals with PMS.

283 ditional MFC sensors, PMMFCs integrated with PMS exhibit the distinct advantages of tight paper-packe

284 tent, TNT denitration was also observed with PMS+ and PES+ in the presence of NAD(P)H.

285 82 [173] nM; P = .04), whereas patients with PMS displayed markedly reduced levels compared with heal

286 severe COVID-19 was higher in patients with PMS than in those with RRMS.

287 or natalizumab), and eligible patients with PMS were those younger than 70 years with an Expanded Di

288 Among patients with PMS, 19.0% and 15.5% of patients treated and not treated

289 le accurate differentiation of patients with PMS, having values below the threshold, from those with

290 In patients with PMS, there was no association between anti-CD20 therapie

291 rologic disability and younger patients with PMS.

292 s a safe therapeutic approach in people with PMS, where it holds the promise of healing the injured C

293 hypertension was reported by 342 women with PMS and 541 women without.

294 er risk factors for hypertension, women with PMS had a hazard ratio for hypertension of 1.4 (95% conf

295 trual syndrome (PMS), but whether women with PMS have a higher risk of subsequently developing hypert

296 Women with PMS often are counseled to minimize caffeine intake, alt

297 r more symptomatic cycles from 16 women with PMS were analyzed.

298 ugh smoking may be more common in women with PMS, it is unknown whether smoking is involved in PMS et

299 stable symptoms in this group of women with PMS.

300 erval: 1.2, 1.6) compared with women without PMS.