ライフサイエンスコーパス: PND

1 PND occurs in nearly one fifth of patients with intracer

2 In monkeys lesioned between the 9th and 12th PND, neurons in the deafferented hand region did not res

3 o the dorsal hippocampus of postnatal day 2 (PND 2) Ts65Dn pups to explore the feasibility of early p

4 rom gestational day 12 to post-natal day 20; PND 20).

5 but becomes impassable by postnatal day 30 (PND 30).

6 ded from hippocampus of pre-pubertal (~28-32 PND) and pubertal (~35-44 PND) female wild-type or alpha

7 eyes but were younger than postnatal day 35 (PND 35) exhibited modestly increased direction selectivi

8 elopment and into adulthood (PND 16-25, 35%; PND 35-80, 54%; PND 80-120, 66%; PND 175-225, 73%), prov

9 e-pubertal (~28-32 PND) and pubertal (~35-44 PND) female wild-type or alpha4-/- mice.

10 predominantly microglia at postnatal day 5 (PND 5) to predominantly oligodendrocytes by PND 30.

11 o adulthood (PND 16-25, 35%; PND 35-80, 54%; PND 80-120, 66%; PND 175-225, 73%), providing evidence t

12 ects (HLHS: n=1171, 92% PND; TGA: n=691, 58% PND) were submitted by 21 centers (19 in the United Stat

13 In monkeys lesioned between the 3rd and 5th PND, neurons in the entire hand region of area 3b and th

14 16-25, 35%; PND 35-80, 54%; PND 80-120, 66%; PND 175-225, 73%), providing evidence that synaptic circ

15 ally (0, 100, 300 or 1000 microg/day; GD 9 - PND 21) to assess activity and anxiety-like behaviors.

16 Data on 1862 subjects (HLHS: n=1171, 92% PND; TGA: n=691, 58% PND) were submitted by 21 centers (

17 Morphine administration typically abolished PND and reduced the discharge rate of most ccRTN neurons

18 ticle, we review some classic concepts about PND and recent clinical and immunological developments,

19 nt [postnatal day (PND) 28], mid-adolescent (PND 35), or adult male rats (PND 70) were surgically imp

20 ually during development and into adulthood (PND 16-25, 35%; PND 35-80, 54%; PND 80-120, 66%; PND 175

21 Even after PND 15,D-betaHB blocked morphological changes produced b

22 types prior to (PND 5 and PND 15) and after (PND 30 and PND 60) the period when pathway receptivity i

23 At later ages (PNDs 12 and 15), a clear patchy distribution of intrinsi

24 fluoride levels were determined on ED 20 and PND 11.

25 sion in the visual cortex between PND 28 and PND 49, and 3) an increased ratio of vimentin:GFAP-label

26 to (PND 5 and PND 15) and after (PND 30 and PND 60) the period when pathway receptivity is lost.

27 levels are significantly lower at PND 4 and PND 12 than in adult animals.

28 target tissue phenotypes prior to (PND 5 and PND 15) and after (PND 30 and PND 60) the period when pa

29 ed to posit common causes of early birth and PND other than the study exposure.

30 Here the evolution of relaxor phases and PNDs with thickness is explored in prototypical thin rel

31 ncreases in direction selectivity in animals PND 35 or older were explained by decreases in responses

32 ore slowly than the complexes formed by anti-PND IgG.

33 Pretreatment of the anti-PND IgG with a haptenic electrophilic phosphonate compou

34 At PND 15, adding D-betaHB to the media allowed robust long

35 At PND 15, some oligodendrocytes in the subcortical white m

36 At PND 30, vimentin, collagen IV, and fibronectin were abse

37 At PND 5, microglia are found throughout gray and white mat

38 At PND 5-15, both tissues also expressed vimentin, collagen

39 At PND 55-62, the animals were tested in an interchangeable

40 At PND 62, the only remaining loss was of the dendritic mar

41 At PND 90, MIA-exposed offspring had higher total white blo

42 at Ucn 1-immunoreactivity (ir) was absent at PND 1, while CART-ir was already apparent in pIIIu at bi

43 nucleus and posterodorsal medial amygdala at PND 20 and 25, respectively.

44 ratio of vimentin:GFAP-labeled astrocytes at PND 49 with reduced GFAP cell body area.

45 ) mice displayed 1) evidence of blindness at PND 49, with visual deficits detected at PND 35, 2) redu

46 alpha mRNA expression in the mouse cortex at PND 25 was significantly reduced as compared to PND 1 (p

47 wed by the secondary somatosensory cortex at PND 7.

48 at PND 49, with visual deficits detected at PND 35, 2) reduced GFAP mRNA expression in the visual co

49 d pulmonary function and lung development at PND 21.

50 status and became significantly different at PND 16.

51 ERbeta mRNA expression at PND 25 was significantly increased as compared to PND 1

52 ncreased during ontogeny and was greatest at PND 20.

53 measured together is significantly higher at PND 60 in kat2-/- mice than those of wild-type mice indi

54 Offspring were killed at PND 36, 110, 450 and 650.

55 g preexposure impairs contextual learning at PND 23.

56 reased with age, approaching adult levels at PND 16.

57 Ucn 1 mRNA levels are significantly lower at PND 4 and PND 12 than in adult animals.

58 chromatography-mass spectrometry (LC-MS) at PND 22 or at 7 weeks of age.

59 ic currents measured in layer 2/3 neurons at PND 8, just after these neurons ceased to migrate, revea

60 lood samples collected from the offspring at PND 90 to quantify immune cell profiles.

61 y but not completely co-localized in pIII at PND 24.

62 was absent in CART-positive cells of pIII at PND 4 and that Ucn 1 and CART are strongly but not compl

63 exposed to PPD had reduced levels of PPI at PND 56, but not PND 35, suggesting the emergence of a se

64 ure modulated offspring cytokine profiles at PND 30, 90, and 180, as indicated by persistent changes

65 Starting at PND 90, behaviour was assessed at weekly intervals in th

66 ound to be not long lasting; rats trained at PND 60, after neonatally receiving the original high dos

67 onary alveolarization and vascularization at PND 21; however, there were no differences in the degree

68 ch day on PNDs 2-14 and recorded in vitro at PNDs 15-21.

69 ing circuit, which are not functional before PND 18-25.

70 ferentially expressed (P-adj < 0.05) between PND 2 and 16.

71 tigate the bidirectional association between PND and AD.

72 mRNA expression in the visual cortex between PND 28 and PND 49, and 3) an increased ratio of vimentin

73 in sensitivity to MK-801 were found between PND 21 and 30.

74 a similar shift of the relationship between PND and end-tidal CO(2).

75 l ischemia were observed in all mice at both PND-17 and -20.

76 ion activated the ccRTN neurons normally but PND activation and the central respiratory modulation of

77 By PND 2, ARC ERalpha and Kiss1 levels were abundant, sexua

78 By PND 30, the predominant ferritin-containing cell type wi

79 ices became more intense and well defined by PND 9.

80 (PND 5) to predominantly oligodendrocytes by PND 30.

81 rrangement of connections had taken place by PND 6.

82 5; center-to-center approximately 750 pm) by PND 6.

83 th higher levels in females, but reversed by PND 4 due to declining levels in females.

84 t 2DG uptake contralateral to stimulation by PND 6, followed by the secondary somatosensory cortex at

85 which antigen-specific T cell stimulation by PND APCs triggers IFNgamma, followed by CXCL10 productio

86 PAR) paradigm, young rats at post-natal day (PND) 16 were found to exhibit a performance deficit that

87 l analyses were conducted at post-natal day (PND) 21 and 55.

88 0%kcal from fat) starting at post-natal day (PND) 30.

89 ion, with catch up growth by post-natal day (PND)21.

90 e prepared from rat brains at postnatal day (PND) 0-2 and were cultured for up to 60 d in vitro (DIV)

91 ors isolated from pups on the postnatal day (PND) 1 and 21.

92 From postnatal day (PND) 1 to 14, litters were separated from the dam, but n

93 ue taken from C57BL/6 mice on postnatal day (PND) 1, 4, 10, 18 and 25 and expression levels were dete

94 ygdala lesions made at either Postnatal Day (PND) 10 or PND40 were tested on a series of reversal tas

95 Six female C57BL/6 mice at postnatal day (PND) 10 were administered a single gavage dose of alpha-

96 By postnatal day (PND) 12, communities separated based on exposure status

97 Females were bred at postnatal day (PND) 120.

98 ission electron microscopy of postnatal day (PND) 14 Rho(P23H/+) mouse retina revealed disordered sag

99 age: it is most marked before postnatal day (PND) 14.

100 restored EPSPs in slices from postnatal day (PND) 15 rats but not in slices from PND 30 or 120 rats.

101 were killed 48 hours later on postnatal day (PND) 15, 20, and 25.

102 from embryonic day (ED) 6 to postnatal day (PND) 15.

103 he somatosensory cortex, from postnatal day (PND) 16 to PND 25 spine retractions exceeded additions,

104 fference that persisted until postnatal day (PND) 19 except in the ARC.

105 vaginal swab samples taken on postnatal day (PND) 2 and 16 in gilts to determine if temporal changes

106 from gestation day (GD) 8 to postnatal day (PND) 2 and compared results to those we previously repor

107 whole-brain radiation dose on postnatal day (PND) 21 and were randomized to 0.24% Li2CO(3) chow or no

108 throughout development, from postnatal day (PND) 21 to adult (N=174 reliable observations).

109 ) 6 until pups were weaned on postnatal day (PND) 21.

110 cularization were analyzed at postnatal day (PND) 21.

111 D) 9 to birth and from GD9 to postnatal day (PND) 21.

112 the stress of handling) from Postnatal Day (PND) 22 to PND 40 and determined the effects of daily lo

113 required by weanling rats at postnatal day (PND) 23 to demonstrate contextual fear conditioning.

114 In the present study, we fed postnatal day (PND) 24 weanling female rats an SPI diet for 30 d [short

115 using the forced-swim test on postnatal day (PND) 25 in rats either weaned on PND 21, or left with th

116 Early adolescent [postnatal day (PND) 28], mid-adolescent (PND 35), or adult male rats (P

117 cnn1b-Tg(+)) mice to SHS from postnatal day (PND) 3-21 and lung phenotypes were examined at PND22.

118 of the parental generation to postnatal day (PND) 6 of the F2 generation to a realistically proportio

119 expression starts as early as postnatal day (PND) 7 and peaks in adult.

120 the LE and developing GE from postnatal day (PND) 7 to PND 56.

121 cted between the 3rd and 12th postnatal day (PND), and the somatosensory cortex was mapped when the m

122 oved from the F1 offspring on postnatal day (PND)-1 and various analyses were performed.

123 ted to 75% to 85% oxygen from postnatal day (PND)-7 to -12 and then were abruptly placed in room air.

124 clc gene neared completion by postnatal day (PND)14, and loss of GCLC protein was complete by PND21.

125 rom gestational day 8 through postnatal day (PND)16.

126 sure regimes, one stopping at postnatal day (PND)21 (stop-dose) the other continuing until tissue har

127 ppocampus and white matter at postnatal day (PND)7 with IL-1Ra being protective.

128 e upper lip in neonatal rats (postnatal day [PND] 1-30).

129 Both juvenile (postnatal day [PND] 21) and adult (PND112) offspring of obese dams exhi

130 vehicle) twice/day for 11 consecutive days (PND 45-55).

131 ss was administered between post-natal days (PND) 25-27, EE from PND 35 to early adulthood, when beha

132 ions in the visual cortex of postnatal days (PND) 1 and 21 rats.

133 .1 to 5,000 mug BPA/kg BW on postnatal days (PND) 1, 3, and 5.

134 cal and apoptotic changes on postnatal days (PND) 1, 3, and 7.

135 and brains were collected at postnatal days (PND) 1, 4, 8, 12, 16, 24 and 45.

136 ere depleted of serotonin on postnatal days (PND) 10-20 by treating with the tryptophan hydroxylase i

137 audiovisual stimulation from postnatal days (PND) 10-40 and assessing anxiety-like behavior, social m

138 bination (ABX cocktail) from postnatal days (PND) 14 to 21, followed by ad libitum, low-dose individu

139 diation during infancy, from postnatal days (PND) 2-11 in the rat, results in severe hippocampal gran

140 atal alcohol exposure during postnatal days (PND) 2-6 in rats (third trimester human equivalent) lead

141 On postnatal days (PND) 21, 26, or 30, rats were trained on spatial discrim

142 On postnatal days (PND) 3-15, male and female Long-Evans rats underwent 3 h

143 The pups were tested at postnatal days (PND) 35 and 56 for PPI.

144 indlimb extensors of rats on Postnatal Days (PND) 5, 10, 15, or 20, during episodes of coordinated L-

145 pups were injected daily on postnatal days (PND) 7-19, with MK-801 (MK+) or the less active isomer o

146 mice were exposed to GEN on postnatal days (PND)1-5 and uterine tissues collected on PND5, PND22-26,

147 (5-7/group) were assessed on postnatal days (PNDs) 0, 2, 4, 7, and 19 for ER alpha (ERalpha; Esr1), b

148 Subjects were tested at postnatal days (PNDs) 0-9 and 21.

149 On postnatal days (PNDs) 1 and 3, a diffuse distribution of axons and label

150 ormoxia or hyperoxia through postnatal days (PNDs) 1 to 14, and the hyperoxia-exposed mice were allow

151 g of LPS or vehicle daily on postnatal days (PNDs) 3 to 5.

152 s.c.) on alternate days from postnatal days (PNDs) 3-13 and killed on PNDs 15, 30 and 60.

153 System or to filtered air on postnatal days (PNDs) 4-7 and 10-13, and the animals were euthanized eit

154 tional age-specific risk of perinatal death (PND) can be decomposed as the product of the gestational

155 Promoted NOx decomposition (PND) technology for real-world automotive applications i

156 e paraneoplastic neurological degenerations (PNDs) are remarkable examples of naturally occurring tum

157 env chimeric clone with a partially deleted PND and did not altered the fitness of the virus in vivo

158 theoretical probability of neighbor density (PND) as a robust tool to discern protein oligomeric stat

159 rectional link between perinatal depression (PND) and AD is largely unexplored.

160 tract at area postrema level desynchronized PND from ventilation, eliminated the lung inflation-sync

161 d with prehospital neurologic deterioration (PND) are unknown.

162 g to the principal neutralizing determinant (PND) of human immunodeficiency virus type-1 (HIV) gp120

163 n in adults and animals late in development (PND 30-89).

164 ve in animals lesioned later in development (PND 30-89).

165 critical period of hippocampal development (PNDs 2-14).

166 d as a cause of permanent neonatal diabetes (PND).

167 rd genetics, interest in prenatal diagnosis (PND) for deafness, and preference for having deaf or hea

168 piratory responses [phrenic nerve discharge (PND) and AP] caused by injecting dl-homocysteic acid (DL

169 SND, phrenic nerve discharge (PND) and putative sympathoexcitatory vasomotor neurons o

170 rst just before the phrenic nerve discharge (PND) and rebound after inspiration (pre-I neurons).

171 sed blood pressure, phrenic nerve discharge (PND) and the firing rate of ccRTN neurons in isoflurane-

172 imol eliminated the phrenic nerve discharge (PND) at rest, during hyperoxic hypercapnia (10% CO(2)),

173 nstantly eliminated phrenic nerve discharge (PND) but normal PND could usually be elicited by strong

174 eded to inhibit the phrenic nerve discharge (PND).

175 nchronized with the phrenic nerve discharge (PND).

176 ic SNA, and rate of phrenic nerve discharge (PND; P<0.05).

177 pected paraneoplastic neurological disorder (PND) may be difficult because of the limitations of conv

178 The paraneoplastic neurologic disorders (PND) are a rare group of neurologic syndromes that arise

179 Paraneoplastic neurologic disorders (PND) are autoimmune diseases associated with cancer and

180 ment of paraneoplastic neurologic disorders (PND) include the detection of new antineuronal antibodie

181 Paraneoplastic neurologic disorders (PNDs) are believed to be autoimmune neuronal degeneratio

182 Paraneoplastic neurologic disorders (PNDs) offer an uncommon opportunity to study human tumor

183 ber concentrations (PNCs) and distributions (PNDs) in the 5-1000 nm range close to a busy roadside, c

184 acids) in the principal neutralizing domain (PND) of gp90 during the third febrile episode.

185 heterologous principal neutralizing domains (PND).

186 activation with minocycline treatment during PND 2-6 alcohol exposure ameliorated the hormonal and mi

187 HIV complexes formed by anti-E-PND IgG dissociated noticeably more slowly than the comp

188 log containing electrophilic phosphonates (E-PND) neutralized a homologous HIV strain (MN) approximat

189 utralizing domain variants of groups 1 (EIAV(PND-1)) and 5 (EIAV(PND-5)), respectively; however, the

190 riants of groups 1 (EIAV(PND-1)) and 5 (EIAV(PND-5)), respectively; however, the neutralization-resis

191 ; however, the neutralization-resistant EIAV(PND-5) variant was less infectious in single-round repli

192 01 (MK-) (0.25 mg/kg), and trained at either PND 22 or 60.

193 ocked the sympathetic baroreflex, eliminated PND at rest and during chemoreceptor stimulation but did

194 tzinger region, also called CVLM) eliminated PND while increasing the stimulatory effect of CO(2) on

195 rostral ventral respiratory group eliminated PND but did not change RTN neuron response to either lun

196 roventricular (i.c.v.) kynurenate eliminated PND and the response of RTN neurons to lung inflation bu

197 atory column 1.5 mm caudal to RTN eliminated PND and the respiratory modulation of RTN neurons.

198 patient antisera to clone the genes encoding PND antigens has led to new insight into the mechanism o

199 ased and parabasal decreased (P < 0.05) from PND 0 to 16.

200 oteins have been cloned using antiserum from PND patients.

201 d to 0.24% Li2CO(3) chow or normal chow from PND 49 to 77.

202 ehavior, and spatial learning/cognition from PND 50-60.

203 ne glycol-400 (PEG-400) was given daily from PND-14 to -16, and mice were killed on PND-17 to form gr

204 (100 mg/kg) or PEG-400 was given daily from PND-17 to -19, and mice were killed on PND-20.

205 the PMCo, ERalpha expression decreased from PND 2 and remained low through PND 19.

206 between post-natal days (PND) 25-27, EE from PND 35 to early adulthood, when behavioural testing and

207 atal life (PND 1-21) or throughout life from PND 1 until the end of the study.

208 tal day (PND) 15 rats but not in slices from PND 30 or 120 rats.

209 otypic explant cultures of cortex taken from PND 3 mice.

210 group was subject to gavage with water from PND 14 to 21 and received drinking water till the time o

211 cipants said that they would consider having PND, and, of these, 29% said that they would prefer to h

212 h neutralizing activity against heterologous PND variants can prevent lentivirus infection and clinic

213 In PND patients, common tumors such as breast, ovarian and

214 te hypoxia promoted exaggerated increases in PND amplitude after CIH (P<0.05).

215 e effect of FK506 on CSF chemokine levels in PND patients.

216 uring 2001-2013, we included 55,299 incident PND, their unaffected full sisters, and 10 unaffected ma

217 Future work to increase PND should be considered in these specific populations.

218 onide were quantitated in sera of individual PND 3 pups collected 1 hr postexposure utilizing ultra-h

219 HD, before Ritalin, on PSA, after infantile (PND 2-15) exposure to x-irradiation.

220 50 mg Mn/kg/day during early postnatal life (PND 1-21) or throughout life from PND 1 until the end of

221 0.64-0.95]) were also associated with lower PND in TGA.

222 ted phrenic nerve discharge (PND) but normal PND could usually be elicited by strong peripheral chemo

223 an familiar shaving odors on PND 10, but not PND 14.

224 had reduced levels of PPI at PND 56, but not PND 35, suggesting the emergence of a sensorimotor gatin

225 utations are the second most common cause of PND and a rare cause of MODY.

226 data acquired, mechanisms of OH oxidation of PND and CND were proposed.

227 tilely evoked cerebellar field potentials of PND 30-40 animals compared with neonates and adults, sug

228 We sought to determine the prevalence of PND among patients with intracerebral hemorrhage during

229 rs need to be aware of the increased risk of PND among women with ADs and vice versa.

230 l age-specific risk of birth and the risk of PND conditional on birth at a given gestational age.

231 On PND 40, F1 offspring (n = 10/group/sex) were exposed to

232 On PND 40, locomotor activity levels were not significantly

233 On PND's 30 and 62, animals were perfused for immunodensito

234 t for 30 d [short-term SPI (ST-SPI)], and on PND 55, we switched SPI diet to control Cas diet until a

235 Hippocampal neurogenesis was assessed on PND 77, 91, and 105.

236 Rats were bred on PND 72.

237 vagina was swabbed using a cytology brush on PND 0, 2 and 16 and slides were prepared.

238 the central nucleus of the amygdala (CeA) on PND 198.

239 ary tract nucleus (commNTS) had no effect on PND or SND activation by CO(2).

240 x difference was lost and then re-emerged on PND 19, with females having more than males.

241 both synaptic markers in the hippocampus on PND 30.

242 from PND-14 to -16, and mice were killed on PND-17 to form group A.

243 from PND-17 to -19, and mice were killed on PND-20.

244 ession of ERbeta in the MePD was observed on PND 0, with higher levels in females, but reversed by PN

245 cage nest to clean familiar shaving odors on PND 10, but not PND 14.

246 slower in performing the righting reflex on PND 4 and negative geotaxis compared with WKY and Spragu

247 roteins isolated from vaginal swabs taken on PND 2 and 16 from six gilts across three litters were me

248 maternal bonding in a return to dam task on PND 17 (p<0.05).

249 Rats treated with MK+ or MK- and trained on PND 22 were significantly impaired in PSA when compared

250 tnatal day (PND) 25 in rats either weaned on PND 21, or left with their mother until PND 25 (non-wean

251 n clusters than in the intercluster zones on PND 6.

252 we separated pups from dams once each day on PNDs 2-14 and recorded in vitro at PNDs 15-21.

253 rom postnatal days (PNDs) 3-13 and killed on PNDs 15, 30 and 60.

254 time with the dam during the active phase on PNDs 15-17 (p<0.05) and experienced decreased maternal b

255 Peak PNDs appeared at approximately 12 nm, showing an unusual

256 sisters, and 10 unaffected matched women per PND case.

257 Aqueous oxidation of pinanediol (PND) and camphanediol (CND) by hydroxyl radical (OH) was

258 nts of the uteri were analyzed at postnatal (PND) day 21, 90, and 365.

259 Preweanling (PND 17-18) rats, which suffer such hippocampal granule-c

260 Young Sprague Dawley rats (PND 21) were assigned to environmentally enriched, pair-

261 mid-adolescent (PND 35), or adult male rats (PND 70) were surgically implanted with a guide cannula a

262 rapezoid nucleus (RTN) eliminated or reduced PND, respectively, but did not change the effect of CO(2

263 bryo lacking all GPI-linked proteins rescues PND migration in a dose-dependent fashion, (2) showing t

264 erebral hemorrhage, 22 patients (22%) showed PND during Emergency Medical Services transport, with a

265 tes of 43 unselected patients with suspected PND referred for FDG-PET scanning to determine how usefu

266 of small tumours in patients with suspected PND.

267 irection selectivity in animals younger than PND 35 were explained by increases in responses to the p

268 Here we report that PND patients harbor high levels of the chemokine CXCL10

269 These data suggest that PND pathfinding is accomplished by migration up a gradie

270 al in SAP- and SSP-SAP-treated rats, but the PND rate was slightly elevated in SSP-SAP-treated rats.

271 ms for vesicular biogenesis, mediated by the PND.

272 es regions, V1 to V8, with V3 containing the PND region.

273 F-binding activity, and (3) showing that the PND will migrate toward a GDNF-soaked bead in vivo, but

274 Abs from mice immunized with the PND analog containing electrophilic phosphonates (E-PND)

275 pproaches the length of the long axis of the PNDs (25-30 nm), electrostatically driven phase instabil

276 25, 0.05, 0.1, 0.15, and 0.20 mg/kg) through PND days 22-25.

277 ecreased from PND 2 and remained low through PND 19.

278 nsory cortex, from postnatal day (PND) 16 to PND 25 spine retractions exceeded additions, resulting i

279 of handling) from Postnatal Day (PND) 22 to PND 40 and determined the effects of daily low-dose admi

280 developing GE from postnatal day (PND) 7 to PND 56.

281 25 was significantly reduced as compared to PND 1 (p<0.01).

282 5 was significantly increased as compared to PND 1 (p<0.05).

283 thway and target tissue phenotypes prior to (PND 5 and PND 15) and after (PND 30 and PND 60) the peri

284 ongitudinal neurobehavioral assessment until PND 60 followed by brain harvesting for multiregional ol

285 Kiss1 was not detectable until PND 11 in the anterior hypothalamus, but expression leve

286 N ERbeta levels were higher in females until PND 19.

287 Dietary regimen was maintained until PND 30 when all offspring were switched to CD.

288 Separate cohorts were maintained until PND 50 and tested for learning and memory using Morris w

289 d on PND 21, or left with their mother until PND 25 (non-weaned).

290 Animals that had opened their eyes and were PND 35 or older exhibited increased direction selectivit

291 With PND, EGR is no longer needed.

292 vident herniation seem to be associated with PND.

293 ly than control Abs from mice immunized with PND.

294 Patients with PND harbor high-titer antibodies and T cells in their se

295 In 279 patients with PND, the frequency of KCNJ11, ABCC8, and INS gene mutati

296 Aqueous OH reaction with PND can produce up to 0.3 (Tg x yr(-1)) of (aq)SOA, assu

297 cids (TAs) from the aqueous OH reaction with PND was confirmed with authentic standards.

298 so activated SND in bursts synchronized with PND.

299 mpared to their matched controls, women with PND were at higher risk of subsequent PMDs (HR 1.81, 95%

300 artment among both patients with and without PND.